Search results for "Calbindin"

showing 10 items of 26 documents

Ikaros-1 couples cell cycle arrest of late striatal precursors with neurogenesis of enkephalinergic neurons

2010

et al.

Cyclin-Dependent Kinase Inhibitor p21CalbindinsEnkephalinNeurogenesiseducationCentral nervous systemCell Cycle ProteinsStriatumSubstance PBiologyEfferent PathwaysCalbindinIkaros Transcription FactorMiceS100 Calcium Binding Protein GmedicineAnimalsProgenitor cellTranscription factorhealth care economics and organizationsHomeodomain ProteinsMice KnockoutNeuronsStem CellsGeneral NeuroscienceNeurogenesisCell DifferentiationEnkephalinsCell cycleCorpus StriatumGenes cdcMice Inbred C57BLmedicine.anatomical_structurenervous systemTrans-ActivatorsNeuroscienceTranscription FactorsThe Journal of Comparative Neurology
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Alterations in the organization of the isocortical layer I in trisomy 22.

1999

The isocortical layer I of human fetal brains obtained from different cases of chromosomal abnormalities (trisomy 18, 21, 22) and controls without pathological disturbances were investigated histologically and immunohistochemically by using the antibodies SMI 311, SMI 35 and SMI 81 (SNAP 25) as well as antibodies against GAP 43 and calretinin. In cases of trisomy 22 the Cajal-Retzius cells in Nissl-sections and in SMI 311-immunopreparations do not reveal any alterations regarding their location or morphology. However, the axonal plexus, selectively labelled with SMI 35, normally located in layer Ib, is malpositioned in Ia. Likewise, SNAP 25- and GAP 43-immunoreactive structures, which were …

Down syndromePathologymedicine.medical_specialtyChromosomes Human Pair 21Chromosomes Human Pair 22SynaptogenesisChromosome DisordersNerve Tissue ProteinsTrisomyTrisomy 22FetusGAP-43 ProteinS100 Calcium Binding Protein GmedicineHumansGap-43 proteinChromosome AberrationsPlexusbiologyGeneral NeuroscienceSnapBrainGeneral MedicineAnatomymedicine.diseasenervous systemCalbindin 2biology.proteinCalretininDown SyndromeTrisomyChromosomes Human Pair 18Neuroscience research
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Ontogeny of the human amygdala.

2003

Data on the fetal development of the human amygdala is reviewed with special reference to major ontogenetic events. In the fifth gestational month, the inferior portion of the amygdala reveals cell-dense columns merging with the ganglionic eminence (proliferative zone) in Nissl-stained sections. These columns contain vimentin-positive fibers and can therefore be regarded as migrational routes. In the sixth and seventh months, distinct reorganization of the cytoarchitectonics takes place. The sequential occurrence of afferens can be visualized using anti-GAP-43; moreover, outgrowing axons appear to reach the periphery of the ganglionic eminence. The latter may thus represent an intermediate …

Ganglionic eminenceGeneral NeuroscienceGlutamate receptorGestational AgeNerve Tissue ProteinsAnatomyBiologyAmygdalaCalbindinAmygdalaGeneral Biochemistry Genetics and Molecular BiologyEmbryonic and Fetal Developmentmedicine.anatomical_structurenervous systemHistory and Philosophy of ScienceCytoarchitecturePostsynaptic potentialmedicineHumansCalretininNeuroscienceImmunostainingBiomarkersAnnals of the New York Academy of Sciences
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Calretinin/PSA-NCAM immunoreactive granule cells after hippocampal damage produced by kainic acid and DEDTC treatment in mouse.

2003

There is a dramatic increase in the number of lightly immunoreactive calretinin cells in the granular layer of the dentate gyrus of the mouse hippocampus 1 day after excitotoxic injury using kainic acid combined with the zinc chelator diethyldithiocarbamate. At 7 days after treatment, these cells are strongly immunoreactive for calretinin and for the polysialated form of the glycoprotein neural cell adhesion molecule (PSA-NCAM). The reexpression of calretinin and PSA-NCAM after treatment corresponds well with the loss of input from the damaged hilar mossy cells. These cells could be considered immature granule cells since they are immunoreactive to markers for immature cells such as PSA-NCA…

