Search results for "Catalase"

showing 10 items of 165 documents

Quality and antioxidant response of gilthead seabream (Sparus aurata L.) to dietary supplements of fenugreek (Trigonella foenum graecum) alone or com…

2017

The present study was conducted to determine the potential effect of the dietary intake of fenugreek (Trigonella foenum graecum) seeds alone or in combination with Bacillus licheniformis, Lactobacillus plantarum or B. subtilis on gilthead seabream quality and antioxidant response after 2 and 3 weeks of experimental feeding. The results showed that the supplements did not affect the percentage of the fatty acid profiles of muscle, demonstrating that all the additives tested can be administrated without any negative effect on biochemical composition and quality of gilthead seabream. The quantification of thiobarbituric acid reactive substances in muscle demonstrated the significant beneficial…

0301 basic medicineTrigonellaFenugreek seedThiobarbituric acidAquatic ScienceProbioticTeleostsGilthead seabream (Sparus aurata L.)law.inventionSuperoxide dismutase03 medical and health sciencesIngredientchemistry.chemical_compoundProbioticRandom AllocationAdjuvants ImmunologicSettore AGR/20 - ZoocolturelawEnvironmental ChemistryAnimalsBacillus licheniformisFood scienceSettore BIO/06 - Anatomia Comparata E Citologiachemistry.chemical_classificationbiologyPlant ExtractsAntioxidant statuProbioticsFatty acid04 agricultural and veterinary sciencesGeneral Medicinebiology.organism_classificationAnimal FeedImmunity InnateSea BreamDietAntioxidant-related gene expression030104 developmental biologyTrigonellachemistryBiochemistryCatalaseDietary Supplements040102 fisheriesbiology.protein0401 agriculture forestry and fisheriesLactobacillus plantarumBacillus subtilisLactobacillus plantarumFishshellfish immunology
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Erwinia amylovoracatalases KatA and KatG are virulence factors and delay the starvation-induced viable but non-culturable (VBNC) response

2017

The life cycle of the plant pathogen Erwinia amylovora comprises periods inside and outside the host in which it faces oxidative stress caused by hydrogen peroxide (H2 O2 ) and other compounds. The sources of this stress are plant defences, other microorganisms and/or exposure to starvation or other environmental challenges. However, the functional roles of H2 O2 -neutralizing enzymes, such as catalases, during plant-pathogen interactions and/or under starvation conditions in phytopathogens of the family Erwiniaceae or closely related families have not yet been investigated. In this work, the contribution of E. amylovora catalases KatA and KatG to virulence and survival in non-host environm…

0301 basic medicinechemistry.chemical_classificationbiology030106 microbiologyMutantSoil ScienceVirulencePlant ScienceErwiniabiology.organism_classificationmedicine.disease_causeVirulence factorMicrobiology03 medical and health sciencesEnzymechemistryCatalasebiology.proteinmedicineAgronomy and Crop ScienceMolecular BiologyPathogenOxidative stressMolecular Plant Pathology
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Involvement of hydrogen and lipid peroxides in acute tobacco smoking-induced platelet hyperactivity

1995

Previous studies have established that cigarette smoking results in acute platelet hyperaggregability. We investigated whether changes in plasma oxidative properties could occur after smoking and whether such changes could be responsible for this enhanced platelet activity. In the present work, we report that platelets from nonsmokers become hyperactive after incubation with plasma prepared from blood of smokers obtained 10 min after smoking. This effect was not observed with presmoking plasma and could be inhibited in vitro by adding either catalase or reduced glutathione plus peroxidase to plasma or 2,6-di-tert-butyl-p-cresol (BHT) to platelets before incubation. Comparison of pre- and p…

AdultBlood PlateletsMaleLipid Peroxidesmedicine.medical_specialtyPlatelet AggregationPhysiologymedicine.medical_treatmentFatty Acids NonesterifiedAntioxidantschemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineHumansPlateletPlatelet activationIncubationchemistry.chemical_classificationbiologyVitamin ESmokingThrombinFatty acidHydrogen PeroxideGlutathioneButylated HydroxytolueneMiddle AgedBlood Physiological PhenomenaAdenosine DiphosphateEndocrinologychemistryBiochemistryCatalasebiology.proteinCardiology and Cardiovascular MedicinePeroxidaseAmerican Journal of Physiology-Heart and Circulatory Physiology
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Impairment of antioxidant enzymes, lipid peroxidation and 8-oxo-2'-deoxyguanosine in advanced epithelial ovarian carcinoma of a Spanish community.

