Search results for "Cell line"

showing 10 items of 2924 documents

Role of the cyclic AMP-dependent pathway in free radical-induced cholesterol accumulation in vascular smooth muscle cells.

2000

We have previously reported that free radical-treated vascular smooth muscle cells (SMC) lead to cholesterol accumulation in vitro. In the current study, we investigated the effects of oxidative stress on cyclic AMP concentration and cAMP-dependent enzymes involved in cholesterol homeostasis in A7r5 cells. Under our conditions of a mild oxidative stress, namely with no change in cell viability, we found that free radicals, initiated using azobis-amidinopropane dihydrochloride (AAPH), resulted in a dose-dependent decrease in cellular cAMP which was opposed by vitamin E preincubation. Although the addition of adenylate cyclase activators (carbacyclin and forskolin) increased cAMP levels it di…

medicine.medical_specialtyVascular smooth muscleFree RadicalsSterol O-acyltransferaseAmidinesAdenylate kinaseOxidative phosphorylationmedicine.disease_causeBiochemistryMuscle Smooth VascularCell Linechemistry.chemical_compoundPhysiology (medical)Internal medicineProstaglandins SyntheticmedicineCyclic AMPAnimalsAortaForskolinbiologyCholesterolCell MembraneFatty AcidsOxidantsEpoprostenolCell biologyRatsOxidative StressEndocrinologyCholesterolchemistryBucladesineHMG-CoA reductasebiology.proteinHydroxymethylglutaryl CoA ReductasesCardiology and Cardiovascular MedicineCyclase activityOxidative stressAdenylyl CyclasesSterol O-AcyltransferaseFree radical biologymedicine
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Oxidative stress leads to cholesterol accumulation in vascular smooth muscle cells.

1999

The transformation of macrophages and smooth muscle cells into foam cells by modified low-density lipoproteins (LDL) is one of the key events of atherogenesis. Effects of free radicals have mainly been studied in LDL, and other than toxicity, data dealing with direct action of free radicals on cells are scarce. This study focused on the direct effects of free radicals on cholesterol metabolism of smooth muscle cells. A free radical generator, azobis-amidinopropane dihydrochloride, was used, and conditions for a standardized oxidative stress were set up in vascular smooth muscle cells. After free radical action, the cells presented an accumulation of cholesterol that appeared to be the resul…

medicine.medical_specialtyVascular smooth muscleFree RadicalsSterol O-acyltransferaseAmidinesmedicine.disease_causeBiochemistryMuscle Smooth VascularCell Linechemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineAnimalsHumansViability assayCholesterolIn vitroRatsLipoproteins LDLOxidative StressEndocrinologyCholesterolchemistryCell cultureCholesteryl esterlipids (amino acids peptides and proteins)Cholesterol EstersOxidative stressSterol O-AcyltransferaseFree radical biologymedicine
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Potential role of the neuropeptide CGRP in the induction of differentiation of rat hepatic portal vein wall.

2005

The media of the rat hepatic portal vein is composed of an internal circular muscular layer (CL) and an external longitudinal muscular layer (LL). These two perpendicular layers differentiate progressively from mesenchymal cells within the first month after birth. In this paper, we studied the development of calcitonin gene-related peptide (CGRP) innervation during post-natal differentiation of the vessel. We show that CGRP innervation is already present around the vessel at birth in the future adventitia but far from the lumen of the vessel. Progressively, CGRP immunoreactive fibers reached first LL then CL. CL by itself become only innervated at day 14 after birth. This corresponds to the…

medicine.medical_specialtyVascular smooth musclePhysiologyCalcitonin Gene-Related PeptideRecombinant Fusion ProteinsImmunocytochemistryMyocytes Smooth MuscleGene ExpressionCalcitonin gene-related peptideBiologyTransfectionBiochemistryMuscle Smooth VascularCell LineMuscular layerCellular and Molecular NeuroscienceMiceEndocrinologyInternal medicineAdventitiaMyosinmedicineAnimalsHumansRats WistarLuciferasesPromoter Regions GeneticBinding SitesMyosin Heavy ChainsPortal VeinNeuropeptidesAge FactorsCell DifferentiationImmunohistochemistryRatsEndocrinologymedicine.anatomical_structureLiverConnective TissueDesminHepatic portal veinRabbitsPeptides
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Isolation and confirmation of viral nervous necrosis (VNN) disease in golden grey mullet (Liza aurata) and leaping mullet (Liza saliens) in the Irani…

