Search results for "DAMAGE"

showing 10 items of 1289 documents

Dicer prevents genome instability in response to replication stress

2019

Dicer, an endoribonuclease best-known for its role in microRNA biogenesis and RNA interference pathway, has been shown to play a role in the DNA damage response and repair of double-stranded DNA breaks (DSBs) in mammalian cells. However, it remains unknown whether Dicer is also important to preserve genome integrity upon replication stress. To address this question, we focused our study on common fragile sites (CFSs), which are susceptible to breakage after replication stress. We show that inhibition of the Dicer pathway leads to an increase in CFS expression upon induction of replication stress and to an accumulation of 53BP1 nuclear bodies, indicating transmission of replication-associate…

0301 basic medicineGenome instabilityreplication stressDNA damageChromosomal fragile siteBiologygenomic instabilitycommon fragile siteCell biologySettore BIO/18 - Genetica03 medical and health sciences030104 developmental biology0302 clinical medicineOncology030220 oncology & carcinogenesisFANCD2biology.proteinDicer PathwayMitosiscommon fragile sitesDroshaResearch PaperDicerDicerOncotarget
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Role of the DNA repair glycosylase OGG1 in the activation of murine splenocytes

2017

OGG1 (8-oxoguanine-DNA glycosylase) is the major DNA repair glycosylase removing the premutagenic DNA base modification 8-oxo-7,8-dihydroguanine (8-oxoG) from the genome of mammalian cells. In addition, there is accumulating evidence that OGG1 and its substrate 8-oxoG might function in the regulation of certain genes, which could account for an attenuated immune response observed in Ogg1-/- mice in several settings. Indications for at least two different mechanisms have been obtained. Thus, OGG1 could either act as an ancillary transcription factor cooperating with the lysine-specific demethylase LSD1 or as an activator of small GTPases. Here, we analysed the activation by lipopolysaccaride…

0301 basic medicineGuanineDNA RepairDNA repairp38 mitogen-activated protein kinasesBiologyBiochemistryDNA GlycosylasesMice03 medical and health sciencesAnimalsMolecular BiologyTranscription factorTumor Necrosis Factor-alphaKinaseActivator (genetics)MacrophagesDNACell BiologyBase excision repairMolecular biology030104 developmental biologyGene Expression RegulationDNA glycosylaseTumor necrosis factor alphaSpleenDNA DamageTranscription FactorsDNA Repair
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Free energy profiles for two ubiquitous damaging agents: methylation and hydroxylation of guanine in B-DNA

2017

International audience; DNA methylation and hydroxylation are two ubiquitous reactions in DNA damage induction, yet insights are scarce concerning the free energy of activation within B-DNA. We resort to multiscale simulations to investigate the attack of a hydroxyl radical and of the primary diazonium onto a guanine embedded in a solvated dodecamer. Reaction free energy profiles characterize two strongly exergonic processes, yet allow unprecedented quantification of the barrier towards this damage reaction, not higher than 6 kcal mol−1 and sometimes inexistent, and of the exergonicities. In the case of the [G(C8)-OH]˙ intermediate, we challenge the functional dependence of such simulations…

0301 basic medicineGuanineGuanineDNA damageStereochemistryEntropyGeneral Physics and Astronomy010402 general chemistryHydroxylation01 natural sciencesHydroxylation03 medical and health scienceschemistry.chemical_compoundComputational chemistry[CHIM.ANAL]Chemical Sciences/Analytical chemistryPhysical and Theoretical ChemistryExergonic reactionchemistry.chemical_classificationHydroxyl RadicalBiomoleculeDNA Methylation0104 chemical sciences030104 developmental biologychemistryEnergy TransferDNA methylationHydroxyl radicalDNA B-Form[CHIM.RADIO]Chemical Sciences/RadiochemistryDNA
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Ultrastructural Alterations of Midgut Epithelium, but Not Greater Wing Fluctuating Asymmetry, in Paper Wasps (Polistes dominula) from Urban Environme…

2018

Polistes paper wasps can be used to monitor trace metal contaminants, but the effects of pollution on the health of these insects are still unknown. We evaluated, in a south-eastern area of Spain, whether workers of Polistes dominula collected at urban and rural sites differ in health of midgut tissue and in fluctuating asymmetry, an estimate of developmental noise. We found that wasps collected at the urban sites had abundant lead (Pb)-containing spherites, which were less visible in wasps from the rural sites. Evident ultrastructural alterations in the epithelium of the midgut of the wasps collected at the urban sites included broken and disorganized microvilli, a high amount and density …

0301 basic medicineHeterochromatinDevelopmental noiseWaspsZoologymacromolecular substances010501 environmental sciencesPolistes dominula01 natural sciencesFluctuating asymmetry03 medical and health sciencesMidgutmedicineAnimalsWings AnimalCitiesIntestinal MucosaInstrumentation0105 earth and related environmental sciencesbiologyFluctuating asymmetryfungiUrbanizationMidgutTissue damagesWaspbiology.organism_classificationEpithelium030104 developmental biologymedicine.anatomical_structureLeadSpainUltrastructureEnvironmental PollutantsPolistes
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DNA damage and repair in the differentiation of stem cells and cells of connective cell lineages: A trigger or a complication?

