Search results for "Cytokines"

showing 10 items of 845 documents

LDHA-Associated Lactic Acid Production Blunts Tumor Immunosurveillance by T and NK Cells

2015

Elevated lactate dehydrogenase A (LDHA) expression is associated with poor outcome in tumor patients. Here we show that LDHA-associated lactic acid accumulation in melanomas inhibits tumor surveillance by T and NK cells. In immunocompetent C57BL/6 mice, tumors with reduced lactic acid production (Ldhalow) developed significantly slower than control tumors and showed increased infiltration with IFN-γ-producing T and NK cells. However, in Rag2-/-γc-/- mice, lacking lymphocytes and NK cells, and in Ifng-/- mice, Ldhalow and control cells formed tumors at similar rates. Pathophysiological concentrations of lactic acid prevented upregulation of nuclear factor of activated T cells (NFAT) in T and…

Male0301 basic medicineCell SurvivalPhysiologyT-LymphocytesT cellApoptosisCell CountCD8-Positive T-LymphocytesBiologySodium LactateInterferon-gamma03 medical and health scienceschemistry.chemical_compoundInterleukin 21Downregulation and upregulationCell Line TumormedicineAnimalsHumansLactic AcidImmunologic SurveillanceMelanomaMolecular BiologyCell ProliferationL-Lactate DehydrogenaseNFATC Transcription FactorsNFATCell Biologymedicine.diseaseUp-RegulationLactic acidIsoenzymesKiller Cells NaturalMice Inbred C57BLPhenotype030104 developmental biologymedicine.anatomical_structurechemistryCell cultureImmunologyCancer researchInterleukin 12CytokinesLactate Dehydrogenase 5GlycolysisInfiltration (medical)Cell Metabolism
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Pairing Binge Drinking and a High-Fat Diet in Adolescence Modulates the Inflammatory Effects of Subsequent Alcohol Consumption in Mice

2021

This article belongs to the Special Issue Gut Microbiota and Immunity.

Male0301 basic medicineChemokine CXCL1Self AdministrationBinge drinkingAlcoholStriatumGut floraMicechemistry.chemical_compound0302 clinical medicineBiology (General)BingeSpectroscopybiologyalcoholMicrobiotadigestive oral and skin physiologyAge Factorsfood and beveragesGeneral MedicineComputer Science ApplicationsChemistryHigh-fat diethigh-fat dietCytokinesbingemedicine.symptomAlcoholmedicine.medical_specialtyAlcohol DrinkingQH301-705.5Binge drinkingInflammationDiet High-Fatdigestive systemArticleCatalysisInorganic Chemistry03 medical and health sciencesInternal medicineAnimals Outbred StrainsmicrobiotamedicineAnimalsObesityPhysical and Theoretical ChemistryQD1-999Molecular BiologyNeuroinflammationInflammationEthanolEthanolInterleukin-6business.industryOrganic Chemistrybiochemical phenomena metabolism and nutritionbiology.organism_classificationmedicine.diseaseObesitybinge drinkingcytokinesstomatognathic diseases030104 developmental biologyEndocrinologychemistryinflammationbacteriabusiness030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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Malnutrition impairs mitochondrial function and leukocyte activation

2019

Abstract Background The aim of this study was to evaluate markers of inflammation, oxidative stress and endothelial function in a disease-related malnutrition (DRM) outpatient population. Methods For this cross-sectional study, a total of 83 subjects were included and clustered in 3 groups: 34 with normonutrition (NN), 21 with DRM without inflammation (DRM-I) and 28 with DRM and inflammation (DRM + I). Nutritional diagnosis was conducted for all subjects according to ASPEN. Biochemical parameters, proinflammatory cytokines, reactive oxygen species production, glutathione, mitochondrial membrane potential, oxygen consumption, adhesion molecules and leukocyte-endothelium interactions were eva…

Male0301 basic medicineMedicine (miscellaneous)Mitochondrionmedicine.disease_causechemistry.chemical_compound0302 clinical medicineLeukocytesDisease-related malnutrition030212 general & internal medicineEndothelial dysfunctionlcsh:RC620-627Membrane Potential Mitochondrialchemistry.chemical_classificationeducation.field_of_studyNutrition and DieteticsMiddle AgedGlutathioneMitochondrialcsh:Nutritional diseases. Deficiency diseasesCytokinesFemalelcsh:Nutrition. Foods and food supplymedicine.medical_specialtyPopulationlcsh:TX341-641Proinflammatory cytokine03 medical and health sciencesInternal medicineCell AdhesionmedicineHumanseducationOutpatient populationAgedInflammationReactive oxygen species030109 nutrition & dieteticsbusiness.industryResearchMalnutritionEndothelial functionGlutathionemedicine.diseaseOxygenCross-Sectional StudiesEndocrinologychemistrySpainTransferrinOxidative stressReactive Oxygen SpeciesbusinessOxidative stressNutrition Journal
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Mesenchymal stem cells derived from inflamed dental pulpal and gingival tissue: a potential application for bone formation

