Search results for "Cardiology"

showing 10 items of 6064 documents

Guanxin Danshen Formulation Protects against Myocardial Ischemia Reperfusion Injury-Induced Left Ventricular Remodeling by Upregulating Estrogen Rece…

2017

Background: Guanxin Danshen formulation (GXDSF) is a traditional Chinese herbal recipe recorded in the Chinese Pharmacopoeia since 1995 edition, which consists of Salviae miltiorrhizae Radix et Rhizoma, Notoginseng Radix et Rhizoma and Dalbergiae odoriferae Lignum. Our previous research suggested GXDSF had positive effect on cardiovascular disease. Therefore, the aim of this study was to elucidate the effects of GXDSF on myocardial ischemia reperfusion injury-induced left ventricular remodelling (MIRI-LVR). Methods: The effects of GXDSF on cardiac function were detected by haemodynamics and echocardiograms. The effects of GXDSF on biochemical parameters (AST, LDH and CK-MB) were analysed. H…

0301 basic medicineCardiac function curvemedicine.medical_specialtyGuanxin Danshen formulaEstrogen receptor030204 cardiovascular system & hematologyPharmacologyventricular remodeling03 medical and health sciences0302 clinical medicineFibrosisInternal medicinemedicinenetwork pharmacologyPharmacology (medical)Ventricular remodelingOriginal ResearchPharmacologyPI3K/AktEjection fractionbusiness.industryestrogen receptor βlcsh:RM1-950PHTPPmedicine.diseasemyocardial ischemia reperfusion injury030104 developmental biologylcsh:Therapeutics. PharmacologyCardiologyMyocardial fibrosisbusinessReperfusion injuryFrontiers in Pharmacology
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Pathophysiology of Peripheral Arterial Disease (PAD): A Review on Oxidative Disorders

2020

Peripheral arterial disease (PAD) is an atherosclerotic disease that affects a wide range of the world’s population, reaching up to 200 million individuals worldwide. PAD particularly affects elderly individuals (>65 years old). PAD is often underdiagnosed or underestimated, although specificity in diagnosis is shown by an ankle/brachial approach, and the high cardiovascular event risk that affected the PAD patients. A number of pathophysiologic pathways operate in chronic arterial ischemia of lower limbs, giving the possibility to improve therapeutic strategies and the outcome of patients. This review aims to provide a well detailed description of such fundamental issues as physical exe…

0301 basic medicineCardiovascular eventmedicine.medical_specialtyArterial diseasePopulationPhysical exerciseReview030204 cardiovascular system & hematologyCatalysisInorganic Chemistrylcsh:Chemistry03 medical and health sciences0302 clinical medicineperipheral arterial diseasephysical exerciseInternal medicineHumansMedicineoxidative stressPhysical and Theoretical ChemistryMuscle SkeletaleducationExerciseMolecular Biologylcsh:QH301-705.5Spectroscopypathophysiologyeducation.field_of_studybusiness.industryOrganic ChemistryAtherosclerotic diseaseGeneral Medicineheme oxygenaseArterial Ischemic StrokePathophysiologyComputer Science ApplicationsPeripheralbody regionsperipheral arterial disease; physical exercise; oxidative stress; heme oxygenase; antioxidants; pathophysiologyEarly Diagnosis030104 developmental biologyantioxidantslcsh:Biology (General)lcsh:QD1-999CardiologybusinessBiomarkersHeme Oxygenase-1
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MerTK receptor cleavage promotes plaque necrosis and defective resolution in atherosclerosis

2017

Atherothrombotic vascular disease is often triggered by a distinct type of atherosclerotic lesion that displays features of impaired inflammation resolution, notably a necrotic core and thinning of a protective fibrous cap that overlies the core. A key cause of plaque necrosis is defective clearance of apoptotic cells, or efferocytosis, by lesional macrophages, but the mechanisms underlying defective efferocytosis and its possible links to impaired resolution in atherosclerosis are incompletely understood. Here, we provide evidence that proteolytic cleavage of the macrophage efferocytosis receptor c-Mer tyrosine kinase (MerTK) reduces efferocytosis and promotes plaque necrosis and defective…

