Search results for "DAMAGE"

showing 10 items of 1289 documents

Influence of a Brief Episode of Anesthesia during the Induction of Experimental Brain Trauma on Secondary Brain Damage and Inflammation

2011

It is unclear whether a single, brief, 15-minute episode of background anesthesia already modulates delayed secondary processes after experimental brain injury. Therefore, this study was designed to characterize three anesthesia protocols for their effect on molecular and histological study endpoints. Mice were randomly separated into groups that received sevoflurane (sevo), isoflurane (iso) or an intraperitoneal anesthetic combination (midazolam, fentanyl and medetomidine; comb) prior to traumatic brain injury (controlled cortical impact, CCI; 8 m/s, 1 mm impact depth, 3 mm diameter). Twenty-four hours after insult, histological brain damage, neurological function (via neurological severit…

MaleMouseGeneral AnesthesiaNitric Oxide Synthase Type IIFentanylMiceAnesthesiologyAnesthesiaNeurosurgical CareMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionMicrofilament ProteinsQRAnimal ModelsSurvival RateHead InjuryNeurologyNeurointensive CareAnesthesiaMedicineRegional Anesthesiamedicine.symptomResearch Articlemedicine.drugTraumatic brain injuryScienceBlotting WesternImmunologyBrain damageAnesthetic MechanismsMicrobiologySevofluraneModel OrganismsNeuropharmacologymedicineAnimalsRNA MessengerBiologyInflammationInterleukin-6business.industryCalcium-Binding ProteinsImmunityBrain Contusionmedicine.diseaseMice Inbred C57BLIsofluraneCyclooxygenase 2Brain InjuriesAnestheticMidazolamClinical ImmunologybusinessPLoS ONE
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Comparison of post-activation performance enhancement (PAPE) after isometric and isotonic exercise on vertical jump performance.

2021

Purpose This study aimed to compare the post-activation performance enhancement (PAPE) induced by isometric and isotonic exercise on vertical jump performance. Methods 18 healthy trained men (25.8±2.7 years; 78.4±8.2 kg; 175.7±6.1 cm; 25.4±1.8 BMI; 126.72±10.8 kg squat 1-RM) volunteered for this study. They randomly performed two different PAPE protocols: Isotonic squats (ISOTS), which consisted of 2 sets of 3 repetitions at 75% of one-maximum repetition (1-RM); and isometric squats (ISOMS), which consisted of 2 sets of 4 seconds of submaximal (75% of 1-RM) isometric contraction at 90°-knee flexion. Countermovement jump (CMJ) height was tested at baseline and 4 minutes after each condition…

MaleMuscle PhysiologyPhysiologyKneesIsometric exerciseMaterial FatigueRunningJumpingSkeletal JointsMaterials PhysicsIsotonicMedicine and Health SciencesPublic and Occupational HealthMusculoskeletal SystemMathematicsCross-Over StudiesMultidisciplinaryPhysicsIsotonicQRClassical MechanicsSports SciencePhysical SciencesStrength TrainingLegsMedicineAnatomyPerformance enhancementResearch ArticleMuscle ContractionAdultmedicine.medical_specialtySciencePostureMaterials ScienceSquatAthletic PerformanceVertical jumpPhysical medicine and rehabilitationOsmotic PressureIsometric ContractionPressuremedicineHumansTonicityIsotonic ContractionMuscle StrengthSports and Exercise MedicineMuscle SkeletalExerciseSkeletonDamage MechanicsBiological LocomotionBiology and Life SciencesPhysical ActivityAthletesPhysical FitnessBody LimbsCountermovement jumpPLoS ONE
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Inhibition of myosin light chain kinase reduces brain edema formation after traumatic brain injury.

2010

The role of the endothelial contractile apparatus in the process of brain edema formation after brain trauma is not characterized. Phosphorylation of myosin light chains by myosin light chain kinases (MLCK) activates endothelial contractile elements and results in a rearrangement of the cytoskeleton. This may enhance post-traumatic blood-brain barrier dysfunction. In order to investigate the role of the MLCK on brain edema formation and blood-brain barrier permeability after brain injury, mice were anesthetized and subjected to a controlled cortical impact (CCI). MLCK expression is significantly up-regulated after CCI with a maximum 12 h post-injury. Specific inhibition of MLCK by ML-7 resu…

MaleMyosin light-chain kinaseMyosin Light ChainsTime FactorsEndotheliumIntracranial PressureTraumatic brain injuryCentral nervous systemBrain Edemamacromolecular substancesBrain damageNaphthalenesBlood–brain barrierBiochemistryNeuroprotectionDrug Administration ScheduleFunctional LateralityStatistics NonparametricCerebral edemaCellular and Molecular NeuroscienceMicemedicineAnimalsEnzyme InhibitorsMyosin-Light-Chain KinaseNeurologic Examinationbusiness.industryAzepinesmedicine.diseaseConstrictionCell biologyMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureGene Expression RegulationBlood-Brain BarrierBrain Injuriesmedicine.symptombusinessNeuroscienceEvans BlueJournal of neurochemistry
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Nitrosoureas Modes of Action and Perspectives in the Use of Hormone Receptor Affine Carrier Molecules

