Search results for "Crosses"

showing 10 items of 63 documents

Transcriptomic responses to biotic stresses in Malus x domestica: a meta-analysis study.

2017

AbstractRNA-Seq analysis is a strong tool to gain insight into the molecular responses to biotic stresses in plants. The objective of this work is to identify specific and common molecular responses between different transcriptomic data related to fungi, virus and bacteria attacks in Malus x domestica. We analyzed seven transcriptomic datasets in Malus x domestica divided in responses to fungal pathogens, virus (Apple Stem Grooving Virus) and bacteria (Erwinia amylovora). Data were dissected using an integrated approach of pathway- and gene- set enrichment analysis, Mapman visualization tool, gene ontology analysis and inferred protein-protein interaction network. Our meta-analysis revealed…

0106 biological sciences0301 basic medicineMalusKnowledge BasesArabidopsislcsh:MedicineSecondary MetabolismErwiniaGenes Plant01 natural sciencesArticleTranscriptome03 medical and health sciencesPlant Growth RegulatorsGene Expression Regulation PlantStress PhysiologicalSettore AGR/07 - Genetica AgrariaProtein Interaction Mapslcsh:ScienceSecondary metabolismGeneCrosses GeneticPlant ProteinsGeneticsMultidisciplinarybiologyGene Expression Profilinglcsh:RfungiMalus transcriptomic biotic stressfood and beveragesBiotic stressbiology.organism_classificationSettore AGR/03 - Arboricoltura Generale E Coltivazioni ArboreeGene expression profiling030104 developmental biologyGene OntologyMalustranscriptomic responses biotic stress meta-analysis RNA-seq plantsInactivation Metaboliclcsh:QTranscriptomeApple stem grooving virus010606 plant biology & botanyTranscription FactorsScientific reports
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Bacillus thuringiensis Vip3Aa Toxin Resistance in Heliothis virescens (Lepidoptera: Noctuidae)

2017

ABSTRACT Laboratory selection with Vip3Aa of a field-derived population of Heliothis virescens produced >2,040-fold resistance in 12 generations of selection. The Vip3Aa-selected (Vip-Sel)-resistant population showed little cross-resistance to Cry1Ab and no cross-resistance to Cry1Ac. Resistance was unstable after 15 generations without exposure to the toxin. F 1 reciprocal crosses between Vip3Aa-unselected (Vip-Unsel) and Vip-Sel insects indicated a strong paternal influence on the inheritance of resistance. Resistance ranged from almost completely recessive (mean degree of dominance [ h ] = 0.04 if the resistant parent was female) to incompletely dominant (mean h = 0.53 if the resistan…

0106 biological sciences0301 basic medicineMultifactorial Inheritancemedia_common.quotation_subjectPopulationInsectBiology01 natural sciencesApplied Microbiology and BiotechnologyInsecticide ResistanceLepidoptera genitaliaHemolysin Proteins03 medical and health sciencesBacterial ProteinsBacillus thuringiensisInvertebrate MicrobiologyAnimalsSelection GeneticeducationCrosses Geneticmedia_commonGeneticseducation.field_of_studyBacillus thuringiensis ToxinsEcologyHeliothis virescensfungibiology.organism_classificationSurvival AnalysisEndotoxinsLepidoptera010602 entomology030104 developmental biologyCry1AcPaternal InheritanceNoctuidaeBiological AssayPEST analysisFood ScienceBiotechnologyApplied and Environmental Microbiology
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Degradation of sexual reproduction in Veronica filiformis after introduction to Europe

2011

Abstract Background Baker’s law predicts that self-incompatible plant species are generally poor colonizers because their mating system requires a high diversity of genetically differentiated individuals and thus self-compatibility should develop after long-distance dispersal. However, cases like the introduction of the self-incompatible Veronica filiformis (Plantaginaceae) to Europe constitute an often overlooked alternative to this rule. This species was introduced from subalpine areas of the Pontic-Caucasian Mountains and colonized many parts of Central and Western Europe in the last century, apparently without producing seeds. To investigate the consequences of the absence of sexual rep…

