Search results for "DOT"

showing 10 items of 5147 documents

Platelet-localized FXI promotes a vascular coagulation-inflammatory circuit in arterial hypertension

2017

Multicellular interactions of platelets, leukocytes, and the blood vessel wall support coagulation and precipitate arterial and venous thrombosis. High levels of angiotensin II cause arterial hypertension by a complex vascular inflammatory pathway that requires leukocyte recruitment and reactive oxygen species production and is followed by vascular dysfunction. We delineate a previously undescribed, proinflammatory coagulation-vascular circuit that is a major regulator of vascular tone, blood pressure, and endothelial function. In mice with angiotensin II-induced hypertension, tissue factor was up-regulated, as was thrombin-dependent endothelial cell vascular cellular adhesion molecule 1 ex…

Blood PlateletsMale0301 basic medicinemedicine.medical_specialtyMacrophage-1 AntigenVascular Cell Adhesion Molecule-1Blood Pressure030204 cardiovascular system & hematologyThromboplastinMice03 medical and health sciencesTissue factor0302 clinical medicineThrombinInternal medicinemedicineAnimalsHumansPlateletRats WistarEndothelial dysfunctionBlood CoagulationFactor XIAgedMice Knockoutbusiness.industryAngiotensin IIThrombinGeneral MedicineMiddle AgedOligonucleotides Antisensemedicine.diseaseAngiotensin IIMice Inbred C57BL030104 developmental biologyEndocrinologyBlood pressuremedicine.anatomical_structurePlatelet Glycoprotein GPIb-IX ComplexPathophysiology of hypertensionHypertensionFemalebusinessmedicine.drugBlood vesselScience Translational Medicine
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Large platelets but not putative endothelial progenitor cells are associated with low strut coverage after drug-eluting stent implantation

2015

Objectives This study assessed whether different subsets of circulating endothelial and putative endothelial progenitor cells (CEC and EPC) correlate with stent strut coverage (SSC) using second generation optical coherence tomography (OCT). Background Due to the lack of imaging modalities with a resolution down to the magnitude of a few cells, the influence of EPC on endothelialisation of drug-eluting stents has not been assessed in patients. Methods In 37 patients, SSC of everolimus-eluting stents was assessed by OCT 5-7months after stent implantation. Different subsets of EPC (CD34(+)KDR(+), CD34(+)KDR(+)CD45(dim), CD133(+), CD3(+)CD31(+)), CEC (CD31(+)CD45(-)CD146(+)), and CD31(+)CD45(-…

Blood PlateletsMaleCD31medicine.medical_specialtymedicine.medical_treatmentCD34UrologyFlow cytometryBlood Vessel Prosthesis ImplantationAntigens CDmedicineHumansPlateletEverolimusProgenitor cellAgedEndothelial Progenitor Cellsmedicine.diagnostic_testbusiness.industryStentDrug-Eluting StentsKinase insert domain receptorMiddle AgedFlow CytometryVascular Endothelial Growth Factor Receptor-2Drug-eluting stentImmunologycardiovascular systemFemaleCardiology and Cardiovascular MedicinebusinessImmunosuppressive AgentsTomography Optical CoherenceInternational Journal of Cardiology
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Analysis and comparison of autologous platelet-rich plasma preparation systems used in the treatment of enthesopathies: A preliminary study

2021

Background Autologous platelet-rich plasma (PRP) injection is an alternative but widely accepted method for the treatment of degenerative changes in tendon attachments known as enthesopathies. The PRP is considered a safe source for high concentrations of the growth factors involved in the healing process. Despite initial promising outcomes, many recent studies report conflicting results for this treatment. This may be due to differences in the concentrations of platelets and growth factors in PRPs obtained using different methods. Objectives The aim of this study was to compare PRP preparation systems in terms of morphotic components and selected growth factors to find the most appropriate…

Blood PlateletsMaleVascular Endothelial Growth Factor APlatelet-derived growth factormedicine.medical_treatmentPopulationMedicine (miscellaneous)EnthesopathyGeneral Biochemistry Genetics and Molecular Biologyplatelet-derived growth factorTransforming Growth Factor beta1AndrologyLeukocyte Countchemistry.chemical_compoundEpidermal growth factorgrowth factorsInternal MedicinemedicineHumansPharmacology (medical)PlateleteducationGenetics (clinical)Whole bloodeducation.field_of_studyEpidermal Growth FactorPlatelet-Rich Plasmabusiness.industryGrowth factorVascular endothelial growth factorchemistryPlatelet-rich plasmaReviews and References (medical)businessAdvances in Clinical and Experimental Medicine
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The mechanisms of thrombotic risk induced by hormone replacement therapy.