1989

Mechanisms of DNA adduct formation by antineoplastic 2-chloroethyl-N-nitrosoureas (CNUs) and of DNA damage induced by these compounds are discussed. CNUs are alkylating agents that form DNA-DNA cross-links as well as 2-chloroethylated and 2-hydroxyethylated adducts, the N-7-position of guanine being the predominantly alkylated site. A close correlation exists between the potential of a given compound to induce DNA-DNA cross-links and its antineoplastic effectiveness. However, levels of DNA-DNA cross-linking in bone marrow and extent of myelosuppression as measured in rodents are also closely correlated. The design of new cross-linking analogues capable of directing the antineoplastically re…

MaleNeoplasms Hormone-DependentDNA damageGuaninemedicine.medical_treatmentAntineoplastic AgentsReceptors Cell SurfaceNitrosourea CompoundsAdductStructure-Activity Relationshipchemistry.chemical_compoundBone MarrowmedicineAnimalsHumansRadiology Nuclear Medicine and imagingDrug Carriersbusiness.industryMammary Neoplasms ExperimentalProstatic NeoplasmsEstrogensHematologyGeneral MedicineSteroid hormoneOncologyMechanism of actionBiochemistrychemistryHormone receptormedicine.symptombusinessDNAHormoneActa Oncologica
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Reduction of infarct size by the NO donors sodium nitroprusside and spermine/NO after transient focal cerebral ischemia in rats

2000

Nitric oxide (NO) plays a dual role (neuroprotection and neurotoxicity) in cerebral ischemia. NO promoting strategies may be beneficial shortly after ischemia. Therefore, we have studied the hemodynamic and possible neuroprotective effects of two NO donors, the classical nitrovasodilator sodium nitroprusside (SNP) and the NONOate spermine/NO, after transient focal cerebral ischemia in rats. Parietal cortical perfusion was measured by laser-Doppler flowmetry. The effects of increasing intravenous doses (10-300 microgram) of sodium nitroprusside and spermine/NO on cortical perfusion and arterial blood pressure were assessed. Transient (2 h) focal cerebral ischemia was carried out by the intra…

MaleNitroprussideVasodilator AgentsIschemiaSpermineBlood PressurePerfusion scanningBrain damagePharmacologyNitric oxidechemistry.chemical_compoundAnimalsMedicineNitric Oxide DonorsRats WistarMolecular Biologybusiness.industryCerebral infarctionGeneral NeuroscienceBrainCerebral Infarctionmedicine.diseaseRatschemistryIschemic Attack TransientAnesthesiaSpermineNeurology (clinical)Sodium nitroprussidemedicine.symptombusinessNitrovasodilatorDevelopmental Biologymedicine.drugBrain Research
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Cancer in Children With Fanconi Anemia and Ataxia-Telangiectasia—A Nationwide Register-Based Cohort Study in Germany

2021

PURPOSE Fanconi anemia (FA) and ataxia-telangiectasia (AT) are rare inherited syndromes characterized by abnormal DNA damage response and caused by pathogenic variants in key DNA repair proteins that are also relevant in the pathogenesis of breast cancer and other cancer types. The risk of cancer in children with these diseases is poorly understood and has never been assessed in a population-based cohort before. METHODS We identified 421 patients with FA and 160 patients with AT diagnosed between 1973 and 2020 through German DNA repair disorder reference laboratories. We linked patients' laboratory data with childhood cancer data from the German Childhood Cancer Registry. RESULTS Among 421 …

MaleOncologyRegister basedCancer Researchmedicine.medical_specialtyTime FactorsAdolescentDNA damageAnemiaDNA repairRisk AssessmentAtaxia TelangiectasiaRisk FactorsFanconi anemiaGermanyNeoplasmsInternal medicinemedicineHumansRegistriesChildbusiness.industryIncidenceAge FactorsInfantCancerPrognosismedicine.diseaseFanconi AnemiaOncologyChild PreschoolAtaxia-telangiectasiaFemalebusinessCohort studyJournal of Clinical Oncology
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Alternating current voltammetric determination of DNA damage

1990

Abstract The conditions for alternating current (a.c.) voltammetric DNA determinations have been investigated with respect to its use with alkaline filter elution techniques at low DNA concentrations. In inorganic electrolyte solutions three current peaks can be distinguished: peak I around −1.1 V caused by the reorientation or desorption of DNA segments; peak II around −1.2 V caused by the native DNA (nDNA) form; peak III caused by denatured DNA (dDNA) at −1.4 V. Sonication of nDNA increases the peak current, however not with dDNA. Both dDNA and nDNA give linear peak current increments with DNA increments, their regression lines cutting the concentration axis at the origin. In filter eluti…

MaleOrganic baseChemistryElutionDNA damageSea CucumbersAnalytical chemistryDNAGeneral MedicineHydrogen-Ion ConcentrationToxicologychemistry.chemical_compoundSpectrometry FluorescenceAdsorptionEthanolamineDesorptionElectrochemistryAnimalsDNA damage determination; DNA sonication; alkaline filter elution of DNA; ethanolamine - DNA interactionVoltammetryDNADNA DamageChemico-Biological Interactions
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Utility of Urine Neutrophil Gelatinase-Associated Lipocalin for Worsening Renal Function during Hospitalization for Acute Heart Failure: Primary Find…

