Search results for "Clamp"

showing 10 items of 243 documents

Sensitivity of neuronal nicotinic acetylcholine receptors to the opiate antagonists naltrexone and naloxone: receptor blockade and up-regulation

2003

In HEK293 cells stably expressing alpha4beta2 nAChRs, naltrexone, but not naloxone, blocked alpha4beta2 nAChRs via an open-channel blocking mechanism. In primary hippocampal cultures, naltrexone inhibited alpha7 nAChRs up-regulated by nicotine, and in organotypic hippocampal cultures naltrexone caused a time-dependent up-regulation of functional alpha7 nAChRs that was detected after removal of the drug. These results indicate that naltrexone could be used as a smoking cessation aid.

NicotinePatch-Clamp TechniquesTime FactorsNarcotic AntagonistsClinical BiochemistryGene ExpressionPharmaceutical Science(+)-NaloxoneReceptors NicotinicPharmacologyHippocampal formationSensitivity and Specificitycomplex mixturesBiochemistryNaltrexoneCell LineNicotineStructure-Activity Relationshipmental disordersDrug DiscoverymedicineHumansMolecular BiologyAcetylcholine receptorNeuronsNaloxoneChemistryNarcotic antagonistmusculoskeletal neural and ocular physiologyOrganic ChemistryNaltrexoneUp-RegulationNicotinic agonistnervous systemMechanism of actionMolecular MedicineSmoking Cessationsense organsmedicine.symptommedicine.drugBioorganic & Medicinal Chemistry Letters
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Priekšlaicīgu dzemdību izraisītu grūtniecības komplikāciju izvērtējums.

2017

Cēlonis: Grūtniecības komplikācijas ir galvenie iemesli mātes un augļa mortality.Pregnancy komplikāciju mainās dažādās populāciju, BMI un vecuma grupā. Saskaņā ar Global Health observatoriju, kuri katru dienu 2015. gadā, aptuveni 830 sievietes nomira dēļ komplikācijas grūtniecības un bērna piedzimšanas. Tāpēc ir ļoti svarīgi, lai analizētu grūtniecības komplikācijas gan priekšlaicīgas dzemdības un normālu termiņa piegāde (vadības grupa). Mērķi: Pētījuma mērķis ir analizēt dažas grūtniecības komplikācijas un salīdzināt tos no priekšlaicīgas dzemdības un kontroles grupu, un salīdzināt tos ar vecuma grupu un ĶMI grupu. Materiāli un metodes: Pētījums modelis ietver datus, kas savākti no Rig…

Normal term deliveryPregnancy complicationPreterm deliveryPlacental ProblemsPreeclampsiaMedicīna
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Perforated Patch-clamp Recording of Mouse Olfactory Sensory Neurons in Intact Neuroepithelium: Functional Analysis of Neurons Expressing an Identifie…

2015

Analyzing the physiological responses of olfactory sensory neurons (OSN) when stimulated with specific ligands is critical to understand the basis of olfactory-driven behaviors and their modulation. These coding properties depend heavily on the initial interaction between odor molecules and the olfactory receptor (OR) expressed in the OSNs. The identity, specificity and ligand spectrum of the expressed OR are critical. The probability to find the ligand of the OR expressed in an OSN chosen randomly within the epithelium is very low. To address this challenge, this protocol uses genetically tagged mice expressing the fluorescent protein GFP under the control of the promoter of defined ORs. O…

Olfactory systemPatch-Clamp TechniquesGeneral Chemical EngineeringGene ExpressionSensory systemMice TransgenicBiology[ SDV.BA ] Life Sciences [q-bio]/Animal biologyReceptors OdorantGeneral Biochemistry Genetics and Molecular BiologyOlfactory Receptor Neurons03 medical and health sciencesOlfactory mucosaMice0302 clinical medicineOlfactory Mucosaolfactory sensory neuronsGenes ReportermedicineAnimalsPatch clampissue 101030304 developmental biology0303 health sciencesOlfactory receptorGeneral Immunology and MicrobiologyGeneral Neuroscience[SDV.BA]Life Sciences [q-bio]/Animal biologygene-targeted mouserespiratory systemelectrophysiologytransductionperforated patch-clampEpitheliumSmellmedicine.anatomical_structure[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Odorants[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]sense organspharmacologyNeuroscienceTransduction (physiology)Olfactory epithelium030217 neurology & neurosurgeryNeuroscienceJournal of visualized experiments : JoVE
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Postnatal odorant exposure induces peripheral olfactory plasticity at the cellular level

