Search results for "Channel blocker"

showing 10 items of 157 documents

Pretreatment with potent P-glycoprotein ligands may increase intestinal secretion in rats.

2001

The expression of P-glycoprotein is induced in cell cultures upon exposure to various inducers. Therefore, the aim of the present study was to evaluate the in-vivo relevance of this observation, i.e. the influence of chronic pretreatments with selected drugs -- all of which are ligands to P-glycoprotein (P-gp) as demonstrated in radioligand binding studies and all of which have some or a considerable effect on P-gp expression in Caco-2 cells -- on the effective intestinal permeabilities of the model compound talinolol in rats employing in-situ single-pass intestinal perfusion of three different gut segments. Talinolol was selected, because it shows high selectivity for one of the exsorptive…

MaleColonDuodenumAdrenergic beta-AntagonistsPharmaceutical ScienceBiologyPharmacologyLigandsVinblastineJejunumPropanolamineschemistry.chemical_compoundmedicineAnimalsATP Binding Cassette Transporter Subfamily B Member 1Rats WistarP-glycoproteinIntestinal permeabilityStereoisomerismmedicine.diseaseCalcium Channel BlockersAntineoplastic Agents PhytogenicVinblastineRatsmedicine.anatomical_structureJejunumchemistryBiochemistryVerapamilDuodenumbiology.proteinVerapamilPerfusionTalinololmedicine.drugEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
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Effects of Calcium Antagonists on Rat Normal and Skinned Fundus

1992

Abstract Calcium chloride (CaCl2) (0·1−25 Mm, in K+-depolarized tissue), KCl (10−112 Mm) and acetylcholine (1 × 10−9 m−1 Mm) produced concentration-dependent contractions of rat isolated fundus. Verapamil (0·01−100 μm), cinnarizine (1−100 μm), trifluoperazine (10−500 μm) and dantrolene (50−250 μm) each produced a concentration-related rightward and downward shift of the log concentration-effect curve for CaCl2. The rank order of potencies of these antagonists, measured as the IC50 against Ca2+ (25 Mm)-induced contraction of depolarized fundus, was verapamil (2.5 μm) > cinnarizine (8·7 μm) > trifluoperazine (85·1 μm) > dantrolene (> 250 μm). Cinnarizine (0·5 Mm) a…

MaleContraction (grammar)CinnarizineGuinea PigsPharmaceutical Sciencechemistry.chemical_elementTrifluoperazineIn Vitro TechniquesPharmacologyCalciumDantroleneDantroleneCinnarizinemedicineAnimalsGastric FundusPharmacologyChemistryMuscle SmoothRats Inbred StrainsCalcium Channel BlockersTrifluoperazineRatsVerapamilAnesthesiaVerapamilFemalemedicine.symptomAcetylcholinemedicine.drugMuscle contractionJournal of Pharmacy and Pharmacology
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Effects of K(ATP) channel modulators on acetylcholine release from guinea-pig isolated atria and small intestine.

2002

The effects of K(ATP) channel blockers (glibenclamide, HMR 1883, HMR 1372) and openers (cromakalim, pinacidil, diazoxide) on the electrically-evoked (5 Hz) release of [(3)H]acetylcholine were studied in isolated guinea-pig atria and myenteric plexus-longitudinal muscle preparations which had been preincubated with [(3)H]choline. Atria: Cromakalim (0.3 microM and 1 microM), pinacidil (10 microM) and diazoxide (30 microM) significantly reduced the stimulation-evoked release of [(3)H]acetylcholine. The inhibition produced by cromakalim and pinacidil was prevented by 1 microM of either HMR 1883, HMR 1372 or glibenclamide. The blockers alone significantly increased the release at concentrations …

MaleCromakalimPotassium ChannelsGuinea PigsNeuromuscular JunctionMyenteric PlexusPharmacologyIn Vitro Techniqueschemistry.chemical_compoundGlyburideIntestine SmallmedicineDiazoxidePotassium Channel BlockersAnimalsChannel blockerHeart AtriaPharmacologySulfonamidesPinacidilDiazoxideThioureaPotassium channel blockerMuscle SmoothGeneral Medicinemusculoskeletal systemAtrial FunctionMyocardial ContractionHMR 1883Potassium channelAcetylcholinechemistryAnesthesiaPinacidilcardiovascular systemFemaleCromakalimAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Combination of alpha lipoic acid and gabapentin, its efficacy in the treatment of burning mouth syndrome : a randomized, double-blind, placebo contro…

2010

Burning Mouth Syndrome (BMS) is a disease that manifests as burning in the tongue or in any area of the oral mucosa, in the absence of clinically verifiable injuries. Objectives: To verify the efficacy of alpha lipoic acid (ALA) and gabapentin (GABA), used individually and jointly, to reduce the burning in patients with burning mouth and establish a drug therapy for the BMS. Study Design: During April and May 2008, we conducted a randomized, double-blind, placebo-controlled trial in the Department of Clinical Stomatology, Faculty of Dentistry, Rosario, Argentina. The gathering of patients was between those ones with BMS who were treated in our service between March 2003 and March 2008 witho…