2019

ABSTRACT Background: Worsening renal function (WRF) during acute heart failure (AHF) occurs frequently and has been associated with adverse outcomes, though this association has been questioned. WRF is now evaluated by function and injury. We evaluated whether urine neutrophil gelatinase-associated lipocalin (uNGAL) is superior to creatinine for prediction and prognosis of WRF in patients with AHF. Methods and Results: We performed a multicenter, international, prospective cohort of patients with AHF requiring IV diuretics. The primary outcome was whether uNGAL predicted development of WRF, defined as a sustained increase in creatinine of 0.5 mg/dL or ≥50% above first value or initiation of…

MalePROGNOSISInternationalitymedicine.medical_treatmentUrine030204 cardiovascular system & hematologyKidneyKidney Function TestsGastroenterologyTHERAPYCohort Studieschemistry.chemical_compound0302 clinical medicine030212 general & internal medicineProspective StudiesProspective cohort studyDAMAGERISKAged 80 and overAcute kidney injuryIMPAIRMENTAcute Kidney InjuryMiddle Aged3. Good healthHospitalizationbiomarkerAcute heart failure; biomarker; worsening renal functionFemaleCardiology and Cardiovascular MedicineGlomerular Filtration Ratemedicine.medical_specialtyRenal functionDIAGNOSIS03 medical and health sciencesLipocalin-2Internal medicineSTRATIFICATIONmedicineMANAGEMENTHumansRenal replacement therapyAdverse effectAgedHeart FailureCreatininebusiness.industryMORTALITYAcute heart failuremedicine.diseasechemistryHeart failureworsening renal functionbusinessBiomarkersJournal of cardiac failure
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Influence of Age on Brain Edema Formation, Secondary Brain Damage and Inflammatory Response after Brain Trauma in Mice

2012

After traumatic brain injury (TBI) elderly patients suffer from higher mortality rate and worse functional outcome compared to young patients. However, experimental TBI research is primarily performed in young animals. Aim of the present study was to clarify whether age affects functional outcome, neuroinflammation and secondary brain damage after brain trauma in mice. Young (2 months) and old (21 months) male C57Bl6N mice were anesthetized and subjected to a controlled cortical impact injury (CCI) on the right parietal cortex. Animals of both ages were randomly assigned to 15 min, 24 h, and 72 h survival. At the end of the observation periods, contusion volume, brain water content, neurolo…

MalePathologyAgingAnatomy and PhysiologyCritical Care and Emergency MedicineMouseT-LymphocytesInterleukin-1beta610 MedizinNitric Oxide Synthase Type IISystemic inflammationMiceAnesthesiologyCell Movement610 Medical sciencesEdemaImmune PhysiologyEdemaLungNeurosurgical CareMultidisciplinaryHematologic TestsQRAging and ImmunityAnimal ModelsOrgan SizeHead Injurymedicine.anatomical_structureNeurologyNeurointensive CareCytokinesMedicinemedicine.symptomResearch Articlemedicine.medical_specialtyTraumatic brain injuryScienceImmunologyInflammationBrain damageAtrophyModel OrganismsNeurorehabilitation and TraumamedicineAnimalsRNA MessengerBiologyCerebrumNeuroinflammationInflammationLungbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaImmunityWatermedicine.diseaseMice Inbred C57BLGene Expression RegulationCyclooxygenase 2Immune SystemBrain InjuriesClinical ImmunologybusinessPhysiological ProcessesPLoS ONE
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Glutathione protects metastatic melanoma cells against oxidative stress in the murine hepatic microvasculature.

1998

Calcein-labeled B16 melanoma (B16M) cells were injected intraportally, and in vivo video microscopy was used to study the distribution and damage of cancer cells arrested in the liver microvasculature over a period of 4 hours. The contribution of glutathione (GSH)-dependent antioxidant machinery to the possible oxidative stress-resistance mechanism of B16M cell was determined by in vitro incubation with the selective inhibitor of GSH synthesis L-buthionine (S,R)-sulphoximine (BSO) before B16M cell injection in untreated and 0.5-mg/kg lipopolysaccharide (LPS)-treated mice. In addition, untreated and LPS-treated isolated syngeneic hepatic sinusoidal endothelial cells (HSE) were used to determ…

MalePathologymedicine.medical_specialtyCellMelanoma ExperimentalVideo RecordingVideo microscopyBiologymedicine.disease_causeAndrologychemistry.chemical_compoundMiceIn vivomedicineCell AdhesionTumor Cells CulturedAnimalsEnzyme InhibitorsNeoplasm MetastasisCell damageButhionine SulfoximineHepatologyMicrocirculationLiver NeoplasmsCell PolarityGlutathionemedicine.diseaseGlutathioneMice Inbred C57BLOxidative Stressmedicine.anatomical_structurechemistryLiverCancer cellOxidative stressIntracellularHepatology (Baltimore, Md.)
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