2014

Mammalian olfactory sensory neurons (OSNs) form the primary elements of the olfactory system. Inserted in the olfactory mucosa lining of the nasal cavity, they are exposed to the environment and their lifespan is brief. Several reports say that OSNs are regularly regenerated during the entire life and that odorant environment affects the olfactory epithelium. However, little is known about the impact of the odorant environment on OSNs at the cellular level and more precisely in the context of early postnatal olfactory exposure. Here we exposed MOR23-green fluorescent protein (GFP) and M71-GFP mice to lyral or acetophenone, ligands for MOR23 or M71, respectively. Daily postnatal exposure to …

Olfactory systemPatch-Clamp TechniquessourisReceptors Odorant[ SDV.BA ] Life Sciences [q-bio]/Animal biologybiologie neurosensorielleMembrane Potentials0302 clinical medicinemolecular biology[SDV.BDD]Life Sciences [q-bio]/Development BiologydéveloppementAnimal biology0303 health scienceseducation.field_of_studyNeuronal PlasticityGeneral Neuroscience[SDV.BA]Life Sciences [q-bio]/Animal biologyBiologie du développementArticlesOlfactory BulbDevelopment BiologySmellmedicine.anatomical_structureélectrophysiologie[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]development;développement;electrophysiology;électrophysiologie;mice;souris;molecular biology;biologie moléculaire;olfaction;plasticity;plasticiténeurone récepteur olfactifolfactionmiceGreen Fluorescent ProteinsPopulationMice Transgenicneurone olfactifSensory systemOlfactionBiologybiologie moléculaireOlfactory Receptor Neurons03 medical and health sciencesOlfactory mucosaBiologie animalemedicineAnimalsOlfactory Transduction Pathway[ SDV.BDD ] Life Sciences [q-bio]/Development Biologyeducationdevelopment030304 developmental biologyOlfactory receptorplasticitéNeuroscienceselectrophysiologyElectrooculographyAnimals NewbornGene Expression RegulationNeurons and Cognitionplasticity[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]OdorantsNeuroscienceOlfactory epithelium030217 neurology & neurosurgery
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Polycyclic Aromatic Hydrocarbons Phenanthrene and Retene Modify the Action Potential via Multiple Ion Currents in Rainbow Trout Oncorhynchus mykiss C…

2019

Polycyclic aromatic hydrocarbons (PAHs) are ubiquitous contaminants in aqueous environments. They affect cardiovascular development and function in fishes. The 3-ring PAH phenanthrene has recently been shown to impair cardiac excitation-contraction coupling by inhibiting Ca2+ and K+ currents in marine warm-water scombrid fishes. To see if similar events take place in a boreal freshwater fish, we studied whether the PAHs phenanthrene and retene (an alkylated phenanthrene) modify the action potential (AP) via effects on Na+ (INa ), Ca2+ (ICaL ), or K+ (IKr , IK1 ) currents in the ventricular myocytes of the rainbow trout (Oncorhynchus mykiss) heart. Electrophysiological characteristics of myo…

PAH-yhdisteetHealth Toxicology and MutagenesiscardiotoxicityAction Potentialstoksikologia010501 environmental sciences01 natural sciences03 medical and health scienceschemistry.chemical_compoundmode of actionEnvironmental ChemistryMyocyteAnimalsaquatic toxicologyMyocytes CardiacPatch clampPolycyclic Aromatic HydrocarbonsMode of actionIon channelkalat030304 developmental biology0105 earth and related environmental sciencesvesistöt0303 health sciencesRetenebiologyPhenanthrenePhenanthrenesAryl hydrocarbon receptorhiilivedytchemistryReceptors Aryl Hydrocarbonpolycyclic aromatic hydrocarbons (PAHs)Oncorhynchus mykissbiology.proteinBiophysicsRainbow troutWater Pollutants ChemicalEnvironmental toxicology and chemistry
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Capsaicin differentially modulates voltage-activated calcium channel currents in dorsal root ganglion neurones of rats

