Search results for "Cholinergic"

showing 10 items of 251 documents

Role for NK1 and NK2 receptors in the motor activity in mouse colon

2007

The present study examined the effects induced by endogenous and exogenous activation of NK(1) and NK(2) receptors on the mechanical activity of mouse proximal colon. Experiments were performed in vitro recording the changes in intraluminal pressure from isolated colonic segments. Electrical field stimulation in the presence of atropine and guanethidine produced a small relaxation, followed by nonadrenergic noncholinergic (NANC) contraction. SR140333, NK(1) receptor antagonist, or SR48968, NK(2) receptor antagonist, significantly reduced the contraction, although SR48968 appeared more efficacious. The co-administration of SR140333 and SR48968 virtually abolished the NANC contraction. [Sar(9…

AtropineAgonistmedicine.medical_specialtyContraction (grammar)Colonmedicine.drug_classNeurokinin AMuscarinic AntagonistsTetrodotoxinSubstance PSettore BIO/09 - FisiologiaNK1 receptorNitric oxideMicechemistry.chemical_compoundNeurokinin-1 Receptor Antagonistsnitric oxideInternal medicinemedicineAnimalsNK2 receptorReceptorGuanethidinePharmacologyAntagonistReceptors Neurokinin-2Receptors Neurokinin-1Electric StimulationPeptide FragmentsMice Inbred C57BLEndocrinologychemistryTetrodotoxinNANC contractionCholinergicTachykininMuscle ContractionSodium Channel Blockersmedicine.drugEuropean Journal of Pharmacology
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EMG activity of pigeon oesophagus in vivo.

1982

At rest, the pigeon cervical oesophagus, which is entirely smooth muscle, shows electric activity. This activity consists of bursts of spikes with frequency increasing in the oral-aboral direction. The bursts are un-phase locked, and there are no slow waves (E.C.A.). The surgical transection of the oesophageal muscular wall does not affect the electric activity even in a disconnected segment. After asphyxia electric activity persists, whereas the aboral gradient of frequency disappears. Therefore, the electric activity is thought to be myogenic in origin, and the frequency gradient nervous in origin. Atropine and neostigmine administration suggests that the cholinergic system modulates the …

AtropineElectromyographyPhysiologyAction PotentialsHexamethonium CompoundsAnatomyBiochemistryNeostigmineNeostigmineElectrophysiologyAsphyxiaAtropinechemistry.chemical_compoundEsophagusCervical oesophaguschemistrySmooth muscleIn vivoCholinergic systemmedicineAnimalsHexamethoniumColumbidaemedicine.drug
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PACAP induces bradycardia in guinea-pig heart by stimulation of atrial cholinergic neurones

1996

Based on previous studies which indicated that pituitary adenylate cyclase activating peptide (PACAP) acts as a positive inotropic and chronotropic substance in different species via the cAMP signal transduction pathway, the objective of the present work was to investigate cAMP-regulated myocardial key proteins in response to PACAP in isolated ventricular cells of the guinea pig. Surprisingly, the two molecular forms of PACAP, PACAP(1-27) and PACAP(1-38), showed no effect on intracellular cAMP-levels, L-type Ca2+ channel current or phosphorylation of troponin inhibitor (TnI) and phospholamban (PLB). Additionally, inotropy of isolated guinea-pig ventricular strips was not affected by the neu…

AtropineMaleChronotropicendocrine systemmedicine.medical_specialtyGuinea PigsReceptors Pituitary Adenylate Cyclase-Activating PolypeptideStimulationIn Vitro TechniquesBiologyInternal medicineBradycardiaCyclic AMPmedicineAnimalsHeart AtriaReceptors Pituitary HormonePatch clampNeuronsPharmacologyNeurotransmitter AgentsMyocardiumCalcium-Binding ProteinsNeuropeptidesGeneral MedicineAcetylcholineRatsPhospholambanElectrophysiologyAtropinePituitary adenylate cyclase-activating peptideEndocrinologycardiovascular systemPituitary Adenylate Cyclase-Activating PolypeptideCholinergicFemalehormones hormone substitutes and hormone antagonistsAcetylcholineReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type ISignal Transductionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Inhibition by oxotremorine of acetylcholine resting release from guinea pig-ileum longitudinal muscle strips

1975

1. Longitudinal muscle strips of the guinea-pig ileum were incubated in Tyrode solution containing either DFP or physostigmine as cholinesterase inhibior. After a 90 min preincubation period the acetylcholine resting release into the medium was determined. Acetylcholine was estimated by gas chromatography. 2. The resting release was 0.39 nmol/g×min irrespective of the cholinesterase inhibitor used. In the presence of hexamethonium, or after omission of external calcium, the resting release fell by 50 and 55%, respectively. 3. Oxotremorine (10−5 and 10−4 M) significantly inhibited the resting release of acetylcholine by 25 and 33%, respectively. The inhibitory effect of oxotremorine was comp…

AtropineMalePhysostigminemedicine.medical_specialtyChromatography GasIsoflurophatePhysostigmineGuinea PigsHexamethonium CompoundsIn Vitro Techniqueschemistry.chemical_compoundIleumInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineAnimalsReceptors CholinergicCholinesterasePharmacologybiologyOxotremorineMuscle SmoothGeneral MedicineAcetylcholineAtropineEndocrinologychemistryDepression Chemicalbiology.proteinCholinergicCalciumFemaleHexamethoniumAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Different muscarinic receptor subtypes modulate proliferation of primary human detrusor smooth muscle cells via Akt/PI3K and map kinases.

