Search results for "Cell adhesion"

showing 10 items of 812 documents

Retinoid X receptor agonists impair arterial mononuclear cell recruitment through peroxisome proliferator-activated receptor-γ activation.

2012

Abstract Mononuclear cell migration into the vascular subendothelium constitutes an early event of the atherogenic process. Because the effect of retinoid X receptor (RXR)α on arterial mononuclear leukocyte recruitment is poorly understood, this study investigated whether RXR agonists can affect this response and the underlying mechanisms involved. Decreased RXRα expression was detected after 4 h stimulation of human umbilical arterial endothelial cells with TNF-α. Interestingly, under physiological flow conditions, TNF-α–induced endothelial adhesion of human mononuclear cells was concentration-dependently inhibited by preincubation of the human umbilical arterial endothelial cells with RXR…

medicine.medical_specialtyEndotheliumTetrahydronaphthalenesImmunologyPeroxisome proliferator-activated receptorDown-RegulationVascular Cell Adhesion Molecule-1Cell CommunicationRetinoid X receptorBiologyPeripheral blood mononuclear cellUmbilical ArteriesCell LineInternal medicinemedicineImmunology and AllergyHumansReceptorMuscle SkeletalBexarotenechemistry.chemical_classificationRetinoid X Receptor alphaTumor Necrosis Factor-alphaMicrocirculationIntercellular Adhesion Molecule-1Cell biologyPPAR gammaEndocrinologymedicine.anatomical_structureNuclear receptorchemistryBexaroteneCell Migration InhibitionLeukocytes MononuclearEndothelium VascularMononuclear cell migrationmedicine.drugJournal of immunology (Baltimore, Md. : 1950)
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Adhesion molecules, endothelial products, and microalbuminuria in essential hypertension

2001

medicine.medical_specialtyEndotheliumbiologybusiness.industryCell adhesion moleculeIntercellular Adhesion Molecule-1Essential hypertensionmedicine.diseaseEndothelin 1medicine.anatomical_structureEndocrinologyInternal medicineE-selectinInternal Medicinemedicinebiology.proteinMicroalbuminuriaEndothelial dysfunctionbusinessAmerican Journal of Hypertension
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ABSTRACTS: 9
Phenotype characterization of the anti-inflammatory behavior of decidual endothelial cells

2008

Problem:  During pregnancy, an adequate inflammatory control is essential for normal intrauterine foetus development. Vascular endothelium is known to play a critical role in regulation of inflammatory processes. We hypothesize a different ability of decidual endothelial cells (DEC) compared to endothelium from other districts to respond to inflammatory stimuli. Material and Methods:  DEC were isolated and activated with inflammatory stimuli and their response in term of adhesion molecules expression, vascular leakage and cytokines production was evaluated. These data were compared to the data obtained with HUVEC and microvascular endothelial cells isolated from human skin (ADMEC) and from …

medicine.medical_specialtyFetusEndotheliumCell adhesion moleculemedicine.drug_classImmunologyObstetrics and GynecologyHuman skinStimulationBiologyVascular leakagePhenotypeAnti-inflammatoryCell biologymedicine.anatomical_structureEndocrinologyReproductive MedicineInternal medicinemedicineImmunology and AllergyAmerican Journal of Reproductive Immunology
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Diacylglycerol kinase α mediatses 17-β-estradiol-induced proliferation, motility, and anchorage-independent growth of Hec-1A endometrial cancer cell …

2011

Increased levels of endogenous and/or exogenous estrogens are one of the well known risk factors of endometrial cancer. Diacylglycerol kinases (DGKs) are a family of enzymes which phosphorylate diacylglycerol (DAG) to produce phosphatidic acid (PA), thus turning off and on DAG-mediated and PA-mediated signaling pathways, respectively. DGK α activity is stimulated by growth factors and oncogenes and is required for chemotactic, proliferative, and angiogenic signaling in vitro. Herein, using either specific siRNAs or the pharmacological inhibitor R59949, we demonstrate that DGK α activity is required for 17-β-estradiol (E2)-induced proliferation, motility, and anchorage-independent growth of …

