Search results for "Capsazepine"

showing 8 items of 8 documents

Inactivation and tachyphylaxis of heat-evoked inward currents in nociceptive primary sensory neurones of rats.

2000

In contrast to other sensory modalities, pain does not decrease when a noxious stimulus is applied at constant intensity (Greene & Hardy, 1962). From this lack of adaptation on the perceptive level it has traditionally been implied that primary nociceptive afferents also do not adapt upon constant stimulation. This is in contrast to the results of recordings from these afferents, which exhibit pronounced adaptation for physical as well as chemical stimuli (Meyer et al. 1994). Peripheral adaptation of nociceptive nerve endings is compensated by central summation (Mendell & Wall, 1965; Price et al. 1977); this slow summation process of small fibre input to the dorsal horn of the spinal cord i…

Intracellular FluidMaleHot TemperatureTime FactorsPhysiologyStimulationTachyphylaxisStimulus (physiology)Rats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineGanglia SpinalNoxious stimulusAnimalsNeurons AfferentTachyphylaxisCells Cultured030304 developmental biology0303 health sciencesChemistryElectric ConductivityNociceptorsOriginal ArticlesRatsNociceptionNociceptorCalciumFemaleCapsazepineExtracellular SpaceNeuroscienceFree nerve ending030217 neurology & neurosurgeryThe Journal of physiology
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Involvement of TRPV1 channels in the activity of the cannabinoid WIN 55,212-2 in an acute rat model of temporal lobe epilepsy

2016

The exogenous cannabinoid agonist WIN 55,212-2, (R)-(+)-[2,3-dihydro-5-methyl-3-(4-morpholinylmethyl) pyrrolo[1,2,3-de]-1,4-benzoxazin-6-Yl]-1-naphthalenylmethanone (WIN), has revealed to play a role on modulating the hyperexcitability phenomena in the hippocampus. Cannabinoid-mediated mechanisms of neuroprotection have recently been found to imply the modulation of transient receptor potential vanilloid 1 (TRPV1), a cationic channel subfamily that regulate synaptic excitation. In our study, we assessed the influence of pharmacological manipulation of TRPV1 function, alone and on WIN antiepileptic activity, in the Maximal Dentate Activation (MDA) acute model of temporal lobe epilepsy. Our r…

Male0301 basic medicineAgonistCannabinoid Receptor Modulatorsmedicine.drug_classMorpholinesmedicine.medical_treatmentTRPV1TRPV Cation ChannelsHippocampusNaphthalenesPharmacologySettore BIO/09 - FisiologiaNeuroprotection03 medical and health scienceschemistry.chemical_compound0302 clinical medicineReceptor Cannabinoid CB1Hippocampus Temporal lobe epilepsy Cannabinoids TRPV1 Capsaicin ElectrophysiologyMembrane Transport ModulatorsCannabinoid Receptor ModulatorsmedicineAnimalsRats WistarWIN 55212-2ChemistryElectric StimulationBenzoxazinesDisease Models Animal030104 developmental biologyEpilepsy Temporal LobeNeurologyAcute DiseaseAnticonvulsantslipids (amino acids peptides and proteins)Neurology (clinical)CannabinoidCapsaicinCapsazepineNeurosciencepsychological phenomena and processes030217 neurology & neurosurgerymedicine.drugEpilepsy Research
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Inhibition of rapid heat responses in nociceptive primary sensory neurons of rats by vanilloid receptor antagonists.

1999

Recent studies demonstrated that heat-sensitive nociceptive primary sensory neurons respond to the vanilloid receptor (VR) agonist capsaicin, and the first cloned VR is a heat-sensitive ion channel. Therefore we studied to what extent heat-evoked currents in nociceptive dorsal root ganglion (DRG) neurons can be attributed to the activation of native vanilloid receptors. Heat-evoked currents were investigated in 89 neurons acutely dissociated from adult rat DRGs as models for their own terminals using the whole cell patch-clamp technique. Locally applied heated extracellular solution (effective temperature ∼53°C) rapidly activated reversible and reproducible inward currents in 80% (62/80) o…