2013

While acetylcholine (ACh) and muscarinic receptors in the bladder are mainly known for their role in the regulation of smooth muscle contractility, in other tissues they are involved in tissue remodelling and promote cell growth and proliferation. In the present study we have used primary cultures of human detrusor smooth muscle cells (HDSMCs), in order to investigate the role of muscarinic receptors in HDSMC proliferation. Samples were obtained as discarded tissue from men >65 years undergoing radical cystectomy for bladder cancer and cut in pieces that were either immediately frozen or placed in culture medium for the cell culture establishment. HDSMCs were isolated from samples, propagat…

AtropineMalePyrrolidinesMessenger030232 urology & nephrologyGene ExpressionPhosphatidylinositol 3-Kinases0302 clinical medicineAged Atropine; pharmacology Benzofurans; pharmacology Carbachol; pharmacology Cell Proliferation Cells; Cultured Cholinergic Agonists; pharmacology Gene Expression Humans Male Mitogen-Activated Protein Kinases; metabolism Muscarinic Antagonists; pharmacology Myocytes; Smooth Muscle; metabolism Phosphatidylinositol 3-Kinases; metabolism Piperidines; pharmacology Pirenzepine; analogs /&/ derivatives/pharmacology Proto-Oncogene Proteins c-akt; metabolism Pyrrolidines; pharmacology RNA; Messenger; metabolism Receptors; Muscarinic; physiology Urinary Bladder; cytologyPiperidinesSmooth MuscleReceptorsMuscarinic acetylcholine receptor M5Muscarinic acetylcholine receptorCells CulturedCulturedMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2Smooth muscle contractionMuscarinic acetylcholine receptor M1Receptors Muscarinic030220 oncology & carcinogenesisMitogen-Activated Protein KinasesAcetylcholinemedicine.drugmedicine.medical_specialtyCarbacholCellsMyocytes Smooth MuscleUrinary BladderMuscarinic AntagonistsBiologyCholinergic Agonists03 medical and health sciencesInternal medicineMuscarinicmedicineHumansRNA MessengerAgedBenzofuransCell ProliferationPharmacologyMyocytesPirenzepineEndocrinologyphysiologycytologyRNACarbacholanalogs /&/ derivatives/pharmacologymetabolismProto-Oncogene Proteins c-aktPharmacological research
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Antimuscarinic action of quinidine on the heart? A study in myocardial preparations from cat hearts

1984

Quinidine exerts anticholinergic effects which have been ascribed to atropine-like properties of the drug. We have examined the effects of acetylcholine on the force of contraction in isolated heart muscle preparations from cats and compared the inhibitory effects of atropine with those of quinidine. The effects of acetylcholine were antagonized competitively in the presence of atropine. The Schild-plot yielded a straight line; the slope was not significantly different from unity. In the presence of quinidine, the concentration-response curve of acetylcholine was shifted to the right as with atropine, however, the Schild-plot yielded a regression line which was not linear; the slope was sta…

AtropineMaleQuinidineInotropemedicine.medical_specialtymedicine.drug_classAction PotentialsIn Vitro TechniquesPharmacologyParasympatholyticInternal medicinemedicineAnticholinergicAnimalsPhosphodiesterase inhibitorPharmacologyPapaverineChemistryCell MembraneParasympatholyticsMyocardial ContractionQuinidineAcetylcholineElectrophysiologyAtropineEndocrinologyCatsFemaleAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Arginine vasopressin, via activation of post-junctional V1 receptors, induces contractile effects in mouse distal colon

2013

The aim of this study was to analyze whether arginine vasopressin (AVP) may be considered a modulator of intestinal motility. In this view, we evaluated, in vitro, the effects induced by exogenous administration of AVP on the contractility of mouse distal colon, the subtype(s) of receptor(s) activated and the action mechanism. Isometric recordings were performed on longitudinal and circular muscle strips of mouse distal colon. AVP (0.001 nM-100 nM) caused concentration-dependent contractile effects only on the longitudinal muscle, antagonized by the V1 receptor antagonist, V-1880. AVP-induced effect was not modified by tetrodotoxin, atropine and indomethacin. Contractile response to AVP was…