2021

The review summarizes literature data on the role of DNA breaks and DNA repair in differentiation of pluripotent stem cells (PSC) and connective cell lineages. PSC, including embryonic stem cells (ESC) and induced pluripotent stem cells (iPSC), are rapidly dividing cells with highly active DNA damage response (DDR) mechanisms to ensure the stability and integrity of the DNA. In PSCs, the most common DDR mechanism is error-free homologous recombination (HR) that is primarily active during S phase of the cell cycle, whereas in quiescent, slow-dividing or non-dividing tissue progenitors and terminally differentiated cells, error-prone non-homologous end joining (NHEJ) mechanism of the double-s…

0301 basic medicineHistologyDNA RepairQH301-705.5DNA repairDNA damageCellular differentiationInduced Pluripotent Stem CellsBiophysicsBiologyArticle03 medical and health sciences0302 clinical medicinestem cellsOsteogenesisAnimalsHumansBiology (General)Induced pluripotent stem cellEmbryonic Stem Cellsconnective tissueConnective Tissue CellsDNA BreaksCell DifferentiationCell BiologydifferentiationEmbryonic stem cellCell biology030104 developmental biology030220 oncology & carcinogenesisStem cellHomologous recombinationReprogrammingChondrogenesisEuropean Journal of Histochemistry : EJH
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N-(2-methyl-indol-1H-5-yl)-1-naphthalenesulfonamide : a novel reversible antimitotic agent inhibiting cancer cell motility

2016

Este es el post-print que se ha publicado de forma definitiva en: https://www.sciencedirect.com/science/article/abs/pii/S0006295216301423 A series of compounds containing the sulfonamide scaffold were synthesized and screened for their in vitro anticancer activity against a representative panel of human cancer cell lines, leading to the identification of N-(2-methyl-1H-indol-5-yl)-1-naphthalenesulfonamide (8e) as a compound showing a remarkable activity across the panel, with IC50 values in the nanomolar-to-low micromolar range. Cell cycle distribution analysis revealed that 8e promoted a severe G2/M arrest, which was followed by cellular senescence as indicated by the detection of senescen…

0301 basic medicineIndolesSulfonamides - Therapeutic use.MotilityApoptosisAntimitotic AgentsMicrotubulesBiochemistryJurkat Cells03 medical and health sciences0302 clinical medicineCell MovementTubulinMicrotubuleCélulas cancerosas - Motilidad.Apoptosis.HumansSulfamidas - Uso terapéutico.MitosisCell ProliferationPharmacologySulfonamidesMolecular StructurebiologyCancer cells - Motility.Cell cycleCell biologyMitosis.030104 developmental biologyTubulinCell cultureApoptosis030220 oncology & carcinogenesisCancer cellMCF-7 Cellsbiology.proteinDrug Screening Assays AntitumorDNA Damage
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Molecular Biology of Atherosclerotic Ischemic Strokes

2020

Among the causes of global death and disability, ischemic stroke (also known as cerebral ischemia) plays a pivotal role, by determining the highest number of worldwide mortality, behind cardiomyopathies, affecting 30 million people. The etiopathogenetic burden of a cerebrovascular accident could be brain ischemia (~80%) or intracranial hemorrhage (~20%). The most common site when ischemia occurs is the one is perfused by middle cerebral arteries. Worse prognosis and disablement consequent to brain damage occur in elderly patients or affected by neurological impairment, hypertension, dyslipidemia, and diabetes. Since, in the coming years, estimates predict an exponential increase of people w…

0301 basic medicineInflammasomesCerebral arteriesmicrogliaDiseaseReviewneuroinflammationBrain ischemialcsh:Chemistry0302 clinical medicineatherosclerosiStrokelcsh:QH301-705.5SpectroscopymicroRNAGeneral MedicineMKEYDKK-3Computer Science ApplicationsmicroRNAsBlood-Brain BarrierCardiologymedicine.symptomDectin-1medicine.medical_specialtyIschemiaBrain damageCatalysisInorganic Chemistry03 medical and health sciencesInternal medicineDiabetes mellitusmedicineischemic strokeAnimalsHumansPhysical and Theoretical ChemistryMolecular Biologybusiness.industryOrganic ChemistryAFmedicine.diseaseImmunity InnateNLRP3 inflammasome030104 developmental biologylcsh:Biology (General)lcsh:QD1-999atherosclerosisbusinessBBB030217 neurology & neurosurgeryDyslipidemiaCD200-CD200R
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Exposure to environmental radionuclides alters mitochondrial DNA maintenance in a wild rodent