2017

Background Chronic periodontal disease is an infectious disease consisting of prolonged inflammation of the supporting tooth tissue and resulting in bone loss. Guided bone regeneration procedures have become common and safe treatments in dentistry, and in this context dental stem cells would represent the ideal solution as autologous cells. In this study, we verified the ability of dental pulp mesenchymal stem cells (DPSCs) and gingival mesenchymal stem cells (GMSCs) harvested from periodontally affected teeth to produce new mineralized bone tissue in vitro, and compared this to cells from healthy teeth. Methods To characterize DPSCs and GMSCs, we assessed colony-forming assay, immunophenot…

Male0301 basic medicinePathologyCellular differentiationGingivaMedicine (miscellaneous)Bone tissue0302 clinical medicineOsteogenesisMedicinelcsh:QD415-436Pulpal and gingival mesenchymal stem cellsCells CulturedStem cell transplantation for articular cartilage repairlcsh:R5-920Heat shock proteinCell DifferentiationADFsMiddle AgedGingivitismedicine.anatomical_structureBone formationMolecular MedicineFemaleStem celllcsh:Medicine (General)Adultmedicine.medical_specialtyAdolescentBiochemistry Genetics and Molecular Biology (miscellaneous)Proinflammatory cytokinelcsh:Biochemistry03 medical and health sciencesstomatognathic systemHumansPeriodontitisBone regenerationDental PulpAgedProinflammatory cytokinesInflammationbusiness.industryResearchMesenchymal stem cellMesenchymal Stem Cells030206 dentistryCell BiologyDental diseaseInflammation Dental disease Pulpal and gingival mesenchymal stem cells Bone formation Heat shock protein ADFs Proinflammatory cytokinesstomatognathic diseases030104 developmental biologyCancer researchPulp (tooth)businessStem Cell Research & Therapy
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Anti-Inflammatory Effects of the Mediterranean Diet in the Early and Late Stages of Atheroma Plaque Development

2017

Objective.To evaluate the long-term effects of a Mediterranean diet (MeDiet) intervention on the plasma concentrations of inflammatory and plaque stability-related molecules in elderly people at high risk for cardiovascular disease.Design and Setting. 66 participants from primary care centers affiliated with the Hospital Clinic of Barcelona were randomized into 3 groups: MeDiet plus extra virgin olive oil (EVOO) or nuts and a low-fat diet (LFD). At baseline and at 3 and 5 years, we evaluated the changes in the plasma concentrations of 24 inflammatory biomarkers related to the different stages of the atherosclerotic process by Luminex®.Results.At 3 and 5 years, both MeDiet groups showed a si…

Male0301 basic medicinePathologyMediterranean dietHigh-risk030204 cardiovascular system & hematologyDiet MediterraneanDieta mediterràniaGastroenterology0302 clinical medicineRisk FactorsElderly peopleChemokine CCL4Chemokine CCL5Diet Fat-RestrictedAged 80 and overBioquímica y tecnologíaInterleukin-18Middle AgedInflammatory biomarkersPlaque AtheroscleroticInterleukin-10Lipoproteins LDLBiochemistry and technologyCardiovascular diseasesCardiovascular DiseasesPlasma concentrationCytokinesFemalelcsh:RB1-214medicine.medical_specialtyArticle Subjectmedicine.drug_classImmunologyVascular Cell Adhesion Molecule-1ActivationPrimary careBioquímica i biotecnologiaLow-grade inflammationAnti-inflammatory03 medical and health sciencesMediterranean cookingElderly personsCardiovascular-diseaseInternal medicineCuina mediterrànialcsh:PathologymedicineHumansAgedMarkers030109 nutrition & dieteticsInterleukin-6Malalties cardiovascularsbusiness.industryPatternCell Biologymedicine.diseaseAtherosclerosisAtheromaClinical Study0962-9351Placa ateroscleròticabusiness
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H-ferritin and CD68+/H-ferritin+ monocytes/macrophages are increased in the skin of adult-onset Still's disease patients and correlate with the multi…