0106 biological sciencesDNA PlantEvolutionIntroduced speciesFlowersBiologymedicine.disease_cause010603 evolutionary biology01 natural sciences03 medical and health sciencesGenetics (medical genetics to be 30107 and agricultural genetics to be 40402)PollenBotanyQH359-425medicineAmplified Fragment Length Polymorphism AnalysisOvuleCrosses GeneticEcology Evolution Behavior and Systematics030304 developmental biologyOvuleAnalysis of VarianceEvolutionary Biology0303 health sciencesGeographyEcologyObligateReproductionVeronica filiformisBotanyGenetic VariationSelf-Incompatibility in Flowering Plantsfood and beverages15. Life on landMating systembiology.organism_classificationVeronicaSexual reproductionEuropeSeedsPollenBiological dispersalIntroduced SpeciesResearch ArticleBMC Evolutionary Biology
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The dominance of the herbicide resistance cost in several Arabidopsis thaliana mutant lines

2004

Abstract Resistance evolution depends upon the balance between advantage and disadvantage (cost) conferred in treated and untreated areas. By analyzing morphological characters and simple fitness components, the cost associated with each of eight herbicide resistance alleles (acetolactate synthase, cellulose synthase, and auxin-induced target genes) was studied in the model plant Arabidopsis thaliana. The use of allele-specific PCR to discriminate between heterozygous and homozygous plants was used to provide insights into the dominance of the resistance cost, a parameter rarely described. Morphological characters appear more sensitive than fitness (seed production) because 6 vs. 4 differen…

0106 biological sciencesDNA PlantGenotypeArabidopsisDrug ResistanceDrug resistance[SDV.GEN] Life Sciences [q-bio]/GeneticsGenes Plant01 natural sciences03 medical and health sciencesGene FrequencyArabidopsisGenotypeGeneticsAlleleGeneCrosses GeneticComputingMilieux_MISCELLANEOUSGenes Dominant030304 developmental biologyDominance (genetics)Genetics[SDV.GEN]Life Sciences [q-bio]/Genetics0303 health sciencesAcetolactate synthaseBase SequencebiologyHerbicidesbiology.organism_classificationPhenotypeMutationbiology.proteinUnderdominanceResearch Article010606 plant biology & botany
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IL-17A/F in Leishmania major-resistant C57BL/6 mice.

2019

Proinflammatory IL-17 plays an important role in various diseases and defence against extracellular microorganisms. Healing of leishmaniasis is promoted by Th1/Tc1 cells, whereas Th2/Treg are associated with worsened disease outcome. In addition, high expression of IL-17A in Leishmania-susceptible BALB/c and artificial overexpression of IL-17A in T cells in resistant C57BL/6 mice worsened disease outcome. Since C57BL/6 mice lacking only IL-17A exhibited no phenotype, and IL-17A and IL-17F share similar receptors, but differentially regulate chemokine secretion, we studied mice lacking both IL-17A and IL-17F (IL-17A/F-/- ) in infections with Leishmania major. Interestingly, lesion volumes an…

0301 basic medicineC57BL/6CD4-Positive T-LymphocytesMaleDermatologyBiochemistryProinflammatory cytokineLesion030207 dermatology & venereal diseases03 medical and health sciencesMice0302 clinical medicineTh2 CellsmedicineAnimalsSecretionLeishmania majorReceptorMolecular BiologyIntraepithelial LymphocytesLeishmaniasisCrosses GeneticLeishmaniaMice Inbred BALB CbiologyInterleukin-17Th1 Cellsbiology.organism_classificationPhenotypeMice Inbred C57BL030104 developmental biologyPhenotypeChemokine secretionImmunologyDisease ProgressionCytokinesFemalemedicine.symptomExperimental dermatology
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A dual role of caspase-8 in triggering and sensing proliferation-associated DNA damage, a key determinant of liver cancer development.