0301 basic medicineCarotid Artery DiseasesMalePathologymedicine.medical_specialtyNecrosisCardiology030204 cardiovascular system & hematologyBiologyC-Mer Tyrosine KinaseProinflammatory cytokine03 medical and health sciencesMiceNecrosis0302 clinical medicineProto-Oncogene ProteinsmedicineAnimalsHumansEfferocytosisMice Knockoutc-Mer Tyrosine KinaseBrief ReportFibrous capReceptor Protein-Tyrosine KinasesGeneral MedicineMERTKPlaque Atherosclerotic030104 developmental biologymedicine.anatomical_structureReceptors LDLApoptosisProteolysisFemalemedicine.symptomTyrosine kinase
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Effects of gaseous and solid constituents of air pollution on endothelial function

2018

Abstract Ambient air pollution is a leading cause of non-communicable disease globally. The largest proportion of deaths and morbidity due to air pollution is now known to be due to cardiovascular disorders. Several particulate and gaseous air pollutants can trigger acute events (e.g. myocardial infarction, stroke, heart failure). While the mechanisms by which air pollutants cause cardiovascular events is undergoing continual refinement, the preponderant evidence support rapid effects of a diversity of pollutants including all particulate pollutants (e.g. course, fine, ultrafine particles) and gaseous pollutants such as ozone, on vascular function. Indeed alterations in endothelial function…

0301 basic medicineClinical ReviewFine particulateAir pollutionAir pollution030204 cardiovascular system & hematologymedicine.disease_cause03 medical and health sciencesGreenhouse Gases0302 clinical medicineOzoneRisk FactorsEnvironmental healthMedicineAnimalsHumansEndothelial dysfunctionNitrogen dioxidePollutantInflammationAir PollutantsHuman studiesAmbient air pollutionbusiness.industryEnvironmental ExposureParticulates3. Good healthEditor's Choice030104 developmental biology13. Climate actionCardiovascular DiseasesOxidative stressEndothelium VascularCurrent (fluid)Cardiology and Cardiovascular MedicinebusinessVascular functionParticulate matterTranslational MedicineEuropean Heart Journal
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BIOMARKERS OF CORONARY MICROVASCULAR DYSFUNCTION IN PATIENTS WITH MICROVASCULAR ANGINA: A NARRATIVE REVIEW

2021

The current gold standard for diagnosis of coronary microvascular dysfunction (CMD) in the absence of myocardial diseases, whose clinical manifestation is microvascular angina (MVA), is reactivity testing using adenosine or acetylcholine during coronary angiography. This invasive test can be difficult to perform, expensive, and harmful. The identification of easily obtainable blood biomarkers which reflect the pathophysiology of CMD, characterized by high reliability, precision, accuracy, and accessibility may reduce risks and costs related to invasive procedures and even facilitate the screening and diagnosis of CMD. In this review, we summarized the results of several studies that have i…

0301 basic medicineCoronary angiographymedicine.medical_specialtyClinical manifestationCoronary Artery Disease030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicinemicrovascular dysfunction (CMD) oxidative stress inflammation endothelial dysfunction microvascular angina (MVA)Internal medicineCoronary CirculationMedicineHumansIn patientEndothelial dysfunctionMicrovascular Anginabusiness.industryMicrocirculationMicrovascular anginaReproducibility of ResultsGold standard (test)medicine.disease030104 developmental biologyCardiologyNarrative reviewCardiology and Cardiovascular MedicinebusinessBiomarkersReactivity Testing
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Airborne disease: a case of a Takotsubo cardiomyopathie as a consequence of nighttime aircraft noise exposure.

