Search results for "Cell communication"

showing 10 items of 153 documents

From non-excitable single-cell to multicellular bioelectrical states supported by ion channels and gap junction proteins: Electrical potentials as di…

2019

Endogenous bioelectric patterns within tissues are an important driver of morphogenesis and a tractable component of a number of disease states. Developing system-level understanding of the dynamics by which non-neural bioelectric circuits regulate complex downstream cascades is a key step towards both, an evolutionary understanding of ion channel genes, and novel strategies in regenerative medicine. An important capability gap is deriving rational modulation strategies targeting individual cells' bioelectric states to achieve global (tissue- or organ-level) outcomes. Here, we develop an ion channel-based model that describes multicellular states on the basis of spatio-temporal patterns of …

Gap Junction Proteins030303 biophysicsCellBiophysicsCell CommunicationRegenerative medicineModels BiologicalConnexinsIon ChannelsCell membrane03 medical and health sciencesmedicineMorphogenesisAnimalsHumansMolecular BiologyIon channelPhysics0303 health sciencesCell potentialElectrical potentialsGap JunctionsElectrophysiological PhenomenaMulticellular organismmedicine.anatomical_structureSingle-Cell AnalysisNeuroscienceSignal TransductionProgress in biophysics and molecular biology
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Simultaneous Aurora-A/STK15 overexpression and centrosome amplification induce chromosomal instability in tumour cells with a MIN phenotype

2007

Abstract Background Genetic instability is a hallmark of tumours and preneoplastic lesions. The predominant form of genome instability in human cancer is chromosome instability (CIN). CIN is characterized by chromosomal aberrations, gains or losses of whole chromosomes (aneuploidy), and it is often associated with centrosome amplification. Centrosomes control cell division by forming a bipolar mitotic spindle and play an essential role in the maintenance of chromosomal stability. However, whether centrosome amplification could directly cause aneuploidy is not fully established. Also, alterations in genes required for mitotic progression could be involved in CIN. A major candidate is represe…

Genome instabilityCancer ResearchCellular differentiationAneuploidyApoptosisCell CommunicationSpindle ApparatusBiologyProtein Serine-Threonine Kinaseslcsh:RC254-282Aurora KinasesChromosome instabilityChromosomal InstabilitymedicineTumor Cells CulturedGeneticsHumansRNA Small InterferingMitosisIn Situ Hybridization FluorescenceAurora Kinase ACentrosomePloidiesReverse Transcriptase Polymerase Chain ReactionAurora-A centrosomes amplification aneuploidyCell Differentiationlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseAneuploidyCell biologySpindle apparatusUp-RegulationSettore BIO/18 - GeneticaCell Transformation NeoplasticPhenotypeMicroscopy FluorescenceOncologyCentrosomeColonic NeoplasmsEctopic expressionMicrosatellite InstabilityResearch ArticleBMC Cancer
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New Vocabulary for Bacterial Communication

2019

Abstract Quorum sensing (QS) is widely accepted as a procedure that bacteria use to converse. However, prevailing thinking places acyl homoserine lactones (AHLs) at the forefront of this communication pathway in Gram‐negative bacteria. With the advent of high‐throughput genomics and the subsequent influx of bacterial genomes, bioinformatics analysis has determined that the genes encoding AHL biosynthesis, originally discovered to be indispensable for QS (LuxI‐like proteins and homologues), are often absent in QS‐capable bacteria. Instead, the sensing protein (LuxR‐like proteins) is present with an apparent inability to produce any outgoing AHL signal. Recently, several signals for these Lux…

GenomicsCell CommunicationBacterial genome sizeComputational biologyAcyl-Butyrolactones010402 general chemistry01 natural sciencesBiochemistryDNA sequencing570 Life sciencesGram-Negative Bacteriabacterial communicationMolecular BiologyGeneAcyl-Homoserine Lactonesgene sequencingbiology010405 organic chemistryOrganic Chemistryquorum sensingfood and beveragesMinireviewsbiochemical phenomena metabolism and nutritionbiology.organism_classification0104 chemical sciencesQuorum sensingQuorum Quenchingquorum quenchingMolecular MedicineMinireviewbiosynthesisBacteria570 BiowissenschaftenChemBioChem
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Isolation and characterization of a 60-70-kD plasma membrane glycoprotein involved in the contact-dependent inhibition of growth

1990

Previous studies have shown that plasma membrane compounds are involved in the contact-dependent inhibition of growth of human diploid fibroblasts. The purification of the active plasma membrane glycoprotein is described in this report. The glycoprotein has an apparent molecular mass of 60-70 kD and, due to differential sialylation, isoelectric points between pH 5.5. and 6.2. Treatment with sialidase yielded one spot in two-dimensional gel electrophoresis with an isoelectric point of 6.3. After removal of the N-glycosidically linked oligosaccharide chains, the apparent molecular mass is reduced by approximately 22 kD. Treatment was diluted NaOH, which removes the O-glycosidically linked por…

