Search results for "Cellular differentiation"

showing 10 items of 482 documents

Evaluation of the stromal vascular fraction of adipose tissue as the basis for a stem cell-based tissue-engineered vascular graft

2017

Abstract Objective One of the rate-limiting barriers within the field of vascular tissue engineering is the lengthy fabrication time associated with expanding appropriate cell types in culture. One particularly attractive cell type for this purpose is the adipose-derived mesenchymal stem cell (AD-MSC), which is abundant and easily harvested from liposuction procedures. Even this cell type has its drawbacks, however, including the required culture period for expansion, which could pose risks of cellular transformation or contamination. Eliminating culture entirely would be ideal to avoid these concerns. In this study, we used the raw population of cells obtained after digestion of human lipo…

Adult0301 basic medicinePathologymedicine.medical_specialtyTime FactorsCellular differentiationMyocytes Smooth MusclePopulationAdipose tissueCell Separation030204 cardiovascular system & hematologyMesenchymal Stem Cell TransplantationMuscle Smooth VascularArticleBlood Vessel Prosthesis Implantation03 medical and health sciences0302 clinical medicineLipectomyCell MovementBlood vessel prosthesisAnimalsHumansMedicineAorta AbdominaleducationCells CulturedBioprosthesiseducation.field_of_studyTissue EngineeringTissue Scaffoldsbusiness.industryAngiotensin IIMesenchymal stem cellCell DifferentiationMesenchymal Stem CellsAnatomyStromal vascular fractionAngiotensin IIBlood Vessel ProsthesisPhenotype030104 developmental biologyAdipose TissueRats Inbred LewFemaleSurgeryStromal CellsStem cellbusinessCardiology and Cardiovascular Medicine
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Mesenchymal stem cells of Systemic Sclerosis patients, derived from different sources, show a profibrotic microRNA profiling

2019

AbstractSystemic Sclerosis (SSc) is a disease with limited therapeutic possibilities. Mesenchymal stem cells (MSCs)-therapy could be a promising therapeutic option, however the ideal MSCs source has not yet been found. To address this problem, we perform comparison between bone marrow (BM)-MSCs and adipose (A)-MSCs, by the miRs expression profile, to identify the gene modulation in these two MSCs source. MicroRNAs (miRs) are RNAs sequences, regulating gene expression and MSCs, derived from different tissues, may differently respond to the SSc microenvironment. The miRs array was used for the miRs profiling and by DIANA-mirPath tool we identified the biological functions of the dysregulated …

Adult0301 basic medicineTherapeutic gene modulationAutoimmune diseasesCellular differentiationGene regulatory networklcsh:MedicineBone Marrow CellsBiologyRegenerative medicineArticle03 medical and health sciences0302 clinical medicinemicroRNAmedicineHumansGene Regulatory Networkslcsh:ScienceCells CulturedSystemic SclerosiCell ProliferationRegulation of gene expressionScleroderma SystemicMultidisciplinarySequence Analysis RNAGene Expression ProfilingMesenchymal stem celllcsh:RCell DifferentiationMesenchymal Stem CellsSettore MED/16 - ReumatologiaMicroRNAs030104 developmental biologymedicine.anatomical_structureAdipose TissueGene Expression RegulationCancer researchSystemic sclerosisFemalelcsh:QBone marrow030217 neurology & neurosurgeryScientific Reports
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The transcription factor ZEB1 (deltaEF1) promotes tumour cell dedifferentiation by repressing master regulators of epithelial polarity.

