Search results for "Cell Hypoxia"

showing 10 items of 84 documents

Mineralization of bone-related SaOS-2 cells under physiological hypoxic conditions

2015

Inorganic polyphosphate (polyP) is a physiological energy-rich polymer with multiple phosphoric anhydride bonds. In cells such as bone-forming osteoblasts, glycolysis is the main pathway generating metabolic energy in the form of ATP. In the present study, we show that, under hypoxic culture conditions, the growth/viability of osteoblast-like SaOS-2 cells is not impaired. The addition of polyP to those cells, administered as amorphous calcium polyP nanoparticles (aCa-polyP-NP; approximate size 100 nm), significantly increased the proliferation of the cells. In the presence of polyP, the cells produce significant levels of lactate, the end product of anaerobic glycolysis. Under those conditi…

Calcium Phosphates0301 basic medicineCell SurvivalSurface PropertiesBicarbonatechemistry.chemical_element02 engineering and technologyBiologyCalciumBiochemistryMineralization (biology)Cell LineStructure-Activity Relationship03 medical and health scienceschemistry.chemical_compoundCalcification PhysiologicAntigens NeoplasmCarbonic anhydraseHumansLactic AcidParticle SizeCarbonic Anhydrase IXMolecular BiologySaos-2 cellsCarbonic AnhydrasesCell ProliferationOsteoblastsPolyphosphateCell Biology021001 nanoscience & nanotechnologyCell HypoxiaOxygen030104 developmental biologyBiochemistrychemistryAnaerobic glycolysisCell culturebiology.proteinBiophysicsNanoparticles0210 nano-technologyFEBS Journal
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The role of hypoxia-induced factors in tumor progression.

2004

Abstract Learning Objectives After completing this course, the reader will be able to: Describe hypoxia-induced mechanisms for cell survival. Discuss hypoxia-induced gene expression. Relate hypoxia and glucose metabolism. Access and take the CME test online and receive 1 hour of AMA PRA category 1 credit atCME.TheOncologist.com Hypoxia is a common characteristic of locally advanced solid tumors that has been associated with diminished therapeutic response and, more recently, with malignant progression, that is, an increasing probability of recurrence, locoregional spread, and distant metastasis. Emerging evidence indicates that the effect of hypoxia on malignant progression is mediated by a…

Cancer ResearchAngiogenesisCell SurvivalRegulatorBiologyNeoplasmsmedicineHumansNuclear proteinSelection GeneticTranscription factorG alpha subunitRegulation of gene expressionHelix-Loop-Helix MotifsNuclear ProteinsHypoxia (medical)Hypoxia-Inducible Factor 1 alpha SubunitCell HypoxiaDNA-Binding ProteinsGene Expression Regulation NeoplasticCell Transformation NeoplasticGlucoseOncologyTumor progressionImmunologyCancer researchDisease ProgressionHypoxia-Inducible Factor 1medicine.symptomTranscription FactorsThe oncologist
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Tumor Hypoxia: Causative Factors, Compensatory Mechanisms, and Cellular Response

2004

Abstract Learning Objectives After completing this course, the reader will be able to: Explain the effect of hypoxia on resistance to treatment. Describe the causes of tumor hypoxia. Characterize cellular response to hypoxia. Access and take the CME test online and receive 1 hour of AMA PRA category 1 credit at CME.TheOncologist.com Hypoxia is a characteristic feature of locally advanced solid tumors resulting from an imbalance between oxygen (O2) supply and consumption. Major causative factors of tumor hypoxia are abnormal structure and function of the microvessels supplying the tumor, increased diffusion distances between the nutritive blood vessels and the tumor cells, and reduced O2 tra…

Cancer ResearchCell SurvivalAnemiamedicine.medical_treatmentPhotodynamic therapyDiseaseNeoplasmsmedicineHumansNeovascularization PathologicTumor hypoxiabusiness.industryAnemiaTumor OxygenationHypoxia (medical)medicine.diseaseAdaptation PhysiologicalCell HypoxiaOxygenRadiation therapyCell Transformation NeoplasticOncologyDrug Resistance NeoplasmImmunologyDisease ProgressionCancer researchHemoglobinmedicine.symptombusinessThe Oncologist
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Calibration of misonidazole labeling by simultaneous measurement of oxygen tension and labeling density in multicellular spheroids

1995

To correlate misonidazole concentrations and oxygen pressures (Po2) at identical locations within EMT6/Ro multi-cell spheroids (mean diameters +/- SD: 867 +/- 20 microns), Po2 measurements were performed with oxygen-sensitive microelectrodes during incubation of these spheroids with tritiated misonidazole (10 mg/I; 445 microCi/mg). In each individual spheroid, Po2 profiles were correlated with the corresponding spatial distribution of misonidazole as quantified by conventional autoradiography and grain counting. To compare the oxygenation status of spheroids in the measuring chamber with that of spheroids in spinner culture, misonidazole labeling was performed in both environments following…

