Search results for "Complement System Proteins"

showing 10 items of 56 documents

Complement pore genesis observed in erythrocyte membranes by fluorescence microscopic single-channel recording

1991

The formation and opening of single complement pores could be directly observed in erythrocyte ghosts by confocal laser-scanning microscopy employing the recently introduced method of fluorescence microscopic single-channel recording. Resealed sheep erythrocyte ghosts were incubated with human complement. By limiting the concentration of C8, the eighth component of complement, the fraction of cells rendered permeable for the small polar fluorescent probe Lucifer Yellow was varied between 0.50 and 0.90. Under each condition the flux rate, k, of Lucifer Yellow was determined for a substantial number of ghosts. By analysing the sample population distribution of k the flux rate k1 of ghosts wit…

Lucifer yellowPhotolysisSheepScanning electron microscopeConfocalErythrocyte MembraneAnalytical chemistryComplement System ProteinsCell BiologyModels TheoreticalIsoquinolinesBiochemistryFluorescenceKineticschemistry.chemical_compoundMonomerMembraneMicroscopy FluorescencechemistryMicroscopyFluorescence microscopeAnimalsMolecular BiologyFluorescent DyesResearch ArticleBiochemical Journal
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Effect of in vivo stimulation of mice on the secretion of factor B of the alternative complement pathway by peritoneal macrophages

1977

After in vivo treatment of mice with thioglycollate medium, the amount of native factor B which could be detected in vitro in culture supernatants of peritoneal macrophages was much lower than that found in supernatants of macrophages taken from untreated mice. However, when the macrophages from thioglycollate medium-treated mice were cultured on a plastic surface covered with glutardialdehyde-linked bovine serum albumin, the culture supernatants contained larger quantities of native factor B than culture supernatants of macrophages from untreated mice under the same conditions. Thus, the effect of in vivo thioglycollate medium treatment on the in vitro secretion of factor B by peritoneal m…

MacrophagesGuinea PigsImmunologyCell CountSerum Albumin BovineStimulationComplement System ProteinsBiologyComplement factor BIn vitroMicrobiologyMiceGlutaralIn vivobiology.proteinAlternative complement pathwayAnimalsImmunology and AllergySecretionFactor DBovine serum albuminPlasticsCells CulturedEuropean Journal of Immunology
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Genetic prediction of ICU hospitalization and mortality in COVID‐19 patients using artificial neural networks

2021

There is an unmet need of models for early prediction of morbidity and mortality of Coronavirus disease-19 (COVID-19). We aimed to a) identify complement-related genetic variants associated with the clinical outcomes of ICU hospitalization and death, b) develop an artificial neural network (ANN) predicting these outcomes and c) validate whether complement-related variants are associated with an impaired complement phenotype. We prospectively recruited consecutive adult patients of Caucasian origin, hospitalized due to COVID-19. Through targeted next-generation sequencing, we identified variants in complement factor H/CFH, CFB, CFH-related, CFD, CD55, C3, C5, CFI, CD46, thrombomodulin/THBD, …

Male0304 Medicinal and Biomolecular Chemistry 0601 Biochemistry and Cell Biology 1103 Clinical SciencesBiochemistry & Molecular BiologyGreeceModels GeneticThrombomodulinCOVID-19Complement System ProteinsCell BiologyMiddle AgedPolymorphism Single NucleotideHospitalizationSettore ICAR/09 - Tecnica Delle CostruzioniIntensive Care UnitsComplement Factor HHumansMolecular MedicineFemaleNeural Networks ComputerMorbidityartificial intelligence complement complement inhibition COVID-19 genetic susceptibility SARS-CoV2Complement ActivationJournal of Cellular and Molecular Medicine
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Vascular Leakage in Severe Dengue Virus Infections: A Potential Role for the Nonstructural Viral Protein NS1 and Complement

2006

Background Vascular leakage and shock are the major causes of death in patients with dengue hemorrhagic fever (DHF) and dengue shock syndrome (DSS). Thirty years ago, complement activation was proposed to be a key underlying event, but the cause of complement activation has remained unknown. Methods The major nonstructural dengue virus (DV) protein NS1 was tested for its capacity to activate human complement in its membrane-associated and soluble forms. Plasma samples from 163 patients with DV infection and from 19 patients with other febrile illnesses were prospectively analyzed for viral load and for levels of NS1 and complement-activation products. Blood and pleural fluids from 9 patient…

