Search results for "Cell Differentiation"

showing 10 items of 907 documents

Differentiation-regulated loss of the polysialylated embryonic form and expression of the different polypeptides of the neural cell adhesion molecule…

1989

The expression of the neural cell adhesion molecule (N-CAM) on cultured murine oligodendrocytes, their precursors, and myelin was examined by indirect immunofluorescence, biosynthetic radiolabeling followed by immunoprecipitation and Western blot analysis, using antibodies specific for various forms of the molecule. In all culture systems studied, whether the oligodendrocytes were cultured as an enriched fraction containing precursor cells or in the presence of astrocytes and neurons, a similar differentiation-stage-related expression of N-CAM was seen. At early developmental stages many tetanus toxin receptor- and A2B5 antigen-positive putative oligodendrocyte precursors with bipolar morph…

Polydendrocytesanimal structuresFluorescent Antibody TechniqueMice Inbred StrainsBiologyMiceCellular and Molecular NeuroscienceMyelinCerebellumCell AdhesionmedicineAnimalsProtein PrecursorsCells CulturedMyelin SheathMembrane GlycoproteinsCell adhesion moleculeAntibodies MonoclonalCell DifferentiationEmbryo MammalianEmbryonic stem cellOligodendrocyteCell biologyOligodendrogliamedicine.anatomical_structureCell cultureType C PhospholipasesAntigens SurfaceSialic AcidsNeurogliaNeural cell adhesion moleculeCell Adhesion MoleculesNeurogliaNeuroscienceJournal of Neuroscience Research
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Uptake of polymeric nanoparticles in a human induced pluripotent stem cell-based blood-brain barrier model: Impact of size, material, and protein cor…

2021

The blood–brain barrier (BBB) maintains the homeostasis of the central nervous system, which is one of the reasons for the treatments of brain disorders being challenging in nature. Nanoparticles (NPs) have been seen as potential drug delivery systems to the brain overcoming the tight barrier of endothelial cells. Using a BBB model system based on human induced pluripotent stem cells (iPSCs), the impact of polymeric nanoparticles has been studied in relation to nanoparticle size, material, and protein corona. PLGA [poly(lactic-co-glycolic acid)] and PLLA [poly(d,l-lactide)] nanoparticles stabilized with Tween® 80 were synthesized (50 and 100 nm). iPSCs were differentiated into human brain m…

PolymersInduced Pluripotent Stem CellsStatic ElectricityGeneral Physics and AstronomyNanoparticleProtein Corona02 engineering and technology010402 general chemistryBlood–brain barrier01 natural sciencesModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyBiomaterialschemistry.chemical_compoundPolylactic Acid-Polyglycolic Acid CopolymermedicineElectric ImpedanceHumansGeneral Materials ScienceParticle SizeInduced pluripotent stem cellEndothelial CellsCell DifferentiationGeneral ChemistryHuman brain021001 nanoscience & nanotechnologyDynamic Light ScatteringFractionation Field Flow0104 chemical sciencesPLGAmedicine.anatomical_structurechemistryBlood-Brain BarrierSelective adsorptionDrug deliveryCalibrationBiophysicsNanoparticlesPolystyrenesProtein Corona0210 nano-technologyBiointerphases
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Differentiation-associated apoptosis of neural stem cells is effected by Bcl-2 overexpression: impact on cell lineage determination

2001

Apoptosis is an integral part of neural development. To elucidate the importance of programmed cell death on cell lineage determination we utilized murine PCC7-Mzl cells, a model system for neural differentiation. Treatment of pluripotent PCC7-Mzl stem cells with 0.1 microM all-trans retinoic acid (RA) causes a cease of proliferation and an initiation of differentiation into neurons, glial cells and fibroblasts. Simultaneously, a fraction of the cell culture (ca. 25%) dies within 24 h by apoptosis. We transfected PCC7-Mzl cells with the human bcl-2 cDNA and generated PCC7-Mz-Bcl-2 cell lines expressing two- to tenfold higher levels of Bcl-2 than parental cells. Overexpression of Bcl-2 resul…

Programmed cell deathDNA ComplementaryHistologyCellular differentiationApoptosisTretinoinBiologyCeramidesTransfectionPathology and Forensic MedicineMiceNeurosphereTumor Cells CulturedAnimalsCell LineageElectrophoresis Agar GelNeuronsCaspase 8Stem CellsCell DifferentiationCell BiologyGeneral MedicineFibroblastsMolecular biologyCaspase 9Neural stem cellCell biologyP19 cellProto-Oncogene Proteins c-bcl-2Cell cultureCaspasesStem cellNeurogliaBiomarkersCell DivisionAdult stem cellEuropean Journal of Cell Biology
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Knockout of myeloid cell leukemia-1 induces liver damage and increases apoptosis susceptibility of murine hepatocytes

