Search results for "Chromosome"

showing 10 items of 1175 documents

Easy One-Step Amplification and Labeling Procedure for Copy Number Variation Detection.

2019

Abstract Background The specific characteristics of copy number variations (CNVs) require specific methods of detection and characterization. We developed the Easy One-Step Amplification and Labeling procedure for CNV detection (EOSAL-CNV), a new method based on proportional amplification and labeling of amplicons in 1 PCR. Methods We used tailed primers for specific amplification and a pair of labeling probes (only 1 labeled) for amplification and labeling of all amplicons in just 1 reaction. Products were loaded directly onto a capillary DNA sequencer for fragment sizing and quantification. Data obtained could be analyzed by Microsoft Excel spreadsheet or EOSAL-CNV analysis software. We d…

0301 basic medicineDNA Copy Number VariationsClinical BiochemistryComputational biologyPolymerase Chain Reaction03 medical and health sciences0302 clinical medicineHumansMultiplexMultiplex ligation-dependent probe amplificationCopy-number variationIn Situ Hybridization FluorescenceFluorescent DyesChemistryBiochemistry (medical)Sequence Analysis DNAAmpliconChromosome 17 (human)MSH6DNA sequencer030104 developmental biologyReceptors LDLMSH2030220 oncology & carcinogenesisDNA ProbesMultiplex Polymerase Chain ReactionClinical chemistry
researchProduct

Contribution of allelic imbalance to colorectal cancer

2018

Point mutations in cancer have been extensively studied but chromosomal gains and losses have been more challenging to interpret due to their unspecific nature. Here we examine high-resolution allelic imbalance (AI) landscape in 1699 colorectal cancers, 256 of which have been whole-genome sequenced (WGSed). The imbalances pinpoint 38 genes as plausible AI targets based on previous knowledge. Unbiased CRISPR-Cas9 knockout and activation screens identified in total 79 genes within AI peaks regulating cell growth. Genetic and functional data implicate loss of TP53 as a sufficient driver of AI. The WGS highlights an influence of copy number aberrations on the rate of detected somatic point muta…

0301 basic medicineDenmarkLoss of HeterozygosityGeneral Physics and AstronomyAllelic ImbalanceLoss of heterozygosityGenotypeddc:576.5RNA Small Interferinglcsh:ScienceRNA Small Interfering/geneticsGeneticsMultidisciplinaryQGenomicsPhenotype3. Good healthGENOMEPhenotypesyöpägeenitAllelic ImbalanceTumor Suppressor Protein p53/geneticsColorectal NeoplasmsChromosomes Human Pair 8GENESDNA Copy Number VariationsGenotypeScienceTranscription Factors/geneticsGenomicscolorectal cancerBiologyArticleGeneral Biochemistry Genetics and Molecular BiologyProto-Oncogene Proteins p21(ras)Proto-Oncogene Proteins p21(ras)/genetics03 medical and health sciencesmedicineHumansPoint MutationGenetic Predisposition to DiseaseGenepaksusuolisyöpäChromosome AberrationsWhole Genome SequencingHUMAN-COLONGene Expression ProfilingPoint mutationCancerGeneral Chemistrymedicine.diseaseColorectal Neoplasms/geneticsENHANCERS030104 developmental biologyCELLSlcsh:Q3111 BiomedicineTumor Suppressor Protein p53CRISPR-Cas SystemsmutaatiotTranscription FactorsMicrosatellite Repeats
researchProduct

Identification of novel drug resistance mechanisms by genomic and transcriptomic profiling of glioblastoma cells with mutation-activated EGFR.

