Search results for "Cytokines"

showing 10 items of 845 documents

Nanosecond pulsed electric field inhibits malignant melanoma growth by inducing the change of systemic immunity

2019

Background Nanosecond pulsed electric fields (nsPEFs) showed an inhibitory effect on proliferation of malignant melanoma. In this study, the growth of melanoma were inhibited by changing the systemic immunity. Material and Methods C57BL/6 mice with B16 malignant were exposed to 200 pulses of 100 ns duration, 30kV/cm. The mice were executed four days later. T lymphocyte has been extracted from spleen. Cell viability was evaluated by CCK-8 assay. CD3+CD4+ T cells, CD3+CD8+ T cells, regulatory T cells (Treg) and myeloid-derived suppressor cells (MDSC) were analyzed by flow cytometry. TNF-α, IL-2, IL-10, TGF-β, IFN– γ levels in supernatants were assessed by ELISA. Results C57 malignant melanoma…

Skin NeoplasmsCD3T-LymphocytesSpleenFlow cytometry03 medical and health sciencesMice0302 clinical medicineImmune systemmedicineAnimalsViability assayGeneral DentistryMelanomabiologymedicine.diagnostic_testChemistryMelanomaResearch030206 dentistryT lymphocytemedicine.disease:CIENCIAS MÉDICAS [UNESCO]Molecular biologyMice Inbred C57BLmedicine.anatomical_structureOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASbiology.proteinCytokinesSurgeryOral SurgeryCD8
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Post-Transcriptional Regulation of Human Inducible Nitric-Oxide Synthase Expression by the Jun N-terminal Kinase

2007

Human inducible nitric-oxide synthase (iNOS) expression is regulated both at transcriptional and post-transcriptional levels. In the present study, the effect of Jun N-terminal kinase (JNK) on human iNOS expression was investigated. In A549/8 human alveolar epithelial cells, both the inhibition of JNK by a pharmacological inhibitor anthra[1,9-cd]pyrazol-6(2H)-one1,9-pyrazoloanthrone (SP600125) and small interfering RNA (siRNA)-mediated down-regulation of JNK led to a reduction of iNOS mRNA and protein expression. iNOS promoter activity was not affected by these treatments. Hence, JNK seems to regulate iNOS expression through post-transcriptional mechanisms by stabilizing iNOS mRNA. Our labo…

Small interfering RNARNA Stabilityp38 mitogen-activated protein kinasesDown-RegulationNitric Oxide Synthase Type IIRNA-binding proteinNitric Oxidep38 Mitogen-Activated Protein KinasesGene Expression Regulation EnzymologicCell LineTristetraprolinHumansPhosphorylationRNA Small InterferingPromoter Regions GeneticPost-transcriptional regulationAnthracenesPharmacologyRegulation of gene expressionMessenger RNAbiologyChemistryKinaseJNK Mitogen-Activated Protein KinasesEpithelial Cellsrespiratory systemMolecular biologyPulmonary AlveoliNitric oxide synthasebiology.proteinCytokinesMolecular MedicineSignal TransductionMolecular Pharmacology
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Sorafenib perpetuates cellular anti-cancer effector functions by modulating the cross talk between macrophages and natural killer cells.

2012

Alternatively polarized macrophages (Mϕ) shape the microenvironment of hepatocellular carcinoma (HCC) and temper anticancer immune responses. We investigated if sorafenib alters the HCC microenvironment by restoring classical macrophage polarization and triggering tumor-directed natural killer (NK) cell responses. In vivo experiments were conducted with sorafenib (25 mg/kg)-treated C57BL/6 wildtype as well as hepatitis B virus (HBV) and lymphotoxin transgenic mice with and without HCC. Monocyte-derived Mϕ or tumor-associated macrophages (TAM) isolated from HCC tissue were treated with sorafenib (0.07-5.0 μg/mL) and cocultured with autologous NK cells. Mϕ and NK cell activation was analyzed …

SorafenibNiacinamideCarcinoma Hepatocellularmedicine.medical_treatmentMacrophage polarizationDrug Evaluation PreclinicalAntineoplastic AgentsApoptosisBiologyMiceliver cancer; therapy; microenvironment; immunology; HCCmedicineAnimalsHumansneoplasmsHepatologyMacrophagesPhenylurea CompoundsLiver NeoplasmsDegranulationNF-kappa BInterleukinMacrophage ActivationSorafenibdigestive system diseasesKiller Cells NaturalMice Inbred C57BLCytokineLymphotoxinImmunologyCancer researchInterleukin 12CytokinesInterleukin 18medicine.drug
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TAFA4 relieves injury-induced mechanical hypersensitivity through LDL receptors and modulation of spinal A-type K+ current