Cancer ResearchMisonidazolePartial PressureOxygenechemistry.chemical_elementTritiumOxygenMicechemistry.chemical_compoundLaboratory flaskTumor Cells CulturedAnimalsMisonidazolecomputer.programming_languagebusiness.industryChemistrySpheroidMammary Neoplasms ExperimentalOxygenationPartial pressureCell HypoxiaOxygen tensionOxygenOncologyCalibrationembryonic structuresBiophysicsAutoradiographyFeasibility StudiesNuclear medicinebusinessMicroelectrodescomputerCell DivisionInternational Journal of Cancer
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Erythropoietin restores the anemia-induced reduction in radiosensitivity of experimental human tumors in nude mice

2003

Abstract Purpose The effect of recombinant human erythropoietin (rhEPO) on the radiosensitivity of human tumor xenografts growing in anemic and nonanemic nude mice was studied. Methods and materials Anemia was induced by total body irradiation ([TBI], 2 × 4 Gy) of mice before tumor implantation into the subcutis of the hind leg. The development of anemia was prevented by rhEPO (750 U/kg s.c.) given 3 times weekly starting 2 weeks before TBI. Fourteen days after fractionated TBI (tumor volume of approx. 40 mm 3 ), single-dose irradiation of the tumor with varying doses was performed so that in full dose–response relationship for the probability of tumor cure was obtained. Results Radiation-i…

Cancer ResearchPathologymedicine.medical_specialtyAnemiamedicine.medical_treatmentTransplantation HeterologousDrug Evaluation PreclinicalUrologyMice NudeHindlimbRadiation ToleranceHemoglobinsMicemedicineAnimalsHumansRadiology Nuclear Medicine and imagingRadiosensitivityddc:610ErythropoietinRadiationbusiness.industryAnemiaSarcomaHypoxia (medical)Total body irradiationmedicine.diseaseCell HypoxiaRecombinant ProteinsRadiation therapyRadiation Injuries ExperimentalOncologyErythropoietinDose Fractionation RadiationHemoglobinmedicine.symptombusinessNeoplasm TransplantationWhole-Body Irradiationmedicine.drug
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Comparative study of human colonic tumor-derived endothelial cells (HCTEC) and normal colonic microvascular endothelial cells (HCMEC): Hypoxia-induce…

2009

Colorectal carcinoma growth and progression is dependent on the vasculature of the tumor microenvironment. Tumor-derived endothelial cells differ functionally from their normal counterpart. For this reason we isolated microvascular endothelial cells from human colon cancer tissue (HCTEC) and compared them with endothelial cells from normal colonic tissue (HCMEC) of the same donor. Since hypoxia is a universal hallmark of carcinomas, we examined its effects on HCTEC of five patients in comparison with the corresponding HCMEC, with respect to the secretion of the soluble form of the two important vascular endothelial growth factor (VEGF) receptors, VEGFR-1 and -2. After dissociation by dispas…

Cancer ResearchPathologymedicine.medical_specialtyEndotheliumColonEnzyme-Linked Immunosorbent AssayCell SeparationBiologychemistry.chemical_compoundmedicineHumansCells CulturedTumor microenvironmentVascular Endothelial Growth Factor Receptor-1OncogeneMicrocirculationEndothelial CellsGeneral MedicineVascular Endothelial Growth Factor Receptor-2Cell HypoxiaEndothelial stem cellVascular endothelial growth factormedicine.anatomical_structureOncologychemistryApoptosisTumor progressionColonic NeoplasmsCancer researchTumor necrosis factor alphaOncology Reports
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GLUT-1 staining of squamous cell carcinomas of the uterine cervix identifies a novel element of invasion.

2010

Perturbation of the normal tissue architecture in solid malignant tumors is perceived to be the consequence of actively migrating cancer cells which invade the adjacent normal host tissue. The opposite, invasion of cancer cell clusters by a vascularized stroma, has not been considered. The latter process should, however, be expected to occur since the hypoxic cores of tumor cell aggregates, under the control of HIF-1, are known to secrete cytokines (e.g., bFGF, VEGF) which attract fibroblasts and induce blood vessel formation. In this study, the expression of glucose transporter (GLUT)-1, a major HIF-1 target gene, was examined in 51 squamous cell carcinomas of the uterine cervix by immunoh…