2016

Summary Adult-onset Still's disease (AOSD) patients may show an evanescent salmon-pink erythema appearing during febrile attacks and reducing without fever. Some patients may experience this eruption for many weeks. During AOSD, exceptionally high serum levels of ferritin may be observed; it is an iron storage protein composed of 24 subunits, heavy (H) subunits and light (L) subunits. The ferritin enriched in L subunits (L-ferritin) and the ferritin enriched in H subunits (H-ferritin) may be observed in different tissues. In this work, we aimed to investigate the skin expression of both H-and L-ferritin and the number of macrophages expressing these molecules from AOSD patients with persist…

Male0301 basic medicinePathologymedicine.medical_specialtyAdult-onset Still's diseaseDermal immune systemErythemaMacrophageBiopsyImmunologyAntigens Differentiation MyelomonocyticGene ExpressionDiseaseAdult-onset Still's diseaseMonocytesH-Ferritin03 medical and health sciences0302 clinical medicineAntigens CDadult-onset Still's disease; dermal immune system; ferritin; hyperferritinaemic syndrome; macrophagemedicineHumansImmunology and AllergyMonocytes macrophagesMacrophageRNA MessengerSkin030203 arthritis & rheumatologyFerritinbiologyCD68MacrophagesOriginal ArticlesFerritinSettore MED/16 - Reumatologia030104 developmental biologyApoferritinsImmunologyLeukocytes Mononuclearbiology.proteinCytokinesFemaleInflammation Mediatorsmedicine.symptomHyperferritinaemic syndromeStill's Disease Adult-OnsetBiomarkersClinical and Experimental Immunology
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Regulatory T cell deficient scurfy mice exhibit a Th2/M2-like inflammatory response in the skin

2017

Abstract Background Scurfy mice have a functional defect in regulatory T cells (Treg), which leads to lethal multi-organ inflammation. The missing Treg function results in uncontrolled autoimmune cellular and humoral inflammatory responses. We and others have previously shown that during the course of disease scurfy mice develop severe skin inflammation and autoantibodies including anti-nuclear autoantibodies (ANA). Objective Autoimmune skin inflammation and ANA are hallmarks for the diagnosis of autoimmune connective tissue diseases; therefore we analyzed scurfy mice for typical signs of these diseases. Methods Indirect immunofluorescence was used to specify the ANA pattern in scurfy mice.…

Male0301 basic medicinePathologymedicine.medical_specialtyRegulatory T cellCD3Fluorescent Antibody TechniqueConnective tissueDermatitisInflammationDermatologyT-Lymphocytes RegulatoryBiochemistrySclerodermaAutoimmune DiseasesMice03 medical and health sciencesMixed connective tissue diseaseFibrosismedicineAnimalsMolecular BiologySkinCell NucleusScleroderma SystemicTissue Inhibitor of Metalloproteinase-1biologybusiness.industryMacrophagesAutoantibodyForkhead Transcription FactorsMacrophage ActivationFlow Cytometrymedicine.diseaseFibrosisUp-Regulation030104 developmental biologymedicine.anatomical_structureAntibodies AntinuclearImmunologybiology.proteinCytokinesFemaleCollagenmedicine.symptombusinessJournal of Dermatological Science
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Differential impact of high and low penetrance TNFRSF1A gene mutations on conventional and regulatory CD4+ T cell functions in TNFR1-associated perio…

2015

Abstract TNFR-associated periodic syndrome is an autoinflammatory disorder caused by autosomal-dominant mutations in TNFRSF1A, the gene encoding for TNFR superfamily 1A. The lack of knowledge in the field of TNFR-associated periodic syndrome biology is clear, particularly in the context of control of immune self-tolerance. We investigated how TNF-α/TNFR superfamily 1A signaling can affect T cell biology, focusing on conventional CD4+CD25− and regulatory CD4+CD25+ T cell functions in patients with TNFR-associated periodic syndrome carrying either high or low penetrance TNFRSF1A mutations. Specifically, we observed that in high penetrance TNFR-associated periodic syndrome, at the molecular le…