2021

Pain, whether acute or persistent, is a serious medical problem worldwide. However, its management remains unsatisfactory, and new analgesic molecules are required. We show here that TAFA4 reverses inflammatory, postoperative, and spared nerve injury (SNI)-induced mechanical hypersensitivity in male and female mice. TAFA4 requires functional low-density lipoprotein receptor-related proteins (LRPs) because their inhibition by RAP (receptor-associated protein) dose-dependently abolishes its antihypersensitive actions. SNI selectively decreases A-type K+ current (IA) in spinal lamina II outer excitatory interneurons (L-IIo ExINs) and induces a concomitant increase in IA and decrease in hyperpo…

Spinal Cord Dorsal HornSNiSpinal neuron[SDV]Life Sciences [q-bio]AnalgesicPainCHO CellsPharmacologyInhibitory postsynaptic potentialGeneral Biochemistry Genetics and Molecular BiologyTAFA4Mice03 medical and health sciencesCricetulus0302 clinical medicineAnimalsHumansMedicine030304 developmental biologyLDL-receptors0303 health sciencesbusiness.industryIA and IhNerve injury3. Good healthHEK293 CellsRAW 264.7 CellsReceptors LDLHyperalgesiainjury-induced mechanical painLDL receptorPotassiumExcitatory postsynaptic potentialCytokinesgating neuronsmedicine.symptomRAPbusiness030217 neurology & neurosurgeryLipoproteinCell Reports
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3D Morphology, ultrastructure and development of Ceratomyxa puntazzi stages: first insights into the mechanisms of motility and budding in the Myxozo…

2012

Free, amoeboid movement of organisms within media as well as substrate-dependent cellular crawling processes of cells and organisms require an actin cytoskeleton. This system is also involved in the cytokinetic processes of all eukaryotic cells. Myxozoan parasites are known for the disease they cause in economical important fishes. Usually, their pathology is related to rapid proliferation in the host. However, the sequences of their development are still poorly understood, especially with regard to pre-sporogonic proliferation mechanisms. The present work employs light microscopy (LM), electron microscopy (SEM, TEM) and confocal laser scanning microscopy (CLSM) in combination with specific…

SporesIndolesPhalloidineParasitic Diseases AnimalBiophysicsMotilitylcsh:MedicineBiologyBiochemistryFish DiseasesMicroscopy Electron TransmissionCell MovementMolecular Cell BiologyOxazinesAnimalsBilePseudopodiaMyxozoaCytoskeletonlcsh:ScienceBiologyCell ProliferationAmoeboid movementBuddingLife Cycle StagesMultidisciplinaryMicroscopy ConfocalStaining and LabelingPhysicslcsh:RProteinsCell BiologyActin cytoskeletonCellular StructuresSea BreamCell biologyUltrastructureMicroscopy Electron Scanninglcsh:QFilopodiaZoologyCytokinesisCell DivisionResearch ArticleDevelopmental BiologyPLoS ONE
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Selective killing of human monocytes and cytokine release provoked by sphingomyelinase (beta-toxin) of Staphylococcus aureus.

1996

The best-known activity of Staphylococcus aureus sphingomyelinase C, alias beta-toxin, is as a hemolysin that provokes hot-cold lysis of erythrocytes which contain substantial amounts of sphingomyelin in the plasma membrane. Sheep erythrocytes are most susceptible, and we found that one hemolytic unit, representing the toxin concentration that elicits 50% hemolysis of 2.5 X 10(8) erythrocytes per ml, corresponds to 0.05 enzyme units or to approximately 0.25 microg of sphingomyelinase per ml. The cytotoxic action of beta-toxin on nucleated cells has not been described in any detail before, and the present investigation was undertaken to fill this information gap. We now identify beta-toxin a…

Staphylococcus aureusTime FactorsLipopolysaccharideCD14ImmunologyBacterial ToxinsLipopolysaccharide ReceptorsExotoxinsMicrobiologyMonocytesMicrobiologychemistry.chemical_compoundHemolysin ProteinsPhospholipase A2Antigens CDmedicineHumansbiologyCell DeathDose-Response Relationship DrugCytotoxinsMonocyteHemolysinReceptors Interleukinmedicine.diseaseReceptors Interleukin-6HemolysisInfectious Diseasesmedicine.anatomical_structureSphingomyelin PhosphodiesteraseMechanism of actionchemistrybiology.proteinCytokinesParasitologymedicine.symptomSphingomyelinResearch ArticleInterleukin-1
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Reversible effect of MR and ELF magnetic fields (0.5 T and 0.5 mT) on human lymphocyte activation patterns.

