Search results for "Cell adhesion molecule"

showing 10 items of 469 documents

Generation of human pulmonary microvascular endothelial cell lines.

2001

The limited lifespan of human microvascular endothelial cells in cell culture represents a major obstacle for the study of microvascular pathobiology. To date, no endothelial cell line is available that demonstrates all of the fundamental characteristics of microvascular endothelial cells. We have generated endothelial cell lines from human pulmonary microvascular endothelial cells (HPMEC) isolated from adult donors. HPMEC were cotransfected with a plasmid encoding the catalytic component of telomerase (hTERT) and a plasmid encoding the simian virus 40 (SV40) large T antigen. Cells transfected with either plasmid alone had an extended lifespan, but the cultures eventually entered crisis aft…

CD31AdultLipopolysaccharidesPathologymedicine.medical_specialtyPulmonary CirculationTime FactorsEndotheliumAngiogenesisCell SurvivalCell TransplantationAntigens Polyomavirus TransformingTransplantation HeterologousMice NudeNeovascularization PhysiologicBiologyTransfectionPathology and Forensic MedicineCell LineMiceCatalytic DomainmedicineAnimalsHumansMolecular BiologyTelomeraseCells CulturedMatrigelPlatelet Endothelial Cell Adhesion MoleculeCell adhesion moleculeMicrocirculationCell BiologyCell biologyEndothelial stem cellDNA-Binding Proteinsmedicine.anatomical_structurePhenotypeCell cultureEndothelium VascularInflammation MediatorsBiomarkersCell DivisionPlasmidsLaboratory investigation; a journal of technical methods and pathology
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Tumorigenic conversion of endothelial cells.

2003

Tumors of endothelial origin develop rarely. Until now, only two angiosarcoma (AS)-derived endothelial cell lines have been be isolated, ISO-HAS and AS-M. Both AS-derived endothelial cell lines presented the typical endothelial characteristics, such as the expression of CD31 and von Willebrand factor, but differed from normal endothelial cells in a nuclear expression of p53, in a delayed angiogenic reaction, and a reduced expression of caveolin. In addition, differences in the expression of cytokines and cell adhesion molecules responsive to proinflammatory stimuli were observed. While AS-M showed an expression pattern similar to that of human umbilical vein endothelial cells (HUVEC), ISO-H…

CD31AdultLipopolysaccharidesTelomerasePathologymedicine.medical_specialtyClinical BiochemistryCaveolin 1Vascular Cell Adhesion Molecule-1BiologyCaveolinsPathology and Forensic Medicinevon Willebrand FactormedicineCell AdhesionHumansMolecular BiologyTelomeraseCells CulturedCell NucleusCell adhesion moleculeReverse Transcriptase Polymerase Chain ReactionGranulocyte-Macrophage Colony-Stimulating FactorTelomereIntercellular Adhesion Molecule-1Cell biologyVascular endothelial growth factor BEndothelial stem cellDNA-Binding ProteinsPlatelet Endothelial Cell Adhesion Molecule-1Vascular endothelial growth factor ACell Transformation NeoplasticVascular endothelial growth factor CCell cultureEndothelium VascularTumor Suppressor Protein p53Experimental and molecular pathology
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PECAM-1/CD31 in infarction and longevity.

2007

: Inflammation has recently proven to be associated with the pathogenesis of atherosclerosis and inflammatory genes are good candidates for the risk of developing atherosclerosis. The early phase of atherosclerosis involves the recruitment of inflammatory cells from the circulation and their transendothelial migration. This process is mainly mediated by cellular adhesion molecules, which are expressed by the vascular endothelium and by circulating leukocytes in response to several inflammatory stimuli. Adhesion of circulating cells to the arterial surface is among the first detectable events in atherogenesis. Cellular adhesion molecules, expressed by the vascular endothelium and by circulat…

CD31MaleGenotypePopulationLongevityMyocardial InfarctionSingle-nucleotide polymorphismInflammationCoronary DiseaseBiologyPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologypolymorphismSex FactorsHistory and Philosophy of ScienceKEYWORDS: centenarianmedicineCell AdhesionSNPHumansGenetic Predisposition to DiseaseCell adhesioneducationSettore MED/04 - Patologia GeneraleAged 80 and overInflammationeducation.field_of_studyPolymorphism GeneticCell adhesion moleculeGeneral NeurosciencePlatelet Endothelial Cell Adhesion Molecule-1ItalyCase-Control StudiesImmunologycardiovascular systemCentenarianmedicine.symptomAnnals of the New York Academy of Sciences
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Implanted neonatal human dermal fibroblasts influence the recruitment of endothelial cells in mice