MaleAgonistHot TemperaturePatch-Clamp TechniquesPhysiologymedicine.drug_classReceptors DrugRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDorsal root ganglionGanglia SpinalmedicineAnimalsNeurons AfferentPatch clamp030304 developmental biology0303 health sciencesDose-Response Relationship DrugChemistryGeneral NeuroscienceNociceptorsRuthenium RedRatsElectrophysiologySolutionsElectrophysiologymedicine.anatomical_structureNociceptionCapsaicinBiophysicsNociceptorFemaleCapsaicinCapsazepineNeuroscience030217 neurology & neurosurgerySignal Transduction
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Inhibition by Anandamide and Synthetic Cannabimimetics of the Release of [3H]d-Aspartate and [3H]GABA from Synaptosomes Isolated from the Rat Hippoca…

2004

Cannabinoids (CB) can act as retrograde synaptic mediators of depolarization-induced suppression of inhibition or excitation in hippocampus. This mechanism may underlie the impairment of some cognitive processes produced by these compounds, including short-term memory formation in the hippocampus. In this study, we investigated several compounds known to interact with CB receptors, evaluating their effects on K +-evoked release of [ 3H]d-aspartate ([ 3H]d-ASP) and [ 3H]GABA from superfused synaptosomes isolated from the rat hippocampus. [ 3H]d-ASP and [ 3H]GABA release were inhibited to different degrees by the synthetic cannabinoids WIN 55,212-2; CP 55,940, and arachidonyl-2′- chloroethyla…

MaleCannabinoid receptorSettore BIO/14 - FARMACOLOGIAPolyunsaturated Alkamidesmedicine.medical_treatmentHippocampusArachidonic AcidsPharmacologyHippocampal formationDepolarization-induced suppression of inhibitionHippocampusBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundglutamate releasemedicineAnimalsRats WistarCannabinoidgamma-Aminobutyric AcidCannabinoid Receptor AgonistsAspartic AcidCannabinoidsChemistryGeneral MedicineAnandamideCyclohexanolsgaba releaseEndocannabinoid systemRatsKineticsnervous systemBiochemistryAnimals Arachidonic Acids Aspartic Acid Calcium Cannabinoids Capsaicin Cyclohexanols gamma-Aminobutyric Acid Hippocampus Kinetics Polyunsaturated Alkamides Potassium Rats Receptors Cannabinoid SynaptosomesPotassiumCalciumlipids (amino acids peptides and proteins)CannabinoidCapsaicinCapsazepineEndocannabinoidsSynaptosomesNeurochemical Research
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Changes in cytosolic calcium in response to noxious heat and their relationship to vanilloid receptors in rat dorsal root ganglion neurons.

2001

Heat transduction mechanisms in primary nociceptive afferents have been suggested to involve a vanilloid receptor channel with high calcium permeability. To characterize the changes in free cytosolic calcium evoked by noxious heat stimuli (< or =51 degrees C, 10s), we performed microfluorometric measurements in acutely dissociated small dorsal root ganglion neurons (< or =32.5 microm) of adult rats using the dye FURA-2. Only neurons that responded with a reversible increase in intracellular calcium to high potassium were evaluated. Heat-induced calcium transients (exceeding mean + 3S.D. of the temperature dependence of the dye) were found in 66 of 105 neurons. These transients increased non…

MaleHot Temperaturemedicine.drug_classReceptors Drugchemistry.chemical_elementPainCalcium channel blockerCalciumCalcium in biologyRats Sprague-Dawleychemistry.chemical_compoundCytosolGanglia SpinalmedicineAnimalsThermosensingCalcium SignalingNeurons AfferentCells CulturedFluorescent DyesCalcium metabolismVoltage-dependent calcium channelGeneral NeuroscienceMyocardiumT-type calcium channelNociceptorsRatschemistryBiochemistryCapsaicinBiophysicsPotassiumCalciumFemaleCalcium ChannelsCapsaicinCapsazepineFura-2Signal TransductionNeuroscience
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Differential effects of anandamide on acetylcholine release in the guinea-pig ileum mediated via vanilloid and non-CB1 cannabinoid receptors