2016

Chronic noise exposure (in particular nighttime noise) leads to disturbances of activities, sleep, and communication. As a consequence, emotional responses or annoyance will lead to stress reactions characterized by an activation of the sympathetic nervous system and/or increased levels of circulating stress hormones. We report here a case of a patient, who was exposed to heavy …

0301 basic medicineCoronary angiographymedicine.medical_specialtySympathetic nervous systemAircraft noiseAircraftAnnoyance030204 cardiovascular system & hematologyCoronary Angiography03 medical and health sciences0302 clinical medicineNoise exposureTakotsubo CardiomyopathyInternal medicinemedicineHumansbusiness.industryEnvironmental exposureEnvironmental Exposuremedicine.diseaseAirborne diseaseNoise030104 developmental biologymedicine.anatomical_structureEchocardiographyNoise TransportationCardiologyMedical emergencyCardiology and Cardiovascular MedicinebusinessEuropean heart journal
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Changes in the spatial distribution of the Purkinje network after acute myocardial infarction in the pig

2018

Purkinje cells (PCs) are more resistant to ischemia than myocardial cells, and are suspected to participate in ventricular arrhythmias following myocardial infarction (MI). Histological studies afford little evidence on the behavior and adaptation of PCs in the different stages of MI, especially in the chronic stage, and no quantitative data have been reported to date beyond subjective qualitative depictions. The present study uses a porcine model to present the first quantitative analysis of the distal cardiac conduction system and the first reported change in the spatial distribution of PCs in three representative stages of MI: an acute model both with and without reperfusion; a subacute …

0301 basic medicineCritical Care and Emergency MedicineSwinemedicine.medical_treatmentMyocardial InfarctionInfarction030204 cardiovascular system & hematologyPathology and Laboratory MedicineVascular MedicinePurkinje Cells0302 clinical medicineAnimal CellsIschemiaMedicine and Health SciencesTissue DistributionMyocardial infarctionNeuronsCardiomyocytesMultidisciplinaryQRHeartInfarctionDisease ProgressionCardiologyMedicineCellular TypesAnatomyElectrical conduction system of the heartResearch Articlemedicine.medical_specialtyHistologyScienceCardiologyMuscle TissueIschemiaMyocardial Reperfusion InjuryCatheter ablation03 medical and health sciencesSigns and SymptomsHeart Conduction SystemDiagnostic MedicineInternal medicinemedicineAnimalscardiovascular diseasesEndocardiumMuscle Cellsbusiness.industryBiology and Life SciencesCell Biologymedicine.diseaseElectrophysiologyBiological Tissue030104 developmental biologyVacuolizationCellular NeuroscienceReperfusionCardiovascular AnatomyNerve NetbusinessEndocardiumNeuroscience
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SGLT-2 (Sodium-Glucose Cotransporter 2) Inhibition Reduces Ang II (Angiotensin II)-Induced Dissecting Abdominal Aortic Aneurysm in ApoE (Apolipoprote…

2019

Objective: Abdominal aortic aneurysm (AAA) is a pathological condition of permanent vessel dilatation that predisposes to the potentially fatal consequence of aortic rupture. SGLT-2 (sodium-glucose cotransporter 2) inhibitors have emerged as powerful pharmacological tools for type 2 diabetes mellitus treatment. Beyond their glucose-lowering effects, recent studies have shown that SGLT-2 inhibitors reduce cardiovascular events and have beneficial effects on several vascular diseases such as atherosclerosis; however, the potential effects of SGLT-2 inhibition on AAA remain unknown. This study evaluates the effect of oral chronic treatment with empagliflozin—an SGLT-2 inhibitor—on dissecting …

