Search results for "Combination Therapy"

showing 10 items of 162 documents

Sequential Therapy with Azelastine in Seasonal Allergic Rhinitis

2011

A sequential therapy treatment with azelastine (Allergodil (r) ) in seasonal allergic rhinitis is introduced. In the critical early stage, treatment begins with a combination of azelastine tablets (azelastine hydrochloride, CAS 79307-93-0) and azelastine nasal spray (azelastine, CAS 58581-89-8), and after five days only the nasal spray is administered. This sequential therapy model aims at achieving the quickest and most complete effect without reducing the tolerability. The investigation was carried out as a randomized, controlled double-blind phase IV study of parallel group design with 300 patients during 14 days. In the first five days, one group was given one puff (0.14 mg) of azelasti…

AllergyAzelastine HydrochlorideCombination therapybusiness.industrymedicine.medical_treatmentPlacebomedicine.diseaseAzelastineTolerabilityNasal sprayOral administrationAnesthesiaDrug Discoverymedicinebusinessmedicine.drugArzneimittelforschung
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Effect of tiotropium/olodaterol combination therapy on long-term heart rate and blood pressure in COPD patients

2018

Introduction: Cardiovascular (CV) comorbidities are common in COPD, and associated with poor prognosis. LABAs and LAMAs are established COPD treatments whose pharmacology would suggest the potential to increase heart rate (HR) and impact blood pressure (BP). However, previous studies indicate that HR and BP are not negatively influenced by tiotropium (Tio) or olodaterol (Olo) monotherapy. Aims: To determine the effect of dual bronchodilation with Tio/Olo (T/O) on HR and BP. Methods: The 52-week, Phase III TONADO® studies (NCT01431274/NCT01431287) evaluated T/O 5/5 µg, Tio 5 µg or Olo 5 µg, via the Respimat® inhaler, in GOLD 2–4 COPD patients. In this post hoc analysis, long-term changes fro…

COPDmedicine.medical_specialtyCombination therapybusiness.industryCopd patientsInhalerOlodaterolmedicine.disease3. Good health03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBlood pressure030228 respiratory systemchemistryInternal medicineHeart ratePost-hoc analysismedicineCardiology030212 general & internal medicinebusinessAirway pharmacology and treatment
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BIOM-08. DNA METHYLATION-BASED SUBGROUPING PREDICTS SURVIVAL BENEFIT FROM LOMUSTINE/TEMOZOLOMID COMBINATION THERAPY IN MGMT PROMOTOR-METHYLATED GLIOB…

2021

Abstract BACKGROUND The CeTeG/NOA-09 trial showed that lomustine/temozolomide chemotherapy prolongs survival for newly diagnosed MGMT-methylated glioblastoma patients. Previous reports on temozolomide monotherapy suggested, that the survival benefit of temozolomide in MGMT-methylated tumors may be restricted to the RTK II methylation subgroup and absent in RTK I and MES subgroups. To identify patients with a particularly strong benefit from CCNU/TMZ, we explored the association of methylation subgroups with outcome after lomustine/temozolomide therapy. METHODS All patients from the CeTeG/NOA-09 trial with sufficiently available tumor tissue (n = 98) underwent 850K methylation array analysis…

Cancer ResearchCombination therapybusiness.industryMedizinPromoterLomustine26th Annual Meeting & Education Day of the Society for Neuro-Oncologymedicine.diseaseSurvival benefitOncologyDNA methylationmedicineCancer researchNeurology (clinical)Temozolomidbusinessmedicine.drugGlioblastomaNeuro-Oncology
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Temporal molecular and biological assessment of an erlotinib-resistant lung adenocarcinoma model reveals markers of tumor progression and treatment r…

2012

Abstract Patients with lung cancer with activating mutations in the EGF receptor (EGFR) kinase, who are treated long-term with tyrosine kinase inhibitors (TKI), often develop secondary mutations in EGFR associated with resistance. Mice engineered to develop lung adenocarcinomas driven by the human EGFR T790M resistance mutation are similarly resistant to the EGFR TKI erlotinib. By tumor volume endpoint analysis, these mouse tumors respond to BIBW 2992 (an irreversible EGFR/HER2 TKI) and rapamycin combination therapy. To correlate EGFR-driven changes in the lung with response to drug treatment, we conducted an integrative analysis of global transcriptome and metabolite profiling compared wit…