2016

The present study was conducted on 428 moribund mullet fish samples to isolate and identify the causative agent of a mysterious acute mortality which recently occurred in wild mullets in Iranian waters of Caspian Sea, suspected to be due to viral nervous necrosis (VNN) disease. Disease investigation was carried out employing various diagnostic procedures such as virology, bacteriology, parasitology, haematology, histopathology, IFAT, IHC and nested RT-PCR. Brain and eye samples of affected fishes were collected in sterile conditions and then kept at -80 °C for cell culture isolation and nested RT-PCR detection of the causative agent. Other tissue samples were also collected and fixed for hi…

medicine.medical_specialtyVeterinary medicinePathology040301 veterinary sciencesCaspian sea; Golden grey mullet (Liza aurata); Iran; Leaping mullet (Liza saliens); Viral nervous necrosisOceans and SeasViral nervous necrosisBetanodavirusSettore BIO/05 - ZoologiaBiologyIranMicrobiologyPolymerase Chain ReactionMulletCell Line0403 veterinary scienceTissue cultureFish DiseasesRNA Virus InfectionsMicroscopy Electron TransmissionmedicineCaspian seaAnimalsNodaviridaeFluorescent Antibody Technique IndirectGeneral VeterinaryOutbreak04 agricultural and veterinary sciencesGeneral Medicinebiology.organism_classificationImmunohistochemistrySurvival AnalysisSmegmamorphaParasitology040102 fisheries0401 agriculture forestry and fisheriesHistopathologyGolden grey mullet (Liza aurata)Leaping mullet (Liza saliens)Golden grey mulletLeaping mulletVeterinary microbiology
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Inhibition of growth of measles virus by mycoplasma in cell-cultures and the restoring effect of arginine

1970

Presence of mycoplasma in MK cell cultures caused an inhibition of measles virus, as revealed by a lower plating efficiency. When arginine is supplemented to the cell-system, mycoplasma contaminated cells yield plaque titers as high as those of uncontaminated cells. The essential role of arginine for plaque formation of measles virus is suggested also by experiments in which arginine added to the agar overlay restored the plating efficiency of measles virus in cultures maintained in a minimal nutrient medium.

medicine.medical_specialtyVirus CultivationPlating efficiencyArginineBiologyArginineKidneymedicine.disease_causeMeaslesCell LineMicrobiologyMeasles virusMycoplasmaMedical microbiologyCulture TechniquesVirologymedicineAnimalsHaplorhiniGeneral MedicineMycoplasmabiology.organism_classificationmedicine.diseaseVirologyCulture MediaTiterMeasles virusCell cultureArchiv f�r die gesamte Virusforschung
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Chemosensory signalling pathways involved in sensing of amino acids by the ghrelin cell

2015

AbstractTaste receptors on enteroendocrine cells sense nutrients and transmit signals that control gut hormone release. This study aimed to investigate the amino acid (AA) sensing mechanisms of the ghrelin cell in a gastric ghrelinoma cell line, tissue segments and mice. Peptone and specific classes of amino acids stimulate ghrelin secretion in the ghrelinoma cell line. Sensing of L-Phe occurs via the CaSR, monosodium glutamate via the TAS1R1-TAS1R3 while L-Ala and peptone act via 2 different amino acid taste receptors: CaSR & TAS1R1-TAS1R3 and CaSR & GPRC6A, respectively. The stimulatory effect of peptone on ghrelin release was mimicked ex vivo in gastric but not in jejunal tissue …

medicine.medical_specialty[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEnteroendocrine cellGPRC6ANutrient sensingBiologyArticleReceptors G-Protein-CoupledMice03 medical and health sciences0302 clinical medicineGlucagon-Like Peptide 1Receptor-Interacting Protein Serine-Threonine Kinase 2Taste receptorCell Line TumorInternal medicinemedicineFood and NutritionAnimalsAmino AcidsReceptor030304 developmental biology0303 health sciencesMultidisciplinarydigestive oral and skin physiologyGhrelinEndocrinologySomatostatinReceptor-Interacting Protein Serine-Threonine KinasesAlimentation et NutritionGhrelin[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryGhrelin secretionhormones hormone substitutes and hormone antagonistsSignal TransductionScientific Reports
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Signaling through BMPR-IA regulates quiescence and long-term activity of neural stem cells in the adult hippocampus.

2010

SummaryNeural stem cells (NSCs) in the adult hippocampus divide infrequently, and the molecules that modulate their quiescence are largely unknown. Here, we show that bone morphogenetic protein (BMP) signaling is active in hippocampal NSCs, downstream of BMPR-IA. BMPs reversibly diminish proliferation of cultured NSCs while maintaining their undifferentiated state. In vivo, acute blockade of BMP signaling in the hippocampus by intracerebral infusion of Noggin first recruits quiescent NSCs into the cycle and increases neurogenesis; subsequently, it leads to decreased stem cell division and depletion of precursors and newborn neurons. Consistently, selective ablation of Bmpr1a in hippocampal …

medicine.medical_specialtyanimal structuresGenetic VectorsHippocampal formationBiologyBone morphogenetic proteinHippocampusModels BiologicalMOLNEUROCell LineMiceNeural Stem CellsInternal medicineGeneticsmedicineAnimalsHumansNogginBone Morphogenetic Protein Receptors Type ICells Culturedreproductive and urinary physiologySmad4 ProteinNeuronsReverse Transcriptase Polymerase Chain ReactionStem CellsCell CycleLentivirusNeurogenesisCentral-nervous-system; Bone morphogenetic protein; Dentate gyrus; Progenitor cells; Neurogenesis; Expression; Receptor; Noggin; Brain; DifferentiationCell BiologyFlow CytometrySTEMCELLRats Inbred F344BMPR1ANeural stem cellRatsCell biologyEndocrinologyStem cell divisionnervous systemembryonic structuresMolecular MedicineStem cellbiological phenomena cell phenomena and immunityCarrier ProteinsSignal Transduction
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OP0309 Intestinal sclerostin/serotonin axis is modulated by dysbiosis and regulates ilc3 expansion in as patients