2006

In the present study, we describe the changes of antioxidant enzyme activities and other oxidative stress-related parameters in a mediterranean cohort of women affected with epithelial ovarian carcinoma (EOC). For that purpose, the most representative enzymatic activities, such as superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GPx) and the oxidized/reduced glutathione (GSSG/GSH) ratio have been analyzed in tumor tissue biopsies and compared with the normal tissue of the same patient. As oxidation products, the levels of malondialdehyde (MDA) as an indication of lipid peroxidation, and the DNA damaged base 8-oxo-2'-deoxyguanosine (8-oxo-dG) have been also measured. Ad…

AdultCancer Research8-Oxo-2'-deoxyguanosinemedicine.disease_causeAndrologyLipid peroxidationSuperoxide dismutasechemistry.chemical_compoundmedicineHumansNeoplasms Glandular and EpithelialAgedNeoplasm Stagingchemistry.chemical_classificationAged 80 and overOvarian NeoplasmsGlutathione PeroxidasebiologySuperoxide DismutaseGlutathione peroxidase8-Hydroxy-2'-deoxyguanosineDeoxyguanosineGlutathioneMiddle AgedMalondialdehydeCatalaseGlutathioneOncologyBiochemistrychemistry8-Hydroxy-2'-Deoxyguanosinebiology.proteinFemaleLipid PeroxidationOxidative stressDNA DamageCancer letters
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Induction of heme oxygenase-1 and adaptive protection against the induction of DNA damage after hyperbaric oxygen treatment.

2000

Hyperbaric oxygen (HBO) treatment of human subjects (i.e. exposure to 100% oxygen at a pressure of 2.5 ATA for a total period of 3 x 20 min) caused clear and reproducible DNA damage in lymphocytes, as detected with the comet assay (single cell gel electrophoresis). Induction of DNA damage was found only after the first HBO exposure and not after further treatments of the same individuals. Furthermore, blood taken 24 h after HBO treatment was significantly protected against the induction of DNA damage by hydrogen peroxide (H(2)O(2)) in vitro, indicating that adaptation occurred due to induction of antioxidant defenses. The cells were not significantly protected against the genotoxic effects …

AdultCancer ResearchDNA RepairDNA repairDNA damageCarbon-Oxygen LyasesBiologymedicine.disease_causeSuperoxide dismutasemedicineDNA-(Apurinic or Apyrimidinic Site) LyaseHumansLymphocytesDNA Polymerase betachemistry.chemical_classificationReactive oxygen speciesHyperbaric OxygenationSuperoxide DismutaseMembrane ProteinsGeneral MedicineHydrogen PeroxideCatalaseMolecular biologyDNA-(apurinic or apyrimidinic site) lyaseAdaptation PhysiologicalDeoxyribonuclease IV (Phage T4-Induced)Comet assayOxidative StresschemistryBiochemistryCatalaseEnzyme InductionHeme Oxygenase (Decyclizing)biology.proteinOxidative stressHeme Oxygenase-1DNA DamageCarcinogenesis
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Active paraplegics are protected against exercise-induced oxidative damage through the induction of antioxidant enzymes

2016

Exercise improves functional capacity in spinal cord injury (SCI). However, exhaustive exercise, especially when sporadic, is linked to the production of reactive oxygen species that may have a detrimental effect on SCI. We aimed to study the effect of a single bout of exhaustive exercise on systemic oxidative stress parameters and on the expression of antioxidant enzymes in individuals with paraplegia. The study was conducted in the Physical Therapy department and the Physical Education and Sports department of the University of Valencia. Sixteen paraplegic subjects were submitted to a graded exercise test (GET) until volitional exhaustion. They were divided into active or non-active group…

AdultMale0301 basic medicinePathologymedicine.medical_specialtyAntioxidantNeurologyEnzimasmedicine.medical_treatmentEnzimaPharmacologyAntioxidantsProtein CarbonylationOxidative damage03 medical and health sciences0302 clinical medicineMalondialdehydeAccelerometrymedicineHumansRNA MessengerExerciseSpinal cord injuryAgedParaplegiachemistry.chemical_classificationGlutathione PeroxidaseParaplejíaSuperoxide Dismutasebusiness.industryGeneral MedicineMiddle AgedCatalasemedicine.disease030104 developmental biologyEnzymeGene Expression RegulationNeurologychemistryExercise TestLeukocytes MononuclearFemaleLipid PeroxidationNeurology (clinical)ParaplegiabusinessEnfermedad030217 neurology & neurosurgery
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Antioxidant Activities and Oxidative Stress Byproducts in Human Hypertension

2003

The objective was to study oxidative status, antioxidant activities, and reactive oxygen species byproducts in whole blood and mononuclear peripherals cells and their relationship with blood pressure. Sixty-six hypertensive patients and 16 normotensive volunteers as a control group were studied. In both, whole blood and peripheral mononuclear cells oxidized/reduced glutathione ratio and malondialdehyde was significantly higher, and the activity of superoxide dismutase, catalase, and glutathione peroxidase was significantly lower in hypertensive patients when compared with normal subjects. The content of damaged base 8-oxo-2′-deoxyguanosine in nuclear and mitochondrial deoxyribonucleoprotein…

