Search results for "Colony-Stimulating Factor"

showing 10 items of 174 documents

Effect of Granulocyte-Macrophage Colony-Stimulating Factor on Neutropenia and Related Morbidity Induced by Myelotoxic Chemotherapy

1990

Myelosuppression-related neutropenia is the major side effect of most anticancer chemotherapy. Despite considerable improvements in supportive care due to the advent of a variety of new antibiotic combinations, infection remains the main risk arising during the neutropenic period that follows intensive chemotherapy for cancer [1]. In addition, neutropenia is the major obstacle to dose escalation, frequency of cytoreductive treatment, and thus to improved cancer control. Regarding reduction of the period of neutropenia and increase of the maximum tolerated dose of effective anticancer agents, autologous bone marrow transplantation (ABMT) has recently offered new promise. However, as many as …

Oncologymedicine.medical_specialtyChemotherapySide effectbusiness.industrymedicine.medical_treatmentCancerImmunotherapyNeutropeniamedicine.diseaseColony-stimulating factorLeukemiaGranulocyte macrophage colony-stimulating factorInternal medicineImmunologymedicinebusinessmedicine.drug
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Stem cell-secreted factor therapy regenerates the ovarian niche and rescues follicles.

2021

Background Ovarian senescence is a normal age-associated phenomenon, but increasingly younger women are affected by diminished ovarian reserves or premature ovarian insufficiency. There is an urgent need for developing therapies to improve ovarian function in these patients. In this context, previous studies suggest that stem cell–secreted factors could have regenerative properties in the ovaries. Objective This study aimed to test the ability of various human plasma sources, enriched in stem cell–secreted factors, and the mechanisms behind their regenerative properties, to repair ovarian damage and to promote follicular development. Study Design In the first phase, the effects of human pla…

Ovarian CortexOvaryBone Marrow CellsPrimary Ovarian InsufficiencyPremature ovarian insufficiencyHematopoietic Cell Growth Factors03 medical and health sciencesMicePlasma0302 clinical medicineOvarian FollicleMice Inbred NODGranulocyte Colony-Stimulating FactorMedicineAnimalsHumans030212 general & internal medicinePlatelet activationOvarian ReserveStem Cell Factor030219 obstetrics & reproductive medicinebusiness.industryCell growthStem CellsOvaryInfant NewbornObstetrics and GynecologyBone Marrow Stem CellCell cycleFetal BloodDisease Models Animalmedicine.anatomical_structureCancer researchHeterograftsFemaleStem cellbusinessAmerican journal of obstetrics and gynecology
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Dupuytren's contracture: an update of biomolecular aspects and therapeutic perspectives.

2005

The so-called fibrogenic cytokines, able to induce the growth of fibroblasts and their differentiation into myofibroblasts and to stimulate their production of extracellular matrix, are involved in the genesis of Dupuytren’s contracture. Although many studies have been made of biomolecular aspects of palmar fibromatosis, practical applications from them are still far from imminent because of the real difficulty of blocking their action in vivo, even in a chronic, progressive lesion such as Dupuytren’s disease. Consequently, surgical excision of the palmar fascia still remains the treatment of choice.

Pathologymedicine.medical_specialtyGENETIC SUSCEPTIBILITYFIBRONECTINBioinformaticsDISEASEExtracellular matrixTransforming Growth Factor betamedicineFIBROSISHumansGenetic Predisposition to DiseaseDupuytren's contracturePlatelet-Derived Growth FactorTransplantationEpidermal Growth Factorbusiness.industryGROWTH-FACTOR-BETANONOPERATIVE TREATMENTSTEROIDSFibromatosisGranulocyte-Macrophage Colony-Stimulating FactorTissue Inhibitor of MetalloproteinasesFasciaASSOCIATIONmedicine.diseaseHandCOLLAGENFasciotomyFibronectinsbody regionsDupuytren Contracturemedicine.anatomical_structureMetalloproteasesSurgeryContracturemedicine.symptombusinessPalmar fasciaMyofibroblastMATRIXPalmar FibromatosisJournal of hand surgery (Edinburgh, Scotland)
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Biomarkers of periodontal tissue remodeling during orthodontic tooth movement in mice and men: overview and clinical relevance

2013

Biologically active substances are expressed by cells within the periodontium in response to mechanical stimuli from orthodontic appliances. Several possible biomarkers representing biological modifications during specific phenomena as simile-inflammatory process, bone resorption and formation, periodontal ligament changes, and vascular and neural responses are proposed. Citations to potentially published trials were conducted by searching PubMed, Cochrane databases, and scientific textbooks. Additionally, hand searching and contact with experts in the area were undertaken to identify potentially relevant published and unpublished studies. Selection criteria were as follows: animal models i…