2005

It is discussed whether capsaicin, an agonist of the pain mediating TRPV1 receptor, decreases or increases voltage-activated calcium channel (VACC) currents (ICa(V)). ICa(V) were isolated in cultured dorsal root ganglion (DRG) neurones of rats using the whole cell patch clamp method and Ba 2+ as charge carrier. In large diameter neurones (>35Am), a concentration of 50AM was needed to reduce ICa(V) (activated by depolarizations to 0 mV) by 80%, while in small diameter neurones (30Am), the IC50 was 0.36 AM. This effect was concentration dependent with a threshold below 0.025 AM and maximal blockade (>80%) at 5AM. The current–voltage relation was shifted to the hyperpolarized direction with an…

Pain ThresholdPatch-Clamp TechniquesTRPV1TRPV Cation ChannelsN-type calcium channelSecond Messenger SystemsMembrane PotentialsGanglia SpinalAnimalsL-type calcium channelPatch clampRats WistarMolecular BiologyCell SizeNeuronsDose-Response Relationship DrugVoltage-dependent calcium channelChemistryGeneral NeuroscienceCalcium channelT-type calcium channelCalciseptineAnatomyRatsRats Inbred LewBiophysicsCalcium ChannelsNeurology (clinical)CapsaicinSignal TransductionDevelopmental BiologyBrain Research
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Alterations in membrane and firing properties of layer 2/3 pyramidal neurons following focal laser lesions in rat visual cortex.

2013

Focal cortical injuries are well known to cause changes in function and excitability of the surviving cortical areas but the cellular correlates of these physiological alterations are not fully understood. In the present study we employed a well established ex vivo-in vitro model of focal laser lesions in the rat visual cortex and we studied membrane and firing properties of the surviving layer 2/3 pyramidal neurons. Patch-clamp recordings, performed in the first week post-injury, revealed an increased input resistance, a depolarized spike threshold as well as alterations in the firing pattern of neurons in the cortex ipsilateral to the lesion. Notably, the reported lesion-induced alteratio…

Patch-Clamp TechniquesAction PotentialsGABAB receptorBiologyIn Vitro TechniquesSynaptic TransmissionGlutamatergicchemistry.chemical_compoundCortex (anatomy)Biological neural networkmedicineDNQXAnimalsRats Long-EvansVisual CortexMembrane potentialNeuronsGABAA receptorGeneral NeuroscienceLasersPyramidal CellsCell MembraneElectrophysiological PhenomenaRatsVisual cortexmedicine.anatomical_structurechemistryData Interpretation StatisticalSynapsesNeuroscienceNeuroscience
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Modelling the spatial and temporal constrains of the GABAergic influence on neuronal excitability

2021

GABA (γ-amino butyric acid) is an inhibitory neurotransmitter in the adult brain that can mediate depolarizing responses during development or after neuropathological insults. Under which conditions GABAergic membrane depolarizations are sufficient to impose excitatory effects is hard to predict, as shunting inhibition and GABAergic effects on spatiotemporal filtering of excitatory inputs must be considered. To evaluate at which reversal potential a net excitatory effect was imposed by GABA (EGABAThr), we performed a detailed in-silico study using simple neuronal topologies and distinct spatiotemporal relations between GABAergic and glutamatergic inputs. These simulations revealed for GABAe…