AdultMaleAntioxidantmedicine.medical_treatmentBlood PressureOxidative phosphorylationPharmacologymedicine.disease_causeDNA MitochondrialAntioxidantsMalondialdehydeInternal MedicinemedicineHumansWhole bloodchemistry.chemical_classificationGlutathione PeroxidaseReactive oxygen speciesGlutathione DisulfideSuperoxide DismutaseChemistryDeoxyguanosine8-Hydroxy-2'-deoxyguanosineDNAMetabolismMiddle AgedCatalaseGlutathioneDNA metabolismOxidative StressBiochemistry8-Hydroxy-2'-DeoxyguanosineHypertensionFemaleReactive Oxygen SpeciesOxidative stressHypertension
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Inadequate Cytoplasmic Antioxidant Enzymes Response Contributes to the Oxidative Stress in Human Hypertension

2006

Untreated hypertensive patients show increased oxidative stress and decreased antioxidant enzyme activity in mononuclear cells. Therefore, the objective of this study was to determine whether or not the low antioxidant enzyme activity observed in mononuclear cells of hypertensive subjects is in part dependent on a defective activity of antioxidant mechanisms. Activity and mRNA level of antioxidant enzymes, CuZn- and Mn-superoxide dismutases, catalase, glutathione peroxidase type 1, and glutathione reductase were simultaneously measured in mononuclear cells of controls (n = 38) and hypertensive subjects (n = 35), in the absence of and during antihypertensive treatment. An increase in oxidati…

AdultMaleCytoplasmmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentGlutathione reductasemedicine.disease_causeAntioxidantsSuperoxide dismutasechemistry.chemical_compoundGlutathione Peroxidase GPX1Internal medicineInternal MedicinemedicineHumansRNA MessengerAntihypertensive Agentschemistry.chemical_classificationGlutathione PeroxidasebiologySuperoxide Dismutasebusiness.industryGlutathione peroxidaseNADPH OxidasesGlutathioneMiddle AgedCatalaseOxidative StressGlutathione ReductaseEndocrinologychemistryCase-Control StudiesHypertensionbiology.proteinFemaleDismutaseOxidoreductasesbusinessOxidative stressPeroxidaseAmerican Journal of Hypertension
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Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohnʼs disease

2009

Background: Oxidative stress is considered a potential etiological factor for Crohn's disease (CD). We characterized the reactive oxygen species (ROS) generated in immune peripheral cells of CD patients, as well as their antioxidant enzyme status and the presence of oxidative damage. In addition, mitochondrial function (ΔΨm) was analyzed to detect the possible origin of ROS. Methods: Cells were obtained from patients at the onset of disease, prior to any treatment. Experiments were repeated when patients were in clinical remission. A set of experiments was carried out in a group of CD patients in persistent morphological remission. Controls were healthy volunteers who were not receiving any…

AdultMaleMitochondrial DiseasesMitochondrionBiologyPharmacologymedicine.disease_causeInflammatory bowel diseaseAntioxidantsMonocytesNitric oxideSuperoxide dismutasechemistry.chemical_compoundCrohn DiseaseMalondialdehydemedicineHumansImmunology and AllergyLymphocytesMembrane Potential Mitochondrialchemistry.chemical_classificationReactive oxygen speciesSuperoxide DismutaseSuperoxideGastroenterologyDeoxyguanosineHydrogen PeroxideCatalaseOxidantsmedicine.diseaseOxidative Stresschemistry8-Hydroxy-2'-DeoxyguanosineCatalaseCase-Control StudiesImmunologybiology.proteinFemaleReactive Oxygen SpeciesOxidative stressGranulocytesInflammatory Bowel Diseases
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Oxidant and antioxidant status in mothers and their newborns according to birthweight

2008

The aim of this study is to determine the oxidant and antioxidant status in Algerian mothers and their newborns according to birth weight.Subjects for the study were consecutively recruited from Tlemcen hospital. 139 pregnant women and their newborns were included. The plasma total antioxidant activity (ORAC), vitamins A, C, E, hydroperoxides, carbonyl proteins, and erythrocyte antioxidant enzyme activities (catalase, glutathione peroxidase, glutathione reductase and superoxide dismutase) were measured on mothers and their newborns. Lipid and lipoprotein parameters were also determined. The results were assessed in accordance with small for gestational age (SGA), appropriate (AGA) and large…

AdultMalePediatricsmedicine.medical_specialtyErythrocytesAntioxidantLipoproteinsmedicine.medical_treatmentBirth weightGlutathione reductaseIntrauterine growth restrictionPhysiologyAscorbic AcidAntioxidantsPregnancymedicineBirth WeightHumansVitamin EVitamin Achemistry.chemical_classificationGlutathione PeroxidaseFetal Growth RetardationSuperoxide Dismutasebusiness.industryVitamin EGlutathione peroxidaseInfant NewbornObstetrics and GynecologyCatalaseOxidantsmedicine.diseaseAscorbic acidLipidsOxidative StressGlutathione ReductaseReproductive MedicinechemistryInfant Small for Gestational AgeSmall for gestational ageFemalebusinessEuropean Journal of Obstetrics & Gynecology and Reproductive Biology
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