MaleAdolescentvirusesComplement C5aComplement Membrane Attack ComplexViral Nonstructural ProteinsDengue virusBiologyAntibodies Viralmedicine.disease_causeVirusCell LineDengue feverDenguemedicineHumansImmunology and AllergyAnaphylatoxinVascular DiseasesChildGlycoproteinsPleural Cavityvirus diseasesComplement System ProteinsDengue VirusViral Loadmedicine.diseaseVirologyComplement systemInfectious DiseasesCase-Control StudiesChild PreschoolImmunologybiology.proteinRNA ViralFemaleAntibodyComplement membrane attack complexViral loadThe Journal of Infectious Diseases
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Proteome analysis of myocardial tissue following ischemia and reperfusion--effects of complement inhibition.

2006

Myocardial ischemia-reperfusion injury can be related to complement activation with generation of chemotactic mediators, release of cytokines, leukocyte accumulation, and subsequent severe tissue injury. In this regard, activation of transcription factors (i.e., NFkappaB) and de novo protein synthesis or inflammatory protein degradation seems to play an important role. In the present study, we analyzed the cardiac protein expression following myocardial ischemia (60 min) and reperfusion (180 min) in a rabbit model utilizing two-dimensional electrophoresis and nanoHPLC/ESI-MS/MS for biochemical protein identification. To achieve cardioprotective effects, we used a novel highly selective smal…

MaleProteomeG proteinNeutrophilsMolecular Sequence DataBiophysicsIschemiaMyocardial IschemiaMyocardial Reperfusion InjuryProtein degradationComplement C1 Inactivator ProteinsBiochemistryAnalytical ChemistrySuperoxide dismutaseClassical complement pathwayElectrocardiographyNecrosismedicineProtein biosynthesisAnimalsAmino Acid SequenceMolecular BiologyCreatine KinasebiologySuperoxide DismutaseMyocardiumalpha-Crystallin B ChainComplement System Proteinsmedicine.diseaseMolecular biologyComplement systembiology.proteinCreatine kinaseRabbitsMicrotubule-Associated ProteinsBiomarkersBiochimica et biophysica acta
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Major histocompatibility complex (MHC) class III genetics in two Amerindian tribes from southern Brazil: the Kaingang and the Guarani.

1997

Population genetic studies of the major histocompatibility complex (MHC) class III region, comprising C2, BF and C4 phenotypes, and molecular genetic data are rarely available for populations other than Caucasoids. We have investigated three Amerindian populations from Southern Brazil: 131 Kaingang from Ivaí (KIV), 111 Kaingang (KRC) and 100 Guarani (GRC) from Rio das Cobras. Extended MHC haplotypes were derived after standard C2, BF, C4 phenotyping and restriction fragment length polymorphism (RFLP) analysis with TaqI, together with HLA data published previously by segregation analysis. C2 and BF frequencies corresponded to other Amerindian populations. C4B*Q0 frequency was high in the GRC…

MaleTaqIPopulationLocus (genetics)Human leukocyte antigenBiologyMajor Histocompatibility Complexchemistry.chemical_compoundGene FrequencyGeneticsHumanseducationChildGenetics (clinical)Geneticseducation.field_of_studyHistocompatibility TestingIndians South AmericanHaplotypeComplement C4Complement System ProteinsComplement C2Genetic distancechemistryHaplotypesGenetic markerFemaleSteroid 21-HydroxylaseRestriction fragment length polymorphismBrazilPolymorphism Restriction Fragment LengthComplement Factor BHuman genetics
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Complement Activation in Peritoneal Dialysis–Induced Arteriolopathy

2017

Cardiovascular disease (CVD) is the leading cause of increased mortality in patients with CKD and is further aggravated by peritoneal dialysis (PD). Children are devoid of preexisting CVD and provide unique insight into specific uremia- and PD-induced pathomechanisms of CVD. We obtained peritoneal specimens from children with stage 5 CKD at time of PD catheter insertion (CKD5 group), children with established PD (PD group), and age-matched nonuremic controls (n=6/group). We microdissected omental arterioles from tissue layers not directly exposed to PD fluid and used adjacent sections of four arterioles per patient for transcriptomic and proteomic analyses. Findings were validated in omenta…

