Search results for "Cholinergic"

showing 10 items of 251 documents

Urinary Incontinence in Chronic Obstructive Pulmonary Disease: A Common Co-morbidity or a Typical Adverse Effect?

2019

Urinary incontinence (UI) is defined as a loss of bladder control and is characterized by the complaint of any involuntary leakage of urine. Evidence suggests that the prevalence of UI is higher in subjects with chronic obstructive pulmonary disease (COPD) than in age-matched controls in both sexes. UI is classified as stress, urge, and mixed, and has a considerable impact on quality of life. However, the prevalence of UI in individuals with COPD is mostly unexplored in clinical research and often underestimated in clinical practice. Interestingly, although the involuntary leakage of a small amount of urine during coughing (e.g., stress UI) is among the most plausible causes of UI in patien…

Malemedicine.medical_specialtyUrinary incontinenceAnticholinergic agentsComorbidity03 medical and health sciencesPulmonary Disease Chronic Obstructive0302 clinical medicineQuality of lifeInternal medicineSurveys and QuestionnairesPrevalenceMedicineHumansCOPDPharmacology (medical)030212 general & internal medicineAdverse effectbladderAgedCOPDbusiness.industryUrinary retentionmedicine.diseaseComorbidityClinical researchUrinary IncontinenceQuality of LifeFemaleGeriatrics and Gerontologymedicine.symptombusiness030217 neurology & neurosurgery
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Functional evidence for different roles of GABAA and GABAB receptors in modulating mouse gastric tone

2010

Abstract The aims of the present study were to investigate, using mouse whole stomach in vitro , the effects of γ-aminobutyric acid (GABA) and GABA receptor agonists on the spontaneous gastric tone, to examine the subtypes of GABA receptors involved in the responses and to determine the possible site(s) of action. GABA induced gastric relaxation, which was antagonized by the GABA A -receptor antagonist, bicuculline, potentiated by phaclofen, GABA B -receptor antagonist, but not affected by 1,2,5,6-Tetrahydropyridin-4-yl methylphosphinic acid hydrate (TPMPA), GABA C -receptor antagonist. Muscimol, GABA A -receptor agonist, mimicked GABA effects inducing relaxation, which was significantly re…

Malemedicine.medical_specialtymedicine.drug_classMuscle RelaxationIn Vitro TechniquesGABAB receptorApaminSettore BIO/09 - FisiologiaMicePotassium Channels Calcium-ActivatedGABACellular and Molecular Neurosciencechemistry.chemical_compoundPhaclofenReceptors GABAGABA receptorNANC inhibitory nerves.GABA receptorInternal medicinemedicineAnimalsGABA-A Receptor AgonistsGABA-A Receptor Antagonistsgamma-Aminobutyric AcidPharmacologyGABAA receptorMuscle SmoothBicucullineReceptors GABA-AReceptor antagonistMice Inbred C57BLEndocrinologyReceptors GABA-Bnervous systemMuscimolchemistryGABA-B Receptor AgonistsMuscle Tonuscholinergic excitatory nerveNitric Oxide SynthaseGABA-B Receptor Antagonistsstomachmedicine.drugNeuropharmacology
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Nerves projecting from the intrinsic cardiac ganglia of the pulmonary veins modulate sinoatrial node pacemaker function

2013

Rationale: Autonomic nerves from sinoatrial node (SAN) ganglia are known to regulate SAN function. However, it is unclear whether remote pulmonary vein ganglia (PVG) also modulate SAN pacemaker rhythm. Objective: To investigate whether in the mouse heart PVG modulate SAN function. Methods and Results: In hearts from 45 C57BL and 7 Connexin40+/GFP mice, we used tyrosine-hydroxylase (TH) and choline-acetyltransferase (ChAT) immunofluorescence labeling to characterize adrenergic and cholinergic elements, repectively, within the PVG and SAN. PVG project postganglionic nerves to the SAN. TH and ChAT stained nerves, enter the SAN as an extensive, dense mesh-like neural network. Neurons in PVG are…

