Search results for "DAMPs"

showing 5 items of 5 documents

Mechanisms of Defence to Pathogens : Biochemistry and Physiology

2014

SPE IPM; International audience; Plant defences comprise both pre-existing barriers as well as defences induced upon perception of pathogen-associated molecular patterns (PAMPs) or microbe-associated molecular patterns (MAMPs) or molecules produced from damage as a result of infection (damage-associated molecular patterns (DAMPs)). This chapter focuses on the induced mechanisms of defence. The inducibility of phytoalexin biosynthesis has probably been favoured in the course of evolution by biological constraints such as metabolic costs and functional side-effects associated with chemical defence. Historically, the term ‘hypersensitive’ refers to the rapid and localized cell death induced in…

0106 biological sciences0303 health sciences[SDV]Life Sciences [q-bio]plant defencesfood and beveragespathogenspathogenesis-related (PR) proteinsBiology01 natural sciencesPhytoalexin biosynthesisMicrobiologymicrobe-associated molecular patterns (MAMPs)03 medical and health scienceshypersensitive response (HR)Biochemistrypathogen-associated molecular patterns (PAMPs)[SDE]Environmental Sciences[SDV.BV]Life Sciences [q-bio]/Vegetal Biologydamage-associated molecular patterns (DAMPs)phytoalexin biosynthesis030304 developmental biology010606 plant biology & botany
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Ageing: from inflammation to cancer.

2018

Abstract Ageing is the major risk factor for cancer development. Hallmark of the ageing process is represented by inflammaging, which is a chronic and systemic low-grade inflammatory process. Inflammation is also a hallmark of cancer and is widely recognized to influence all cancer stages from cell transformation to metastasis. Therefore, inflammaging may represent the biological phenomena able to couple ageing process with cancer development. Here we review the molecular and cellular pathway involved in age-related chronic inflammation along with its potential triggers and their connection with cancer development.

0301 basic medicineSenescencelcsh:Immunologic diseases. AllergyAgingCellImmunologyAgeing; Cancer; DAMPs; Inflammation; MiRna; Microbiota; Obesity; SenescenceInflammationReviewlcsh:GeriatricsSenescenceMetastasis03 medical and health sciencesmicroRNAMedicineDAMPObesityCancerSettore MED/04 - Patologia GeneraleInflammationDAMPsbusiness.industryAgeing; Cancer; DAMPs; Inflammation; Microbiota; MiRna; Obesity; Senescence; Immunology; AgingMicrobiotaCancermedicine.diseaselcsh:RC952-954.6Ageing030104 developmental biologymedicine.anatomical_structureAgeingCancer researchSettore MED/26 - NeurologiaCancer developmentmedicine.symptombusinesslcsh:RC581-607MiRna
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Whole-Blood Mitochondrial DNA Copies Are Associated With the Prognosis of Acute Respiratory Distress Syndrome After Sepsis

2021

Acute respiratory distress syndrome (ARDS) is an inflammatory process of the lungs that develops primarily in response to pulmonary or systemic sepsis, resulting in a disproportionate death toll in intensive care units (ICUs). Given its role as a critical activator of the inflammatory and innate immune responses, previous studies have reported that an increase of circulating cell-free mitochondrial DNA (mtDNA) is a biomarker for fatal outcome in the ICU. Here we analyzed the association of whole-blood mtDNA (wb-mtDNA) copies with 28-day survival from sepsis and sepsis-associated ARDS. We analyzed mtDNA data from 687 peripheral whole-blood samples within 24 h of sepsis diagnosis from unrelat…

MaleMitochondrial DNAARDSTime FactorsMedicinaImmunologyDNA MitochondrialRisk AssessmentsurvivalSepsisPathogenesisPredictive Value of TestsRisk FactorsIntensive careSepsismedicineImmunology and AllergyHumansHospital MortalityOriginal ResearchAgedRespiratory Distress SyndromeDAMPsProportional hazards modelbusiness.industrymtDNAHazard ratiowhole bloodMiddle AgedRC581-607medicine.diseasePrognosismitochondriaSpainImmunologyBiomarker (medicine)FemaleARDSImmunologic diseases. AllergybusinessBiomarkersFrontiers in Immunology
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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Are the xyloglucans new elicitors of plant immunity ?

2017

Damaged-Associated Molecular Patterns (DAMPs) are endogenous molecules released from the plant cell wall after wounding by pathogens. DAMPs are recognized by Pattern- Recognition Receptors (PRRs) that play a key role in plant immunity by mediating defense responses. The plant cell wall-derived oligogalacturonides (OG) are well characterized DAMPs that elicit plant immune responses such as MAPK activation, [Ca2+]cyt variations, H2O2 production, defense-related gene expression and enhanced resistance against Botrytis cinerea. Our study focused on a new polysaccharide component of the plant cell wall called xyloglucans (Xh) and compared the immune events triggered by OG and Xh in Arabidopsis t…

[SDE] Environmental SciencesDAMPs[SDV]Life Sciences [q-bio]fungifood and beveragesxyloglucansimmunity[SDV] Life Sciences [q-bio]A. thaliana[SDE]Environmental Sciencesoligogalacturonides[SDV.BV]Life Sciences [q-bio]/Vegetal Biology[SDV.BV] Life Sciences [q-bio]/Vegetal Biologysignaling
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