AtropineMaleReceptors Vasopressinmedicine.medical_specialtyVasopressinCarbacholNifedipineColonPhysiologyIndomethacinClinical BiochemistryMuscarinic AntagonistsTetrodotoxinCholinergic AgonistsIn Vitro TechniquesBiologyBiochemistryContractilityMiceCellular and Molecular Neurosciencechemistry.chemical_compoundPhosphoinositide Phospholipase CEndocrinologyInternal medicinemedicineAnimalsCyclooxygenase InhibitorsReceptorVasopressin receptorPhospholipase CArginine vasopressin receptor 1AMuscle SmoothCalcium Channel BlockersArginine vasopressinIntestinalcontractility V1 receptorsPhospholipase C Mouse colonArginine VasopressinEnzyme ActivationMice Inbred C57BLEndocrinologychemistryCarbacholGastrointestinal MotilityCyclopiazonic acidhormones hormone substitutes and hormone antagonistsMuscle ContractionSignal Transductionmedicine.drugRegulatory Peptides
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Muscarine receptor types mediating autoinhibition of acetylcholine release and sphincter contraction in the guinea-pig iris.

1990

The potencies of several muscarine receptor antagonists in blocking either the autoinhibition of acetylcholine release or the muscarinic contraction of the sphincter muscle upon acetylcholine release were investigated in the guinea-pig iris. The agonist at pre- or postjunctional muscarine receptors was acetylcholine released upon field stimulation (5.5 Hz, 2 min) of the irides preloaded with 14C-choline. The stimulation-evoked 14C-overflow was doubled in the presence of atropine 0.1 mumol/l but unaffected by the agonist (+/-)-methacholine (50 mumol/l). Thus, under the present stimulation conditions, the autoinhibition of acetylcholine release on the guinea-pig iris cholinergic nerves was ne…

AtropineMalemedicine.medical_specialtyGuinea PigsNeuromuscular JunctionIrisBiologyDiaminesIn Vitro Techniqueschemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineMethoctramineAnimalsMethacholine CompoundsMethacholine ChloridePharmacologyMuscarineGallamine TriethiodideGallamine triethiodideMuscle SmoothGeneral MedicinePirenzepinePirenzepineReceptors MuscarinicAcetylcholineElectric StimulationEndocrinologychemistryCholinergicFemalemedicine.symptomAcetylcholineMuscle contractionmedicine.drugMuscle ContractionNaunyn-Schmiedeberg's archives of pharmacology
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Safety and feasibility of atropine added in patients with sub-maximal heart rate during exercise myocardial perfusion SPECT.

2006

Failure to reach 80% of maximal predicted heart rate (HR) during exercise may render a myocardial perfusion single photon emission computed tomography (SPECT) study non-diagnostic for ischemia detection. We sought to investigate the injection of atropine in patients who fail to achieve 80% of age-predicted HR during exercise performed for myocardial perfusion SPECT (MPS), defining its safety and efficacy to raise HR to adequate levels as well as its effect on MPS interpretation.Between January 2002 and December 2004, we studied 3,150 consecutive patients (2,253 men and 897 women, mean age 55 +/- 6 years) who were referred to a single office-based nuclear cardiology laboratory for MPS using …

AtropineMalemedicine.medical_specialtyIschemiaMyocardial IschemiaBlood PressureSingle-photon emission computed tomographySeverity of Illness IndexMetabolic equivalentCholinergic AntagonistsOrganophosphorus CompoundsHeart RateInternal medicineSpect imagingCoronary CirculationHeart rateMedicineHumansRadiology Nuclear Medicine and imagingTomography Emission-Computed Single-Photonexercise testingmedicine.diagnostic_testmaximal predicted heart ratebusiness.industrymyocardial perfusion SPECTArrhythmias CardiacOrganotechnetium CompoundsMiddle Agedmedicine.diseaseAtropineRate pressure productResearch DesignCardiologyExercise TestFeasibility StudiesFemaleRadiopharmaceuticalsCardiology and Cardiovascular MedicinebusinessPerfusionmedicine.drugThe international journal of cardiovascular imaging
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Muscarinic inhibition of potassium-induced noradrenaline release and its dependence on the calcium concentration.

1975

1. Noradrenaline release from the isolated rabbit heart was evoked by perfusion with a medium containing 135 mM potassium and 17 mM sodium ions (high K+-low Na+). 2. The noradrenaline output in response to high K+-low Na+ was dose-dependently decreased by methacholine (0.625-320 muM) and this effect was reserved by atropine 1.44 mM. 3. Lowering the calcium concentration of high K+-low Na+ from 1.8-0.1125 mM decreased the noradrenaline output by 85%. The effect of methacholine, expressed as % inhibition of noradrenaline release, was potentiated by lowering of the calcium concentration. 4. Both at normal and lowered calcium concentrations the inhibitory action of methacholine was larger from …

AtropineMalemedicine.medical_specialtyReserpineTime FactorsSodiumPotassiumchemistry.chemical_elementAdrenergicCalciumchemistry.chemical_compoundNorepinephrineInternal medicineMuscarinic acetylcholine receptormedicinePressureAnimalsMethacholine CompoundsReceptors CholinergicPharmacologyCalcium metabolismMuscarineChemistryMyocardiumOsmolar ConcentrationSodiumGeneral MedicinePerfusionEndocrinologyPotassiumMethacholineCalciumFemaleRabbitsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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