Search results for "Cytoarchitecture"

showing 7 items of 7 documents

p73 is required for ependymal cell maturation and neurogenic SVZ cytoarchitecture

2015

The adult subventricular zone (SVZ) is a highly organized microenvironment established during the first postnatal days when radial glia cells begin to transform into type B-cells and ependymal cells, all of which will form regenerative units, pinwheels, along the lateral wall of the lateral ventricle. Here, we identify p73, a p53 homologue, as a critical factor controlling both cell-type specification and structural organization of the developing mouse SVZ. We describe that p73 deficiency halts the transition of the radial glia into ependymal cells, leading to the emergence of immature cells with abnormal identities in the ventricle and resulting in loss of the ventricular integrity. p73-de…

0301 basic medicineEpendymal CellCiliumNeurogenesisSubventricular zoneBiology03 medical and health sciencesCellular and Molecular NeuroscienceLateral ventricles030104 developmental biologymedicine.anatomical_structureDevelopmental NeuroscienceCytoarchitectureCiliogenesismedicineskin and connective tissue diseasesEpendymaneoplasmsNeuroscienceDevelopmental Neurobiology
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Characterization of the canine rostral ventricular-subventricular zone: Morphological, immunohistochemical, ultrastructural, and neurosphere assay st…

2017

The mammalian ventricular-subventricular zone (V-SVZ) presents the highest neurogenic potential in the brain of the adult individual. In rodents, it is mainly composed of chains of neuroblasts. In humans, it is organized in layers where neuroblasts do not form chains. The aim of this study is to describe the cytoarchitecture of canine V-SVZ (cV-SVZ), to assess its neurogenic potential, and to compare our results with those previously described in other species. We have studied by histology, immunohistochemistry (IHC), electron microscopy and neurosphere assay the morphology, cytoarchitecture and neurogenic potential of cV-SVZ. Age groups of animals were performed. Histological and ultrastru…

0301 basic medicineMalePathologymedicine.medical_specialtyanimal diseasesSubventricular zoneBiology03 medical and health sciences0302 clinical medicineDogsNeuroblastNeural Stem CellsSpecies SpecificityNeurospheremedicineSubependymal zoneAnimalsStem Cell NicheCells CulturedGeneral NeuroscienceNeurogenesisBrainHistologyImmunohistochemistryMicroscopy Electron030104 developmental biologymedicine.anatomical_structurenervous systemCytoarchitectureImmunohistochemistryFemale030217 neurology & neurosurgeryThe Journal of comparative neurology
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Effects of Nrf2 deficiency on bone microarchitecture in an experimental model of osteoporosis

2014

Objective. Redox imbalance contributes to bone fragility. We have evaluated the in vivo role of nuclear factor erythroid derived 2-related factor-2 (Nrf2), an important regulator of cellular responses to oxidative stress, in bone metabolism using a model of postmenopausal osteoporosis. Methods. Ovariectomy was performed in both wild-type and mice deficient in Nrf2 (Nrf2-/-). Bone microarchitecture was analyzed by CT. Serum markers of bone metabolism were also measured. Reactive oxygen species production was determined using dihydrorhodamine 123. Results. Sham-operated or ovariectomized Nrf2 -/- mice exhibit a loss in trabecular bone mineral density in femur, accompanied by a reduction in co…

Agingmedicine.medical_specialtycytoarchitectureArticle SubjectNF-E2-Related Factor 2MedicinaOsteoporosisOsteoclastsBone Marrow Cellsprotein deficiencymedicine.disease_causeenvironment and public healthBiochemistryBone resorptionBone remodelingMiceIn vivoInternal medicinemedicineAnimalsFemurcontrolled studyFemurlcsh:QH573-671Cells CulturedMice Knockoutchemistry.chemical_classificationReactive oxygen specieslcsh:CytologyChemistrybone densityCell DifferentiationCell BiologyGeneral Medicinerespiratory systemmedicine.diseaseosteoporosisMice Inbred C57BLDisease Models AnimalOxidative StressEndocrinologyOvariectomized ratReactive Oxygen SpeciesTomography X-Ray ComputedBiomarkersOxidative stressResearch Article
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Purkinje cell loss and motor coordination defects in profilin1 mutant mice.