2021

Abstract Aims Epidermal growth factor receptor (EGFR) is not only involved in carcinogenesis, but also in chemoresistance. We characterized U87.MGΔEGFR glioblastoma cells with constitutively active EGFR due to deletion at the ligand binding domain in terms of gene expression profiling and chromosomal aberrations. Wild-type U87.MG cells served as control. Materials and methods RNA sequencing and network analyses (Ingenuity Pathway Analysis) were performed to identify novel drug resistance mechanisms related to expression of mutation activated EGFR. Chromosomal aberrations were characterized by multicolor fluorescence in situ hybridization (mFISH) and array comparative genomic hybridization (…

0301 basic medicineDown-RegulationBiologymedicine.disease_cause030226 pharmacology & pharmacyGeneral Biochemistry Genetics and Molecular BiologyTranscriptome03 medical and health sciences0302 clinical medicineCell Line TumormedicineHumansGene Regulatory NetworksProtein Interaction MapsGeneral Pharmacology Toxicology and PharmaceuticsGeneTranscription factorMetaphaseChromosome AberrationsMutationmedicine.diagnostic_testBrain NeoplasmsGene Expression ProfilingGeneral MedicineGenomicsUp-RegulationGene expression profilingErbB ReceptorsGene Expression Regulation Neoplastic030104 developmental biologyDrug Resistance NeoplasmMutationCancer researchCarcinogenesisGlioblastomaTranscriptomeComparative genomic hybridizationFluorescence in situ hybridizationSignal TransductionLife sciences
researchProduct

Partial resistance to homologous challenge infections of the digenean Echinostoma caproni in ICR mice

2015

AbstractIn the present paper, we analyse the effect of a primary infection of ICR mice with Echinostoma caproni (Trematoda: Echinostomatidae) on the generation of resistance against homologous challenge infections. In ICR mice, E. caproni induces chronic infections concomitantly with strong responses characterized by the development of T-helper 1 (Th1)-type local immune responses with elevated levels of local interferon-gamma (IFN-γ) and inflammatory and antibody responses. Here, the effect of the response generated against a primary infection with E. caproni in the generation of resistance against subsequent homologous infections was analysed. For this purpose, ICR mice were challenged wit…

0301 basic medicineEchinostoma caproniAntibodies HelminthMicrobiologyInterferon-gamma03 medical and health sciencesImmune systemEchinostomaHomologous chromosomeAnimalsParasite hostingDisease ResistanceInflammationEchinostomiasisMice Inbred ICRbiologyGeneral MedicineTh1 Cellsbiology.organism_classificationDisease Models Animal030104 developmental biologyAntibody responseAnimal Science and ZoologyParasitologyTrematodaIcr miceJournal of Helminthology
researchProduct

Genome wide DNA methylation profiling identifies specific epigenetic features in high-risk cutaneous squamous cell carcinoma

2019

ABSTRACTCutaneous squamous cell carcinoma (cSCC) is the second most common skin cancer. Although most cSCCs have good prognosis, a subgroup of high-risk cSCC has a higher frequency of recurrence and mortality. Therefore, the identification of molecular risk factors associated with this aggressive subtype is of major interest. In this work we carried out a global-scale approach to investigate the DNA-methylation profile in patients at different stages, from premalignant actinic keratosis to low-risk invasive and high-risk non-metastatic and metastatic cSCC. The results showed massive non-sequential changes in DNA-methylome and identified a minimal methylation signature that discriminates bet…

0301 basic medicineEpigenomicsMaleSkin NeoplasmsDiseaseBiochemistryActinic KeratosisGenomeEpigenesis Genetic0302 clinical medicineRisk FactorsMedicine and Health SciencesSkin TumorsAged 80 and overMultidisciplinaryDNA methylationQRSquamous Cell CarcinomasMethylationMiddle AgedPrognosisChromatinNucleic acidsGene Expression Regulation NeoplasticKeratosis ActinicOncology030220 oncology & carcinogenesisDNA methylationCarcinoma Squamous CellDisease ProgressionMedicineEpigeneticsFemaleDNA modificationChromatin modificationResearch ArticleChromosome biologyCell biologyCutaneous squamous cell carcinomaKeratosisScienceDermatologyBiologyCarcinomas03 medical and health sciencesDiagnostic MedicineCarcinomaGeneticsCancer Detection and DiagnosismedicineHumansEpigeneticsAgedNeoplasm StagingTreatment GuidelinesHealth Care PolicyBiology and life sciencesActinic keratosisCancers and NeoplasmsDNAmedicine.diseaseDNA FingerprintingDna methylation profilingHealth Care030104 developmental biologyCancer researchGene expressionNeoplasm Recurrence LocalSkin cancerGenome-Wide Association Study
researchProduct

