Search results for "Cellular Reprogramming"

showing 4 items of 34 documents

Transcriptional Mechanisms of Proneural Factors and REST in Regulating Neuronal Reprogramming of Astrocytes

2015

Summary Direct lineage reprogramming induces dramatic shifts in cellular identity, employing poorly understood mechanisms. Recently, we demonstrated that expression of Neurog2 or Ascl1 in postnatal mouse astrocytes generates glutamatergic or GABAergic neurons. Here, we take advantage of this model to study dynamics of neuronal cell fate acquisition at the transcriptional level. We found that Neurog2 and Ascl1 rapidly elicited distinct neurogenic programs with only a small subset of shared target genes. Within this subset, only NeuroD4 could by itself induce neuronal reprogramming in both mouse and human astrocytes, while co-expression with Insm1 was required for glutamatergic maturation. Cu…

Transcription GeneticRepressorNerve Tissue ProteinsCell fate determinationBiologyDNA-binding proteinArticleMiceGlutamatergicBasic Helix-Loop-Helix Transcription FactorsGeneticsAnimalsHumansPromoter Regions GeneticTranscription factorCells CulturedNeuronsCell BiologyCellular ReprogrammingMolecular biologyCell biologyDNA-Binding ProteinsRepressor ProteinsASCL1Astrocytesembryonic structuresMolecular MedicineGABAergicReprogrammingTranscription FactorsCell Stem Cell
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Improving mRNA-Based Therapeutic Gene Delivery by Expression-Augmenting 3' UTRs Identified by Cellular Library Screening.

2019

Synthetic mRNA has emerged as a powerful tool for the transfer of genetic information, and it is being explored for a variety of therapeutic applications. Many of these applications require prolonged intracellular persistence of mRNA to improve bioavailability of the encoded protein. mRNA molecules are intrinsically unstable and their intracellular kinetics depend on the UTRs embracing the coding sequence, in particular the 3′ UTR elements. We describe here a novel and generally applicable cell-based selection process for the identification of 3′ UTRs that augment the expression of proteins encoded by synthetic mRNA. Moreover, we show, for two applications of mRNA therapeutics, namely, (1) …

Untranslated regionCellular differentiationRNA StabilityInduced Pluripotent Stem CellsBlood DonorsComputational biologyGene deliveryBiologyCancer Vaccines03 medical and health sciencesMice0302 clinical medicineDrug DiscoveryGeneticsCoding regionAnimalsHumansRNA MessengerInduced pluripotent stem cellMolecular BiologyGene3' Untranslated RegionsCells Cultured030304 developmental biologyGene LibraryPharmacology0303 health sciencesMessenger RNAMice Inbred BALB CVaccinationGene Transfer TechniquesGenetic TherapyFibroblastsCellular Reprogramming030220 oncology & carcinogenesisMolecular MedicineFemaleOriginal ArticleReprogrammingHalf-LifeMolecular therapy : the journal of the American Society of Gene Therapy
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Regulation of T-Cell Immune Responses by Pro-Resolving Lipid Mediators

2021

Both the initiation and the resolution of inflammatory responses are governed by the sequential activation, migration, and control/suppression of immune cells at the site of injury. Bioactive lipids play a major role in the fine-tuning of this dynamic process in a timely manner. During inflammation and its resolution, polymorphonuclear cells (PMNs) and macrophages switch from producing pro-inflammatory prostaglandins and leukotrienes to specialized pro-resolving lipid mediators (SPMs), namely, lipoxins, resolvins, protectins, and maresins, which are operative at the local level to limit further inflammation and tissue injury and restore homeostasis. Accumulating evidences expand now the rol…

chronic inflammationT cellT-LymphocytesImmunologyInflammationReviewBiologymedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunity03 medical and health sciencesImmunomodulating Agents0302 clinical medicineImmune systemmedicineHumansImmunology and Allergy030304 developmental biology0303 health sciencestherapyEffectorautoimmunityresolutionT cellLipid signalingadaptive immunityRC581-607Acquired immune systemCellular Reprogramming3. Good healthLipoxinsmedicine.anatomical_structureEicosapentaenoic AcidImmunologyspecialized pro-resolving lipid mediators (SPMs)Eicosanoidsmedicine.symptomImmunologic diseases. AllergyInflammation MediatorsHomeostasis030215 immunologyFrontiers in Immunology
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In vitro reprogramming of pancreatic alpha cells towards a beta cell phenotype following ectopic HNF4α expression

2015

There is currently a shortage of organ donors available for pancreatic beta cell transplantation into diabetic patients. An alternative source of beta cells is pre-existing pancreatic cells. While we know that beta cells can arise directly from alpha cells during pancreatic regeneration we do not understand the molecular basis for the switch in phenotype. The aim of the present study was to investigate if hepatocyte nuclear factor 4 alpha (HNF4α), a transcription factor essential for a normal beta cell phenotype, could induce the reprogramming of alpha cells towards potential beta cells. We utilised an in vitro model of pancreatic alpha cells, the murine αTC1-9 cell line. We initially chara…

medicine.medical_specialtyBiologyBiochemistryAlpha cellCell LineMiceEndocrinologyInsulin-Secreting CellsInternal medicinemedicineAnimalspancreatic alpha cellsMolecular Biologyreprogramming3T3-L1Cellular ReprogrammingAntigens Differentiationbeta cellCell biologyEndocrinologyHepatocyte Nuclear Factor 4Glucagon-Secreting CellsCell cultureHepatocyte nuclear factor 4 alphaPAX4Ectopic expressionBeta cellReprogrammingMolecular and Cellular Endocrinology
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