MaleVascular Endothelial Growth Factor A0301 basic medicinePathologyProteomemedicine.medical_treatmentComplement Membrane Attack ComplexSmad2 ProteinSeverity of Illness IndexTransforming Growth Factor betaMedicinePhosphorylationChildComplement ActivationCatheter insertionGeneral MedicineArteriosclerosisArteriolesComplement C3dNephrologyChild PreschoolFemaleOmentumPeritoneal DialysisSignal Transductionmedicine.medical_specialtyAdolescentPeritoneal dialysis03 medical and health sciencesDownregulation and upregulationClinical ResearchTGF beta signaling pathwayHumansSmad3 ProteinVascular DiseasesUremiabusiness.industryVascular diseaseComplement C1qInfant NewbornInfantComplement System Proteinsmedicine.diseaseUremiaComplement systemGene Ontology030104 developmental biologyCase-Control StudiesKidney Failure ChronicTranscriptomebusinessJournal of the American Society of Nephrology
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Seminal plasma-induced suppression of the respiratory burst of polymorphonuclear leukocytes and monocytes.

1984

Summary: Based on recent findings indicating suppression of lymphocytic functions by seminal plasma (SP) we tested the effects of SP from men with normo- and oligozoospermia (n = 7, each) on the generation of luminol-enhanced chemiluminescence (CL) of polymorphonuclear leukocytes (PMN) and monocytes (Mo) stimulated in vitro with zymosan. We found a complete suppression of CL of PMN and Mo by undiluted SP's, 1,000-fold dilutions still induced ≥ 20 percent inhibition. There was no difference between normo- and oligozoospermic men in inhibition of CL both with PMN and Mo. Protein concentrations of SP's were closely the same; all SP were free of the complement components C4 and C3c. After dialy…

Malemedicine.medical_specialtyNeutrophilsUrologyPercent InhibitionMonocytesComplement componentschemistry.chemical_compoundEndocrinologyOxygen ConsumptionSemenInternal medicinemedicineHumansImmunosuppression TherapyMonocyteZymosanProteinsGeneral MedicineComplement System ProteinsOligospermiaMolecular biologyRespiratory burstEndocrinologymedicine.anatomical_structurechemistryGenital tractLuminescent MeasurementsAndrologia
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Distinct Roles of Classical and Lectin Pathways of Complement in Preeclamptic Placentae

2022

Pre-eclampsia is a pregnancy complication characterized by defective vascular remodeling in maternal decidua responsible for reduced blood flow leading to functional and structural alterations in the placenta. We have investigated the contribution of the complement system to decidual vascular changes and showed that trophoblasts surrounding unremodeled vessels prevalent in preeclamptic decidua fail to express C1q that are clearly detected in cells around remodeled vessels predominant in control placenta. The critical role of C1q is supported by the finding that decidual trophoblasts of femaleC1qa-/-pregnant mice mated toC1qa+/+male mice surrounding remodeled vessels express C1q of paternal …

Malepre-eclampsiavascular remodelingComplement System ProteinPlacentaImmunologyMannose-Binding Protein-Associated Serine ProteaseSettore MED/08 - Anatomia PatologicaPre-Eclampsia.MicePregnancyLectinsficolin-3Immunology and AllergyAnimalsHumansSettore MED/05 - Patologia Clinicacomplement system; pre-eclampsia; vascular remodeling; C1q; ficolin-3C1qcomplement systemAnimalComplement C1qEndothelial CellsComplement System ProteinsMannose-Binding Protein-Associated Serine ProteasesFemale
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Research on complement: old issues revisited and a novel sphere of influence

2003

Immunology in recent years has taken a somewhat surprising turn, expressed by a renewed interest in innate immunity. Especially intriguing is the regulatory role exerted by the innate components on the adaptive response, with Toll receptors and complement components being the most investigated. This function has been firmly established for complement protein CR2 (CD21) as part of the BCR co-receptor CD19/CD21/CD81. New findings are now providing a broader picture of complement and its tuning of the immune response; for example, complement proteins have been implicated in the control of T-cell-mediated responses. We will review some of these data here and summarize new discoveries in areas o…

Membrane GlycoproteinsInnate immune systemT-LymphocytesImmunologychemical and pharmacologic phenomenaComplement System ProteinsComplement C1 Inactivator ProteinsBiologyImmunity InnateComplement componentsComplement systemComplement (complexity)Membrane Cofactor ProteinImmune systemAntigens CDComplement Factor HImmunologyAnimalsHumansImmunology and AllergyKidney DiseasesSphere of influenceComplement C1 Inhibitor ProteinSerpinsTrends in Immunology
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