Male0301 basic medicinePenetranceAutoimmunitymedicine.disease_causeT-Lymphocytes RegulatoryImmune toleranceSettore MED/38 - Pediatria Generale E SpecialisticaTRAPS; Tconvs; Tregs; autoimmunity; immune toleranceImmunology and AllergyIL-2 receptorChildGeneticsMutationTconvTOR Serine-Threonine Kinaseshemic and immune systemsMiddle AgedAcquired immune systemPenetranceTregSTAT Transcription Factorsmedicine.anatomical_structureReceptors Tumor Necrosis Factor Type ICytokinesFemalebiological phenomena cell phenomena and immunitySignal TransductionAdultAdolescentFeverT cellAutoimmunity; Immune tolerance; Tconvs; Tregs; TRAPS; Cell Biology; ImmunologyImmunologyReceptors Antigen T-CellContext (language use)Tregs[object Object]BiologyImmunophenotypingYoung Adult03 medical and health sciencesImmune systemmedicineHumansAgedCell ProliferationDemographyTconvsImmune toleranceHereditary Autoinflammatory DiseasesTRAPSCell Biologybiological factors030104 developmental biologyMutationCancer research
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Relationships of growth factors, proinflammatory cytokines, and anti-inflammatory cytokines with long-term clinical results of autologous bone marrow…

2017

Aim The aim of the study was to test the hypothesis suggesting that the pre-intervention levels of proinflammatory cytokines, anti-inflammatory cytokines, and angiogenic growth factors predict the long-term clinical results of autologous bone marrow-derived mononuclear cell (ABMMC) transplantation in patients with primary ST elevation myocardial infarction (STEMI). Methods and results From 2003 to 2006, a total of 62 patients with primary STEMI were enrolled in an open randomized study registered under the title ESTABOMA. Patients were randomized into two groups: group 1 included patients treated with percutaneous coronary intervention (PCI) and ABMMC transplantation (n = 28); group 2 compr…

Male0301 basic medicinePhysiologyCardiovascular ProceduresCell Transplantationmedicine.medical_treatmentMyocardial InfarctionSocial Scienceslcsh:Medicine030204 cardiovascular system & hematologyPathology and Laboratory MedicineVascular MedicineGastroenterologyAnginaEndocrinology0302 clinical medicineImmune PhysiologyMedicine and Health SciencesBlood and Lymphatic System ProceduresPsychologyMyocardial infarctionlcsh:ScienceImmune ResponseBone Marrow TransplantationInnate Immune SystemMultidisciplinaryCardiac TransplantationAnginaMiddle AgedBrain natriuretic peptidesurgical procedures operativeCytokinesIntercellular Signaling Peptides and ProteinsFemaleInflammation MediatorsResearch Articlemedicine.medical_specialtySocial PsychologyImmunologyCardiologySurgical and Invasive Medical ProceduresTransplantation AutologousProinflammatory cytokine03 medical and health sciencesSigns and SymptomsDiagnostic MedicineGrowth FactorsInternal medicineчрескожное коронарное вмешательствоmedicineHumanscardiovascular diseasesInflammationTransplantationEndocrine Physiologybusiness.industrylcsh:RBiology and Life SciencesPercutaneous coronary interventionOrgan TransplantationMolecular Developmentmedicine.diseaseпровоспалительные цитокиныинфаркт миокардаTransplantation030104 developmental biologyImmune SystemHeart failureImmunologyConventional PCIтрансплантация мононуклеарных клетокlcsh:Qbusinessаутологичная трансплантация костного мозгаDevelopmental BiologyStem Cell Transplantation
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Identification of periplakin as a major regulator of lung injury and repair in mice

2018

IF 12.784 (2016); International audience; Periplakin is a component of the desmosomes that acts as a cytolinker between intermediate filament scaffolding and the desmosomal plaque. Periplakin is strongly expressed by epithelial cells in the lung and is a target antigen for autoimmunity in idiopathic pulmonary fibrosis. The aim of this study was to determine the role of periplakin during lung injury and remodeling in a mouse model of lung fibrosis induced by bleomycin. We found that periplakin expression was downregulated in the whole lung and in alveolar epithelial cells following bleomycin-induced injury. Deletion of the Ppl gene in mice improved survival and reduced lung fibrosis developm…

Male0301 basic medicinePulmonologylcsh:MedicineMouse modelsMiceIdiopathic pulmonary fibrosischemistry.chemical_compoundFibrosisPeriplakinMice KnockoutLung InjuryGeneral Medicinerespiratory system3. Good healthmedicine.anatomical_structureCytokinesmedicine.symptomSignal TransductionResearch ArticleCell signalingDown-RegulationInflammationRespiratory MucosaLung injuryBleomycinBleomycin03 medical and health sciencesmedicineAnimalsHumansInflammationLung030102 biochemistry & molecular biologybusiness.industryMacrophagesPlakinslcsh:Rmedicine.diseaseFibrosisIdiopathic Pulmonary Fibrosisrespiratory tract diseasesMice Inbred C57BLDisease Models Animal030104 developmental biologychemistryAlveolar Epithelial CellsCancer researchbusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyJCI Insight
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