2008

Apoptosis, or programmed cell death, regulates tissue development and homeostasis in multi-cellular organisms. Extrinsic or intrinsic death signals activate pro-apoptotic pathways, resulting in the activation of caspases and finally in cell death. An important event during apoptosis process is the permeabilization of the outer mitochondrial membrane (OMM). Integrity of the OMM is regulated by the Bcl-2 protein family, which is divided into three groups: anti-apoptotic members Bcl-2, Bcl-xL and myeloid cell leukemia-1 (Mcl-1), pro-apoptotic multidomain members Bax and Bak, and pro-apoptotic BH3-only proteins. Mitochondrial activation is regulated by selective interactions of Bcl-2 proteins v…

Programmed cell deathGenotypeCellular differentiation610 Medicine & healthApoptosisBiologyPolymerase Chain ReactionArticleMiceimmune system diseases10049 Institute of Pathology and Molecular Pathologyhemic and lymphatic diseasesmedicineAnimalsAspartate AminotransferasesneoplasmsDNA PrimersHepatologyCaspase 3Alanine TransaminaseCell DifferentiationDNAFas receptorCell biologyMyeloid Cell Leukemia Sequence 1 ProteinHaematopoiesisGene Expression RegulationLiverProto-Oncogene Proteins c-bcl-2ApoptosisHepatocytesMyeloid Cell Leukemia Sequence 1 ProteinRNA2721 HepatologyHepatocyte growth factorStem cellmedicine.drugHepatology
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Natural and induced apoptosis during lymphocyte development in the axolotl

1999

Lymphocytes apoptosis was characterized in a urodele amphibian, the axolotl, by morphology using electron microscopy and by flow cytometry after propidium iodide staining, as well as by biochemical criteria with the detection of DNA ladders after glucocorticoid treatment. The morphological and biochemical features observed in treated axolotls are in accordance with the criteria of apoptosis found in different models of mammalian lymphocyte programmed cell death. The onset of natural apoptosis was then detected by DNA fragmentation in thymus and in spleen during lymphocyte development and ontogenesis. A typical DNA ladder characteristic of apoptosis is detectable in the thymus as early as 5 …

Programmed cell deathHydrocortisoneT-LymphocytesLymphocyteImmunologyApoptosisBiologyAmbystomaFlow cytometryEnterotoxinschemistry.chemical_compoundAxolotlmedicineSuperantigenAnimalsLymphocytesPropidium iodideSuperantigensmedicine.diagnostic_testCell Differentiationbiology.organism_classificationMolecular biologymedicine.anatomical_structurechemistryApoptosisLarvaDNA fragmentationDevelopmental BiologyDevelopmental & Comparative Immunology
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Influence of organophosphate poisoning on human dendritic cells.

2013

Organophosphourus compounds (OPC, including nerve agents and pesticides) exhibit acute toxicity by inhibition of acetylcholinesterase. Lung affections are frequent complications and a risk factor for death. In addition, epidemiological studies reported immunological alterations after OPC exposure. In our experiments we investigated the effects of organophosphourus pesticides dimethoate and chlorpyrifos on dendritic cells (DC) that are essential for the initial immune response, especially in the pulmonary system. DC, differentiated from the monocyte cell line THP-1 by using various cytokines (IL-4, GM-CSF, TNF-α, Ionomycin), were exposed to organophosphourus compounds at different concentrat…

Programmed cell deathInsecticidesMAP Kinase Signaling Systemmedicine.medical_treatmentPoison controlPharmacologyToxicologyMonocytesProinflammatory cytokineCell Linechemistry.chemical_compoundOrganophosphate PoisoningmedicineHumansDimethoatePesticidesProtein kinase BCell Deathbusiness.industryMonocyteCell DifferentiationGeneral MedicineDendritic cellDendritic Cellsmedicine.anatomical_structureCytokinechemistryImmunologyIonomycinCytokinesChlorpyrifosbusinessChemico-biological interactions
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Excitotoxin-induced changes in transglutaminase during differentiation of cerebellar granule cells

2002

Excitotoxicity induced by NMDA receptor stimulation is able to increase the activity of many enzymes involved in neuronal cell death. Primary cultures of rat cerebellar granule cells were used to elucidate the role of transglutaminase reaction in the excitotoxic cell response, and to evaluate the role of glutamate receptors in cell survival and degeneration. Granule neurons, maintained in vitro for two weeks, were exposed to NMDA at different stages of differentiation. Following NMDA receptor activation, increases in transglutaminase activity were observed in cell cultures. The levels of enzyme activity were higher in cells at 5 days in vitro than in those at 8-9 or 13-14 days in vitro. Mor…

Programmed cell deathN-MethylaspartateTime FactorsCell SurvivalTissue transglutaminaseNeurotoxinsClinical BiochemistryExcitotoxicityStimulationmedicine.disease_causeReceptors N-Methyl-D-AspartateBiochemistryCerebellummedicineAnimalsRats WistarNeuronsTransglutaminasesbiologyOrganic ChemistryGlutamate receptorCell DifferentiationIn vitroRatsCell biologyAnimals Newbornnervous systemApoptosisNerve Degenerationbiology.proteinNMDA receptorTransglutaminase – Excitotoxicity – Neurodegenerative diseases – Apoptosis – Glutamate – Cerebellar granule neuronsAmino Acids
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Extracorporeal shock wave-mediated changes in proliferation, differentiation, and gene expression of human osteoblasts.