2007

Epithelial to mesenchymal transition (EMT) is implicated in the progression of primary tumours towards metastasis and is likely caused by a pathological activation of transcription factors regulating EMT in embryonic development. To analyse EMT-causing pathways in tumouri-genesis, we identified transcriptional targets of the E-cadherin repressor ZEB1 in invasive human cancer cells. We show that ZEB1 repressed multiple key determinants of epithelial differentiation and cell–cell adhesion, including the cell polarity genes Crumbs3, HUGL2 and Pals1-associated tight junction protein. ZEB1 associated with their endogenous promoters in vivo, and strongly repressed promotor activities in reporter …

AdultCancer ResearchChromatin ImmunoprecipitationCellular differentiationImmunoblottingDown-RegulationBreast NeoplasmsBiologymedicine.disease_causeEpitheliumArticleCell polarityGeneticsmedicineTumor Cells CulturedHumansNeoplasm InvasivenessEpithelial–mesenchymal transitionCell adhesionPromoter Regions GeneticMolecular BiologyTranscription factorEpithelial polarityAgedOligonucleotide Array Sequence AnalysisHomeodomain ProteinsMembrane GlycoproteinsReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingCell PolarityMembrane ProteinsZinc Finger E-box-Binding Homeobox 1Cell DifferentiationMiddle AgedCadherinsCytoskeletal ProteinsMicroscopy FluorescenceCancer cellColonic NeoplasmsCancer researchDisease ProgressionSnail Family Transcription FactorsCarcinogenesisNucleoside-Phosphate KinaseTranscription FactorsOncogene
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Mesenchymal stromal cells and rheumatic diseases: new tools from pathogenesis to regenerative therapies

2015

In recent years, mesenchymal stromal cells (MSCs) have been largely investigated and tested as a new therapeutic tool for several clinical applications, including the treatment of different rheumatic diseases. MSCs are responsible for the normal turnover and maintenance of adult mesenchymal tissues as the result of their multipotent differentiation abilities and their secretion of a variety of cytokines and growth factors. Although initially derived from bone marrow, MSCs are present in many different tissues such as many peri-articular tissues. MSCs may exert immune-modulatory properties, modulating different immune cells in both in vitro and in vivo models, and they are considered immune-…

AdultCancer ResearchpathogenesiCellular differentiationImmunologyCell- and Tissue-Based TherapyBone Marrow CellsMesenchymal Stem Cell TransplantationRegenerative MedicineRegenerative medicineAutoimmune DiseaseAutoimmune DiseasesChondrocytesImmune systemIn vivoBone MarrowRheumatic DiseasesmedicineHumansImmunology and Allergyrheumatic diseaseGenetics (clinical)TransplantationOsteoblastsMesenchymal Stromal Cellbusiness.industryOsteoblastMesenchymal stem cellMesenchymal Stem CellsCell DifferentiationCell BiologyChondrocyteClinical trialmedicine.anatomical_structureregenerative therapyOncologymesenchymal stromal cells; pathogenesis; regenerative therapy; rheumatic disease; Adult; Autoimmune Diseases; Bone Marrow; Bone Marrow Cells; Cell Differentiation; Cell- and Tissue-Based Therapy; Chondrocytes; Humans; Mesenchymal Stem Cell Transplantation; Mesenchymal Stromal Cells; Osteoblasts; Regenerative Medicine; Rheumatic DiseasesImmunologyBone Marrow CellBone marrowStem cellbusinessHuman
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Tetramer visualization of gut-homing gluten-specific T cells in the peripheral blood of celiac disease patients

2007

Tetramers of MHC–peptide complexes are used for detection and characterization of antigen-specific T cell responses, but they require knowledge about both antigenic peptide and the MHC restriction element. The successful application of these reagents in human diseases involving CD4 + T cells is limited. Celiac disease, an intestinal inflammation driven by mucosal CD4 + T cells recognizing wheat gluten peptides in the context of disease-associated HLA-DQ molecules, is an ideal model to test the potential clinical use of these reagents. We investigated whether gluten-specific T cells can be detected in the peripheral blood of celiac disease patients using DQ2 tetramers. Nine DQ2 + patients a…

