Search results for "Corpus Callosum"

showing 10 items of 66 documents

Impaired anatomical connectivity and related executive functions: differentiating vulnerability and disease marker in bipolar disorder.

2012

Background Bipolar 1 disorder (BD1) has been associated with impaired set shifting, increased risk taking, and impaired integrity of frontolimbic white matter. However, it remains unknown to what extent these findings are related to each other and whether these abnormalities represent risk factors or consequences of the illness. Methods We addressed the first question by comparing 19 patients with BD1 and 19 healthy control subjects (sample 1) with diffusion tensor imaging, the Intra-Extra Dimensional Set Shift Task, and the Cambridge Gambling Task. The second question we approached by applying the same protocol to 22 healthy first-degree relatives of patients with BD1 and 22 persons withou…

AdultMalemedicine.medical_specialtyInternal capsuleBipolar DisorderAdolescentStatistics as TopicUncinate fasciculusAudiologyNeuropsychological TestsCorpus callosumNerve Fibers MyelinatedCorpus CallosumWhite matterExecutive FunctionYoung AdultFractional anisotropymedicineHumansFamilyBipolar disorderFirst-degree relativesPsychiatryBiological PsychiatryPsychiatric Status Rating ScalesCognitive flexibilityBrainMiddle Agedmedicine.diseasemedicine.anatomical_structureDiffusion Magnetic Resonance ImagingAnisotropyFemalePsychologyCognition DisordersBiological psychiatry
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Corpus callosum involvement: a useful clue for differentiating Fabry Disease from Multiple Sclerosis.

2017

PURPOSE: Multiple sclerosis (MS) has been proposed as a possible differential diagnosis for Fabry disease (FD). The aim of this work was to evaluate the involvement of corpus callosum (CC) on MR images and its possible role as a radiological sign to differentiate between FD and MS. METHODS: In this multicentric study, we retrospectively evaluated the presence of white matter lesions (WMLs) on the FLAIR images of 104 patients with FD and 117 patients with MS. The incidence of CC-WML was assessed in the two groups and also in a subgroup of 37 FD patients showing neurological symptoms. RESULTS: WMLs were detected in 50 of 104 FD patients (48.1%) and in all MS patients. However, a lesion in the…

AdultMalemedicine.medical_specialtyPathologyNeurologySettore MED/09 - Medicina InternaAdolescentCorpus callosumFluid-attenuated inversion recoveryCorpus callosumCorpus callosum; Fabry disease; MRI; Multiple sclerosis030218 nuclear medicine & medical imagingDiagnosis DifferentialMultiple sclerosis03 medical and health sciences0302 clinical medicinemedicineHumansRadiology Nuclear Medicine and imagingAgedRetrospective StudiesNeuroradiologyFabry diseasebusiness.industryMultiple sclerosisMiddle Agedmedicine.diseaseMagnetic Resonance ImagingFabry diseaseHyperintensityCorpus callosum; Fabry disease; MRI; Multiple sclerosis.FemaleNeurology (clinical)RadiologyDifferential diagnosisCardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgeryMRI
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Corpus callosum area in patients with bipolar disorder with and without psychotic features: an international multicentre study

2015

Background Previous studies have reported MRI abnormalities of the corpus callosum (CC) in patients with bipolar disorder (BD), although only a few studies have directly compared callosal areas in psychotic versus nonpsychotic patients with this disorder. We sought to compare regional callosal areas in a large international multicentre sample of patients with BD and healthy controls. Methods We analyzed anatomic T-1 MRI data of patients with BD-I and healthy controls recruited from 4 sites (France, Germany, Ireland and the United States). We obtained the mid-sagittal areas of 7 CC subregions using an automatic CC delineation. Differences in regional callosal areas between patients and contr…

AdultMaleoasis brain databasePediatricsmedicine.medical_specialtyBipolar DisorderNeuroimagingshapeLithiumCorpus callosumearly alzheimers-diseasesizeCorpus CallosumGermanyImage Processing Computer-AssistedmedicineHumansPharmacology (medical)In patientBipolar disorderPsychiatryCognitive impairmentmriBiological Psychiatrycognitive impairmentreliabilitymedicine.diagnostic_testExtramuralbusiness.industryMagnetic resonance imagingMiddle Agedmedicine.diseaseMagnetic Resonance ImagingWhite MatterUnited States3. Good healthDiagnostic and Statistical Manual of Mental DisordersPsychiatry and Mental healthMulticenter studyLinear ModelsFemaleabnormalitiesFrancei disorderbusinessrating-scaleIrelandResearch PaperAntipsychotic AgentsJournal of Psychiatry and Neuroscience
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A novel L1CAM mutation in a fetus detected by prenatal diagnosis