Malemedicine.medical_specialtysinoatrial nodepulmonary veinsPhysiologyAdrenergicMice TransgenicStimulationIn Vitro TechniquesMiceFetal HeartBiological ClocksHeart Conduction SystemHeart RatePhysiology (medical)Internal medicineAtrial FibrillationHeart ratemouse heartmedicineAnimalsHumansSinus rhythmIntrinsic cardiac gangliaSinoatrial NodeSinoatrial nodebusiness.industryOriginal ArticlesMiddle AgedElectric StimulationElectrophysiological PhenomenaMice Inbred C57BLoptical mappingAtropinemedicine.anatomical_structureEndocrinologyPulmonary Veinscardiac arrhythmiasCatheter AblationCardiologyCholinergicFemaleGangliaElectrical conduction system of the heartCardiology and Cardiovascular Medicinebusinessmedicine.drugCardiovascular Research
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The contribution of acetylcholine and dopamine to subprocesses of visual working memory--what patients with amnestic mild cognitive impairment and Pa…

2014

Attentional selection, i.e. filtering out of irrelevant sensory input and information storage are two crucial components of working memory (WM). It has been proposed that the two processes are mediated by different neurotransmitters, namely acetylcholine for attentional selection and dopamine for memory storage. However, this hypothesis has been challenged by others, who for example linked a lack in dopamine levels in the brain to filtering deficits. Here we tested the above mentioned hypothesis in two patient cohorts which either served as a proxy for a cholinergic or a dopaminergic deficit. The first group comprised 18 patients with amnestic mild cognitive impairment (aMCI), the second 22…

Malephysiopathology [Cognitive Dysfunction]Parkinson's diseaseCognitive NeuroscienceDopamineModels NeurologicalExperimental and Cognitive Psychologyphysiopathology [Brain]Neuropsychological TestsCohort StudiesBehavioral Neurosciencechemistry.chemical_compoundDopaminemedicineHumansAttentionCognitive Dysfunctionddc:610metabolism [Dopamine]NeurotransmitterAgedWorking memoryDopaminergicBrainCognitionParkinson Diseasephysiopathology [Amnesia]Middle Agedphysiology [Visual Perception]medicine.diseaseAcetylcholineMemory Short-Termchemistryphysiology [Memory Short-Term]physiology [Attention]Visual PerceptionCholinergicFemalephysiopathology [Parkinson Disease]AmnesiaPsychologyNeurosciencemetabolism [Acetylcholine]Acetylcholinemedicine.drugNeuropsychologia
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Drug‐refractory myasthenia gravis: Clinical characteristics, treatments, and outcome

2022

[Objective] To describe the clinical characteristics and outcomes in patients with refractory myasthenia gravis (MG) and to determine the effectiveness and side effects of the drugs used for their treatment.

Maleprogressive multifocal leukoencepdiarrheacholinergic receptorplasma exchangemiddle agedadultimmunologic factornauseaanemiahypertrichosisageddrug withdrawaldiabetes mellitusdisease severityTRIALsafetycorticosteroidhypertensionImmunologyMiastenia gravismethotrexateArticlebulbar paralysispancytopeniaMuscular DiseasescompulsionMyasthenia Gravischolinesterase inhibitorcross-sectional studyHumansImmunologic FactorshumanRITUXIMABarthralgiaNeurologíaMalalties muscularsAgedRetrospective Studiesmyasthenia gravisleukopeniaabdominal painDrug testingmajor clinical studyCross-Sectional StudiesDrug side effectscyclophosphamideobservational studyNeurology (clinical)immunoglobulinFEATURESefficacyclinical outcomeelectrophysiological procedurescomputer assisted tomographyDOUBLE-BLINDTratamiento médicorituximabOutcome Assessment Health CareImmunologiamuscle specific tyrosine kinaseRegistriestacrolimusazathioprineMedicamentoGeneral Neurosciencenephrotoxicitygeneral condition deteriorationhyperplasiatrialMiddle Agedliver toxicitydrug toxicityunclassified drugfemaleEfectes secundaris dels medicamentsSAFETYFemaledouble-blindheadacheblindnessAdultAssaigs clínics de medicamentsmalefeaturesfollow uppneumoniacyclosporinemycophenolate mofetilprotein tyrosine kinaseimmunosuppressive agentallergyalopeciaEFFICACYclinical featureosteopeniaSpainprednisonehyperglycemiaautoantibodyFollow-Up Studies
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Impact of tiotropium + olodaterol on physical functioning in COPD: results of an open-label observational study