GlycanCell CommunicationCell LineAnimalsHumansPolyacrylamide gel electrophoresisCells CulturedCytoskeletonGel electrophoresischemistry.chemical_classificationMembrane GlycoproteinsbiologyMolecular massContact InhibitionCell MembraneContact inhibitionCell BiologyArticlesFibroblastsMolecular biologyMolecular WeightMicroscopy ElectronIsoelectric pointchemistryBiochemistryCell culturebiology.proteinChromatography GelElectrophoresis Polyacrylamide GelGlycoproteinCell DivisionThe Journal of Cell Biology
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Contact-dependent inhibition of growth of normal diploid human fibroblasts by plasma membrane glycoproteins.

1988

Homeostasis in vivo is maintained by a highly complex network of positive and negative signals. At the cellular level, this regulatory microenvironment can be divided, in a simplified fashion, into two major compartments: the humoral compartment, including compounds such as hormones, growth factors and nutrients, and the contact-environment compartment, including cell-cell and cell-matrix interactions. At least in cultures of diploid, non-transformed cells, cell-cell and cell-matrix interactions have been shown to be of major importance for the regulation of growth as well as of differentiation. Although until now the glycoprotein involved in the contact-dependent inhibition of growth has n…

GlycanCell CommunicationPlatelet Membrane GlycoproteinsBiochemistrychemistry.chemical_compoundmedicineCompartment (development)AnimalsHumansReceptors ImmunologicFibroblastReceptorCells Culturedchemistry.chemical_classificationMembrane GlycoproteinsbiologyContact InhibitionCell MembraneAntibodies MonoclonalBiological activityGeneral MedicineFibroblastsMembrane glycoproteinsmedicine.anatomical_structureCell Transformation NeoplasticchemistryBiochemistryPlatelet Glycoprotein GPIb-IX Complexbiology.proteinGrowth inhibitionGlycolipidsGlycoproteinCell DivisionBiochimie
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Identification and Functional Characterization of Human Cd4+Cd25+ T Cells with Regulatory Properties Isolated from Peripheral Blood

2001

A subpopulation of peripheral human CD4(+)CD25(+) T cells that expresses CD45RO, histocompatibility leukocyte antigen DR, and intracellular cytotoxic T lymphocyte-associated antigen (CTLA) 4 does not expand after stimulation and markedly suppresses the expansion of conventional T cells in a contact-dependent manner. After activation, CD4(+)CD25(+) T cells express CTLA-4 on the surface detectable for several weeks. These cells show a G1/G0 cell cycle arrest and no production of interleukin (IL)-2, IL-4, or interferon (IFN)-gamma on either protein or mRNA levels. The anergic state of CD4(+)CD25(+) T cells is not reversible by the addition of anti-CD28, anti-CTLA-4, anti-transforming growth fa…

Immunoconjugateshuman regulatory T cellsT cellCTLA-4 expressionImmunologychemical and pharmacologic phenomenaCell CommunicationBiologyLymphocyte ActivationAbataceptMiceInterleukin 21Antigens CDT-Lymphocyte SubsetsCD4+CD25+ T cellsImmune TolerancemedicineAnimalsHumansImmunology and AllergyCytotoxic T cellCTLA-4 AntigenIL-2 receptorAntigen-presenting cellInterleukin 3toleranceCD28Receptors Interleukin-2hemic and immune systemsNatural killer T cellAntigens DifferentiationMolecular biologymedicine.anatomical_structureT cell inhibitionCD4 AntigensCytokinesLeukocyte Common AntigensOriginal ArticleJournal of Experimental Medicine
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Mast cells enhance proliferation of B lymphocytes and drive their differentiation toward IgA-secreting plasma cells.

2010

AbstractThe evidence of a tight spatial interaction between mast cells (MCs) and B lymphocytes in secondary lymphoid organs, along with the data regarding the abundance of MCs in several B-cell lymphoproliferative disorders prompted us to investigate whether MCs could affect the proliferation and differentiation of B cells. To this aim, we performed coculture assays using mouse splenic B cells and bone marrow–derived MCs. Both nonsensitized and activated MCs proved able to induce a significant inhibition of cell death and an increase in proliferation of naive B cells. Such proliferation was further enhanced in activated B cells. This effect relied on cell-cell contact and MC-derived interle…

