Search results for "Cerebral blood flow."

showing 10 items of 138 documents

Effects of the exercise in the cerebral blood flow and metabolism: A review

2015

In recent years it has been shown that cerebral blood flow is affected by intense exercise, what may even lead to a reduction in the cognitive capacity. This statement is contrary to the traditional belief that cerebral blood flood remains constant and unaltered even when exercise is performed. During physical exercise of moderate intensity, cerebral blood flow increases in the cerebral areas responsible for movement. Moreover, recent studies have observed that cerebral blood flow decreases during high-intensity exercise as a consequence of a local hyperventilation and vasoconstriction of the areas with lower cerebral activity. Traditionally, the glucose has been considered as the main and …

Hyperthermiamedicine.medical_specialtyGlucose uptakeGlucose uptakePhysical Therapy Sports Therapy and RehabilitationPhysical exerciseOxygen consumptionInternal medicineHyperventilationEducación Física y DeportivaMedicineHyperthermialcsh:Sports medicinebusiness.industryBrainMetabolismmedicine.diseaseCerebral activityCerebral blood flowCardiologyLactatemedicine.symptombusinessBlood circulationlcsh:RC1200-1245NeuroscienceVasoconstrictionJournal of Human Sport and Exercise
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Pathophysiology of traumatic brain injury.

2007

The knowledge of the pathophysiology after traumatic head injury is necessary for adequate and patient-oriented treatment. As the primary insult, which represents the direct mechanical damage, cannot be therapeutically influenced, target of the treatment is the limitation of the secondary damage (delayed non-mechanical damage). It is influenced by changes in cerebral blood flow (hypo- and hyperperfusion), impairment of cerebrovascular autoregulation, cerebral metabolic dysfunction and inadequate cerebral oxygenation. Furthermore, excitotoxic cell damage and inflammation may lead to apoptotic and necrotic cell death. Understanding the multidimensional cascade of secondary brain injury offers…

InflammationNecrosisTraumatic brain injurybusiness.industryInflammationVasospasmBrain Edemamedicine.disease_causemedicine.diseaseBioinformaticsPathophysiologyOxidative StressAnesthesiology and Pain MedicineOxygen ConsumptionCerebral blood flowAnesthesiaBrain InjuriesCerebrovascular CirculationmedicineHumansmedicine.symptombusinessCell damageOxidative stressBritish journal of anaesthesia
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Prevention and treatment of intracranial hypertension

2007

Intracranial pressure (ICP) is the pressure exerted by cranial contents on the dural envelope. It comprises the partial pressures of brain, blood and cerebrospinal fluid (CSF). Normal intracranial pressure is somewhere below 10 mmHg; it may increase as a result of traumatic brain injury, stroke, neoplasm, Reye's syndrome, hepatic coma, or other pathologies. When ICP increases above 20 mmHg it may damage neurons and jeopardize cerebral perfusion. If such a condition persists, treatment is indicated. Control of ICP requires measurement, which can only be performed invasively. Standard techniques include direct ventricular manometry or measurement in the parenchyma with electronic or fiberopti…

Intracranial PressureTraumatic brain injurymedicine.medical_treatmentBrain EdemaCerebral autoregulationNeurosurgical ProceduresmedicineHumansCerebral perfusion pressureIntracranial pressurePostoperative Carebusiness.industrymedicine.diseaseRespiration ArtificialHydrocephalusHypertonic salineAnesthesiology and Pain MedicineCerebral blood flowBrain InjuriesAnesthesiaPneumocephalusDrainageDecompressive craniectomyIntracranial HypertensionbusinessHydrocephalusBest Practice & Research Clinical Anaesthesiology
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High-intensity transient signals during laparoscopic surgery in children.

2009

Background Laparoscopic interventions in children gain increasing popularity. Pneumoperitoneum as applied during laparoscopic surgery can induce gas emboli formation, but it is unclear whether this is associated with cerebral embolic events. To investigate the hypothesis that pneumoperitoneum causes cerebral emboli in children, the number and intensity of high-intensity transient signals (HITS) detected using transcranial Doppler ultrasonography were assessed before and after induction of pneumoperitoneum. Methods Twenty children were monitored during laparoscopic surgery. General anaesthesia was performed using sevoflurane and sufentanil or alfentanil. Pressure-controlled ventilation was a…