2016

Rüdiger Sauer,1 Michaela Hänsel,2 Roland Buhl,3 Roman A Rubin,4 Marcel Frey,5 Thomas Glaab2,3 1Lung Centre Ulm, Ulm, Germany; 2Boehringer Ingelheim Pharma GmbH & Co. KG, Ingelheim, Germany; 3Pulmonary Department, Mainz University Hospital, Mainz, Germany; 4Pulmonary Specialist Practice, Wiesbaden, Germany; 5Biometrics, Alcedis GmbH, Gießen, Germany Background: Maintaining and improving physical functioning is key to mitigating the cycle of deconditioning associated with chronic obstructive pulmonary disease (COPD). We evaluated the impact of free combination of the long-acting anticholinergic tiotropium plus the long-acting β2-agonist ol…

Malereal-worldTime FactorsnoninterventionalHealth StatusSeverity of Illness IndexCholinergic AntagonistsPulmonary Disease Chronic Obstructivechemistry.chemical_compound0302 clinical medicinetiotropiumDeconditioningPhysical functioningSurveys and QuestionnairesProspective Studies030212 general & internal medicineLungOriginal ResearchCOPDOlodaterolGeneral MedicineTiotropium bromideMiddle AgedBronchodilator AgentsDrug CombinationsTreatment OutcomeFemalemedicine.drugmedicine.medical_specialtymedicine.drug_classInternational Journal of Chronic Obstructive Pulmonary Diseasechronic obstructive pulmonary disease03 medical and health sciencesAdministration InhalationSeverity of illnessmedicineAnticholinergicphysical functioningHumansTiotropium BromideIntensive care medicineAdrenergic beta-2 Receptor AgonistsAgedolodaterolbusiness.industryNebulizers and VaporizersRecovery of Functionmedicine.diseaseBenzoxazines030228 respiratory systemchemistryPhysical therapyObservational studybusinessInternational Journal of Chronic Obstructive Pulmonary Disease
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Long-Term Response to Cholinesterase Inhibitor Treatment Is Related to Functional MRI Response in Alzheimer's Disease.

2015

<b><i>Background:</i></b> Treatment of Alzheimer's disease (AD) with cholinesterase inhibitors (ChEI) enhances cholinergic activity and alleviates clinical symptoms. However, there is variation in the clinical response as well as system level changes revealed by functional MRI (fMRI) studies. <b><i>Methods:</i></b> We investigated 18 newly diagnosed mild AD patients with fMRI using a face recognition task after a single oral dose of rivastigmine, a single dose of placebo and 1-month treatment with rivastigmine. The clinical follow-up took place at 6 and 12 months. <b><i>Results:</i></b> MMSE score difference between bas…

Malevsual processingCognitive NeuroscienceRivastigmineDiseasePharmacologyNeuropsychological TestsAlzheimerin tautimmoryta3112behavioral disciplines and activitiesBrain mappingAlzheimer DiseasemedicineHumansCholinesteraseAgedRivastigmineAged 80 and overmuistisairaudetBrain Mappingmedicine.diagnostic_testbiologyBrainMagnetic resonance imagingMiddle Agedmedicine.diseaseMagnetic Resonance Imagingta3124Psychiatry and Mental healthLong term responsecolinesterase inhibitorbiology.proteinCholinergicFemaleCholinesterase InhibitorsGeriatrics and GerontologyAlzheimer's diseasePsychologyAlzheimer’s diseaseNeurosciencepsychological phenomena and processesmgnetic resonance imagingmedicine.drugDementia and geriatric cognitive disorders
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Involvement of the cholinergic basal forebrain nuclei in spinocerebellar ataxia type 2 (SCA2)