Patch-Clamp TechniquesAction potentialPhysiologyAction PotentialsSynaptic TransmissionNervous SystemBiochemistryMiceNerve FibersAnimal CellsMedicine and Health SciencesGABAergic NeuronsBiology (General)gamma-Aminobutyric AcidNeuronsMembrane potentialEcologyChemistryPyramidal CellsDepolarizationNeurochemistryNeurotransmittersCA3 Region HippocampalElectrophysiologyReceptors GlutamateComputational Theory and MathematicsModeling and SimulationExcitatory postsynaptic potentialGABAergicAnatomyCellular TypesShunting inhibitionResearch Articlemedicine.drugQH301-705.5Models NeurologicalNeurophysiologyAMPA receptorMembrane Potentialgamma-Aminobutyric acidCellular and Molecular NeuroscienceGlutamatergicSpatio-Temporal AnalysisGeneticsmedicineAnimalsComputer SimulationReceptors AMPAReversal potentialMolecular BiologyEcology Evolution Behavior and SystematicsComputational BiologyBiology and Life SciencesNeural InhibitionDendritesCell BiologyNeuronal DendritesAxonsMice Inbred C57BLAnimals Newbornnervous systemCellular NeuroscienceSynapsesDepolarizationNeuroscienceNeurosciencePLOS Computational Biology
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GABA-A Receptors Regulate Neocortical Neuronal Migration In Vitro and In Vivo

2006

The cortical migration process depends on a number of trophic factors and on the activation of different voltage- and ligand-gated channels. We investigated the role of gamma-aminobutyric acid (GABA) type A receptors in the neuronal migration process of the newborn rat parietal cortex in vivo and in vitro. Local in vivo application of the GABA-A antagonist bicuculline methiodide (BMI) or the agonist muscimol via cortical surface Elvax implants induced prominent alterations in the cortical architecture when compared with untreated or sham-operated controls. BMI- and muscimol-treated animals revealed heterotopic cell clusters in the upper layers and a complete loss of the cortical lamination …

Patch-Clamp TechniquesAntimetabolitesCognitive NeuroscienceNeocortexIn Vitro TechniquesBiologyBicucullineReceptors N-Methyl-D-Aspartategamma-Aminobutyric acidGABA AntagonistsCellular and Molecular Neurosciencechemistry.chemical_compoundCell MovementPostsynaptic potentialGlial Fibrillary Acidic ProteinmedicineAnimalsRats WistarReceptorGABA AgonistsDrug ImplantsNeuronsMuscimolGABAA receptorBicucullineReceptors GABA-AImmunohistochemistryRatsCell biologyElectrophysiologyKineticsmedicine.anatomical_structureAnimals NewbornBromodeoxyuridinenervous systemMuscimolchemistryCerebral cortexGABAergicCalciumNeurosciencemedicine.drugCerebral Cortex
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Postsynaptic NO/cGMP Increases NMDA Receptor Currents via Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels in the Hippocampus

2013

The nitric oxide (NO)/cyclic guanosine monophosphate (cGMP) signaling cascade participates in the modulation of synaptic transmission. The effects of NO are mediated by the NO-sensitive cGMP-forming guanylyl cyclases (NO-GCs), which exist in 2 isoforms with indistinguishable regulatory properties. The lack of long-term potentiation (LTP) in knock-out (KO) mice deficient in either one of the NO-GC isoforms indicates the contribution of both NO-GCs to LTP. Recently, we showed that the NO-GC1 isoform is located presynaptically in glutamatergic neurons and increases the glutamate release via hyperpolarization-activated cyclic nucleotide (HCN)-gated channels in the hippocampus. Electrophysiologi…

Patch-Clamp TechniquesCognitive NeuroscienceLong-Term PotentiationIn Vitro TechniquesNeurotransmissionNitric OxideReceptors N-Methyl-D-AspartateMiceCellular and Molecular Neurosciencechemistry.chemical_compoundCyclic nucleotidePostsynaptic potentialHyperpolarization-Activated Cyclic Nucleotide-Gated ChannelsHCN channelAnimalsAnesthetics LocalCA1 Region HippocampalCyclic GMPCyclic guanosine monophosphateMice KnockoutNeuronsbiologyLidocaineTetraethylammoniumLong-term potentiationHyperpolarization (biology)Electric StimulationPyrimidinesAnimals Newbornnervous systemchemistryGuanylate CyclaseBiophysicsbiology.proteinNMDA receptorExcitatory Amino Acid AntagonistsNeuroscienceCerebral Cortex
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