2017

Summary Concomitant hepatocyte apoptosis and regeneration is a hallmark of chronic liver diseases (CLDs) predisposing to hepatocellular carcinoma (HCC). Here, we mechanistically link caspase-8-dependent apoptosis to HCC development via proliferation- and replication-associated DNA damage. Proliferation-associated replication stress, DNA damage, and genetic instability are detectable in CLDs before any neoplastic changes occur. Accumulated levels of hepatocyte apoptosis determine and predict subsequent hepatocarcinogenesis. Proliferation-associated DNA damage is sensed by a complex comprising caspase-8, FADD, c-FLIP, and a kinase-dependent function of RIPK1. This platform requires a non-apop…

0301 basic medicineGenome instabilityMaleliver; Hepatocellular carcinoma; DNA damage response; replication stress; apoptosisCancer ResearchDNA RepairCarcinogenesisFas-Associated Death Domain ProteinApoptosisurologic and male genital diseasesDNA damage responseDna Damage Response ; Apoptosis ; Hepatocellular Carcinoma ; Liver ; Replication StressHistonesMice0302 clinical medicineRisk FactorsFADDPhosphorylationCellular SenescenceCaspase 8biologyLiver Neoplasmshepatocellular carcinomaLiver regeneration3. Good healthHistoneOncologyReceptors Tumor Necrosis Factor Type I030220 oncology & carcinogenesisReceptor-Interacting Protein Serine-Threonine KinasesFemalebiological phenomena cell phenomena and immunityCell agingCarcinoma HepatocellularDNA damageDNA repairreplication stressCaspase 8liverArticleGenomic Instability03 medical and health sciencesAnimalsHepatectomyHumansCrosses GeneticCell ProliferationJNK Mitogen-Activated Protein KinasesCell BiologyLiver Regeneration030104 developmental biologyImmunologyChronic Diseasebiology.proteinCancer researchHepatocytesMyeloid Cell Leukemia Sequence 1 ProteinDNA Damage
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Epigenetic Control of Phenotypic Plasticity in the Filamentous Fungus Neurospora crassa

2016

Abstract Phenotypic plasticity is the ability of a genotype to produce different phenotypes under different environmental or developmental conditions. Phenotypic plasticity is a ubiquitous feature of living organisms, and is typically based on variable patterns of gene expression. However, the mechanisms by which gene expression is influenced and regulated during plastic responses are poorly understood in most organisms. While modifications to DNA and histone proteins have been implicated as likely candidates for generating and regulating phenotypic plasticity, specific details of each modification and its mode of operation have remained largely unknown. In this study, we investigated how e…

0301 basic medicineRNA-interferenssiGenotypeInvestigationsQH426-470MethylationModels BiologicalHistone methylationEpigenesis GeneticNeurospora crassaHistonesGene Knockout Techniques03 medical and health sciencesRNA interferenceHistone demethylationGene Expression Regulation FungalHistone methylationGeneticshistone deacetylationEpigeneticshistone methylationGenetikMolecular BiologyGeneCrosses GeneticGenetic Association StudiesGenetics (clinical)Histone deacetylationGeneticsAnalysis of VariancePhenotypic plasticityModels StatisticalDNA methylationNeurospora crassabiologyAcetylationbiology.organism_classificationDNA-metylaatioPhenotype030104 developmental biologyHistonereaction normMutationDNA methylationbiology.proteinta1181fungisienetAlgorithmsG3: Genes, Genomes, Genetics
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Insulin Dissociates the Effects of Liver X Receptor on Lipogenesis, Endoplasmic Reticulum Stress, and Inflammation

2016

IF 4.258; International audience; Diabetes is characterized by increased lipogenesis as well as increased endoplasmic reticulum (ER) stress and inflammation. The nuclear hormone receptor liver X receptor (LXR) is induced by insulin and is a key regulator of lipid metabolism. It promotes lipogenesis and cholesterol efflux, but suppresses endoplasmic reticulum stress and inflammation. The goal of these studies was to dissect the effects of insulin on LXR action. We used antisense oligonucleotides to knock down Lxr alpha in mice with hepatocytespecific deletion of the insulin receptor and their controls. We found, surprisingly, that knock-out of the insulin receptor and knockdown of Lxr alpha …