2001

Abstract Objective : To review the available information on the action of hormones on the mechanisms involved in thrombotic risk. Results and Conclusions : Thrombosis plays a crucial role in the genesis and progression of both coronary heart disease (CHD) and venous thromboembolic disease (VTED), the two main forms of cardiovascular disease. Two main determinants of the thromboembolic phenotype, hypercoagulable state and altered endothelium, accumulate much of the work performed on the influence of hormones on thrombosis. Information has accumulated mainly for oestrogens, but increasing evidences support a role for progestogens. The sensitivity of each of the three components of the hemosta…

Blood PlateletsMalemedicine.medical_specialtyArteriosclerosismedicine.medical_treatmentCoronary DiseaseDiseaseBioinformaticsFibrinogenGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundRisk FactorsInternal medicineFibrinolysismedicineHumansHormone replacement therapyEndothelial dysfunctionAgedVenous ThrombosisHemostasisFactor VIIbusiness.industryAntithrombinEstrogen Replacement TherapyObstetrics and GynecologyEstrogensMiddle Agedmedicine.diseasePostmenopauseEndocrinologyPhenotypechemistryHemostasisFemaleEndothelium Vascularbusinessmedicine.drugMaturitas
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Platelet-endothelial interaction in tumor angiogenesis and microcirculation

2003

Activated platelets release angiogenic growth factors and have therefore been proposed to contribute to tumor angiogenesis within a potentially prothrombotic tumor microcirculation. The aim of the study was to investigate interactions of platelets with the angiogenic microvascular endothelium of highly vascularized solid tumors during growth and in response to endothelial stimulation in comparison with normal subcutaneous tissue. Experiments were performed in the dorsal skinfold chamber preparation of C57BL/6J mice bearing the Lewis lung carcinoma (LLC-1) or methylcholanthrene-induced fibrosarcoma (BFS-1). Fluorescently labeled rolling and adherent platelets, red blood cell velocity, and ve…

Blood PlateletsMalemedicine.medical_specialtyEndotheliumAngiogenesisFibrosarcomaImmunologyBiologyBiochemistrySkin Window TechniqueNeovascularizationCarcinoma Lewis LungMicePlatelet AdhesivenessCell MovementPlatelet adhesivenessInternal medicinemedicineAnimalsPlateletPlatelet activationCalcimycinIonophoresNeovascularization PathologicMicrocirculationVideotape RecordingLewis lung carcinomaCell BiologyHematologyPlatelet ActivationMice Inbred C57BLEndothelial stem cellmedicine.anatomical_structureEndocrinologyImmunologyCalciumEndothelium Vascularmedicine.symptomBlood Flow VelocityMethylcholanthreneBlood
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A single loading dose of clopidogrel causes dose-dependent improvement of endothelial dysfunction in patients with stable coronary artery disease: Re…

2006

Clinical studies have demonstrated beneficial effects for clopidogrel in patients with atherothrombotic disease. Recent in vitro studies identified stimulating effects of clopidogrel on endothelial cells, pointing towards mechanisms of action beyond the inhibition of platelet aggregation. We hypothesized that in vivo use of clopidogrel improves endothelial dysfunction in patients with coronary artery disease (CAD). Fifty-eight patients with CAD were randomly assigned to double-blinded oral administration of one single dose of clopidogrel 300 mg (C300) or 600 mg (C600), respectively. Endothelial function was assessed by measurement of flow-mediated dilation (FMD) of the brachial artery befor…

Blood PlateletsMalemedicine.medical_specialtyTiclopidineCoronary Artery DiseaseLoading doseCoronary artery diseaseP2Y12Double-Blind MethodSuperoxidesmedicine.arteryInternal medicinePurinergic P2 Receptor AntagonistsHumansMedicinePlateletcardiovascular diseasesBrachial arteryEndothelial dysfunctionAgedbusiness.industryVascular diseaseMicrofilament ProteinsMiddle AgedPhosphoproteinsmedicine.diseaseClopidogrelReceptors Purinergic P2Y12ClopidogrelAnesthesiaCardiologyFemaleEndothelium VascularCardiology and Cardiovascular MedicinebusinessCell Adhesion MoleculesDilatation Pathologiccirculatory and respiratory physiologymedicine.drugAtherosclerosis
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Blood flow patterns spatially associated with platelet aggregates in murine colitis.