2020

AbstractMitochondria are sensitive to oxidative stress, including that derived from ionizing radiation. To quantify the effects of exposure to environmental radionuclides on mitochondrial DNA (mtDNA) dynamics in wildlife, bank voles (Myodes glareolus) were collected from the chernobyl exclusion zone (CEZ), where animals are exposed to elevated levels of radionuclides, and from uncontaminated areas within the CEZ and elsewhere in Ukraine. Brains of bank voles from outside the CEZ were characterized by low mtDNA copy number and low mtDNA damage; by contrast, bank voles within the CEZ had high mtDNA copy number and high mtDNA damage, consistent with putative damaging effects of elevated radiat…

0301 basic medicineIonizing radiationMitochondrial DNARodentmetsämyyrämitokondriotZoologyMyodes glareolusBiologyMitochondrionmedicine.disease_cause03 medical and health sciences0302 clinical medicinebiology.animalMyodes glareolusmedicineGeneEcology Evolution Behavior and Systematicsmitokondrio-DNACopy numberionisoiva säteilyDNAMitochondria030104 developmental biologyMitochondrial biogenesisAnimal ecology030220 oncology & carcinogenesisDNA damage[SDE.BE]Environmental Sciences/Biodiversity and EcologyOxidative stress
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Comparison of time and dose dependent gene expression and affected pathways in primary human fibroblasts after exposure to ionizing radiation

2020

Abstract Background Exposure to ionizing radiation induces complex stress responses in cells, which can lead to adverse health effects such as cancer. Although a variety of studies investigated gene expression and affected pathways in human fibroblasts after exposure to ionizing radiation, the understanding of underlying mechanisms and biological effects is still incomplete due to different experimental settings and small sample sizes. Therefore, this study aims to identify the time point with the highest number of differentially expressed genes and corresponding pathways in primary human fibroblasts after irradiation at two preselected time points. Methods Fibroblasts from skin biopsies of…

0301 basic medicineIonizing radiationTime FactorsDNA damageCellHigh doseIonizing radiationlcsh:BiochemistryGene-radiation interaction03 medical and health sciences0302 clinical medicineRadiation IonizingGene expressionGeneticsmedicineHumanslcsh:QD415-436IrradiationMolecular BiologyGeneGenetics (clinical)Gene-radiation interaction ; RNA sequencing ; Childhood cancer ; High dose ; Fibroblasts ; Low dose ; Second primary neoplasm ; IPA ; Ionizing radiationCells CulturedChemistryGene Expression Profilinglcsh:RM1-950Second primary neoplasmCancerComputational BiologyRNA sequencingDose-Response Relationship RadiationFibroblastsmedicine.diseaseCell biology030104 developmental biologymedicine.anatomical_structurelcsh:Therapeutics. PharmacologyLow doseGene Expression Regulation030220 oncology & carcinogenesisIPACase-Control StudiesMolecular MedicineSignal transductionChildhood cancerResearch ArticleSignal TransductionMolecular Medicine
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Molecular mechanisms underlying the neuroprotective role of atrial natriuretic peptide in experimental acute ischemic stroke

2018

Abstract Along with its role in regulating blood pressure and fluid homeostasis, the natriuretic peptide system could be also part of an endogenous protective mechanism against brain damage. We aimed to assess the possibility that exogenous atrial natriuretic peptide (ANP) could protect against acute ischemic stroke, as well as the molecular mechanisms involved. Three groups of rats subjected to transient middle cerebral artery occlusion (tMCAO, intraluminal filament technique, 60 min) received intracerebroventricular vehicle, low-dose ANP (0.5 nmol) or high-dose ANP (2.5 nmol), at 30 min reperfusion. Neurofunctional condition, and brain infarct and edema volumes were measured at 24 h after…

0301 basic medicineMAPK/ERK pathwayMalePotassium ChannelsSignaling pathwaysmedicine.drug_classMAP Kinase Signaling SystemAcute ischemic strokeDown-RegulationApoptosisBrain damagePharmacologyBiochemistryNeuroprotectionBrain Ischemia03 medical and health sciencesPhosphatidylinositol 3-Kinases0302 clinical medicineEndocrinologyAtrial natriuretic peptideNatriuretic peptideMedicineAnimalsDNA CleavageRats WistarReceptorAtrial natriuretic peptideMolecular BiologyProtein kinase BPI3K/AKT/mTOR pathwayInjections Intraventricularbusiness.industryCaspase 3Natriuretic peptide receptorsBrainInfarction Middle Cerebral ArteryStroke030104 developmental biologyNeuroprotective AgentsReperfusion InjuryK+ channelsmedicine.symptombusinessProto-Oncogene Proteins c-aktReceptors Atrial Natriuretic Factor030217 neurology & neurosurgeryAtrial Natriuretic Factorhormones hormone substitutes and hormone antagonists
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