Cancer ResearchLung NeoplasmsCombination therapyAfatinibGene ExpressionAdenocarcinoma of LungCell Growth ProcessesAdenocarcinomaAfatinibArticleErlotinib HydrochlorideMiceAntineoplastic Combined Chemotherapy ProtocolsmedicineAnimalsEpidermal growth factor receptorLung cancerErlotinib HydrochlorideProtein Kinase InhibitorsSirolimusbiologymedicine.diseaserespiratory tract diseasesErbB ReceptorsOncologyTumor progressionDrug Resistance NeoplasmCancer researchbiology.proteinDisease ProgressionQuinazolinesErlotinibTyrosine kinasemedicine.drugTranscription FactorsCancer research
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Bcl-2 and glutathione depletion sensitizes B16 melanoma to combination therapy and eliminates metastatic disease.

2007

Abstract Purpose: Advanced melanoma resists all current therapies, and metastases in the liver are particularly problematic. Prevalent resistance factors include elevated glutathione (GSH) and increased expression of bcl-2 in melanoma cells. GSH has pleiotropic effects promoting cell growth and broad resistance to therapy, whereas Bcl-2 inhibits the activation of apoptosis and contributes to elevation of GSH. This study determined the in vivo efficacy of combination therapies administered while GSH and Bcl-2 were individually and simultaneously decreased in metastatic melanoma lesions. Experimental Design: Highly metastatic murine B16 melanoma (B16M-F10) cells have elevated levels of both G…

Cancer Researchmedicine.medical_specialtySkin NeoplasmsCombination therapyPaclitaxelGlutamineMelanoma ExperimentalBiologyMetastasischemistry.chemical_compoundMiceIn vivoInternal medicinemedicineAnimalsNeoplasm MetastasisAcivicinHematologyTumor Necrosis Factor-alphaMelanomaX-RaysGlutathioneThionucleotidesmedicine.diseaseAntineoplastic Agents PhytogenicCombined Modality TherapyGlutathioneTreatment OutcomeOncologychemistryProto-Oncogene Proteins c-bcl-2ToxicityCancer researchClinical cancer research : an official journal of the American Association for Cancer Research
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Increased circulating and intrahepatic T-cell-specific chemokines in chronic hepatitis C: relationship with the type of virological response to pegin…

2004

Summary Aims : To determine the serum and intrahepatic levels of T-helper-1-associated chemokines in patients with chronic hepatitis C before, during and after peginterferon plus ribavirin combination therapy and to search for correlations with baseline characteristics of hepatitis C virus-related chronic liver disease and type of therapeutic response. Methods : Serum chemokine levels were determined by enzyme-linked immunosorbent assays and intrahepatic chemokine messenger RNA and protein levels were tested by ribonuclease protection assay and immunohistochemistry. Results : Serum and intrahepatic chemokine levels were elevated in all patients with chronic hepatitis C and showed a marked d…

ChemokineHepatologybiologyCombination therapybusiness.industryRibavirinGastroenterologyHepatitis Cmedicine.diseaseChronic liver diseasechemistry.chemical_compoundBasal (phylogenetics)chemistryFibrosisImmunologyGenotypebiology.proteinMedicinePharmacology (medical)businessAlimentary Pharmacology & Therapeutics
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SDF-1/CXCR4 inhibition prevents paradoxical generation of cisplatin-induced pro-metastatic niches

2020

AbstractPlatinum-based chemotherapy remains widely used in advanced non-small cell lung cancer (NSCLC) despite its ineffectiveness in long-term control of metastasis.Here, we uncover the interconnected pathways subtending cisplatin-induced metastasis promotion.We report that cisplatin treatment of tumor-free mice results in bone-marrow expansion of CCR2+CXCR4+Ly6Chigh inflammatory monocytes (IM) concomitantly with increased levels in the lungs of stromal SDF-1, the CXCR4 ligand. In experimental metastasis assays, cisplatin-induced IM favor tumor cells extravasation and expansion of CD133+CXCR4+ metastasis initiating cells (MICs), facilitating lung metastasis formation. At the primary tumor,…