2010

X-linked hydrocephalus is due to mutations in the L1 neuronal cell adhesion molecule (L1CAM) gene. L1 protein plays a key role in neurite outgrowth, axonal guidance, and pathfinding during the development of the nervous system. We report on a familial case diagnosed by prenatal ultrasonographic examination, with cerebellar hypoplasia, agenesis of the corpus callosum, and the bilateral overlapping of the second and third fingers of the hand. Sequencing of the L1CAM gene showed a novel missense mutation in exon 14: transition of a guanine to cytosine at position 1777 (c.1777G > C), which led to an amino acid change of alanine to proline at position 593 (Ala593Pro) in the sixth immunoglobulin …

Adultmedicine.medical_specialtyPathologyL1Neural Cell Adhesion Molecule L1Prenatal diagnosismedicine.disease_causeL1CAM L1-desease prenatal diagnosis hydrocephalus HSAS CRASH syndromeExonSettore MED/38 - Pediatria Generale E SpecialisticaPregnancyPrenatal DiagnosisInternal medicinemedicineHumansPoint MutationMissense mutationAgenesis of the corpus callosumUltrasonographyMutationbusiness.industrymedicine.diseasePedigreeFetal DiseasesEndocrinologyKaryotypingPediatrics Perinatology and Child HealthFemaleNeural cell adhesion moleculeCerebellar hypoplasia (non-human)businessHydrocephalus
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A simplified framework to optimize MRI contrast preparation

2018

PURPOSE This article proposes a rigorous optimal control framework for the design of preparation schemes that optimize MRI contrast based on relaxation time differences. METHODS Compared to previous optimal contrast preparation schemes, a drastic reduction of the optimization parameter number is performed. The preparation scheme is defined as a combination of several block pulses whose flip angles, phase terms and inter-pulse delays are optimized to control the magnetization evolution. RESULTS The proposed approach reduces the computation time of B 0 -robust preparation schemes to around a minute (whereas several hours were required with previous schemes), with negligible performance loss. …

AgingMultiple Sclerosis[SDV.IB.IMA]Life Sciences [q-bio]/Bioengineering/ImagingComputer scienceComputationContrast MediaContext (language use)HippocampusCorpus Callosum030218 nuclear medicine & medical imagingReduction (complexity)Magnetics03 medical and health sciences0302 clinical medicineThalamusAlzheimer Disease[INFO.INFO-IM]Computer Science [cs]/Medical ImagingAnimalsHumansComputer SimulationRadiology Nuclear Medicine and imagingPoint (geometry)Gray MatterComputingMilieux_MISCELLANEOUSBlock (data storage)Flexibility (engineering)Phantoms ImagingBrainContrast (statistics)Models TheoreticalOptimal controlMagnetic Resonance ImagingRatsFemaleAlgorithmAlgorithms030217 neurology & neurosurgeryMagnetic Resonance in Medicine
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Recessive mutations in EPG5 cause Vici syndrome, a multisystem disorder with defective autophagy

2013

Vici syndrome is a recessively inherited multisystem disorder characterized by callosal agenesis, cataracts, cardiomyopathy, combined immunodeficiency and hypopigmentation. To investigate the molecular basis of Vici syndrome, we carried out exome and Sanger sequence analysis in a cohort of 18 affected individuals. We identified recessive mutations in EPG5 (previously KIAA1632), indicating a causative role in Vici syndrome. EPG5 is the human homolog of the metazoan-specific autophagy gene epg-5, encoding a key autophagy regulator (ectopic P-granules autophagy protein 5) implicated in the formation of autolysosomes. Further studies showed a severe block in autophagosomal clearance in muscle a…

BiopsyVesicular Transport ProteinsAutophagy-Related ProteinsGenes RecessiveConsanguinityBiologymedicine.disease_causeArticleCataract03 medical and health sciencesConsanguinity0302 clinical medicineCataractsAntigens NeoplasmGeneticsmedicineAutophagyHumansVici syndromeExomeFamilyMuscle SkeletalExomeImmunodeficiency030304 developmental biologyGenetics0303 health sciencesMutationAutophagyIntracellular Signaling Peptides and ProteinsLysosome-Associated Membrane GlycoproteinsProteinsmedicine.diseaseMutationAutophagy Protein 5Agenesis of Corpus CallosumLysosomes030217 neurology & neurosurgeryNature genetics
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CNS involvement in OFD1 syndrome: a clinical, molecular, and neuroimaging study

2014

Background Oral-facial-digital type 1 syndrome (OFD1; OMIM 311200) belongs to the expanding group of disorders ascribed to ciliary dysfunction. With the aim of contributing to the understanding of the role of primary cilia in the central nervous system (CNS), we performed a thorough characterization of CNS involvement observed in this disorder. Methods A cohort of 117 molecularly diagnosed OFD type I patients was screened for the presence of neurological symptoms and/or cognitive/behavioral abnormalities on the basis of the available information supplied by the collaborating clinicians. Seventy-one cases showing CNS involvement were further investigated through neuroimaging studies and neur…