2012

The vascularization of new tissue within a reasonable time is a crucial prerequisite for the success of different cell- and material-based strategies. Considering that angiogenesis is a multi-step process involving humoral and cellular regulatory components, only in vivo assays provide the adequate information about vessel formation and the recruitment of endothelial cells. The present study aimed to investigate if neonatal human dermal fibroblasts could influence in vivo neovascularization. Results obtained showed that fibroblasts were able to recruit endothelial cells to vascularize the implanted matrix, which was further colonized by murine functional blood vessels after one week. The ve…

CD31MalePathologymedicine.medical_specialtyAngiogenesisCell TransplantationBiomedical EngineeringCD34Medicine (miscellaneous)Neovascularization PhysiologicInflammationAntigens CD34BiologyNitric OxideRegenerative MedicineBiomaterialsNeovascularizationHemoglobinsMiceTissue engineeringMicroscopy Electron TransmissionIn vivoReportmedicineAnimalsHumansRegenerationSkinInflammationMatrigelNeovascularization PathologicTissue EngineeringEndothelial CellsGeneral MedicineFibroblastsMice Inbred C57BLPlatelet Endothelial Cell Adhesion Molecule-1Drug CombinationsPhenotypeProteoglycansCollagenLamininmedicine.symptom
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Antibodies to PECAM-1 and glucocorticoids reduce leukocyte adhesion in adjuvant arthritis of the rat knee synovium in vivo.

2002

Objective and design: To demonstrate the effect of monoclonal antibodies to the adhesion-molecule PECAM-1 (CD31) and of prednisolone on leukocyte adhesion in rat adjuvant arthritis.¶Material: Adjuvant arthritis was induced in male CD®-rats (five groups of n=6) 18 days prior to measurements.¶Treatment: Mouse-monoclonal antibody to rat CD-31 at 200 μg/kg or prednisolone at 24 mg/kg were administered i.v. 15 minutes prior to measurements.¶Methods: Venules within the intact rat-knee synovium were focused by confocal laser scanning microscopy. Numbers of rolling and adherent leukocytes were assessed in vivo.¶Results: Induction of arthritis significantly increased rolling and adherent leukocytes …

CD31Malemedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentPrednisoloneImmunologyAnti-Inflammatory AgentsArthritisMonoclonal antibodyLeukocyte CountIn vivoInternal medicineSynovial FluidmedicineCell AdhesionLeukocytesAnimalsAntibodies BlockingGlucocorticoidsPharmacologyMicroscopy Confocalbusiness.industryAntibodies Monoclonalmedicine.diseaseArthritis ExperimentalRheumatologyRatsPlatelet Endothelial Cell Adhesion Molecule-1Rheumatoid arthritisImmunologyPrednisoloneJointsbusinessAdjuvantmedicine.drugInflammation research : official journal of the European Histamine Research Society ... [et al.]
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Heterogeneous expression of cell adhesion molecules by endothelial cells in ARDS

2002

ARDS (acute respiratory distress syndrome) can be associated with septic shock and multiple organ failure caused by an uncontrolled systemic inflammatory response to Gram-negative bacterial infection. While in animal models the key role of the endothelial adhesion molecules ICAM-1, E-selectin, and VCAM in ARDS has been extensively studied, there are scarcely any corresponding pathomorphological studies of human lung tissue. Hence, little is known about whether there is a comparable, or even heterogeneous, expression pattern of these molecules in the human pulmonary vasculature. This study was therefore undertaken to investigate the immunohistochemical expression of the constitutively expres…

CD31Pathologymedicine.medical_specialtyARDSLungEndotheliumSeptic shockCell adhesion moleculeBiologymedicine.diseasePathology and Forensic MedicineEndothelial stem cellmedicine.anatomical_structuremedicineSelectinThe Journal of Pathology
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Paracrine Effects Influenced by Cell Culture Medium and Consequences on Microvessel-Like Structures in Cocultures of Mesenchymal Stem Cells and Outgr…

2011

Mesenchymal stem cells (MSC) from bone marrow and outgrowth endothelial cells (OEC) from peripheral blood are considered as attractive cell types for applications in regenerative medicine aiming to build up complex vascularized tissue-engineered constructs. MSC provide several advantages such as the potential to differentiate to osteoblasts and to support the neovascularization process by release of proangiogenic factors. On the other hand, the neovascularization process can be actively supported by OEC forming perfused vascular structures after co-implantation with other cell types. In this study the formation of angiogenic structures in vitro was investigated in cocultures of MSC and OEC,…

CD31medicine.medical_treatmentCellular differentiationBiomedical EngineeringFluorescent Antibody TechniqueEnzyme-Linked Immunosorbent AssayBioengineeringCD146 AntigenBiologyPolymerase Chain ReactionBiochemistryBiomaterialsParacrine signallingchemistry.chemical_compoundmedicineHumansCells CulturedGrowth factorMesenchymal stem cellEndothelial CellsCell DifferentiationMesenchymal Stem CellsFlow CytometryCoculture TechniquesCulture MediaCell biologyPlatelet Endothelial Cell Adhesion Molecule-1Vascular endothelial growth factorchemistryCell cultureImmunologyCD146Tissue Engineering Part A
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Role of genetic factors on the effect of additional loading doses and two maintenance doses used to overcome clopidogrel hyporesponsiveness.