MaleGabapentinCyclohexanecarboxylic AcidsPlacebo-controlled studyBurning Mouth SyndromePlaceboGroup BPharmacotherapyDouble-Blind MethodTonguemedicineHumansOral mucosaAminesGeneral Dentistrygamma-Aminobutyric AcidThioctic Acidbusiness.industryBurning mouth syndromeMiddle AgedCalcium Channel Blockers:CIENCIAS MÉDICAS [UNESCO]medicine.anatomical_structureOtorhinolaryngologyAnesthesiaUNESCO::CIENCIAS MÉDICASSurgeryDrug Therapy CombinationFemalemedicine.symptomGabapentinbusinessmedicine.drug
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Changes in cytosolic calcium in response to noxious heat and their relationship to vanilloid receptors in rat dorsal root ganglion neurons.

2001

Heat transduction mechanisms in primary nociceptive afferents have been suggested to involve a vanilloid receptor channel with high calcium permeability. To characterize the changes in free cytosolic calcium evoked by noxious heat stimuli (< or =51 degrees C, 10s), we performed microfluorometric measurements in acutely dissociated small dorsal root ganglion neurons (< or =32.5 microm) of adult rats using the dye FURA-2. Only neurons that responded with a reversible increase in intracellular calcium to high potassium were evaluated. Heat-induced calcium transients (exceeding mean + 3S.D. of the temperature dependence of the dye) were found in 66 of 105 neurons. These transients increased non…

MaleHot Temperaturemedicine.drug_classReceptors Drugchemistry.chemical_elementPainCalcium channel blockerCalciumCalcium in biologyRats Sprague-Dawleychemistry.chemical_compoundCytosolGanglia SpinalmedicineAnimalsThermosensingCalcium SignalingNeurons AfferentCells CulturedFluorescent DyesCalcium metabolismVoltage-dependent calcium channelGeneral NeuroscienceMyocardiumT-type calcium channelNociceptorsRatschemistryBiochemistryCapsaicinBiophysicsPotassiumCalciumFemaleCalcium ChannelsCapsaicinCapsazepineFura-2Signal TransductionNeuroscience
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Synthesis, configuration, and calcium modulatory properties of enantiomerically pure 5-oxo-1,4,5,6,7,8-hexahydroquinoline-3-carboxylates.

1992

Enantiomerically pure hexahydroquinolinones of the structural type 9 were prepared by a variation of the Hantzsch synthesis in which an optically active acetoacetate served as a chiral auxiliary reagent. Determinations of the de and ee values are described. The absolute configurations of the optically pure products were characterized by single-crystal X-ray analysis. The antipodes 9a and 9b exhibited calcium antagonistic activities on smooth musculature; the (S)-(-)-enantiomer 9b was the more potent compound with regard to the EC50 values which differed by a factor of 100; the intrinsic activity of 9b was 1.2, compared with a value of 0.54 for 9a. On the other hand, R-(+)-9a exerted positiv…

MaleIntrinsic activityGuinea PigsMolecular Conformationchemistry.chemical_elementCalciumQuinolonesMedicinal chemistrychemistry.chemical_compoundX-Ray DiffractionIleumDrug DiscoveryAnimalsHeart AtriaAortaChiral auxiliaryBicyclic moleculeMolecular StructureEnantioselective synthesisAbsolute configurationBiological activityStereoisomerismPapillary MusclesAtrial FunctionCalcium Channel BlockersElectric StimulationchemistryReagentMolecular MedicineFemaleMuscle ContractionJournal of medicinal chemistry
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Prenatal exposure to the CB1 receptor agonist WIN 55,212-2 causes learning disruption associated with impaired cortical NMDA receptor function and em…

2005

The aim of this study was to investigate whether prenatal exposure to the cannabinoid CB1 receptor agonist WIN 55,212-2 (WIN) at a daily dose devoid of overt signs of toxicity and/or gross malformations (0.5 mg/kg, gestation days 5-20), influences cortical glutamatergic neurotransmission, learning and emotional reactivity in rat offspring. Basal and K+-evoked extracellular glutamate levels were significantly lower in cortical cell cultures obtained from pups exposed to WIN during gestation with respect to those measured in cultures obtained from neonates born from vehicle-treated dams. The addition of NMDA to cortical cell cultures from neonates born from vehicle-treated dams concentration-…