PeriodontiumVascular Endothelial Growth Factor APeriodontal tissuemedicine.medical_specialtyTooth Movement TechniquesInterleukins; biomarkers; tooth movementsDentistrylcsh:MedicineReview Articlelcsh:TechnologyGeneral Biochemistry Genetics and Molecular BiologyBone resorptionCrevicular fluidMiceMedicinePeriodontal fiberAnimalsHumansClinical significancelcsh:ScienceGeneral Environmental Sciencebusiness.industrylcsh:TInterleukinsMacrophage Colony-Stimulating FactorRANK Ligandlcsh:RGeneral MedicinePeriodontiumperiodontal tissue remodelingorthodontic tooth movementtooth movementTooth movementOrthopedic surgeryCytokinestooth movementslcsh:Qperiodontal tissueTooth MobilitybusinessBiomarkers
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Mast cell-derived mediators promote murine neutrophil effector functions

2013

Mast cells are able to trigger life-saving immune responses in murine models for acute inflammation. In such settings, several lines of evidence indicate that the rapid and protective recruitment of neutrophils initiated by the release of mast cell-derived pro-inflammatory mediators is a key element of innate immunity. Herein, we investigate the impact of mast cells on critical parameters of neutrophil effector function. In the presence of activated murine bone marrow-derived mast cells, neutrophils freshly isolated from bone marrow rapidly lose expression of CD62L and up-regulate CD11b, the latter being partly driven by mast cell-derived TNF and GM-CSF. Mast cells also strongly enhance neu…

PhagocytosisImmunologyApoptosisInflammation610 Medicine & healthmast cellsBiology142-005 142-005Neutrophil ActivationlungMiceImmune systemPhagocytosisneutrophilsmedicineAnimalsImmunology and AllergyCells CulturedMice Knockout2403 ImmunologyInnate immune systemTumor Necrosis Factor-alpharodentGranulocyte-Macrophage Colony-Stimulating FactorPneumoniaGeneral MedicineFlow CytometryMast cellMice Mutant StrainsCell biologycell activationMice Inbred C57BLInterleukin 33medicine.anatomical_structureinflammationImmunology2723 Immunology and AllergyTumor necrosis factor alphamedicine.symptomCell activation
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Primary proliferating immature myeloid cells from CML patients are not resistant to induction of apoptosis by DNA damage and growth factor withdrawal.

1996

Induction of apoptosis by growth factor deprivation or gamma-irradiation-induced DNA damage was directly studied in proliferating primary haemopoietic cells derived from CD34-positive cells of 13 CML patients and 12 normal controls. CD34-positive cells were cultured in the presence of appropriate concentrations of SCF and G-CSF for 5–7 d. After gamma irradiation with 500 rad or growth factor deprivation, the fraction of apoptotic cells was assessed by two independent methods applying either measurement of cells incorporating FITC-labelled dUTP by terminal transferase or assessment of the fraction of cells with a less than 2N DNA content in flow cytometry. Proliferating CML cells were not re…

Programmed cell deathDNA damagemedicine.medical_treatmentFusion Proteins bcr-ablApoptosisBiologyFlow cytometrychemistry.chemical_compoundhemic and lymphatic diseasesGranulocyte Colony-Stimulating FactormedicineHumansStem Cell Factormedicine.diagnostic_testGrowth factorHematologyHematopoietic Stem CellsIn vitroTerminal deoxynucleotidyl transferasechemistryApoptosisGamma RaysImmunologyLeukemia Myeloid Chronic-PhaseCancer researchDNACell DivisionDNA DamageBritish journal of haematology
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Effect of cilomilast (Ariflo) on TNF-α, IL-8, and GM-CSF release by airway cells of patients with COPD

2003

Background: Inflammation in chronic obstructive pulmonary disease (COPD) is characterised by increased neutrophilic infiltration of the airways. Cilomilast, a novel selective phosphodiesterase 4 inhibitor in clinical development for COPD treatment, exerts anti-inflammatory effects. The ability of cilomilast to inhibit the release of neutrophil chemoattractants such as tumour necrosis factor (TNF)-α, interleukin (IL)-8, and granulocyte-macrophage colony stimulating factor (GM-CSF) by bronchial epithelial cells and sputum cells isolated from 10 patients with COPD, 14 normal controls, and 10 smokers was investigated. Methods: Bronchial epithelial cells obtained by bronchial brushing and sputum…

Pulmonary and Respiratory MedicineAdultMaleSputum CytologyCyclohexanecarboxylic AcidsPhosphodiesterase Inhibitorsmedicine.medical_treatmentChronic Obstructive Pulmonary DiseaseCarboxylic AcidsCell CountEnzyme-Linked Immunosorbent AssayRespiratory MucosaBronchial brushingPulmonary Disease Chronic ObstructiveNitrilesmedicineHumansInterleukin 8Cells CulturedAgedbusiness.industryTumor Necrosis Factor-alphaCilomilastInterleukin-8SputumGranulocyte-Macrophage Colony-Stimulating FactorEpithelial CellsMiddle Agedrespiratory tract diseasesBronchodilator AgentsChemotaxis LeukocyteCytokineGranulocyte macrophage colony-stimulating factorImmunologySputumTumor necrosis factor alphaFemalemedicine.symptombusinessmedicine.drug
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An open-label, prospective phase I/II study evaluating the immunogenicity and safety of a ras peptide vaccine plus GM-CSF in patients with non-small …