2014

Background and objective: Additional loading doses and higher maintenance doses (MDs) have been used to overcome hyporesponsiveness of clopidogrel. We aimed to investigate whether genetic polymorphisms of two cytochromes (CYP2C19 and CYP2C9) and ABCB1 modify effect of such dose-adjustment strategy.Materials and methods: We enrolled 118 patients undergoing elective or acute percutaneous coronary intervention (PCI) with drug eluting stent (DES). Platelet reactivity index (PRI) was measured using the vasodilator-stimulated phosphoprotein (VASP) index and a cut-off value of ≥60% was defined as hyporesponsiveness. Polymorphism of two cytochromes (CYP2C19, CYP2C9) and gene ABCB1 were determined. …

CYP2C9MaleMedicine (General)ATP Binding Cassette Transporter Subfamily BTiclopidinemedicine.medical_treatmentCYP2C19PharmacologyR5-920Percutaneous Coronary InterventionmedicinePotencyHumansProspective StudiesCYP2C19AlleleCYP2C9AllelesAgedCytochrome P-450 CYP2C9Medicine(all)Polymorphism GeneticDose-Response Relationship Drugbusiness.industryClopidogrel resistanceMicrofilament ProteinsPercutaneous coronary interventionABCB1Drug-Eluting StentsVASPMiddle AgedClopidogrelPhosphoproteinsClopidogrelCytochrome P-450 CYP2C19Drug-eluting stentPharmacogeneticsAutomotive EngineeringConventional PCIFemalebusinessCell Adhesion MoleculesPlatelet Aggregation InhibitorsClopidogrel resistance; VASP; CYP2C19; ABCB1; CYP2C9medicine.drugMedicina (Kaunas, Lithuania)
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Role of exosomes released by chronic myelogenous leukemia cells in angiogenesis

2012

The present study is designed to assess if exosomes released from Chronic Myelogenous Leukemia (CML) cells may modulate angiogenesis. We have isolated and characterized the exosomes generated from LAMA84 CML cells and demonstrated that addition of exosomes to human vascular endothelial cells (HUVEC) induces an increase of both ICAM-1 and VCAM-1 cell adhesion molecules and interleukin-8 expression. The stimulation of cell-cell adhesion molecules was paralleled by a dose-dependent increase of adhesion of CML cells to a HUVEC monolayer. We further showed that the treatment with exosomes from CML cells caused an increase in endothelial cell motility accompanied by a loss of VE-cadherin and β-ca…

Cancer ResearchAngiogenesisVascular Cell Adhesion Molecule-1BiologyExosomesArticleExosomes Chronic Myelogenous Leukemia Cells Endothelial cells Tumor MicroenvironmentMiceAntigens CDCell Movementhemic and lymphatic diseasesCell Line TumorLeukemia Myelogenous Chronic BCR-ABL PositivemedicineCell AdhesionHuman Umbilical Vein Endothelial CellsTumor MicroenvironmentAnimalsHumansCell adhesionbeta CateninMatrigelTumor microenvironmentNeovascularization PathologicCell adhesion moleculeInterleukin-8medicine.diseaseCadherinsIntercellular Adhesion Molecule-1MicrovesiclesCell biologyEndothelial stem cellDrug CombinationsOncologyGene Expression RegulationCancer researchProteoglycansCollagenLamininChronic myelogenous leukemia
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Migration of renal carcinoma cells is dependent on protein kinase Cδ via β1 integrin and focal adhesion kinase

2008

Migration and adhesion of tumor cells are essential prerequisites for the formation of metastases in malignant diseases. Protein kinase C (PKC) has been shown to regulate cell migration, adhesion and proliferation. In order to identify a connection between PKC isoforms and tumor progression in renal cell carcinoma (RCC), the influence of PKC isoforms on cell migration, adhesion and proliferation and possible influences of the activity of integrins and focal adhesion kinase (FAK) were analyzed in RCC cells. The experiments were performed in the RCC cell line CCF-RC1 after pre-incubation of the cells with the PKC inhibitors GF109203X, GO6976, RO31-8220 and rottlerin. Cell migration and adhesi…

Cancer ResearchCell growthCell adhesion moleculeIntegrinCell migrationCell cycleBiologyCell biologyFocal adhesionOncologyCancer researchbiology.proteinCell adhesionProtein kinase CInternational Journal of Oncology
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