Laparoscopic surgeryMalemedicine.medical_specialtyMean arterial pressureUltrasonography Doppler Transcranialmedicine.medical_treatmentBlood PressurePneumoperitoneummedicineHumansGeneral anaesthesiaAlfentanilChildIntraoperative Complicationsbusiness.industryInfantBlood flowmedicine.diseaseSurgeryTranscranial Dopplerbody regionsAnesthesiology and Pain MedicineCerebral blood flowIntracranial EmbolismAnesthesiaCerebrovascular CirculationChild PreschoolFemaleLaparoscopybusinessPneumoperitoneum ArtificialBlood Flow Velocitymedicine.drugBritish journal of anaesthesia
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Influence of PEEP on cerebral blood flow and cerebrovascular autoregulation in patients with acute respiratory distress syndrome.

2013

High levels of positive end-expiratory pressure (PEEP), as part of the treatment in patients with acute respiratory distress syndrome (ARDS), may prevent alveolar collapse and maintain oxygenation. PEEP potentially reduces cerebral venous return, increases intracranial blood volume, and may, therefore, affect cerebral blood flow (CBF) and cerebrovascular autoregulation (AR). This study investigates the effect of PEEP on CBF and AR in patients with respiratory failure.CBF velocity was measured using transcranial doppler and correlated with the invasive arterial blood pressure curve to calculate the index of AR Mx (Mx0.3 indicates impaired AR). Mx was measured at lower PEEP levels and after i…

MaleARDSmedicine.medical_specialtyCritical CareUltrasonography Doppler TranscranialBlood volumeBlood PressurePreoperative carePositive-Pressure RespirationOxygen ConsumptionInternal medicinePreoperative CaremedicineHomeostasisHumansHypnotics and SedativesIn patientCollapse (medical)AgedAged 80 and overRespiratory Distress SyndromeBlood Volumebusiness.industryOxygenationrespiratory systemMiddle Agedmedicine.diseaserespiratory tract diseasesOxygenAnesthesiology and Pain MedicineCerebral blood flowCerebrovascular CirculationCardiologySurgeryFemaleNeurology (clinical)medicine.symptombusinesstherapeuticsVenous return curvecirculatory and respiratory physiologyJournal of neurosurgical anesthesiology
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Transpulmonary cardiac output measurement in a rat model of cardiac arrest and CPR: Impact of vascular access

2009

Objective: The present study investigated the impact of the vascular access site for cardiac output (CO) measurement by thermodilution on survival and neurohistopathological injury in a rat model of cardiac arrest (CA) and cardiopulmonary resuscitation (CPR). Secondary the influence of the vascular access site on cardiac output measurements was examined. Methods: Rats underwent asphyxial CA and CPR. Thermocouple probes were either placed via the femoral artery into the bifurcation of abdominal aorta/iliac artery (Femoral) or via the carotid artery into the aortic arch (Carotid). CPR was initiated after 9 min CA. Local cerebral blood flow (lCBF) and CO were assessed for 120 min after restora…

MaleAortic archCardiac outputmedicine.medical_treatmentHemodynamicsFemoral arteryEmergency NursingRats Sprague-Dawleymedicine.arteryIntensive caremedicineAnimalsCardiopulmonary resuscitationCardiac Outputbusiness.industryAbdominal aortaCardiopulmonary ResuscitationHeart ArrestRatsDisease Models AnimalCerebral blood flowAnesthesiaHeart Function TestsEmergency MedicineCardiology and Cardiovascular MedicinebusinessResuscitation
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Nitric oxide modulates cerebral blood flow stimulation by acetazolamide in the rat cortex: a laser Doppler scanning study

2001

Abstract The involvement of nitric oxide (NO) in cerebral blood flow (CBF) stimulation by acetazolamide was studied in anaesthetised, mechanically ventilated Wistar rats. CBF was monitored by laser Doppler scanning. Acetazolamide induced a long-lasting significant rCBF-increase. Application of N G -Nitro- l -arginine (L-NNA), an inhibitor of all NO synthetases (NOS), prevented CBF stimulation by acetazolamide. Continuous infusion of the exogenous NO donor SIN-1 (3-morpholinosydnonimine) suppressed L-NNA induced increases of mean arterial blood pressure without effect on rCBF in comparison to baseline. Additional acetazolamide injection then again caused a significant increase of rCBF in spi…