medicine.medical_specialtyGPR30medicine.drug_classCell SurvivalDiacylglycerol kinaseMotilityEstrogen receptorEnzyme AssayEndometrial carcinomaBiologyQuinazolinoneReceptors G-Protein-CoupledPiperidinePiperidinesCell MovementInternal medicineCell Line TumormedicineCell AdhesionHumansEndometrial NeoplasmEnzyme AssaysQuinazolinonesDiacylglycerol kinaseCell ProliferationEstradiolCell growthKinaseCell BiologyDiacylglycerol kinase; Endometrial carcinoma; Estrogen; GPR30; Cell BiologyEstrogenEndometrial NeoplasmsCell biologyEnzyme ActivationLipoprotein LipaseEndocrinologyReceptors EstrogenEstrogenGene Knockdown TechniquesGene Knockdown TechniqueFemaleRNA InterferenceSignal transductionGPERHuman
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Neopterin, cellular adhesion molecules and myeolperoxidase in patients with stable and unstable angina pectoris

2011

Neopterin, cellular adhesion molecules and myeolperoxidase in patients with stable and unstable angina pectoris Recent data indicate that the serum level of neopterin, a marker of inflammation and immune modulator secreted by monocytes/macrophages, is elevated in patients with acute coronary syndrome (ACS) and seems to be a prognostic marker for major cardiovascular events. Soluble cellular adhesion molecules (sCAMs) and myeloperoxidase (MPO) levels are also related to ACS. The aim of the present study was to evaluate differences in serum levels of neopterin, sCAMs and MPO between coronary artery disease and metabolic syndrome (CAD-MetS) patients with stable and unstable angina pectoris (SA…

medicine.medical_specialtyGeneral interestSciencemetabolic syndromecellular adhesion moleculesCoronary artery diseasechemistry.chemical_compoundangina pectorisimmune system diseasesInternal medicinemedicineIn patientMultidisciplinarybiologyUnstable anginaCell adhesion moleculeQNeopterinmedicine.diseasemyeloperoxidaseneopterinchemistryMyeloperoxidasebiology.proteinCardiologyMetabolic syndromecoronary artery diseaseProceedings of the Latvian Academy of Sciences. Section B. Natural, Exact, and Applied Sciences.
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An alternative role of C1q in cell migration and tissue remodeling: contribution to trophoblast invasion and placental development.

2010

Abstract Fetal trophoblast cells invading the decidua in the early phase of pregnancy establish complex interaction with the maternal extracellular matrix. We discovered that C1q was widely distributed in human decidual stroma in the absence of C4 and C3 and was actively synthesized by migrating extravillous trophoblasts. The cells expressed the messages for the three chains of C1q and secreted this complement component that interacted with the proteins of the decidual extracellular matrix. Solid phase-bound C1q promoted trophoblast adhesion and migration, and cell binding to C1q resulted in activation of ERK1/2 MAPKs. Ab inhibition experiments showed that the receptors for the globular hea…

medicine.medical_specialtyImmunologyCellIntegrinImmunoblottingchemical and pharmacologic phenomenaBiologyExtracellular matrixMicePre-Eclampsiaimmune system diseasesPregnancyInternal medicinemedicineCell AdhesionImmunology and AllergyAnimalsHumansImmunoprecipitationskin and connective tissue diseasesReceptorCell adhesionreproductive and urinary physiologyMicroscopy ConfocalC1q placental development.Reverse Transcriptase Polymerase Chain ReactionComplement C1qDeciduaTrophoblastPlacentationImmunohistochemistryPlacentationCell biologyTrophoblastsMice Inbred C57BLChemotaxis Leukocytemedicine.anatomical_structureEndocrinologyembryonic structuresbiology.proteinFemaleJournal of immunology (Baltimore, Md. : 1950)
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Islet beta-cell apoptosis triggered in vivo by interleukin-1beta is not related to the inducible nitric oxide synthase pathway: evidence for mitochon…