0301 basic medicineDissecting Abdominal Aortic AneurysmApolipoprotein EMalemedicine.medical_specialtyInflammation030204 cardiovascular system & hematologyp38 Mitogen-Activated Protein Kinases03 medical and health sciencesMice0302 clinical medicineApolipoproteins EGlucosidesInternal medicinemedicineAnimalsHumansBenzhydryl CompoundsAortic ruptureSodium-Glucose Transporter 2 InhibitorsCells CulturedNeovascularization Pathologicbusiness.industryAngiotensin IINF-kappa Bmedicine.diseaseAngiotensin IIAbdominal aortic aneurysmMatrix MetalloproteinasesMice Inbred C57BLAortic Dissection030104 developmental biologyEndocrinologySodium/Glucose Cotransporter 2Knockout mousemedicine.symptomChemokinesCardiology and Cardiovascular MedicinebusinessAortic Aneurysm AbdominalArteriosclerosis, thrombosis, and vascular biology
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Microparticles harbouring Sonic hedgehog morphogen improve the vasculogenesis capacity of endothelial progenitor cells derived from myocardial infarc…

2019

Aims Endothelial progenitor cells (EPC) play a role in endothelium integrity maintenance and regeneration. Decreased numbers of EPC or their impaired function correlates with an increase in cardiovascular events. Thus, EPC are important predictors of cardiovascular mortality and morbidity. Microparticles carrying Sonic hedgehog (Shh) morphogen (MPShh+) trigger pro-angiogenic responses, both in endothelial cells and in ischaemic rodent models. Here, we propose that MPShh+ regulates EPC function, thus enhancing vasculogenesis, and correcting the defects in dysfunctional EPC obtained from acute myocardial infarction (AMI) patients. Methods and results The mechanisms underlying Shh pathway func…

0301 basic medicineEndotheliumNitric Oxide Synthase Type IIIPhysiologyAngiogenesis[SDV]Life Sciences [q-bio]Myocardial InfarctionMice NudeNeovascularization PhysiologicAcute myocardial infarction030204 cardiovascular system & hematologyMicroparticlesZinc Finger Protein GLI103 medical and health sciences0302 clinical medicineVasculogenesisCell-Derived MicroparticlesPhysiology (medical)Paracrine CommunicationVasculogenesismedicineAnimalsHumansHedgehog ProteinsProgenitor cellSonic hedgehogAngiogenic ProteinsCells CulturedComputingMilieux_MISCELLANEOUSEndothelial progenitor cellsbiologybusiness.industryNitric oxideSmoothened ReceptorHedgehog signaling pathwayPatched-1 ReceptorVascular endothelial growth factor A030104 developmental biologymedicine.anatomical_structureCase-Control StudiesKLF2embryonic structuresCancer researchbiology.proteincardiovascular systemCardiology and Cardiovascular MedicinebusinessSignal Transductioncirculatory and respiratory physiology
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GAS6

2017

Venous thrombosis is determined by the recruitment of monocytes and neutrophils to the inflamed endothelium and is primarily influenced by the plasmatic coagulation system.1 Monocyte tissue factor (TF) was identified as the causative trigger for intraluminal fibrin formation and thrombus load in the inferior vena cava (IVC) stenosis model, resembling human deep vein thrombosis.1 Although monocyte TF is prothrombotic, the TF expressed by activated endothelial cells triggers proinflammatory protease-activated receptor signaling pathways.2,3 See accompanying article on page 1315 In the past years, GAS6 (growth arrest–specific gene-6) was described as a major regulatory protein of prothrombotic…

0301 basic medicineEndotheliumVena Cava Inferior030204 cardiovascular system & hematologyFibrinogenInferior vena cavaMonocytes03 medical and health sciencesTissue factor0302 clinical medicinemedicineHumansPlateletThrombusVenous ThrombosisGAS6business.industryImpaired platelet aggregationmedicine.disease030104 developmental biologymedicine.anatomical_structuremedicine.veinAnesthesiaCancer researchCardiology and Cardiovascular MedicinebusinessGasolinemedicine.drugArteriosclerosis, Thrombosis, and Vascular Biology
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