2009

In the normal murine mucosal plexus, blood flow is generally smooth and continuous. In inflammatory conditions, such as chemically-induced murine colitis, the mucosal plexus demonstrates markedly abnormal flow patterns. The inflamed mucosal plexus is associated with widely variable blood flow velocity as well as discontinuous and even bidirectional flow. To investigate the mechanisms responsible for these blood flow patterns, we used intravital microscopic examination of blood flow within the murine mucosal plexus during dextran sodium sulphate-and trinitrobenzenesulfonic acid-induced colitis. The blood flow patterns within the mucosal plexus demonstrated flow exclusion in 18% of the vessel…

Blood PlateletsPathologymedicine.medical_specialtyHistologyPlatelet AggregationInterleukin-1betaBiologyArticleMicrocirculationchemistry.chemical_compoundMiceIntestinal mucosamedicineAnimalsPlateletColitisIntestinal MucosaEcology Evolution Behavior and SystematicsPlexusMice Inbred BALB CGene Expression ProfilingMicrocirculationDextran SulfateBlood flowmedicine.diseaseColitisMice Inbred C57BLDextranchemistryTrinitrobenzenesulfonic AcidRegional Blood FlowAcute DiseaseEndothelium VascularAnatomyChemokinesIntravital microscopyBiotechnologyAnatomical record (Hoboken, N.J. : 2007)
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Rapid formation of plasma protein corona critically affects nanoparticle pathophysiology

2013

In biological fluids, proteins bind to the surface of nanoparticles to form a coating known as the protein corona, which can critically affect the interaction of the nanoparticles with living systems. As physiological systems are highly dynamic, it is important to obtain a time-resolved knowledge of protein-corona formation, development and biological relevancy. Here we show that label-free snapshot proteomics can be used to obtain quantitative time-resolved profiles of human plasma coronas formed on silica and polystyrene nanoparticles of various size and surface functionalization. Complex time- and nanoparticle-specific coronas, which comprise almost 300 different proteins, were found to …

Blood Plateletsendocrine systemBiomedical EngineeringNanoparticleBioengineeringProtein CoronaNanotechnologyProteomicsCell Lineprotein coronaThrombocyte activationHumansGeneral Materials ScienceElectrical and Electronic EngineeringParticle SizeMicroscopy ConfocalCell DeathChemistrynanoparticleComputational BiologyEndothelial CellsBlood ProteinsCondensed Matter PhysicsHaemolysisSilicon DioxideBlood proteinsAtomic and Molecular Physics and OpticsMicrovesselsBiophysicsSurface modificationNanoparticlesPolystyrenesParticle sizeBiologie
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Increased Platelet Sensitivity toward Platelet Inhibitors during Physical Exercise in Patients with Coronary Artery Disease

1999

Generalized atherosclerosis and coronary artery disease (CAD) are associated with endothelial dysfunction and during acute myocardial ischemia platelet activation has been reported. Activated platelets exert activated fibrinogen receptors (GP IIb/IIIa) and express CD 62p being regarded as reliable marker for platelet activation. Patients with angiographically proven CAD performed a bicycle exercise test until the onset of angina or ST-segment depression. We studied the ischemia-induced alterations in fibrinogen binding to activated platelet GP IIb/IIIa receptors and CD 62p expression. Therefore, the basal fibrinogen binding to GP IIb/IIIa and CD 62p expression and the thrombin-concentration…

Blood Plateletsmedicine.medical_specialtyMyocardial IschemiaCoronary DiseaseProstacyclinPlatelet Glycoprotein GPIIb-IIIa ComplexNitric OxideFibrinogenThrombinRisk FactorsInternal medicinemedicineHumansPlateletcardiovascular diseasesPlatelet activationEndothelial dysfunctionbusiness.industryThrombinFibrinogen bindingHematologyMiddle AgedPlatelet Activationmedicine.diseaseEpoprostenolRadiographyP-SelectinEndocrinologyExercise TestCardiologybusinessPlatelet Aggregation Inhibitorsmedicine.drugIloprostThrombosis Research
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Blood pressure and glycaemia, not insulin-resistance, influence endothelial dysfunction in metabolic syndrome

2005

Blood pressure endothelial dysfunction metabolic syndrome
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