0301 basic medicinemedicine.medical_treatmentLipid-metabolismResistanceBiochemistryHepatitisMESH: HepatitisMESH: Endoplasmic Reticulum Stresspolycyclic compoundsInsulinGene-expressionPhospholipidsLiver X ReceptorsMice KnockoutbiologyMESH : Gene Expression RegulationFatty-acid synthesisfood and beveragesEndoplasmic Reticulum StressOrphan Nuclear ReceptorsCultured-cellsLipidsMESH: Gene Expression RegulationMESH : Endoplasmic Reticulum StressMessenger-rnaLiverMESH: Orphan Nuclear ReceptorsGene Knockdown TechniquesLipogenesisFemalelipids (amino acids peptides and proteins)Signal Transductionliver X receptormedicine.medical_specialtyLxr-alphaMice Transgenicdigestive systemPhospholipid transfer proteinGene Expression Regulation Enzymologic03 medical and health sciencesInsulin resistanceMESH : HepatitisLysophosphatidylcholine acyltransferaseInternal medicinemedicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyLiver X receptorMolecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyCrosses GeneticLipogenesisEndoplasmic reticulumInsulinElement-binding protein-1cMESH : LiverCell Biologymedicine.diseaseMESH : Orphan Nuclear ReceptorsReceptor InsulinMice Inbred C57BLInsulin receptor030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2Gene Expression RegulationNuclear receptorbiology.proteinUnfolded protein responseInsulin ResistanceMESH: Liver
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Genome-wide characterization of chromatin binding and nucleosome spacing activity of the nucleosome remodelling ATPase ISWI.

2010

The evolutionarily conserved ATP-dependent nucleosome remodelling factor ISWI can space nucleosomes affecting a variety of nuclear processes. In Drosophila, loss of ISWI leads to global transcriptional defects and to dramatic alterations in higher-order chromatin structure, especially on the male X chromosome. In order to understand if chromatin condensation and gene expression defects, observed in ISWI mutants, are directly correlated with ISWI nucleosome spacing activity, we conducted a genome-wide survey of ISWI binding and nucleosome positioning in wild-type and ISWI mutant chromatin. Our analysis revealed that ISWI binds both genic and intergenic regions. Remarkably, we found that ISWI…

Adenosine TriphosphatasesMaleChromatin ImmunoprecipitationX ChromosomeD. melanogasterSettore INF/01 - Informaticachromatin remodellingGenomicsChromatin Assembly and DisassemblyArticleNucleosomesDNA-Binding ProteinsISWInucleosome spacingGene Expression RegulationSettore BIO/10 - BiochimicaAnimalsDrosophila ProteinsDrosophilaPromoter Regions GeneticCrosses GeneticProtein BindingTranscription FactorsThe EMBO journal
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RESISTANCE AND TOLERANCE IN A HOST PLANT–HOLOPARASITIC PLANT INTERACTION: GENETIC VARIATION AND COSTS

2002

Host organisms are believed to evolve defense mechanisms (i.e., resistance and/or tolerance) under selective pressures exerted by natural enemies. A prerequisite for the evolution of resistance and tolerance is the existence of genetic variation in these traits for natural selection to act. However, selection for resistance and/or tolerance may be constrained by negative genetic correlations with other traits that affect host fitness. We studied genetic variation in resistance and tolerance against parasitic infection and the potential fitness costs associated with these traits using a novel study system, namely the interaction between a flowering plant and a parasitic plant. In this system…

Analysis of VarianceNatural selectionResistance (ecology)biologyHost (biology)Parasitic plantDefence mechanismsZoologyGenetic VariationUrtica dioicaCuscuta europaeaCuscutabiology.organism_classificationBiological EvolutionNatural population growthBotanyGenetic variationGeneticsParasitologyBiomassGeneral Agricultural and Biological SciencesCrosses GeneticEcology Evolution Behavior and SystematicsEvolution
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