Search results for "DIALYSIS"

showing 10 items of 528 documents

The aminothiol redox status in haemodialysis patients does not improve with folate therapy.

2008

Patients on haemodialysis suffer from high cardiovascular morbidity and mortality, and oxidative stress may play a role in the pathophysiology of cardiovascular disease in these patients. Hyperhomocysteinemia is common in dialysis patients and may have pro-oxidant effects. Moreover, the redox status of the major plasma aminothiols (homocysteine [Hcy], cysteine and cysteinylglycine) may be regarded as a biomarker of oxidative stress. In the present study, we investigated the aminothiol redox status during a period of homocysteine-lowering therapy with folinic acid.In the first part of the study, 32 stable patients receiving maintenance haemodialysis were compared with 32 reference subjects. …

Malemedicine.medical_specialtyHyperhomocysteinemiaHomocysteineClinical Biochemistrymedicine.disease_causeGastroenterologychemistry.chemical_compoundFolinic acidFolic AcidRenal DialysisInternal medicinemedicineHumansSulfhydryl CompoundsAgedbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseRedox statusPathophysiologySurgerychemistryBiomarker (medicine)FemalebusinessOxidation-ReductionOxidative stressCysteinemedicine.drugScandinavian journal of clinical and laboratory investigation
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Environmental enrichment promotes neurogenesis and changes the extracellular concentrations of glutamate and GABA in the hippocampus of aged rats.

2005

The aim of the present study was to investigate the effects of environmental enrichment on the neurogenesis and the extracellular concentrations of glutamate and GABA in the hippocampus of freely moving young and aged rats. Male Wistar rats of 2 (young) and 25 (old) months of age were housed during 8 weeks in an enriched environment; control rats were kept in individual plastic cages during that same period of time. Rats were injected intraperitoneally with bromodeoxyuridine (BrdU; 40 mg/kg; 7 days) during the fourth week of the housing period to detect neurogenesis in the dentate gyrus (DG) of the hippocampus. Rats were sacrified 6 weeks after the last injection of BrdU. During the last we…

Malemedicine.medical_specialtyMicrodialysisAgingMicrodialysisHippocampusGlutamic AcidCell CountWater mazeEnvironmentHippocampusInternal medicineExtracellularmedicineElectrochemistryReaction TimeAnimalsHumansRats WistarChromatography High Pressure Liquidgamma-Aminobutyric AcidNeuronsEnvironmental enrichmentAnalysis of VarianceChemistryGeneral NeuroscienceDentate gyrusNeurogenesisGlutamate receptorImmunohistochemistryRatsEndocrinologynervous systemBromodeoxyuridineExtracellular SpaceNeuroscienceBrain research bulletin
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The neuroprotective effect of lactate is not due to improved glutamate uptake after controlled cortical impact in rats.

2012

For many years lactate was considered to be a waste product of glycolysis. Data are accumulating that suggest that lactate is an important energy substrate for neurons during activation. In severe traumatic brain injury (TBI) glutamate release and ischemic cerebral blood flow (CBF) are major factors for a mismatch between energy demand and supply and for neuronal cell death. Although ATP and behavior could be improved by lactate treatment after TBI, no histological correlate nor any linkage to better astrocytic glutamate uptake or CBF as possible mechanisms have been described. We subjected male rats to a controlled cortical impact (CCI; 5 m/sec, 2.5 mm). To study the effects of lactate tre…

Malemedicine.medical_specialtyMicrodialysisCoumaric AcidsMicrodialysisGlutamic AcidNeuroprotectionRats Sprague-DawleyStereotaxic TechniquesOxygen ConsumptionInternal medicinemedicineAnimalsGlycolysisLactic AcidChromatography High Pressure LiquidBrain ChemistryCerebral CortexSkull FracturesChemistryGlutamate receptorGlutamic acidRatsmedicine.anatomical_structureEndocrinologyNeuroprotective AgentsCerebral blood flowCerebral cortexAnesthesiaBrain InjuriesCerebrovascular CirculationStereotaxic techniqueNeurology (clinical)Extracellular SpaceJournal of neurotrauma
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Mesolimbic dopaminergic system activity as a function of food reward: A microdialysis study

1996

The mesolimbic dopaminergic system (MDS) has been shown to be implicated in feeding behaviors. The present experiment was conducted to examine the effects of the sensory properties of food ingested on MDS activity. Microdialysis coupled to high-performance liquid chromatography with electrochemical detection was employed to measure the extracellular levels of dopamine (DA) and its main metabolites (DOPAC and HVA) in the nucleus accumbens of freely moving rats. During microdialysis sessions rats had access or not to powdered foods varying in palatability: short cakes as highly palatable (HP) food and regular chow as low palatable (LP) food. In the absence of food, there were no alterations i…