AdultGlutensT-LymphocytesT cellCellular differentiationBiologyInterferon-gammaHLA-DQ AntigensmedicineHumansInterferon gammaProtein Structure QuaternaryAgedchemistry.chemical_classificationMultidisciplinaryHLA-DQ Antigennutritional and metabolic diseasesCell DifferentiationBreadBiological SciencesMiddle AgedMHC restrictionGlutendigestive system diseasesStainingGastrointestinal TractCeliac DiseasePhenotypemedicine.anatomical_structurechemistryCase-Control StudiesImmunologyLeukocytes MononuclearHoming (hematopoietic)medicine.drugProceedings of the National Academy of Sciences
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Identification and expansion of human osteosarcoma-cancer-stem cells by long-term 3-aminobenzamide treatment

2009

A novel cancer stem-like cell line (3AB-OS), expressing a number of pluripotent stem cell markers, was irreversibly selected from human osteosarcoma MG-63 cells by long-term treatment (100 days) with 3-aminobenzamide (3AB). 3AB-OS cells are a heterogeneous and stable cell population composed by three types of fibroblastoid cells, spindle-shaped, polygonal-shaped, and rounded-shaped. With respect to MG-63 cells, 3AB-OS cells are extremely smaller, possess a much greater capacity to form spheres, a stronger self-renewal ability and much higher levels of cell cycle markers which account for G1-S/G2-M phases progression. Differently from MG-63 cells, 3AB-OS cells can be reseeded unlimitedly wit…

AdultHomeobox protein NANOGAdolescentPhysiologyCellular differentiationClinical BiochemistryApoptosisBiologyStem cell markerYoung Adultcancer stemm cells osteosarcoma PARP inhibitorsCancer stem cellCell Line TumorSettore BIO/10 - BiochimicaHumansRhodamine 123Enzyme InhibitorsProgenitor cellChildInduced pluripotent stem cellCell ShapeCell potencyFluorescent DyesOsteosarcomaCell DifferentiationCell BiologyCalcium Channel BlockersDrug Resistance MultipleGene Expression Regulation NeoplasticVerapamilBenzamidesImmunologyNeoplastic Stem CellsCancer researchATP-Binding Cassette TransportersBenzimidazolesStem cellBiomarkersJournal of Cellular Physiology
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Special Program of Differentiation Expressed in Keratinocytes of Human Haarscheiben: An Analysis of Individual Cytokeratin Polypeptides

1993

Human haarscheiben, epidermal Merkel cell-rich sensory organs of hairy skin, were studied for the expression of various cytokeratin (CK) polypeptides and other epithelial and neuronal differentiation markers by applying immunoperoxidase and immunofluorescence microscopy to frozen sections and by two-dimensional gel electrophoresis. The basal clusters of Merkel cells were specifically detected by antibodies against CK 20. Haarscheiben keratinocytes were unique mainly by the prominent expression of CK 17 in the lower and middle layers. Further differences as compared to keratinocytes of usual epidermis included the enlargement of the basal compartment, characterized by the expression of CK 5 …

AdultKeratinocytesPathologymedicine.medical_specialtyCellular differentiationDermatologyBiologyBiochemistryCytokeratinKeratinmedicineHumansElectrophoresis Gel Two-DimensionalMolecular BiologyAgedSkinAged 80 and overchemistry.chemical_classificationintegumentary systemImmunoperoxidaseEpidermis (botany)Cell DifferentiationCell BiologyMiddle AgedHair follicleImmunohistochemistryMolecular biologymedicine.anatomical_structureEpidermal CellschemistryKeratinsEpidermisPeptidesKeratinocyteMerkel cellHairJournal of Investigative Dermatology
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Role of p16INK4a and BMI-1 in oxidative stress-induced premature senescence in human dental pulp stem cells

2017

Human dental pulp stem cells (hDPSCs) are a source for cell therapy. Before implantation, an in vitro expansion step is necessary, with the inconvenience that hDPSCs undergo senescence following a certain number of passages, loosing their stemness properties. Long-term in vitro culture of hDPSCs at 21% (ambient oxygen tension) compared with 3–6% oxygen tension (physiological oxygen tension) caused an oxidative stress-related premature senescence, as evidenced by increased β-galactosidase activity and increased lysil oxidase expression, which is mediated by p16INK4a pathway. Furthermore, hDPSCs cultured at 21% oxygen tension underwent a downregulation of OCT4, SOX2, KLF4 and c-MYC factors, w…

