Search results for "Cognition disorders"

showing 10 items of 277 documents

Gender-related effect of clinical and genetic variables on the cognitive impairment in multiple sclerosis

2004

BACKGROUND: Cognitive impairment may occur at any time during the course of multiple sclerosis (MS), and it is often a major cause of disability in patients with the disease. The APOE-epsilon4 allele is the major known genetic risk factor for late onset familial and sporadic Alzheimer's Disease (AD), and it seems to be implicated in cognitive decline in normal elderly persons. OBJECTIVE: To investigate the clinical and genetic variables that can be associated with the cognitive decline in patients with MS. METHODS: Five-hundred and three patients with clinically definite MS underwent a battery of neuropsychological tests and, according to the number of failed tests, were divided into cognit…

Apolipoprotein EAdultMalemedicine.medical_specialtyPediatricsNeurologyMultiple SclerosisMessengerLate onsetDiseaseNeuropsychological TestsApolipoproteins EmedicineOdds RatioHumansRNA MessengerCognitive declineAllelePsychiatrycognitive impairmentAPOE; Cognitive impairment; Multiple sclerosisAnalysis of VarianceSex CharacteristicsChi-Square DistributionReverse Transcriptase Polymerase Chain ReactionMultiple sclerosisCognitive disorderGenetic VariationMiddle Agedmedicine.diseasemultiple sclerosis cognitive impairment gender geneticNeurologyGenetic Variation; Odds Ratio; Analysis of Variance; Sex Characteristics; Chi-Square Distribution; Humans; Apolipoproteins E; Reverse Transcriptase Polymerase Chain Reaction; Cognition Disorders; RNA Messenger; Multiple Sclerosis; Adult; Middle Aged; Neuropsychological Tests; Female; MaleRNAFemaleSettore MED/26 - NeurologiaNeurology (clinical)Psychologymultiple sclerosis · cognitive impairment · APOECognition DisordersAPOE
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The potential use of biomarkers as an adjunctive tool for staging bipolar disorder

2009

Recent data show that biomarkers differ in early and late-stage bipolar disorder (BD). Here we propose a model of staging for bipolar disorder that emphasizes the potential use of biomarkers for differentiating early and late-stage BD patients in the inter-episodic period. The proposed model includes a Latent phase: patients at "ultra-high-risk" for developing BD, characterized by a family history of BD, temperament traits, mood, and anxiety symptoms as well as genetic vulnerability for developing the disorder; Stage I: patients who return to their baseline level of functioning when mood episodes resolve; Stage II: biomarkers and functioning impairment are related to comorbidities or rapid-…

Bipolar Disordermedia_common.quotation_subjectAnxietyModels BiologicalRisk FactorsmedicineHumansNerve Growth FactorsBipolar disorderFamily historyTemperamentBiological Psychiatrymedia_commonPharmacologyCognitive disorderCognitionmedicine.diseaseAffectMoodDisease ProgressionCytokinesAnxietyTemperamentmedicine.symptomCognition DisordersPsychologyManiaBiomarkersClinical psychologyProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Predictors of outcome in acute ischemic cerebrovascular syndromes: The GIFA study

2006

Abstract Background Today it may be more useful to use the term acute ischemic cerebrovascular syndrome (AICS) to define a spectrum of disease ranging from TIA to stroke and that share a similar underlying pathophysiology: cerebral ischemia . The aim of this study is to evaluate the prognostic importance of some demographic, laboratory and clinical variables on the outcome in hospitalized patients with a discharge diagnosis suggestive of acute ischemic cerebral syndrome (AICS). Methods 17,377 Subjects were enrolled in the GIFA study, a multicenter survey of hospitalized older patients. 1878 Subjects with a main discharge diagnosis suggestive of acute ischemic cerebrovascular syndrome (AICS)…

Blood GlucoseMalemedicine.medical_specialtyActivities of daily livingMultivariate analysisIschemiaDiseaseComorbidityBrain IschemiaCentral nervous system diseaseDisability EvaluationLeukocyte CountInternal medicineActivities of Daily LivingOutcome Assessment Health CaremedicineHumansHospital Mortalitystroke fetuin A cytokinesStrokeAgedbusiness.industryVascular diseaseAge Factorsmedicine.diseasePrognosisComorbidityHospitalizationAcute DiseaseMultivariate AnalysisPhysical therapyFemaleCardiology and Cardiovascular MedicinebusinessCognition Disorders
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The COGNITION Study Rationale and Design: Influence of Closed Loop Stimulation on Cognitive Performance in Pacemaker Patients