Malemedicine.medical_specialtyMicrodialysisDopamineMicrodialysisClinical BiochemistryNucleus accumbensToxicologyBiochemistryNucleus AccumbensRats Sprague-DawleyBehavioral Neurosciencechemistry.chemical_compoundRewardDopamineInternal medicineLimbic SystemmedicineAnimalsIngestionPalatabilityNeurotransmitterBiological PsychiatryPharmacologydigestive oral and skin physiologyDopaminergicHomovanillic AcidRatsEndocrinologychemistryFoodCatecholamine34-Dihydroxyphenylacetic Acidmedicine.drugPharmacology Biochemistry and Behavior
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A homeostatic mechanism counteracting K+-evoked choline release in adult brain

2002

Choline (Ch) is an essential nutrient as the biosynthetic precursor of acetylcholine (ACh) and phospholipids. Under resting conditions, the intracellular accumulation of Ch (above 10-fold), which is positively charged, is governed by the membrane potential and follows the Nernst equation. Accordingly, in synaptosomes from adult rats during depolarization, we observed a linear relationship between release of free cytoplasmic Ch and KCl concentration (2.7-120 mm). The K(+) -evoked Ch release was Ca(2+) -independent and did not originate from ACh or phospholipid hydrolysis. In superfused brain slices of adult rats, however, a K(+) -induced Ch efflux was absent. Also, under in vivo conditions, …

Malemedicine.medical_specialtyMicrodialysisMicrodialysisIn Vitro TechniquesHippocampusBiochemistryCholineCellular and Molecular Neurosciencechemistry.chemical_compoundInternal medicinePotassium Channel BlockersmedicineExtracellularAnimalsHomeostasisCholine4-AminopyridineRats WistarNeurotransmitterBrain ChemistrySynaptosomeMembrane potentialDose-Response Relationship DrugBrainBiological TransportDepolarizationHemicholinium 3RatsEndocrinologychemistryPotassiumExtracellular SpaceAcetylcholineSynaptosomesmedicine.drugJournal of Neurochemistry
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Acute Kidney Injury After Percutaneous Edge-to-Edge Mitral Repair.

2020

BACKGROUND In catheter-based procedures, acute kidney injury (AKI) is a frequent, serious complication ranging from 10% to 30%. In MitraClip (Abbott Vascular, Santa Clara, California), a usually contrast-free procedure, there is scarce data about its real incidence and impact. OBJECTIVES This study aimed to evaluate incidence, predictive factors, and midterm outcomes of AKI in patients with significant mitral regurgitation (MR) undergoing transcatheter valve repair with MitraClip. METHODS A total of 721 patients undergoing MitraClip were included. AKI was defined as an absolute or a relative increase in serum creatinine of >0.3 mg/dl or >= 50%, respectively, or the need for hemodialysis dur…

Malemedicine.medical_specialtyMitral Valve Annuloplastymedicine.medical_treatmentPopulationRenal function030204 cardiovascular system & hematologyoutcomesurologic and male genital diseasesrenal insufficiency03 medical and health sciences0302 clinical medicinePostoperative ComplicationsInterquartile rangeInternal medicinemedicineMitraClipHumans030212 general & internal medicinemitral edge-to-edge repaireducationAgedRetrospective StudiesAged 80 and overMitral regurgitationeducation.field_of_studybusiness.industryMitraClipIncidenceEndovascular ProceduresAcute kidney injuryMitral Valve InsufficiencyAcute Kidney InjuryMiddle AgedMitraClip acute kidney injury mitral edge-to-edge repair mortality outcomes renal insufficiencymedicine.diseasemortalityfemale genital diseases and pregnancy complicationsacute kidney injurySpainCardiologyFemaleHemodialysisCardiology and Cardiovascular MedicineComplicationbusiness
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Hippocampal dopamine receptors modulate the motor activation and the increase in dopamine levels in the rat nucleus accumbens evoked by chemical stim…

2005

A number of studies have shown that chemical stimulation (using N-methyl-D-aspartate (NMDA) infusions) or electrical stimulation of the ventral hippocampus (VH) elicits locomotor activation and sustained increases in nucleus accumbens (NAc) dopamine (DA) levels in rodents. How DA neurotransmission in NAc is involved in these effects has also been well established. However, the modulatory role of the DA receptors located in VH is not yet fully understood. The purpose of this study was to characterize the role played by VH D1 and D2 subtype receptors in both the locomotor activation and NAc DA increases induced by NMDA stimulation of the VH. This was assessed by studying how retrodialysis app…