2013

Aims: Spinocerebellar ataxia type 2 (SCA2) belongs to the CAG repeat or polyglutamine diseases. Along with a large variety of motor, behavioural and neuropsychological symptoms the clinical picture of patients suffering from this autosomal dominantly inherited ataxia may also include deficits of attention, impairments of memory, as well as frontal-executive and visuospatial dysfunctions. As the possible morphological correlates of these cognitive SCA2 deficits are unclear we examined the cholinergic basal forebrain nuclei, which are believed to be crucial for several aspects of normal cognition and may contribute to impairments of cognitive functions under pathological conditions. Methods: …

Medial septal nucleusBasal forebrainHistologyAtaxiabusiness.industrySubstantia innominataAnatomymedicine.diseaseDiagonal band of BrocaPathology and Forensic Medicinemedicine.anatomical_structurenervous systemNeurologyPhysiology (medical)medicineSpinocerebellar ataxiaCholinergicNeurology (clinical)medicine.symptomCholinergic neuronbusinessNeuroscienceNeuropathology and Applied Neurobiology
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Temporal coherency between receptor expression, neural activity and AP-1-dependent transcription regulates Drosophila motoneuron dendrite development.

2013

Neural activity has profound effects on the development of dendritic structure. Mechanisms that link neural activity to nuclear gene expression include activity-regulated factors, such as CREB, Crest or Mef2, as well as activity-regulated immediate-early genes, such as fos and jun. This study investigates the role of the transcriptional regulator AP-1, a Fos-Jun heterodimer, in activity-dependent dendritic structure development. We combine genetic manipulation, imaging and quantitative dendritic architecture analysis in a Drosophila single neuron model, the individually identified motoneuron MN5. First, Dα7 nicotinic acetylcholine receptors (nAChRs) and AP-1 are required for normal MN5 dend…

Mef2Transcriptional ActivationEmbryo NonmammalianTime FactorsTranscription GeneticReceptor expressionReceptors NicotinicCREBSynaptic TransmissionAnimals Genetically ModifiedGenes ReporterCa2+/calmodulin-dependent protein kinaseAnimalsDrosophila ProteinsCholinergic synapseCholinergic neuronMolecular BiologyResearch ArticlesCell NucleusDendritic spikeMicroscopy ConfocalbiologyGene Expression Regulation DevelopmentalDendritesImmunohistochemistryCholinergic NeuronsCell biologyEnzyme ActivationTranscription Factor AP-1Drosophila melanogasterMicroscopy Fluorescencebiology.proteinSignal transductionCalcium-Calmodulin-Dependent Protein Kinase Type 2Developmental BiologySignal TransductionDevelopment (Cambridge, England)
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Dual modulation of striatal acetylcholine release by hyperforin, a constituent of St. John's wort.

2002

Extracts of the medicinal plant St. John's wort (Hypericum perforatum) are widely used for the treatment of mild to moderate depression. Hyperforin, a constituent of St. John's wort, is known to inhibit the sodium-dependent uptake of catecholamines and amino acids into synaptic nerve endings, probably by interference with mechanisms controlling the synaptic sodium concentration. Because de novo synthesis of acetylcholine (ACh) is dependent on sodium-dependent high-affinity choline uptake, we studied the effect of hyperforin on choline (Ch) uptake in vitro and on striatal ACh release in vivo using microdialysis. In rat brain synaptosomes, hyperforin inhibited high-affinity choline uptake wit…

MicrodialysisPharmacologyMotor ActivityPhloroglucinolCholineRats Sprague-Dawleychemistry.chemical_compoundBridged Bicyclo CompoundsIn vivomedicineCholineAnimalsReceptors CholinergicIC50PharmacologyChemistryTerpenesHypericum perforatumBiological TransportAcetylcholineCorpus StriatumAnti-Bacterial AgentsRatsHyperforinSystemic administrationMolecular MedicineAcetylcholineHypericummedicine.drugThe Journal of pharmacology and experimental therapeutics
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