AdultMale0301 basic medicineSenescenceAginghDPSCs human dental pulp stem cellsMSC mesenchymal stem cellsAdolescentCellular differentiationClinical BiochemistryCell Culture TechniquesOSKM OCT4 SOX2 KLF4 and c-MYCBiologymedicine.disease_causeBiochemistryCell therapyKruppel-Like Factor 4Young Adult03 medical and health sciencesDental pulp stem cellsmedicineHumansOxygen tensionlcsh:QH301-705.5SIPS stress-induced premature senescenceCells CulturedCellular SenescenceCyclin-Dependent Kinase Inhibitor p16Dental PulpMDA malondialdehydePolycomb Repressive Complex 1lcsh:R5-920Stem CellsOrganic ChemistryCell DifferentiationOxygen tensionCell biologyOxygenOxidative Stress030104 developmental biologylcsh:Biology (General)Cell cultureRegenerative medicineImmunologyFemaleStem celllcsh:Medicine (General)Oxidative stressResearch PaperRedox Biology
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Direct pericyte-to-neuron reprogramming via unfolding of a neural stem cell-like program

2018

Ectopic expression of defined transcription factors can force direct cell-fate conversion from one lineage to another in the absence of cell division. Several transcription factor cocktails have enabled successful reprogramming of various somatic cell types into induced neurons (iNs) of distinct neurotransmitter phenotype. However, the nature of the intermediate states that drive the reprogramming trajectory toward distinct iN types is largely unknown. Here we show that successful direct reprogramming of adult human brain pericytes into functional iNs by Ascl1 and Sox2 encompasses transient activation of a neural stem cell-like gene expression program that precedes bifurcation into distinct…

AdultMale0301 basic medicineSomatic cellCellular differentiationBasic Helix-Loop-Helix Transcription FactorSOXB1 Transcription FactorBiologyArticleYoung Adult03 medical and health sciences0302 clinical medicineNeural Stem CellsSOX2Basic Helix-Loop-Helix Transcription FactorsHumansCell LineageNeural Stem CellAgedPericyteNeuronsSOXB1 Transcription FactorsGeneral NeuroscienceCell DifferentiationMiddle AgedNeuronCellular ReprogrammingNeural stem cellASCL1030104 developmental biologyGene Expression RegulationFemaleEctopic expressionPericytesNeural developmentReprogrammingNeuroscience030217 neurology & neurosurgeryHuman
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Age-Related Lipid Metabolic Signature in HumanLMNA-Lipodystrophic Stem Cell-Derived Adipocytes

2015

Lamin A (LMNA)-linked lipodystrophies belong to a group of clinical disorders characterized by a redistribution of adipose tissue with a variable range of metabolic complications. The leading cause of these disorders is the nonphysiological accumulation of the lamin A precursor, prelamin A. However, the molecular mechanisms by which prelamin A induces the pathology remain unclear.The aim of this study is to use an experimental LMNA-lipodystrophy model based on human mesenchymal stem cell (hMSC)-derived adipocytes that accumulate prelamin A to gain deeper insights into the mechanisms governing these diseases.Prelamin A-induced or -noninduced hMSC-derived adipocytes were obtained from healthy…

AdultMaleAgingcongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyAdolescentLipodystrophyLipolysisEndocrinology Diabetes and MetabolismCellular differentiationClinical BiochemistryAdipose tissueMitochondrionBiologyBiochemistryLMNAYoung AdultEndocrinologyInternal medicineAdipocytesmedicineHumansMetabolomicsLipolysisintegumentary systemStem CellsBiochemistry (medical)Mesenchymal stem cellnutritional and metabolic diseasesCell DifferentiationMiddle AgedLamin Type ALipid MetabolismEndocrinologyMetabolomeFemaleStem cellLaminThe Journal of Clinical Endocrinology & Metabolism
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