2008

Background: Several studies showed the beneficial effect of pacemaker implantation on cognitive performance in patients with bradycardia. But it has never been investigated if patients with chronotropic incompetence may improve their cognitive performance if treated by a rate-adaptive system reacting to mental stress in comparison to the most frequently used accelerometer-driven pacing. Methods: The randomized, single-blind, multicenter COGNITION study evaluates if closed loop stimulation (CLS) offers incremental benefit in the speed of cognitive performance and the overall well-being of elderly patients with bradycardia compared with accelerometer-based pacing. Four hundred chronotropicall…

BradycardiaPacemaker Artificialmedicine.medical_specialtybusiness.industryChronotropic incompetenceBiofeedback PsychologyAtrial fibrillationCognitionGeneral Medicinemedicine.diseaseClosed loop stimulationQuality of lifeGermanyAtrial FibrillationPhysical therapyHumansMedicineEffects of sleep deprivation on cognitive performancemedicine.symptomCognition DisordersCardiology and Cardiovascular MedicinebusinessAdverse effectRandomized Controlled Trials as TopicPacing and Clinical Electrophysiology
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Can Alzheimer disease be a form of type 3 diabetes?

2012

Alzheimer disease (AD) and metabolic syndrome are two highly prevalent pathological conditions of Western society due to incorrect diet, lifestyle, and vascular risk factors. Recent data have suggested metabolic syndrome as an independent risk factor for AD and pre-AD syndrome. Furthermore, biological plausibility for this relationship has been framed within the “metabolic cognitive syndrome” concept. Due to the increasing aging of populations, prevalence of AD in Western industrialized countries will rise in the near future. Thus, new knowledge in the area of molecular biology and epigenetics will probably help to make an early molecular diagnosis of dementia. An association between metabo…

Central Nervous SystemAgingmedicine.medical_specialtySingle-nucleotide polymorphismType 2 diabetesBiologyBioinformaticsPolymorphism Single NucleotideSHIP2 ADAlzheimer DiseaseRisk FactorsDiabetes mellitusInternal medicinemedicineDiabetes MellitusDementiaHumansInsulinEpigeneticsRisk factorLife StyleAgedSettore MED/04 - Patologia GeneraleMetabolic SyndromeInositol Polyphosphate 5-PhosphatasesSyndromeModels Theoreticalmedicine.diseasePhosphoric Monoester HydrolasesEndocrinologySettore MED/26 - NeurologiaGeriatrics and GerontologyAlzheimer's diseaseMetabolic syndromeCognition DisordersSignal TransductionRejuvenation research
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Interplay between glutamatergic and GABAergic neurotransmission alterations in cognitive and motor impairment in minimal hepatic encephalopathy.

2015

The cognitive and motor alterations in hepatic encephalopathy (HE) are the final result of altered neurotransmission and communication between neurons in neuronal networks and circuits. Different neurotransmitter systems cooperate to modulate cognitive and motor function, with a main role for glutamatergic and GABAergic neurotransmission in different brain areas and neuronal circuits. There is an interplay between glutamatergic and GABAergic neurotransmission alterations in cognitive and motor impairment in HE. This interplay may occur: (a) in different brain areas involved in specific neuronal circuits; (b) in the same brain area through cross-modulation of glutamatergic and GABAergic neur…

CerebellumMinimal hepatic encephalopathyGlutamic AcidNeurotransmissionSynaptic TransmissionCellular and Molecular NeuroscienceGlutamatergicmedicineHyperammonemiaAnimalsHumansGlutamatergic neurotransmissionGABAergic neurotransmissionHepatic encephalopathygamma-Aminobutyric AcidHepatic encephalopathyGABAA receptorbusiness.industryGlutamate receptorHyperammonemiaCognitionCell Biologymedicine.diseaseMotor Skills Disordersmedicine.anatomical_structureHepatic EncephalopathyCognitive functionCognition DisordersbusinessNeuroscience
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Cortical astrocytosis in juvenile rhesus monkeys infected with simian immunodeficiency virus

1993

The pattern of expression of GFAP immunoreactivity in astrocytes of the juvenile rhesus monkey cortex was examined following infection with simian immunodeficiency virus (SIV). Blocks of cerebral cortex plus subjacent white matter from saline- and formalin-perfused brain were examined by peroxidase-linked immunochemical and immunofluorescence staining of deparaffinized sections. Strong GFAP immunoreactivity was found in astrocytic cells in both uninfected and SIV-infected juvenile macaque in the subpial cerebral cortex and in subcortical white matter, where GFAP-positive cells were abundant. GFAP staining of cortical layers 2-6 on the other hand was weak or absent in three uninfected contro…

Cerebral CortexbiologyGeneral NeuroscienceSimian Acquired Immunodeficiency SyndromeFluorescent Antibody TechniqueSimian immunodeficiency virusGrey mattermedicine.disease_causeImmunohistochemistryMacaca mulattaMacaqueVirologyWhite mattermedicine.anatomical_structureCerebral cortexAstrocytesbiology.animalGlial Fibrillary Acidic ProteinmedicineAnimalsPrimateAstrocytosisCognition DisordersAstrocyteNeuroReport
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Cognitive impairment in relapsing-remitting multiple sclerosis can be predicted by imaging performed several years earlier.