2007

Mutations of the ras gene have been reported in 20-40% of NSCLC patients. If present, they are critical for the malignant phenotype of these tumors. Therefore, targeting them by specific vaccination is a promising therapeutic approach. In a clinical trial we screened for ras mutations in patients with NSCLC. Patients with ras-positive tumors were immunized six times intradermally with a mixture of seven peptides representing the most common ras mutations. Objectives of the study were the feasibility, efficacy and safety of the vaccination. In addition, the induction of a specific immune reaction was investigated by DTH tests, and the induction of peptide-specific T cells was tested in ex vi…

Pulmonary and Respiratory MedicineOncologyMaleCancer Researchmedicine.medical_specialtyLung Neoplasmsmedicine.medical_treatmentT-LymphocytesCancer VaccinesImmune systemInternal medicineCarcinoma Non-Small-Cell LungCarcinomaMedicineHumansLung cancerCodonAgedNeoplasm StagingImmunity Cellularbusiness.industryImmunogenicityRas PeptideVaccinationGranulocyte-Macrophage Colony-Stimulating FactorImmunotherapyMiddle Agedmedicine.diseaseCombined Modality TherapyPeptide FragmentsRecombinant ProteinsVaccinationOncologyImmunologyMutationras ProteinsFemaleImmunotherapybusinessEx vivoLung cancer (Amsterdam, Netherlands)
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GM-CSF expression by human lung microvascular endothelial cells: in vitro and in vivo findings.

2002

Recently, many findings indicate that granulocyte-macrophage colony-stimulating factor (GM-CSF) plays an important role in the pathogenesis of acute and chronic lung diseases. In the present paper, the production of this cytokine in human pulmonary microvascular endothelial cells (HPMEC) is investigated. In an in vitro study, quiescent HPMEC did not express GM-CSF, either at the transcriptional or at the protein level. After activation for 4 h with tumor necrosis factor (TNF)-α (30/300 U/ml), lipopolysaccharide (LPS; 0.1/1 μg/ml), or interleukin (IL)-1β (100 U/ml), a significant release of GM-CSF was measured by enzyme-linked immunosorbent assay, with a time-dependent increase over 72 h. IL…

Pulmonary and Respiratory MedicinePathologymedicine.medical_specialtyPulmonary CirculationUmbilical VeinsEndotheliumPhysiologymedicine.medical_treatmentHemangiosarcomaEnzyme-Linked Immunosorbent AssayIn Vitro TechniquesPathogenesisIn vivoPhysiology (medical)medicineHumansCells CulturedLungbusiness.industryReverse Transcriptase Polymerase Chain ReactionMicrocirculationRespiratory diseaseGranulocyte-Macrophage Colony-Stimulating FactorCell Biologymedicine.diseaseImmunohistochemistryEndothelial stem cellGranulocyte macrophage colony-stimulating factorCytokinemedicine.anatomical_structureEndothelium Vascularbusinessmedicine.drugAmerican journal of physiology. Lung cellular and molecular physiology
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Human leucocyte antigen-A2 restricted and Mycobacterium tuberculosis 19-kDa antigen-specific CD8+ T-cell responses are oligoclonal and exhibit a T-ce…

2001

CD8+ T cells can be grouped into two different types of secretory T lymphocytes, based on the cytokine-secretion pattern upon antigen exposure: those with a T-cell cytotoxic type 1 response (Tc1), which secrete interferon-gamma (IFN-gamma), or those with a T-cell cytotoxic type 2 response, which secrete interleukin (IL)-4 and IL-10. We examined the CD8+ T-cell response directed against an immunodominant human leucocyte antigen (HLA)-A2-presented peptide derived from a 19-kDa Mycobacterium tuberculosis-associated antigen. T cells were examined by functional analysis and by T-cell receptor (TCR) complementarity-determining region 3 (CDR3)-spectratyping, which defines the complexity of a T-cel…

Receptors Antigen T-Cell alpha-betaT cellImmunologyHuman leukocyte antigenCD8-Positive T-LymphocytesBiologyLymphocyte ActivationEpitopeCell LineInterferon-gammaAntigenHLA-A2 AntigenmedicineHumansImmunology and AllergyCytotoxic T cellAntigen-presenting cellTuberculosis PulmonaryAntigens BacterialImmunodominant EpitopesT-cell receptorGranulocyte-Macrophage Colony-Stimulating FactorMycobacterium tuberculosisOriginal ArticlesComplementarity Determining RegionsMolecular biologyPeptide FragmentsClone Cellsmedicine.anatomical_structureImmunologyInterleukin-4CD8Immunology
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