2008

The goal of this study was to determine whether cell proliferation, differentiation, and gene expression of primary human osteoblasts (hOB) are influenced by shock wave application (SWA).Osteoblast cultures were isolated from cancellous bone fragments and treated with 500 impulses of energy flux densities of 0.06 mJ/mm, 0.18 mJ/mm, 0.36 mJ/mm, and 0.50 mJ/mm. Twenty-four hours and 96 hours after SWA cell proliferation, alkaline phosphatase activity, and mineralization were analyzed. The global gene expression profiling was determined 96 hours after SWA employing Affymetrix HG-U133A microarrays.After 24 hours, hOB showed a dose-dependent increase in cell proliferation from 68.7% (at 0.06 mJ/…

Proliferation differentiationGene ExpressionIn Vitro TechniquesCritical Care and Intensive Care MedicineHigh-Energy Shock WavesBone DensityGene expressionmedicineHumansHigh-Density MicroarrayOligonucleotide Array Sequence AnalysisOsteoblastsCell growthbusiness.industryReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingOsteoblastCell DifferentiationAnatomyExtracorporeal shock waveAlkaline PhosphataseCell biologyGene expression profilingmedicine.anatomical_structureSurgerybusinessCancellous boneCell DivisionThe Journal of trauma
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Cell cycle independent role of Cyclin E during neural cell fate specification in Drosophila is mediated by its regulation of Prospero function

2009

AbstractDuring development, neural progenitor cells or neuroblasts generate a great intra- and inter-segmental diversity of neuronal and glial cell types in the nervous system. In thoracic segments of the embryonic central nervous system of Drosophila, the neuroblast NB6-4t undergoes an asymmetric first division to generate a neuronal and a glial sublineage, while abdominal NB6-4a divides once symmetrically to generate only 2 glial cells. We had earlier reported a critical function for the G1 cyclin, CyclinE (CycE) in regulating asymmetric cell division in NB6-4t. Here we show that (i) this function of CycE is independent of its role in cell cycle regulation and (ii) the two functions are m…

ProsperoNerve Tissue ProteinsStem cellsCyclinEBiologyCell fate determinationNeuroblastNeuroblastsCyclin EAsymmetric cell divisionAnimalsDrosophila ProteinsCell LineageMolecular BiologyNeural cellCell ProliferationSequence DeletionNeuronsCell fate determinationCell CycleNuclear ProteinsCell DifferentiationCell BiologyCell cycleNeural stem cellUp-RegulationCell biologyProtein TransportDrosophila melanogasternervous systemDrosophilaCNSStem cellGanglion mother cellBiomarkersProtein BindingTranscription FactorsDevelopmental BiologyDevelopmental Biology
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The N-glycan processing in HT-29 cells is a function of their state of enterocytic differentiation. Evidence for an atypical traffic associated with …

1991

International audience; When the human colon cancer cells HT-29 undergo enterocytic differentiation, they correctly process their N-glycans, whereas their undifferentiated counterpart are unable to process Man9-8-GlcNAc2 species, the natural substrate of alpha-mannosidase I. As this enzyme is fully active in both HT-29 cell populations, we hypothesize that N-glycoproteins are unable to reach the cis Golgi, the site where alpha-mannosidase I has been localized. We have demonstrated this point by using 1-deoxymannojirimycin, leupeptin, and monensin. In the presence of 1-deoxymannojirimycin, a specific inhibitor of alpha-mannosidase I, differentiated HT-29 cells, as expected, accumulate Man9-8…

Proteases1-DeoxynojirimycinColonLeupeptinsCellular differentiationCellIn Vitro TechniquesBiologyBiochemistry03 medical and health sciencessymbols.namesakechemistry.chemical_compoundPolysaccharidesalpha-Mannosidase[ CHIM.ORGA ] Chemical Sciences/Organic chemistryMannosidasesTumor Cells CulturedmedicineHumansMonensinMolecular Biology030304 developmental biologychemistry.chemical_classificationGlucosamine0303 health sciencesMembrane Glycoproteins[CHIM.ORGA]Chemical Sciences/Organic chemistryEndoplasmic reticulum030302 biochemistry & molecular biologyLeupeptinBiological TransportCell DifferentiationCell BiologyCompartment (chemistry)Golgi apparatus[CHIM.ORGA] Chemical Sciences/Organic chemistrymedicine.anatomical_structureBiochemistrychemistryColonic NeoplasmssymbolsGlycoproteinProtein Processing Post-Translational
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