Immunoglobulin AMAST CELL B LYMPHOCITESCellular differentiationImmunologyNaive B cellCD40 LigandPlasma CellsCell CommunicationImmunoglobulin ELymphocyte ActivationBiochemistryMast cellMiceImmune systemIg isotype switchmedicineAnimalsHumansMast CellsCD40 AntigensCell ProliferationIG-A.B cellB cellsMast cell; B cells; Differentiation; Ig isotype switchCD40biologyCell DeathInterleukin-6Cell DifferentiationCell BiologyHematologyMast cellhumanitiesCell biologyImmunity HumoralImmunoglobulin Amedicine.anatomical_structureGene Expression RegulationDifferentiationImmunologybiology.proteinMAST CELL B LYMPHOCITES; IG-A.Syndecan-1AntibodyBlood
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Cytoskeletal stabilization of inhibitory interactions in immunologic synapses of mature human dendritic cells with natural killer cells

2011

Abstract Human mature dendritic cells (DCs) can efficiently stimulate natural killer (NK)–cell responses without being targeted by their cytotoxicity. To understand this important regulatory crosstalk, we characterized the development of the immunologic synapse between mature DCs and resting NK cells. Conjugates between these 2 innate leukocyte populations formed rapidly, persisted for prolonged time periods and matured with DC-derived f-actin polymerization at the synapse. Polarization of IL-12 and IL-12R to the synapse coincided with f-actin polymerization, while other activating and inhibitory molecules were enriched at the interface between DCs and NK cells earlier. Functional assays re…

Immunological SynapsesImmunologyCell Communicationmacromolecular substancesBiochemistryImmunological synapseNatural killer cell03 medical and health sciences0302 clinical medicineInterleukin-15 Receptor alpha SubunitMicroscopy Electron TransmissionReceptors KIRMHC class ImedicineHumansAntigen-presenting cellCells CulturedCytoskeleton030304 developmental biology0303 health sciencesMicroscopy ConfocalbiologyReceptors Interleukin-12Dendritic CellsCell BiologyHematologyDendritic cellFlow CytometryInterleukin-12Immunological SynapsesActinsCoculture Techniques3. Good healthCell biologyKiller Cells Naturalmedicine.anatomical_structureMicroscopy Fluorescencebiology.proteinInterleukin 12RNA InterferenceK562 CellsMicrotubule-Organizing CenterWiskott-Aldrich Syndrome Protein030215 immunologyK562 cellsBlood
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Cyclic adenosine monophosphate and IL-10 coordinately contribute to nTreg cell-mediated suppression of dendritic cell activation

2010

In humans and mice naturally occurring regulatory T cells (nTregs) are crucial for the maintenance of peripheral tolerance by controlling not only potentially autoreactive T cells but virtually all cells of the adaptive and innate immune system. Here we show that co-culture of murine dendritic cells (DC) and nTregs results in an immediate increase of cAMP in DC, responsible for a rapid down-regulation of co-stimulatory molecules (CD80, CD86). In addition, the inhibitory surface molecule B7-H3 on DC is up-regulated. Subsequently, nTreg-derived IL-10 inhibits the cytokine production (IL-6, IL-12) of suppressed DC therewith preserving their silent phenotype. Hence, our data indicate that nTreg…

ImmunologyDown-RegulationCell CommunicationBiologyT-Lymphocytes RegulatoryImmune toleranceMiceImmune systemCyclic AMPImmune ToleranceAnimalsCD86Innate immune systemInterleukin-6Peripheral toleranceDendritic CellsDendritic cellInterleukin-12Coculture TechniquesInterleukin-10Cell biologyInterleukin 10B7-1 AntigenB7-2 AntigenCD80Signal TransductionCellular Immunology
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Inhibition of human allergic T-helper type 2 immune responses by induced regulatory T cells requires the combination of interleukin-10-treated dendri…

2006

Summary Background In grass pollen-allergic individuals, T cell anergy can be induced by IL-10-treated dendritic cells (IL-10-DC) resulting in the suppression of T helper type 1 (Th1) as well as Th2 cells. This study was performed to analyse whether such IL-10-DC-treated T cells are able to act as regulatory T cells (Treg) suppressing the function of other T cells in the periphery. As transforming growth factor (TGF)-β is also a potential inducer of Treg, we additionally analysed the inhibitory capacity of TGF-β-treated T cells in this system. Materials and Methods Freshly isolated CD4+ or CD4+CD25− T cells from grass pollen-allergic donors were stimulated with autologous mature monocyte-de…

ImmunologyEnzyme-Linked Immunosorbent AssayCell CommunicationBiologyPoaceaeT-Lymphocytes RegulatoryInterleukin 21Interferon-gammaTh2 CellsAntigens CDTransforming Growth Factor betaHypersensitivityImmunology and AllergyCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorAntigen-presenting cellCells CulturedInterleukin 3Cell ProliferationDendritic cellDendritic CellsAllergensNatural killer T cellFlow CytometryAntigens DifferentiationCell biologyInterleukin-10ImmunologyInterleukin 12PollenImmunizationInterleukin-4Interleukin-5Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
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