Cancer ResearchPathologymedicine.medical_specialtyStromal cellAngiogenesisCellUterine Cervical NeoplasmsBiologyMetastasisStromamedicineHumansNeoplasm InvasivenessProspective StudiesCell ShapeGlucose Transporter Type 1Tumor hypoxiaCell cyclemedicine.diseaseImmunohistochemistryCell Hypoxiamedicine.anatomical_structureOncologyCancer cellCarcinoma Squamous CellFemaleStromal CellsInternational journal of oncology
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Tissue gradients of energy metabolites mirror oxygen tension gradients in a rat mammary carcinoma model

2001

Abstract Purpose: It has been shown that oxygen gradients exist in R3230AC tumors grown in window chambers. The fascial surface is better oxygenated than the tumor surface. The purpose of the present study was to determine whether gradients exist for energy metabolites and other end points related to oxygen transport. Methods and Materials: Imaging bioluminescence was used to measure ATP, glucose, and lactate in cryosections of R3230AC tumors. Mean vessel density and hypoxic tissue fraction were assessed using immunohistochemistry. Tumor redox ratio was assessed by redox ratio scanning. Results: Lactate content and hypoxic fraction increased, whereas ATP, glucose, redox ratio, and vessel de…

Cancer ResearchPathologymedicine.medical_specialtychemistry.chemical_elementOxygenMicrocirculationAdenosine TriphosphatemedicineAnimalsPimonidazoleRadiology Nuclear Medicine and imagingLactic AcidRadiationTumor hypoxiabusiness.industryMicrocirculationOxygen transportMammary Neoplasms ExperimentalRadiobiologyOxygenationMetabolismCell HypoxiaRats Inbred F344RatsOxygen tensionOxygenGlucoseOncologychemistryLuminescent MeasurementsBiophysicsDiffusion Chambers CulturebusinessOxidation-ReductionInternational Journal of Radiation Oncology*Biology*Physics
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Nupr1-Aurora Kinase A Pathway Provides Protection against Metabolic Stress-Mediated Autophagic-Associated Cell Death

2012

Abstract Purpose: The limited supply of oxygen and nutrients is thought to result in rigorous selection of cells that will eventually form the tumor. Experimental Design: Nupr1 expression pattern was analyzed in human tissue microarray (TMA) and correlated with survival time of the patient. Microarray analysis was conducted on MiaPaCa2 cells subjected to metabolic stress in Nupr1-silenced conditions. DNA repair and cell cycle–associated gene expression was confirmed by real-time quantitative PCR (qRT-PCR). Nupr1 and AURKA protective role were analyzed using RNA interference (RNAi) silencing or overexpression. DNA damage and autophagy were analyzed by Western blot analysis and immunofluoresc…

Cancer ResearchProgrammed cell deathCell SurvivalDNA damageDNA repairAdenocarcinomaProtein Serine-Threonine KinasesBiologyAurora KinasesStress PhysiologicalCell Line TumorAutophagyBasic Helix-Loop-Helix Transcription FactorsHumansGene silencingAurora Kinase ARegulation of gene expressionGene knockdownMicroarray analysis techniquesAURKA GeneMolecular biologyCell HypoxiaNeoplasm ProteinsCell biologyGene Expression Regulation NeoplasticGlucoseOncologyRNA InterferenceCarcinoma Pancreatic DuctalClinical Cancer Research
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HIF-1 is involved in the negative regulation of AURKA expression in breast cancer cell lines under hypoxic conditions

2013

Numerous microarray-based gene expression studies performed on several types of solid tumors revealed significant changes in key genes involved in progression and regulation of the cell cycle, including AURKA that is known to be overexpressed in many types of human malignancies. Tumor hypoxia is associated with poor prognosis in several cancer types, including breast cancer (BC). Since hypoxia is a condition that influences the expression of many genes involved in tumorigenesis, proliferation, and cell cycle regulation, we performed a microarray-based gene expression analysis in order to identify differentially expressed genes in BC cell lines exposed to hypoxia. This analysis showed that h…

Cancer ResearchSettore MED/06 - Oncologia MedicaDown-RegulationBreast NeoplasmsBiologymedicine.disease_causeAURKA Breast cancer Cell cycle HIF-1a HypoxiaCell Line TumorGene expressionTranscriptional regulationmedicineHumansPromoter Regions GeneticAurora Kinase ARegulation of gene expressionGene knockdownTumor hypoxiaCell cycleHypoxia (medical)Hypoxia-Inducible Factor 1 alpha SubunitMolecular biologyCell HypoxiaGene Expression Regulation NeoplasticOncologyGene Knockdown TechniquesCancer researchFemalemedicine.symptomCarcinogenesisBreast Cancer Research and Treatment
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