Malemedicine.medical_specialtyN-MethylaspartateDopamineMicrodialysisStimulationNucleus accumbensMotor ActivityHippocampusNucleus AccumbensReceptors Dopaminechemistry.chemical_compoundDopamineInternal medicineDopamine receptor D2medicineExcitatory Amino Acid AgonistsAnimalsRats WistarNeurotransmitterPharmacologyRacloprideBrain ChemistrySCH-23390Behavior AnimalChemistryReceptors Dopamine D1BenzazepinesStimulation ChemicalRatsPsychiatry and Mental healthDopamine D2 Receptor AntagonistsEndocrinologyDopamine receptorRacloprideDopamine AntagonistsNeurosciencemedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Hippocampal dopamine receptors modulate cFos expression in the rat nucleus accumbens evoked by chemical stimulation of the ventral hippocampus

2005

Recently, we have shown that D1 and D2 receptors in the ventral hippocampus (VH) modulate both the locomotor activation and the increase in dopamine (DA) levels in the rat nucleus accumbens (NAc) induced by NMDA stimulation of the VH. In the present study we analyze the possible role of VH D1 and D2 receptors in the modulation of the cFos expression in NAc (core and shell subregions) and in dorsal striatum. This was assessed by immunohistochemical analysis of cFos expression in the rat brains after retro-dialysis application of NMDA (50mM, 10 min) into VH, in absence and in presence of either the D1/D5 receptor antagonist SCH 23390 (100 and 250 microM, 60 min) or the D2 receptor antagonist …

Malemedicine.medical_specialtyN-Methylaspartatenucleus accumbensMicrodialysisStriatumNucleus accumbensHippocampusNucleus AccumbensReceptors DopamineCellular and Molecular Neurosciencechemistry.chemical_compoundDopamineDopamine receptor D2Internal medicinemedicineExcitatory Amino Acid AgonistsAnimalsRats WistarPharmacologyRacloprideSCH-23390ChemistryGenes fosBenzazepinesImmunohistochemistryStimulation ChemicalRatsNeostriatumcFosEndocrinologyD2Gene Expression Regulationnervous systemD1NMDADopamine receptorRacloprideNMDA receptorDopamine Antagonistsdopamineventral hippocampusmedicine.drug
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Impact of end-stage renal disease care in planned dialysis start and type of renal replacement therapy--a Spanish multicentre experience.

2006

Background. Timely referral, preparation and initiation of dialysis remain problematic issues. The purpose of this study is to analyse the effect of chronic renal disease care and education on the mode of dialysis start (planned vs non-planned) and on the modality of renal replacement therapy (RRT). Methods. A total of 1504 patients from 35 hospitals started RRT in 2003. Out-patient, scheduled initiation of dialysis with a permanent vascular or peritoneal access was considered planned. Results. About 46% of the patients started non-planned dialysis. Of all the patients, 75% had 3 months of nephrological follow-up, but nearly half were never educated on dialysis options. Haemodialysis (HD) o…

Malemedicine.medical_specialtyPediatricsReferralmedicine.medical_treatmentPeritoneal dialysisEnd stage renal diseasePatient Education as TopicRenal DialysismedicineDiabetes MellitusHumansRenal replacement therapyVascular DiseasesIntensive care medicineDialysisRetrospective StudiesTransplantationbusiness.industryRetrospective cohort studyMiddle Agedmedicine.diseaseNephrologyKidney Failure ChronicFemaleKidney Cortex NecrosisHemodialysisbusinessPeritoneal DialysisKidney diseaseNephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
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Molecular and functional interactions between tumor necrosis factor-alpha receptors and the glutamatergic system in the mouse hippocampus: Implicatio…

2009

Tumor necrosis factor (TNF)-alpha is a proinflammatory cytokine acting on two distinct receptor subtypes, namely p55 and p75 receptors. TNF-alpha p55 and p75 receptor knockout mice were previously shown to display a decreased or enhanced susceptibility to seizures, respectively, suggesting intrinsic modifications in neuronal excitability. We investigated whether alterations in glutamate system function occur in these naive knockout mice with perturbed cytokine signaling that could explain their different propensity to develop seizures. Using Western blot analysis of hippocampal homogenates, we found that p55(-/-) mice have decreased levels of membrane GluR3 and NR1 glutamate receptor subuni…

Malemedicine.medical_specialtyReceptors Kainic acidMicrodialysisAction PotentialsGlutamic AcidKainate receptorAMPA receptorIn Vitro TechniquesBiologyHippocampusReceptors N-Methyl-D-Aspartateelectrophysiology microiontophoresisSettore BIO/09 - FisiologiaMicechemistry.chemical_compoundGlutamatergicReceptors Kainic AcidSeizuresInternal medicinemedicineAnimalsReceptors Tumor Necrosis Factor Type IIReceptors AMPAMice KnockoutNeuronsInflammationTumor Necrosis Factor-alphaGeneral NeuroscienceGlutamate receptorProtein SubunitsEndocrinologymedicine.anatomical_structureReceptors Glutamatenervous systemchemistryReceptors Tumor Necrosis Factor Type IMetabotropic glutamate receptorAstrocytesCytokinesNMDA receptorNBQXDisease SusceptibilityAstrocyte
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