MaleMarijuana AbuseCannabinoid receptoractive avoidance behaviour; basal and K+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationEmotionsReceptor Cannabinoid CB1Pregnancyactive avoidance behaviourWIN 55212-2Cells CulturedCerebral CortexBehavior AnimalGlutamate receptorBraincortical cell culturesCalcium Channel Blockersactive avoidance behaviour; basal and k plus -evoked glutamate levels; basal and k+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationPrenatal Exposure Delayed EffectsChloratesNMDA receptorbasal and K+-evoked glutamate levelsFemaleMicrotubule-Associated Proteinsmedicine.drugAgonistmedicine.medical_specialtyOffspringmedicine.drug_classCognitive NeuroscienceMorpholinesGlutamic Acidmaternal marijuana consumptionNeurotransmissionBiologyNaphthalenesReceptors N-Methyl-D-AspartateCellular and Molecular NeuroscienceGlutamatergicInternal medicinemedicineAvoidance LearningAnimalsRats WistarBenzoxazinesRatsultrasonic vocalizationEndocrinologyAnimals Newbornhoming behaviourVocalization AnimalExtracellular SpaceNeuroscience
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Inhibitory effect of caffeic acid phenethyl ester, a plant-derived polyphenolic compound, on rat intestinal contractility

2009

Abstract Caffeic acid phenethyl ester (CAPE) exerts pharmacological actions (e.g. anti-inflammatory, chemopreventive) which are relevant for potential clinical application in the digestive tract. However, no study has been published on its possible effects on intestinal motility, to date. In the present study, we investigated the effect of this plant-derived polyphenolic compound on the spontaneous contractions of the rat isolated ileum. CAPE reduced (in a tetrodotoxin-insensitive manner) spontaneous ileal contractions and this effect was reduced by the l -type Ca 2+ channel blocker nifedipine and the chelant of calcium ethylenediaminetetraacetic acid. However, the effect of CAPE was not mo…

MaleMuscle Relaxationchemistry.chemical_elementPharmacologyCalciumIn Vitro TechniquesApaminPotassium Chloridechemistry.chemical_compoundPhentolamineCaffeic AcidsPhenolsIleumCaffeic acidmedicineAnimalsChannel blockerRats WistarCaffeic acid phenethyl esterPharmacologyFlavonoidsVoltage-dependent calcium channelPolyphenolsPhenylethyl AlcoholPlantsRatschemistryBiochemistryCyclopiazonic acidmedicine.drugMuscle Contraction
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Role of Ca2+-Activated K+ Channels on Adrenergic Responses of Human Saphenous Vein

2006

Background We studied the participation of K + channels on the adrenergic responses in human saphenous veins as well as the intervention of dihydropyridine-sensitive Ca 2+ channels on modulation of adrenergic responses by K + channels blockade. Methods Saphenous vein rings were obtained from 40 patients undergoing coronary artery bypass surgery. The vein rings were suspended in organ bath chambers for isometric recording of tension. Results Iberiotoxin (10 −7 mol/L), an inhibitor of large conductance Ca 2+ -activated K + channels, and charybdotoxin (10 −7 mol/L), an inhibitor of both large and intermediate conductance Ca 2+ -activated K + channels, enhanced the contractions elicited by elec…

MaleNifedipineCharybdotoxinAdrenergicStimulationIn Vitro TechniquesApaminMuscle Smooth VascularNorepinephrinePotassium Channels Calcium-Activatedchemistry.chemical_compoundInternal MedicinemedicineHumansSaphenous VeinChannel blockerbusiness.industryDihydropyridineMiddle AgedIberiotoxinCalcium Channel BlockersElectric StimulationchemistryVasoconstrictionMuscle TonusAnesthesiaBiophysicsFemalemedicine.symptombusinessMuscle contractionmedicine.drugAmerican Journal of Hypertension
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Mechanisms underlying the nitric oxide inhibitory effects in mouse ileal longitudinal muscle

2005

We investigated the mechanisms involved in the nitric oxide (NO)-induced inhibitory effects on longitudinal smooth muscle of mouse ileum, using organ bath technique. Exogenously applied NO, delivered as sodium nitroprusside (SNP; 0.1–100 µmol/L) induced a concentration-dependent reduction of the ileal spontaneous contractions. 1H-[1,2,4]oxadiazolol[4,3,a]quinoxalin-1-one (ODQ; 1 µmol/L), a guanilyl cyclase inhibitor, reduced the SNP-induced effects. Tetraethylammonium chloride (20 mmol/L), a non-selective K+ channel blocker, and charybdotoxin (0.1 µmol/L), blocker of large conductance Ca2+-dependent K+ channels, significantly reduced SNP-induced inhibitory effects. In contrast, apamin (0.1…

MaleNitroprussideThapsigarginCharybdotoxinPhysiologyMouse ileumIn Vitro TechniquesPharmacologyApaminSettore BIO/09 - FisiologiaPotassium channelsMicePotassium Channels Calcium-Activatedchemistry.chemical_compoundIleumPhysiology (medical)Cyclic GMP-Dependent Protein KinasesPotassium Channel BlockersmedicineAnimalsNitric Oxide DonorsChannel blockerCyclic GMPPharmacologyRyanodineRyanodine receptorCalcium storeMuscle SmoothPotassium channel blockerNitric oxideGeneral MedicineTetraethylammonium chlorideMice Inbred C57BLchemistryCalciumSodium nitroprussideMuscle ContractionSignal Transductionmedicine.drug
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