2006

Purpose: The aim of this study was to investigate the effects of magnetic fields (MF) of different intensity generated by a magnetic resonance (MR) unit (0.5 Tesla) and a double cylindrical coil (0.5 mTesla) on human CD4+ T cell lines. Materials and methods: CD4+ T cells were exposed for two hours under isothermal conditions (37 ± 0.5°C) to the above mentioned MF; a control group was provided for each exposed sample. After exposure, the samples were analysed in the laboratory for the following endpoints: Release of cytokines, expression of surface markers, cell proliferation and levels of cytosolic free-calcium. Results: Exposure to MF for 2 h and subsequent in vitro stimulation in the pres…

T cellBiologyLymphocyte ActivationRadiation DosageCell LineNuclear magnetic resonanceElectricitymedicineHumansRadiology Nuclear Medicine and imagingIL-2 receptorLymphocytesMR Biological effectsCell ProliferationRadiological and Ultrasound Technologymedicine.diagnostic_testCell growthMagnetic resonance imagingDose-Response Relationship RadiationMolecular biologyAdaptation PhysiologicalMagnetic Resonance ImagingIn vitroMagnetic fieldCytosolmedicine.anatomical_structureMitogen-activated protein kinasebiology.proteinCytokines
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Neuronal nitric oxide synthase modulates maturation of human dendritic cells.

2010

AbstractDendritic cells (DCs) are the most potent APCs of the immune system. Understanding the intercellular and intracellular signaling processes that lead to DC maturation is critical for determining how these cells initiate T cell-mediated immune processes. NO synthesized by the inducible NO synthase (iNOS) is important for the function of murine DCs. In our study, we investigated the regulation of the arginine/NO-system in human monocyte-derived DCs. Maturation of DCs induced by inflammatory cytokines (IL-1β, TNF, IL-6, and PGE2) resulted in a pronounced expression of neuronal NOS (nNOS) but only minimal levels of iNOS and endothelial NOS were detected in human mature DCs. In addition, …

T cellCellular differentiationImmunologyImmunoblottingchemical and pharmacologic phenomenaEnzyme-Linked Immunosorbent AssayCell SeparationNitric Oxide Synthase Type IBiologyEndothelial NOSLymphocyte ActivationNitric OxideProinflammatory cytokineCell LineImmune systemmedicineImmunology and AllergyHumansAutocrine signallingMHC class IIReverse Transcriptase Polymerase Chain ReactionCell DifferentiationDendritic CellsFlow CytometryCell biologymedicine.anatomical_structureCell culturebiology.proteinCytokinesJournal of immunology (Baltimore, Md. : 1950)
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Phenotype and function of monocyte derived dendritic cells in chronic hepatitis B virus infection.

2004

The antiviral T cell failure of patients with chronic hepatitis B virus (HBV) infection was suggested to be caused by a T cell stimulation defect of dendritic cells (DC). To address this hypothesis, monocyte derived DC (MDDC) of patients with chronic or resolved acute HBV infection and healthy controls were studied phenotypically by FACS analyses and functionally by mixed lymphocyte reaction, ELISA, ELISpot and proliferation assays of MDDC cultures or co-cultures with an allogeneic HBc-specific Th cell clone. HBV infection of MDDC was studied by quantitative PCR. MDDC from HBV patients seemed to be infected by the HBV, showed a reduced surface expression of HLA DR and CD40 and exhibited a r…

T cellHLA-DR7 Antigenmedicine.disease_causeMonocytesHepatitis B ChronicVirologymedicineHumansCD40 AntigensHepatitis B virusCD40biologyMonocyteELISPOTvirus diseasesDendritic cellDendritic CellsMixed lymphocyte reactionVirologydigestive system diseasesHBcAgmedicine.anatomical_structurePhenotypeImmunologyDNA Viralbiology.proteinCytokinesThe Journal of general virology
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Induction of tumor peptide-specific cytotoxic T cells under serum-free conditions by mature human dendritic cells

2000

Tumor vaccination strategies using antigen-pulsed dendritic cells (DC) are currently under development. We established an in vitro system using cultured DC from HLA-typed volunteers for the induction of tumor peptide-specific CD8+ T cells. The strength and specificity of the resulting CTL responses were investigated. For stimulation of syngeneic CD8+ T cells two well-defined DC populations were generated: CD1a+ immature DC cultured in the presence of GM-CSF and IL-4 and mature CD83+ DC generated by additional stimulation with a cytokine cocktail. Stimulations were performed under serum-free conditions and in the absence of exogenous cytokines. Analysis of T cell responses showed that mature…

T cellImmunoglobulinsPriming (immunology)DermatologyDendritic cell differentiationCD8-Positive T-LymphocytesBiologyCulture Media Serum-FreeInterleukin 21Antigens CDmedicineHumansCytotoxic T cellCells CulturedCellular SenescenceMembrane GlycoproteinsCell DifferentiationDendritic CellsGeneral MedicineDendritic cellMolecular biologyNeoplasm ProteinsDrug CombinationsCTL*medicine.anatomical_structureImmunologyCytokinesCD8T-Lymphocytes Cytotoxic
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