2012

Profilin1 is an actin monomer-binding protein, essential for cytoskeletal dynamics. Based on its broad expression in the brain and the localization at excitatory synapses (hippocampal CA3-CA1 synapse, cerebellar parallel fiber (PF)-Purkinje cell (PC) synapse), an important role for profilin1 in brain development and synapse physiology has been postulated. We recently showed normal physiology of hippocampal CA3-CA1 synapses in the absence of profilin1, but impaired glial cell binding and radial migration of cerebellar granule neurons (CGNs). Consequently, brain-specific inactivation of profilin1 by exploiting conditional mutants and Nestin-mediated cre expression resulted in a cerebellar hyp…

CerebellumPatch-Clamp TechniquesPurkinje cellBiophysicsAction PotentialsParallel fiberMice TransgenicNerve Tissue ProteinsBiologyHippocampal formationIn Vitro TechniquesMotor ActivitySynapseNestinMiceProfilinsPurkinje CellsIntermediate Filament ProteinsmedicineAnimalsGeneral NeuroscienceAge FactorsBrainGene Expression Regulation DevelopmentalLong-term potentiationElectric StimulationDisease Models Animalmedicine.anatomical_structurenervous systemCytoarchitectureAnimals NewbornCerebellar cortexMutationDisease ProgressionPsychomotor DisordersNeuroscienceNeuroscience
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ID4 Is Required for Normal Ependymal Cell Development

2021

Ependymal cells are radial glia-derived multiciliated cells lining the lateral ventricles of the brain and spinal cord. Correct development and coordinated cilia beating is essential for proper cerebrospinal fluid (CSF) flow and neurogenesis modulation. Dysfunctions of ependymal cells were associated with transcription factor deregulation. Here we provide evidence that the transcriptional regulator ID4 is involved in ependymal cell development and maturation. We observed that Id4-deficient mice display altered ventricular cell cytoarchitecture, decreased ependymal cell number and enlarged ventricles. In addition, absence of ID4 during embryonic development resulted in decreased ependymal ce…

Ependymal Cell[SDV]Life Sciences [q-bio]Cèl·lulesbrainNeurosciences. Biological psychiatry. NeuropsychiatryBiology03 medical and health sciencesLateral ventriclesCerebrospinal fluid0302 clinical medicineTranscriptional regulationmedicineNeurociènciesTranscription factordevelopmenttranscription factor030304 developmental biology0303 health sciencesGeneral NeuroscienceCiliumEmbryogenesisNeurogenesisBrief Research ReportSpinal cordCell biology[SDV] Life Sciences [q-bio]medicine.anatomical_structureCytoarchitectureID4030217 neurology & neurosurgeryependymal cellRC321-571Neuroscience
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Ontogeny of the human amygdala.

2003

Data on the fetal development of the human amygdala is reviewed with special reference to major ontogenetic events. In the fifth gestational month, the inferior portion of the amygdala reveals cell-dense columns merging with the ganglionic eminence (proliferative zone) in Nissl-stained sections. These columns contain vimentin-positive fibers and can therefore be regarded as migrational routes. In the sixth and seventh months, distinct reorganization of the cytoarchitectonics takes place. The sequential occurrence of afferens can be visualized using anti-GAP-43; moreover, outgrowing axons appear to reach the periphery of the ganglionic eminence. The latter may thus represent an intermediate …

Ganglionic eminenceGeneral NeuroscienceGlutamate receptorGestational AgeNerve Tissue ProteinsAnatomyBiologyAmygdalaCalbindinAmygdalaGeneral Biochemistry Genetics and Molecular BiologyEmbryonic and Fetal Developmentmedicine.anatomical_structurenervous systemHistory and Philosophy of ScienceCytoarchitecturePostsynaptic potentialmedicineHumansCalretininNeuroscienceImmunostainingBiomarkersAnnals of the New York Academy of Sciences
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Preservation of glial cytoarchitecture from ex vivo human tumor and non-tumor cerebral cortical explants: A human model to study neurological diseases

2007

For the human brain, in vitro models that accurately represent what occurs in vivo are lacking. Organotypic models may be the closest parallel to human brain tissue outside of a live patient. However, this model has been limited primarily to rodent-derived tissue. We present an organotypic model to maintain intraoperatively collected human tumor and non-tumor explants ex vivo for a prolonged period of time (similar to 11 days) without any significant changes to the tissue cytoarchitecture as evidenced through immunohistochemistry and electron microscopy analyses. The ability to establish and reliably predict the cytoarchitectural changes that occur with time in an organotypic model of tumor…

Pathologymedicine.medical_specialtyIndolesTime FactorsbrainMatrix (biology)BiologyModels BiologicalStatistics NonparametricArticleOrgan Culture TechniquesMicroscopy Electron TransmissionIn vivoGlial Fibrillary Acidic ProteinmedicineHumanshumanorganotypicCerebral Cortexelectron microscopyBrain NeoplasmsGeneral NeuroscienceexplantReproducibility of ResultsCell migrationHuman brainMiddle AgedImmunohistochemistrymedicine.anatomical_structureCytoarchitectureImmunohistochemistryFemaleTissue PreservationNervous System DiseasesNeurogliaEx vivoExplant culture
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