Adaptation of gene loci to heterochromatin in the course of Drosophila evolution is associated with insulator proteins.

2020

AbstractPericentromeric heterochromatin is generally composed of repetitive DNA forming a transcriptionally repressive environment. Dozens of genes were embedded into pericentromeric heterochromatin during evolution of Drosophilidae lineage while retaining activity. However, factors that contribute to insusceptibility of gene loci to transcriptional silencing remain unknown. Here, we find that the promoter region of genes that can be embedded in both euchromatin and heterochromatin exhibits a conserved structure throughout the Drosophila phylogeny and carries motifs for binding of certain chromatin remodeling factors, including insulator proteins. Using ChIP-seq data, we demonstrate that ev…

0301 basic medicineEuchromatinHeterochromatinEvolutionMolecular biologyAdaptation Biologicallcsh:MedicineInsulator (genetics)Chromatin remodelingArticleEvolutionary geneticsEvolution Molecular03 medical and health sciences0302 clinical medicineDrosophilidaeHeterochromatinAnimalsDrosophila ProteinsNucleotide Motifslcsh:ScienceEye ProteinsPromoter Regions GeneticGenePericentric heterochromatinPhylogenyGeneticsMultidisciplinarygeenitBinding Sitesbiologylcsh:RfungiChromosome MappingPromoterDNAbiology.organism_classificationChromatinDNA-Binding Proteins030104 developmental biologyGene Expression RegulationGenetic LociChromatin Immunoprecipitation SequencingMolecular evolutionlcsh:QDrosophilaTranscription Initiation SiteTranscription030217 neurology & neurosurgeryProtein BindingScientific reports
researchProduct

A TRAPPC6B splicing variant associates to restless legs syndrome

2016

Abstract INTRODUCTION: RLS is a common movement disorders with a strong genetic component in its pathophysiology, but, up to now, no causative mutation has been reported. METHODS: We re-evaluated the previously described RLS2 family by exome sequencing. RESULTS: We identified fifteen variations in the 14q critical region. The c.485G > A transition of the TRAPPC6B gene segregates with the RLS2 haplotype, is absent in 200 local controls and is extremely rare in 12988 exomes from the Exome Variant Server (EVS). This variant alters a splicing site and hampers the normal transcript processing by promoting exon 3-skipping as demonstrated by minigene transfection and by patient transcripts. CON…

0301 basic medicineExome sequencingMaleVesicular Transport ProteinsLocus (genetics)VariationGene mutationBiologySplicingTransfection03 medical and health sciencesExonGene FrequencyRLSRestless Legs SyndromeAnimalsHumansGenetic Predisposition to DiseaseRNA MessengerRestless legs syndromeExomeExome sequencingMovement disorderCells CulturedGeneticsChromosomes Human Pair 14Family HealthSleep disorderHaplotypeExonsRats030104 developmental biologyAuthors report no disclosureNeurologyHaplotypesRNA splicingMutationFemaleNeurology (clinical)Geriatrics and GerontologyNeurological diseaseMinigene
researchProduct

Genome-wide association study between CNVs and milk production traits in Valle del Belice sheep.