2017

Background Sclerostin is an osteocyte-specific factor that binds to low-density lipoprotein receptor-related protein 5 (LRP5) inhibiting the Wnt signaling pathway and possibly contributing to the pathogenesis of Ankylosing spondylitis (AS). Subclinical gut inflammation observed in AS patients is characterized by the presence of dysbiosis and innate immune alterations. In the gut, LRP5 activation by unknown ligands inhibits serotonin production. Serotonin, by inducing glial derived neurotrophic factor (GDNF), controls ILC3 expansion, in the context of glial–ILC3–epithelial cell unit (GIECU). Sclerostin/serotonin axis has been never studied in AS. Objectives Aim of this study was to evaluate …

medicine.medical_specialtybiologybusiness.industryWnt signaling pathwayLRP5Context (language use)chemistry.chemical_compoundEndocrinologychemistryInternal medicinemedicineGlial cell line-derived neurotrophic factorbiology.proteinEnterochromaffin cellSclerostinSerotonin ProductionSerotoninbusinessOral Presentations
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Statins stimulate the production of a soluble form of the receptor for advanced glycation end products

2013

The beneficial effects of statin therapy in the reduction of cardiovascular pathogenesis, atherosclerosis, and diabetic complications are well known. The receptor for advanced glycation end products (RAGE) plays an important role in the progression of these diseases. In contrast, soluble forms of RAGE act as decoys for RAGE ligands and may prevent the development of RAGE-mediated disorders. Soluble forms of RAGE are either produced by alternative splicing [endogenous secretory RAGE (esRAGE)] or by proteolytic shedding mediated by metalloproteinases [shed RAGE (sRAGE)]. Therefore we analyzed whether statins influence the production of soluble RAGE. Lovastatin treatment of either mouse alveol…

medicine.medical_specialtyendocrine system diseasesADAM10Receptor for Advanced Glycation End ProductsBeta-CyclodextrinsQD415-436PharmacologyBiochemistryCell LineRAGE (receptor)MiceEndocrinologyGlycationInternal medicinediabetic complicationsmedicineAnimalsHumansSecretionLovastatincardiovascular diseasesReceptors ImmunologicReceptorResearch ArticlesDose-Response Relationship DrughypercholesterolemiaChemistrybeta-CyclodextrinsHEK 293 cellsTricarboxylic Acidsnutritional and metabolic diseasesCell BiologyBridged Bicyclo Compounds HeterocyclicADAM 10CholesterolFarnesyl-Diphosphate FarnesyltransferaseEndocrinologySolubilitycardiovascular systemLovastatinHydroxymethylglutaryl-CoA Reductase Inhibitorsatherosclerosishuman activitiesmedicine.drugJournal of Lipid Research
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Increased Connexin 43 Expression as a Potential Mediator of the Neuroprotective Activity of the Corticotropin-Releasing Hormone

2009

CRH is a major central stress mediator, but also a potent neuroprotective effector. The mechanisms by which CRH mediates its neuroprotective actions are largely unknown. Here, we describe that the gap junction molecule connexin43 (Cx43) mediates neuroprotective effects of CRH toward experimentally induced oxidative stress. An enhanced gap junction communication has been reported to contribute to neuroprotection after neurotoxic insults. We show that CRH treatment up-regulates Cx43 expression and gap junctional communication in a CRH receptor-dependent manner in IMR32 neuroblastoma cells, primary astrocytes, and organotypic hippocampal slice cultures. MAPKs and protein kinase A-cAMP response…

medicine.medical_specialtyendocrine systemCorticotropin-Releasing HormoneMAP Kinase Signaling SystemCarbenoxoloneConnexinBiologyNeuroprotectionModels BiologicalArticleRats Sprague-DawleyCorticotropin-releasing hormoneMiceEndocrinologyMediatorInternal medicineCell Line Tumormedicinepolycyclic compoundsAnimalsHumansProtein kinase AMolecular BiologyGap junctionBrainGap JunctionsGeneral MedicineCell biologyRatsEndocrinologyNeuroprotective Agentsnervous systemGene Expression RegulationConnexin 43cardiovascular systemSignal transductionhormones hormone substitutes and hormone antagonistsmedicine.drugSignal Transduction
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