2001

The effects of anandamide on [3H]-acetylcholine release and muscle contraction were studied on the myenteric plexus-longitudinal muscle preparation of the guinea-pig ileum preincubated with [3H]-choline. Anandamide increased both basal [3H]-acetylcholine release (pEC50 6.3) and muscle tone (pEC50 6.3). The concentration-response curves for anandamide were shifted to the right by 1 μM capsazepine (pKB 7.5 and 7.6), and by the combined blockade of NK1 and NK3 tachykinin receptors with the antagonists CP99994 plus SR142801 (each 0.1 μM). The CB1 and CB2 receptor antagonists, SR141716A (1 μM) and SR144528 (30 nM), did not modify the facilitatory effects of anandamide. Anandamide inhibited the e…

Pharmacologymedicine.medical_specialtyCannabinoid receptormedicine.medical_treatmentTRPV1AnandamideMuscarinic agonistchemistry.chemical_compoundEndocrinologychemistryInternal medicinemedicineCannabinoidCapsazepineTachykinin receptorAcetylcholinemedicine.drugBritish Journal of Pharmacology
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'TRPing' synaptic ribbon function in the rat pineal gland: neuroendocrine regulation involves the capsaicin receptor TRPV1.

2009

Synaptic ribbons (SRs) are presynaptic structures thought to regulate and facilitate multivesicular release. In the pineal gland, they display a circadian rhythm with higher levels at night paralleling melatonin synthesis. To gain more insight into the processes involved and the possible functions of these structures, a series of experiments were conducted in rodents. We studied the regional distribution of a molecular marker of pineal SRs, the kinesin motor KIF3A in the gland. Respective immunoreactivity was abundant in central regions of the gland where sympathetic fibers were less dense, and vice versa, revealing that intercellular communication between adjacent pinealocytes is enhanced …

endocrine systemmedicine.medical_specialtyEndocrinology Diabetes and MetabolismTRPV1KinesinsTRPV Cation ChannelsBiologyBradykininPineal GlandCalcium in biologyPinealocyteMembrane PotentialsMelatoninRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundPineal glandNorepinephrineEndocrinologyInternal medicinemedicineAnimalsMelatoninSynaptic ribbonEndocrine and Autonomic SystemsRatsEndocrinologymedicine.anatomical_structurenervous systemchemistrySynapseslipids (amino acids peptides and proteins)CalciumCapsaicinCapsazepineEndocrine glandmedicine.drugNeuroendocrinology
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Proton conductance of human transient receptor potential-vanilloid type-1 expressed in oocytes of Xenopus laevis and in Chinese hamster ovary cells.

2004

Transient receptor potential-vanilloid type-1 (TRPV1) is a ligand-gated cation channel with preference for divalent cations, especially Ca(2+) (sequence of conductances: Ca(2+)Mg(2+)Na(+) approximately/= K(+) approximately/= Cs(+)). In the present study, the two-electrode voltage-clamp technique was used on oocytes of Xenopus laevis expressing TRPV1 to evaluate whether human TRPV1 also conducts protons. In medium devoid of K(+), Na(+), Mg(2+), and Ca(2+), capsaicin 1 microM induced a significant inward current (62% of the current in physiological medium). The effects of capsaicin were abolished in the presence of capsazepine 3 microM. The capsaicin-induced currents in medium devoid of Na(+)…

medicine.medical_specialtyPatch-Clamp TechniquesReceptors DrugTRPV1XenopusHamsterAction PotentialsCHO CellsDivalentchemistry.chemical_compoundXenopus laevisCricetulusInternal medicineCricetinaemedicineAnimalsHumansCloning MolecularReversal potentialchemistry.chemical_classificationMembrane potentialbiologyGeneral NeuroscienceChinese hamster ovary cellbiology.organism_classificationElectrophysiologyEndocrinologychemistryBiophysicsOocyteslipids (amino acids peptides and proteins)FemaleCapsaicinProtonsCapsazepineNeuroscience
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