2019

Copy number variation (CNV) is a major source of genomic structural variation. The aim of this study was to detect genomic CNV regions (CNVR) in Valle del Belice dairy sheep population and to identify those affecting milk production traits. The GO analysis identified possible candidate genes and pathways related to the selected traits. We identified CNVs in 416 individuals genotyped using the Illumina OvineSNP50 BeadChip array. The CNV association using a correlation-trend test model was examined with the Golden Helix SVS 8.7.0 tool. Significant CNVs were detected when their adjusted p-value was <0.01 after false discovery rate (FDR) correction. We identified 7,208 CNVs, which gave 365 C…

0301 basic medicineFalse discovery rateCandidate geneDNA Copy Number VariationsGenotypeSciencePopulationGenomic Structural VariationQuantitative Trait LociGenome-wide association studyQuantitative trait locusBiology03 medical and health sciencesSettore AGR/17 - Zootecnica Generale E Miglioramento GeneticoAnimalsLactationCopy-number variationeducationGeneGeneticseducation.field_of_studyMultidisciplinarySheepBiochemistry Genetics and Molecular Biology (all)QAnimals chromosome mapping dairying female genome-Wide association study genotype lactation sheep DNA copy number variations quantitative trait loci0402 animal and dairy scienceRChromosome Mapping04 agricultural and veterinary sciences040201 dairy & animal scienceDairying030104 developmental biologyAgricultural and Biological Sciences (all)MedicineFemaleGenome-Wide Association StudyResearch ArticlePLoS ONE
researchProduct

"Islands of divergence" in the Atlantic cod genome represent polymorphic chromosomal rearrangements

2016

- In several species genetic differentiation across environmental gradients or between geographically separate populations has been reported to center at “genomic islands of divergence,” resulting in heterogeneous differentiation patterns across genomes. Here, genomic regions of elevated divergence were observed on three chromosomes of the highly mobile fish Atlantic cod (Gadus morhua) within geographically fine-scaled coastal areas. The “genomic islands” extended at least 5, 9.5, and 13 megabases on linkage groups 2, 7, and 12, respectively, and coincided with large blocks of linkage disequilibrium. For each of these three chromosomes, pairs of segregating, highly divergent alleles were id…

0301 basic medicineGene FlowLinkage disequilibriumpopulation genomicsGenomePolymorphism Single NucleotideChromosomesLinkage DisequilibriumDivergenceGene flowPopulation genomics03 medical and health sciencesecological adaptationVDP::Genetikk og genomikk: 474VDP::Genetics and genomics: 474GeneticsGadusAnimalsAllele:Genetikk og genomikk: 474 [VDP]Ecology Evolution Behavior and Systematicschromosomal rearrangementsChromosomal inversionGeneticsmarine organismsGenomebiologystructural polymorphismsbiology.organism_classificationAdaptation Physiological030104 developmental biologyGadus morhuaChromosome InversionMetagenomics:Genetics and genomics: 474 [VDP]Research Article
researchProduct

Topical Review: The Neuronal Ceroid-Lipofuscinoses

1995

The neuronal ceroid-lipofuscinoses, a group of progressive neurodegenerative diseases in children and in adults, have now been recognized for some 90 years, and the childhood forms represent one of the largest groups of progressive neurodegenerative diseases in children. Apart from a core group of major clinical forms—the infantile, the late-infantile, the juvenile, and the adult forms—numerous atypical patients afflicted with neuronal ceroid-lipofuscinosis have now been identified, constituting 10% to 20% of all patients with neuronal ceroid-lipofuscinosis. These "atypical" patients have, over the past 10 years, prompted the suggestion of 15 atypical variants or minor syndromes, many of th…

0301 basic medicineGeneticsPathologymedicine.medical_specialtyATP synthasebiologyProtein subunitChromosomeSphingolipid Activator ProteinsPathogenesis03 medical and health sciences030104 developmental biology0302 clinical medicineCLN3Pediatrics Perinatology and Child Healthbiology.proteinmedicineNeurology (clinical)AlleleGene030217 neurology & neurosurgeryJournal of Child Neurology
researchProduct