2007

Cognitive deficits in multiple sclerosis (MS) are common and correlate with contemporary MRI brain abnormalities, particularly atrophy, but the ability of imaging early in the disease to predict later cognitive impairment remains to be determined. Thirty relapsing—remitting MS patients recruited within three years of the onset of the disease, and in whom MRI had been performed at baseline and a year later, were assessed neuropsychologically five years later. Imaging parameters accounting for significant variance in cognitive performance were identified using multiple regressions, once confounding variables were controlled. Patients performed significantly worse than expected on tests of at…

Cognitive deficits in multiple sclerosisAdultMalePediatricsmedicine.medical_specialtyDevelopmental psychologyCentral nervous system diseaseDisability EvaluationAtrophyDegenerative diseaseMultiple Sclerosis Relapsing-Remittingrelapsing-remitting multiple sclerosiPredictive Value of TestsmedicineHumansMultiple sclerosiCognitive impairmentMultiple sclerosisCognitive disorderCognitionMiddle Agedmedicine.diseaseMagnetic Resonance ImagingEarly DiagnosisNeurologyRelapsing remittingLinear ModelsFemaleNeurology (clinical)AtrophyPsychologyCognition DisordersFollow-Up StudiesMultiple sclerosis (Houndmills, Basingstoke, England)
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Cognitive and behavioural effects induced by social stress plus MDMA administration in mice

2017

Adverse life experiences such as social stress may make an individual more vulnerable to drug addiction and mental disorders associated with drug consumption. The present work aimed to evaluate the effects of stress induced by acute social defeat combined with the administration of 3,4-methylenedioxymethamphetamine (MDMA) on depression-like behaviour, memory function and motor response to drug in late adolescent male mice. Two groups of mice were exposed to social defeat (SD) during four encounters with an aggressive co-specific, which took place on alternate days. Immediately after defeat, animals were treated with saline or MDMA 10mg/kg (SD+SAL and SD+MDMA). In control groups, mice were p…

Dominance-SubordinationMalemedicine.medical_specialtyN-Methyl-34-methylenedioxyamphetaminemedia_common.quotation_subjectPoison controlBehavioral SymptomsMotor ActivityBody TemperatureSocial defeatMice03 medical and health sciencesBehavioral Neurosciencechemistry.chemical_compound0302 clinical medicineCorticosteroneInternal medicinemental disordersAvoidance LearningmedicineAnimalsPsychiatrymedia_commonSocial stressAnalysis of VarianceAddictionRecognition PsychologyMDMAmedicine.diseaseTail suspension test030227 psychiatrySubstance abuseDisease Models AnimalEndocrinologyHindlimb SuspensionchemistryHallucinogensCognition DisordersCorticosteronePsychologyStress Psychologicalpsychological phenomena and processes030217 neurology & neurosurgerymedicine.drugBehavioural Brain Research
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Chronic cocaine exposure impairs progenitor proliferation but spares survival and maturation of neural precursors in adult rat dentate gyrus

2006

Recent observations indicate that drugs of abuse, including alcohol and opiates, impair adult neurogenesis in the hippocampus. We have studied in rats the impact of cocaine treatment (20 mg/kg, daily, i.p.) on cell proliferation, survival and maturation following short-term (8-day) and long-term (24-day) exposure. Using 5'-bromo-2-deoxyuridine (BrdU) and Ki-67 as mitotic markers at the end of the drug treatments, we found that both short- and long-term cocaine exposures significantly reduced cell proliferation in the dentate gyrus (DG) of the hippocampus. By labelling mitotic cells with BrdU pulses before or during the early stages of the drug treatment, we determined that long-term cocaine…

Doublecortin Domain ProteinsMalemedicine.medical_specialtyDoublecortin ProteinCell SurvivalDown-RegulationMitosisHippocampusBiologyHippocampal formationDrug Administration ScheduleCocaine-Related DisordersCocaineDopamine Uptake InhibitorsInternal medicinemedicineAnimalsRats WistarCell ShapeCell ProliferationNeuronsTUNEL assayStem CellsGeneral NeuroscienceDentate gyrusNeuropeptidesNeurogenesisColocalizationCell DifferentiationRatsDoublecortinDisease Models AnimalKi-67 AntigenEndocrinologymedicine.anatomical_structureBromodeoxyuridineChronic DiseaseDentate GyrusMossy Fibers Hippocampalbiology.proteinCognition DisordersMicrotubule-Associated ProteinsNeuroscienceStratum lucidumEuropean Journal of Neuroscience
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