CisplatinChemotherapyStromal cellCombination therapybusiness.industrymedicine.medical_treatmentmedicine.diseaseCXCR4Primary tumorExtravasationMetastasisCancer researchMedicinebusinessmedicine.drug
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Unraveling the extracellular matrix-tumor cell interactions to aid better targeted therapies for neuroblastoma

2021

Treatment in children with high-risk neuroblastoma remains largely unsuccessful due to the development of metastases and drug resistance. The biological complexity of these tumors and their microenvironment represent one of the many challenges to face. Matrix glycoproteins such as vitronectin act as bridge elements between extracellular matrix and tumor cells and can promote tumor cell spreading. In this study, we established through a clinical cohort and preclinical models that the interaction of vitronectin and its ligands, such as αv integrins, are related to the stiffness of the extracellular matrix in high-risk neuroblastoma. These marked alterations found in the matrix led us to speci…

Combination therapyPharmaceutical ScienceCilengitideAntineoplastic AgentsCell CommunicationExtracellular matrixchemistry.chemical_compoundNeuroblastomaNeuroblastomamedicineTumor MicroenvironmentHumansVitronectinEtoposideEtoposidechemistry.chemical_classificationTumor microenvironmentbiologyCilengitidemedicine.diseaseExtracellular MatrixNanomedicinechemistryTumor microenvironmentbiology.proteinCancer researchVitronectinGlycoproteinmedicine.drug
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Synergistic effect of recombinant CD4-immunoglobulin in combination with azidothymidine, dideoxyinosine and 0.5 beta-monoclonal antibody on human imm…

1994

Data are presented which indicate that combinations of rCD4 immunoglobulin with azidothymidine, dideoxyinosine or 0.5 beta mouse monoclonal antibodies directed against the V3 region of HIV-1, were more effective in treatment of acute HIV infection in vitro than each compound alone. It is suggested that combination therapy with these compounds is more beneficial in treatment of HIV-infected patients than monotherapy, especially with respect to a reduction of the known side effects and the formation of resistant HIV strains after treatment with nucleoside analogues.

Combination therapymedicine.drug_classmedicine.medical_treatmentHIV InfectionsBiologyMonoclonal antibodyApplied Microbiology and BiotechnologyVirusZidovudinemedicineHumansLymphocytesCells Culturedvirus diseasesAntibodies MonoclonalDrug SynergismImmunotherapyVirologyIn vitroAnti-Bacterial AgentsDidanosinebiology.proteinDrug Therapy CombinationAntibodyNucleosideZidovudineCD4 Immunoadhesinsmedicine.drugLetters in applied microbiology
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Cardiotoxicity mechanisms of the combination of BRAF-inhibitors and MEK-inhibitors.

2018

Many new drugs have appeared in last years in the oncological treatment scenario. Each drug carries an important set of adverse events, not less, cardiovascular adverse events. This aspect is even more important considering the increasing use of combination therapies with two drugs, or three drugs as in some ongoing clinical trials. Besides it represents a growing problem for Cardiologists, that face it in every day clinical practice and that will face it probably more and more in the coming years. This work reviews the mechanism of action of BRAF-inhibitors and MEK-inhibitors used together, the pathophysiological mechanisms that lead to cardiovascular toxicity. Particularly, it focuses on …

DrugCardiovascular toxicityBRAF inhibitorProto-Oncogene Proteins B-rafmedicine.medical_specialtyCombination therapySettore MED/06 - Oncologia Medicamedia_common.quotation_subjectDecreased ejection fraction030204 cardiovascular system & hematologyCardiovascular System03 medical and health sciences0302 clinical medicineCardiovascular toxicityAntineoplastic Combined Chemotherapy ProtocolsMedicineHumansPharmacology (medical)Intensive care medicineAdverse effectBRAF inhibitor; Cardio-oncology; Cardiovascular toxicity; Decreased ejection fraction; Hypertension; MEK inhibitor; Pharmacology; Pharmacology (medical)MelanomaProtein Kinase Inhibitorsmedia_commonPharmacologyMitogen-Activated Protein Kinase KinasesCardiotoxicityMEK inhibitorClinical Trials as Topicbusiness.industryMEK inhibitorCancermedicine.diseaseCardiotoxicityClinical trialCardio-oncology030220 oncology & carcinogenesisHypertensionbusinessPharmacologytherapeutics
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