MaleArginineVasodilator AgentsHemodynamicsBlood PressureStimulationPharmacologyNitric OxideNitroarginineNitric oxidechemistry.chemical_compoundLaser-Doppler FlowmetrymedicineAnimalsNitric Oxide DonorsRats WistarCarbonic Anhydrase InhibitorsCerebral CortexChemistryGeneral NeuroscienceLaser Doppler velocimetryRatsAcetazolamidemedicine.anatomical_structurenervous systemCerebral blood flowCerebral cortexCerebrovascular CirculationMolsidomineAnesthesiaNitric Oxide SynthaseAcetazolamidecirculatory and respiratory physiologymedicine.drugNeuroscience Letters
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Local Cerebral Blood Flow in a Rat Cortical Vein Occlusion Model

1996

The symptoms following sinus and vein occlusion observed in patients and experimental animals display a considerable variability that so far remains largely unexplained. In a rat cortical vein occlusion model using a photochemical thrombotic technique, we examined changes in the cerebral venous flow pattern by fluorescence angiography and regional cerebral blood flow (rCBF) and cerebral blood volume fraction (CBVF) by a modern laser Doppler “scanning” technique. Brain damage was assessed histologically. Fluorescence angiographic findings fell into two groups: group A, rats with an altered venous flow pattern after occlusion (n = 12), and group B, rats with interruption of blood flow and/or…

MaleCerebral veinsPathologymedicine.medical_specialtyIschemia030218 nuclear medicine & medical imaging03 medical and health sciencesCerebral circulation0302 clinical medicineOcclusionLaser-Doppler FlowmetryAnimalsMedicineFluorescein AngiographyRats WistarCerebral perfusion pressureCerebral CortexBlood Volumebusiness.industryIntracranial Embolism and Thrombosismedicine.diseaseCortical VeinVein occlusionRatsNeurologyCerebral blood flowCerebrovascular CirculationNeurology (clinical)Cardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgeryJournal of Cerebral Blood Flow & Metabolism
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Novel complement C1 inhibitor BSF468248 does not improve brain damage after cortical vein occlusion

2003

BSF468248 is a novel potent complement C1 inhibitor. To determine whether BSF468248 is effective against focal cerebral ischemia, we evaluated the change of cerebral blood flow (CBF) and infarction volume using a photochemically-induced cortical vein occlusion model in rats in blind studies. In 22 Wistar rats, two adjacent cortical veins were occluded by photochemical thrombosis and fiberoptic illumination under controlled anesthesia and ventilation. Just after the occlusion, BSF468248 or physiological saline was administrated. In the low-dose study, a treatment group (n = 7) was administered BSF468248 1 mg/kg bolus and 1 mg/kg continuously for 30 min. The same volume of saline was given to…

MaleCerebral veinsPhotochemistrymedicine.medical_treatmentComplement C1 Inactivator ProteinsRats Sprague-DawleyBolus (medicine)OcclusionmedicineAnimalsRats WistarInfusions IntravenousSalineCerebral Cortexbusiness.industryCerebral InfarctionBlood flowCortical Veinmedicine.diseaseCerebral VeinsThrombosisRatsDisease Models AnimalTreatment OutcomeCerebral blood flowRegional Blood FlowBrain InjuriesCerebrovascular CirculationAnesthesiaIntracranial ThrombosisbusinessOligopeptidesMethods and Findings in Experimental and Clinical Pharmacology
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A fronto-parietal network is mediating improvement of motor function related to repetitive peripheral magnetic stimulation: A PET-H2O15 study.

2006

Repetitive peripheral magnetic stimulation (RPMS) is a focused and painless stimulation method, in which muscle contractions are elicited by depolarization of the terminal motor branches. Clinical-experimental investigations on different disorders of sensorimotor integration in the last decade have shown that RPMS can be used for the rehabilitation of motor functions after stroke. It is supposed that this therapeutic effect is based on the RPMS-induced proprioceptive inflow to the CNS. To analyze the conditioning effects of RPMS on reorganization of the motor system on cortical level positron emission tomography (PET) is used. Regional cerebral blood flow (rCBF) has been measured using H(2)…

MaleCognitive NeurosciencePosterior parietal cortexStimulationBrain mappingPremotor cortexFingersMagneticsParietal LobeMotor systemImage Processing Computer-AssistedMedicineHumansSpasticityAgedBrain MappingMovement DisordersProprioceptionbusiness.industryMiddle AgedFrontal LobeParesismedicine.anatomical_structureNeurologyCerebral blood flowMotor SkillsCerebrovascular CirculationPositron-Emission TomographyFemalemedicine.symptombusinessNeuroscienceNeuroImage
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