Kainic acidTime FactorsNeural Cell Adhesion Molecule L1Granular layerHippocampal formationHippocampuschemistry.chemical_compoundMiceS100 Calcium Binding Protein GExcitatory Amino Acid AgonistsAnimalsMolecular BiologyChelating AgentsNeuronsKainic AcidbiologyGeneral NeuroscienceDentate gyrusNeurogenesisDrug SynergismMolecular biologyImmunohistochemistryMicroscopy Electronnervous systemchemistryCalbindin 2Phosphopyruvate HydrataseImmunologybiology.proteinSialic AcidsNeural cell adhesion moleculeNeurology (clinical)NeuNCalretininDitiocarbDevelopmental BiologyBrain research
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Mice lacking α-synuclein display functional deficits in the nigrostriatal dopamine system

2000

alpha-Synuclein (alpha-Syn) is a 14 kDa protein of unknown function that has been implicated in the pathophysiology of Parkinson's disease (PD). Here, we show that alpha-Syn-/- mice are viable and fertile, exhibit intact brain architecture, and possess a normal complement of dopaminergic cell bodies, fibers, and synapses. Nigrostriatal terminals of alpha-Syn-/- mice display a standard pattern of dopamine (DA) discharge and reuptake in response to simple electrical stimulation. However, they exhibit an increased release with paired stimuli that can be mimicked by elevated Ca2+. Concurrent with the altered DA release, alpha-Syn-/- mice display a reduction in striatal DA and an attenuation of …

MaleCalbindinsNeuroscience(all)DopamineDopamine AgentsLong-Term PotentiationPresynaptic TerminalsSynucleinsGene ExpressionGlutamic AcidSubstantia nigraNerve Tissue ProteinsNeurotransmissionMotor ActivityHippocampusSynaptic TransmissionReuptakechemistry.chemical_compoundMiceS100 Calcium Binding Protein GDopamineDopaminergic CellmedicineAnimalsAutoreceptorsAlpha-synucleinMice KnockoutNeuronsGeneral NeuroscienceRab3A GTP-Binding ProteinCorpus Striatumrab3A GTP-Binding Proteinnervous system diseasesMice Inbred C57BLSubstantia NigraAmphetaminechemistrynervous systemalpha-SynucleinCalciumFemaleBeta-synucleinNeuroscienceLocomotionmedicine.drug
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The dendritic spines of interneurons are dynamic structures influenced by PSA-NCAM expression.

2013

Excitatory neurons undergo dendritic spine remodeling in response to different stimuli. However, there is scarce information about this type of plasticity in interneurons. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) is a good candidate to mediate this plasticity as it participates in neuronal remodeling and is expressed by some mature cortical interneurons, which have reduced dendritic arborization, spine density, and synaptic input. To study the connectivity of the dendritic spines of interneurons and the influence of PSA-NCAM on their dynamics, we have analyzed these structures in a subpopulation of fluorescent spiny interneurons in the hippocampus of glutamic …

MaleDendritic spineTime FactorsInterneuronCognitive NeuroscienceDendritic SpinesGreen Fluorescent ProteinsHippocampusNeuraminidaseMice TransgenicNerve Tissue ProteinsNeural Cell Adhesion Molecule L1BiologyHippocampal formationIn Vitro TechniquesHippocampus03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineOrgan Culture TechniquesInterneuronsmedicineAnimals030304 developmental biology0303 health sciencesPolysialic acidGlutamate DecarboxylaseDendritic filopodiamedicine.anatomical_structurenervous systemAnimals NewbornGene Expression RegulationCalbindin 2Excitatory postsynaptic potentialSialic AcidsNeural cell adhesion moleculeCholecystokininSomatostatinNeuroscience030217 neurology & neurosurgeryVasoactive Intestinal PeptideCerebral cortex (New York, N.Y. : 1991)
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Electrolytic lesion of the nucleus incertus retards extinction of auditory conditioned fear

2013

Fear memory circuits in the brain function to allow animals and humans to recognize putative sources of danger and adopt an appropriate behavioral response; and research on animal models of fear have helped reveal the anatomical and neurochemical nature of these circuits. The nucleus (n.) incertus in the dorsal pontine tegmentum provides a strong GABAergic projection to forebrain ‘fear centers’ and is strongly activated by neurogenic stressors. In this study in adult male rats, we examined the effect of electrolytic lesions of n. incertus on different stages of the fear conditioning-extinction process and correlated the outcomes with anatomical data on the distribution of n. incertus-derive…

MaleElectrolytic lesionConditioning ClassicalNerve Tissue ProteinsAmygdalaExtinction PsychologicalRats Sprague-DawleyBehavioral NeuroscienceS100 Calcium Binding Protein GPonsNeural PathwaysmedicineAnimalsFear conditioningHabituationFear memoryNeuronsFear processing in the brainBehavior AnimalRelaxinExtinctionFearExtinction (psychology)AmygdalaNucleus IncertusRatsFreezing behaviormedicine.anatomical_structureAcoustic StimulationCalbindin 2Relaxin-3PsychologyNucleus incertusNeuroscienceBasolateral amygdalaBehavioural Brain Research
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A "double hit" murine model for schizophrenia shows alterations in the structure and neurochemistry of the medial prefrontal cortex and the hippocamp…