Central nervous systemNeuroimagingNeuropsychological TestsPharmacologyBioinformaticsSettore MED/03 - GENETICA MEDICACiliopathiesCohort Studies03 medical and health sciences0302 clinical medicineNeuroimagingCentral Nervous System DiseasesmedicineHumansGenetics(clinical)Pharmacology (medical)Orofaciodigital type 1Ciliopathies; Neurodevelopmental phenotype; Neuroimaging; OFD1; Central Nervous System Diseases; Cohort Studies; Female; Humans; Magnetic Resonance Imaging; Mutation; Neuropsychological Tests; Orofaciodigital Syndromes; Medicine (all); Genetics (clinical); Pharmacology (medical)Agenesis of the corpus callosumGenetics (clinical)030304 developmental biologyMedicine(all)0303 health sciencesbusiness.industryMedicine (all)ResearchCiliumNeuropsychologyCognitionGeneral MedicineOrofaciodigital Syndromesmedicine.diseasecentral nervous systemMagnetic Resonance ImagingPorencephalyCiliopathies3. Good healthmedicine.anatomical_structureMutationFemaleNeurodevelopmental phenotypeOFD1business030217 neurology & neurosurgeryOrphanet Journal of Rare Diseases
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Contralateral potentials in the suprasylvian gyrus evoked by pulvinar stimulation.

1974

AbstractIn encephale isole cats the transmission of the bioelectrical activity in the suprasylvian gyrus, evoked by contralateral pulvinar stimulation was explored. Through the excitability increase and decrease of the pulvinar (by strychnine or KCl, respectively), the surgical removal of the ipsilateral suprasylvian gyrus and the electrocoagulation of the posterior commissure it appeared that the transmission of the activity in the suprasylvian gyrus evoked by contralateral pulvinar stimulation occurs through both the callosal and the interthalamic pathway.

Cerebral CortexCATSPhysiologybusiness.industryBrainStimulationStrychnineStrychnineBiochemistryElectric StimulationCorpus CallosumPotassium Chloridechemistry.chemical_compoundmedicine.anatomical_structurePosterior commissureGyruschemistryThalamusSurgical removalmedicineCatsAnimalsbusinessNeuroscienceEvoked PotentialsArchives internationales de physiologie et de biochimie
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The role of the claustrum in the bilateral control of frontal oculomotor neurons in the cat.

1991

The effect of claustrum (CL) stimulation on the spontaneous unitary activity of ipsi and contralateral frontal oculomotor neurons, was studied in chloralose-anaesthetized cats. A total of 205 units was bilaterally recorded in the medial oculomotor area, homologous of the primate "frontal eye fields"; 127 neurons were identified as projecting to the superior colliculus; for 33 of these last units stimulation of the ipsilateral CL provoked an excitatory effect lasting 10-25 ms and appearing with a latency of 5-15 ms; on 8 units the excitatory effect was followed by an inhibition lasting 100-250 ms. Ninety-eight of the 127 neurons were also tested through activation of the contralateral CL: 13…

Cerebral CortexNeuronsSuperior ColliculiOculomotor nerveGeneral NeuroscienceSuperior colliculusElectroencephalographyAnatomyFrontal eye fieldsBiologyCorpus callosumClaustrumBasal GangliaElectric StimulationStereotaxic Techniquesmedicine.anatomical_structureOculomotor NerveCerebral cortexStereotaxic techniqueNeural PathwaysmedicineExcitatory postsynaptic potentialCatsAnimalsNeuroscienceExperimental brain research
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Satb2 Regulates Callosal Projection Neuron Identity in the Developing Cerebral Cortex

2008

SummarySatb2 is a DNA-binding protein that regulates chromatin organization and gene expression. In the developing brain, Satb2 is expressed in cortical neurons that extend axons across the corpus callosum. To assess the role of Satb2 in neurons, we analyzed mice in which the Satb2 locus was disrupted by insertion of a LacZ gene. In mutant mice, β-galactosidase-labeled axons are absent from the corpus callosum and instead descend along the corticospinal tract. Satb2 mutant neurons acquire expression of Ctip2, a transcription factor that is necessary and sufficient for the extension of subcortical projections by cortical neurons. Conversely, ectopic expression of Satb2 in neural stem cells m…

Chromatin ImmunoprecipitationNeuroscience(all)Electrophoretic Mobility Shift AssayMice TransgenicNerve Tissue ProteinsDEVBIOBiologyCorpus callosumMOLNEUROMiceNeural PathwaysmedicineAnimalsCells CulturedCerebral CortexNeuronsRegulation of gene expressionStem CellsGeneral NeuroscienceGene Expression Regulation DevelopmentalMatrix Attachment Region Binding ProteinsDNAEmbryo MammalianNeural stem cellChromatinmedicine.anatomical_structureAnimals NewbornBromodeoxyuridinenervous systemCerebral cortexRegulatory sequenceMutationCorticospinal tractEctopic expressionNeuroscienceTranscription Factors
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