2003

IL-1beta is recognized as an effector cytokine contributing to islet beta-cell destruction during diabetes. We have previously shown in vitro that IL-1beta induces nitric oxide (NO) and beta-cell damage. Here, we show that IL-1beta administration in vivo to Wistar rats transiently increases manganese superoxide dismutase activity, whereas inducible NO synthase is not detected, and the levels of nitrate+nitrate do not change. Moreover, a significant decrease of mitochondrial aconitase, leading to a rise of hydroperoxides, and islet beta-cell apoptosis, involving caspase-3 and -8, is observed. Analysis of adhesion molecules in beta-cells showed that intercellular adhesion molecule-1 is highly…

medicine.medical_specialtyLipid PeroxidesNitric Oxide Synthase Type IIApoptosisBiologyMitochondrionIn Vitro TechniquesAconitaseNitric oxidechemistry.chemical_compoundIslets of LangerhansEndocrinologyIn vivoInternal medicinemedicineAnimalsRats WistarNitritesAconitate HydratasegeographyCaspase 8geography.geographical_feature_categoryNitratesCell adhesion moleculeCaspase 3Superoxide DismutaseIsletIntercellular Adhesion Molecule-1Caspase 9Cell biologyMitochondriaRatsNitric oxide synthaseEndocrinologyBiochemistrychemistryApoptosisCaspasesbiology.proteinNitric Oxide SynthaseInterleukin-1Endocrinology
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Surface Topographies of Glaucoma Drainage Devices and Their Influence on Human Tenon Fibroblast Adhesion

2010

PURPOSE. This study was performed to investigate the surface topography of different glaucoma drainage devices and to determine the effects of surface roughness on cell adhesion of cultured human tenon fibroblasts.METHODS. The surface topography of four widely used devices (Ahmed FP7 and Ahmed S-2; New World Medical, Inc., Rancho Cucamonga, CA; Baerveldt BG101-350; Advanced Medical Optics, Irvine, CA; and Molteno S1; Molteno Ophthalmic Ltd., Dunedin, New Zealand) was investigated by scanning electron microscopy, and roughness was quantified by white-light confocal microscopy. Cells were grown for 72 hours on the surfaces of implants affixed to standard culture dishes. The cells were labeled…

medicine.medical_specialtyMaterials scienceScanning electron microscopeConfocalSurface finishProsthesis Designlaw.inventionConfocal microscopylawBone plateMicroscopyCell AdhesionSurface roughnessmedicineHumansFasciaGlaucoma Drainage ImplantsCell adhesionCells CulturedMicroscopy ConfocalFibroblastsSurgeryMicroscopy Electron ScanningCell DivisionProtein BindingBiomedical engineeringInvestigative Opthalmology & Visual Science
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Stabilization of Perivascular Mast Cells by Endothelial CNP (C-Type Natriuretic Peptide)

2020

Objective: Activated perivascular mast cells (MCs) participate in different cardiovascular diseases. Many factors provoking MC degranulation have been described, while physiological counterregulators are barely known. Endothelial CNP (C-type natriuretic peptide) participates in the maintenance of vascular barrier integrity, but the target cells and mechanisms are unclear. Here, we studied whether MCs are regulated by CNP. Approach and Results: In cultured human and murine MCs, CNP activated its specific GC (guanylyl cyclase)-B receptor and cyclic GMP signaling. This enhanced cyclic GMP–dependent phosphorylation of the cytoskeleton-associated VASP (vasodilator-stimulated phosphoprotein) and…

medicine.medical_specialtyMice 129 StrainMedizinMyocardial Reperfusion InjuryCell DegranulationCell LineMicrocirculationCapillary PermeabilityCyclic gmpAdenosine TriphosphateInternal medicineParacrine CommunicationmedicineAnimalsMast CellsPhosphorylationCyclic GMPMice KnockoutChemistryMicrofilament ProteinsDegranulationEndothelial CellsNatriuretic Peptide C-TypeThrombosisPhosphoproteinsMice Inbred C57BLDisease Models AnimalEndocrinologyNeutrophil InfiltrationC-type natriuretic peptideCardiology and Cardiovascular MedicineCell Adhesion MoleculesReceptors Atrial Natriuretic FactorSignal TransductionGuanylate cyclase
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Mutational analysis of E-cadherin, β-catenin and APC genes in synovial sarcomas

2010

medicine.medical_specialtyMutationPathologyHistologyCadherinCell adhesion moleculeCancerAnatomical pathologyGeneral MedicineBiologymedicine.disease_causemedicine.diseaseSynovial sarcomaPathology and Forensic MedicineCateninmedicineCancer researchGeneHistopathology
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