2013

Both alterations in neurodevelopment and aversive experiences during childhood and adolescence seem important risk factors for schizophrenia. Animal models reproducing these alterations mimic some of the symptoms, constituting a valid approach to study the etiopathology of this disorder. Among these models, the perinatal injection of N-methyl-d-aspartate receptor antagonists and the postweaning social isolation rearing are among the most widely used. Our aim is to combine them in a "double hit" model, which should produce a wider spectrum of alterations. Lister Hooded rats have been subjected to a single injection of MK-801 at postnatal day 7 and socially isolated from postweaning to adulth…

MaleInterneuronHippocampusPrefrontal CortexNerve Tissue ProteinsAdult neurogenesisCalbindinHippocampuslcsh:RC321-571InterneuronNeurochemicalPregnancymedicineAnimalsAnimal modelPrefrontal cortexlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNeural Cell Adhesion MoleculesbiologyBody Weightmedicine.diseaseRatsInhibitory neurotransmissionDisease Models Animalmedicine.anatomical_structureNeurologynervous systemAnimals NewbornGene Expression RegulationSocial IsolationSchizophreniabiology.proteinSchizophreniaFemaleCalretininDizocilpine MaleatePsychologyNeuroscienceExcitatory Amino Acid AntagonistsProto-Oncogene Proteins c-fosParvalbuminNeurobiology of disease
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Non-granule PSA-NCAM immunoreactive neurons in the rat hippocampus

2002

The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) continues to be expressed in the adult hippocampus, mainly in a subset of neurons located in the innermost portion of the granule cell layer. PSA-NCAM immunoreactive neurons have also been described outside this layer in humans, where they are severely reduced in schizophrenic brains. Given this important clinical implication, we were interested in finding whether similar neurons existed in the adult rat hippocampus and to characterize their distribution, morphology and phenotype. PSA-NCAM immunocytochemistry reveals labeled neurons in the subiculum, fimbria, alveus, hilus, and stratum oriens, lucidum and radiatum of CA…

MaleInterneuronNeural Cell Adhesion Molecule L1Hippocampal formationHippocampusCalbindinImage Processing Computer-AssistedmedicineAnimalsNeuropeptide YFluorescent Antibody Technique IndirectNeural Cell Adhesion MoleculesMolecular Biologygamma-Aminobutyric AcidNeuronsbiologyGeneral NeuroscienceSubiculumGranule cellImmunohistochemistryRatsPhenotypemedicine.anatomical_structurenervous systemSialic Acidsbiology.proteinNeural cell adhesion moleculeNeurology (clinical)CalretininSomatostatinNeuroscienceParvalbuminDevelopmental BiologyBrain Research
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Infrequent co-existence of nitric oxide synthase and parvalbumin, calbindin and calretinin immunoreactivity in rat pontine neurons.

1995

Neurons in the laterodorsal tegmental nucleus (LDTg), ventrolateral dorsal tegmental nucleus (LDTgV), pedunculopontine tegmental nucleus (PPTg), lateral and medial parabrachial nuclei (LPB and MPB) were immunoreactive to brain nitric oxide synthase (NOS) or isoform I. Double-labeling experiments showed that very few NOS-containing neurons in the pons were immunoreactive to any of the three calcium-binding proteins: calbindin-D 28K (CB-IR), parvalbumin (PV-IR) and calretinin (CR-IR). These findings extend our previous observation in the neocortex and suggest that a population of central NOS-containing neurons can be neurochemically characterized as CB/CR/PV deficient.

Malemedicine.medical_specialtyCalbindinsNerve Tissue ProteinsCalbindinRats Sprague-DawleyS100 Calcium Binding Protein GInternal medicinePonsTegmentummedicineAnimalsPedunculopontine Tegmental NucleusNeuronsParabrachial NucleusbiologyStaining and LabelingChemistryGeneral NeuroscienceCalcium-Binding ProteinsPonsRatsLaterodorsal tegmental nucleusmedicine.anatomical_structureEndocrinologyParvalbuminsnervous systemCalbindin 2biology.proteinImmunologic TechniquesCalmodulin-Binding ProteinsFemaleAmino Acid OxidoreductasesCalretininNitric Oxide SynthaseParvalbuminNeuroscience letters
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