Search results for "DOT"

showing 10 items of 5147 documents

Procalcitonin and long-term prognosis after an admission for acute heart failure

2014

Abstract Background Traditionally, procalcitonin (PCT) is considered a diagnostic marker of bacterial infections. However, slightly elevated levels of PCT have also been found in patients with heart failure. In this context, it has been suggested that PCT may serve as a proxy for underrecognized infection, endotoxemia, or heightened proinflammatory activity. Nevertheless, the clinical utility of PCT in this setting is scarce. We aimed to evaluate the association between PCT and the risk of long-term outcomes. Methods and results We measured at admission PCT of 261 consecutive patients admitted for acute heart failure (AHF) after excluding active infection. Cox and negative binomial regressi…

CalcitoninMalemedicine.medical_specialtyCalcitonin Gene-Related PeptideRenal functionHeart failurePatient ReadmissionProcalcitoninCohort StudiesRisk FactorsInternal medicineNatriuretic Peptide Brainparasitic diseasesInternal MedicineHumansMedicineIn patientLongitudinal StudiesProspective StudiesProtein PrecursorsIntensive care medicineAgedProportional Hazards ModelsAged 80 and overHeart Failurebusiness.industryDiagnostic markerBacterial InfectionsMiddle AgedPrognosisbacterial infections and mycosesmedicine.diseaseInflammatory biomarkersPeptide FragmentsEndotoxinsHospitalizationHeart failureAcute DiseaseCytokinesFemaleRisk of deathbusinessProcalcitoninBiomarkershormones hormone substitutes and hormone antagonistsEuropean Journal of Internal Medicine
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Mr-Proadm Elevation Upon Icu Admission Predicts the Outcome of Septic Patients and is Correlated with Upcoming Fluid Overload.

2017

IF 3.113; International audience; Background: Among septic patients admitted to the intensive care unit (ICU), early recognition of those with the highest risk of death is of paramount importance. We evaluated the prognostic value of Procalcitonin (PCT), mid regional-proadrenomedullin (MR-proADM), copeptine and CT-proendothelin 1 (CT-ProET 1) concentrations. Methods: This was a prospective cohort study, which included 173 septic patient admitted to one ICU. Blood samples for biomarker measure-ments were obtained upon admission and on day 5. The predictive value of each biomarker regarding the risk of death at day 28 was assessed. The fluid balance was evaluated from admission to day 5. Resu…

CalcitoninMalemedicine.medical_specialtyTime FactorsArbitrary unit030204 cardiovascular system & hematologyCritical Care and Intensive Care MedicineProcalcitoninDisease-Free Survivallaw.inventionSepsis03 medical and health sciencesAdrenomedullin0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemlawInternal medicineSepsismedicine[ SDV.MHEP.HEM ] Life Sciences [q-bio]/Human health and pathology/HematologyHumansHospital MortalityProtein PrecursorsProspective cohort studyAgedAged 80 and overEndothelin-1business.industryGlycopeptides[SDV.MHEP.HEM]Life Sciences [q-bio]/Human health and pathology/Hematology030208 emergency & critical care medicine[ SDV.MHEP.CSC ] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemMiddle Agedmedicine.diseaseIntensive care unit3. Good healthSurgeryIcu admissionFluid balanceClinical trialHospitalizationIntensive Care UnitsEmergency MedicineproadrenomedullinBiomarker (medicine)FemalebusinessprocalcitoninBiomarkersShock (Augusta, Ga.)
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Biocompatibility studies of endothelial cells on a novel calcium phosphate/SiO 2 -xerogel composite for bone tissue engineering

2008

The bone biomaterial BONITmatrix®, a nanoporous, granular scaffold composed of hydroxylapatite, calcium phosphate and SiO2, linked by a dense collagen mesh, was tested for its biocompatibility using endothelial cells (EC) in the form of macrovascular HUVEC, microvascular HDMEC and the endothelial cell line ISOHAS-1. Cells were examined for their adherence and growth on the biomaterial and this was followed by confocal laser scanning microscopy after vital staining or immunocytochemical reactions, as well as by scanning electron microscopy. Macro- and microvascular ECs predominantly spread on BONITmatrix®-collagen mesh-covered surfaces and fibres and maintained their typical morphology. As E…

Calcium PhosphatesMaterials scienceBiocompatibilityCell SurvivalBiomedical Engineeringchemistry.chemical_elementBioengineeringCalciumBiomaterialschemistry.chemical_compoundTissue engineeringIn vivoMaterials TestingHumansCells CulturedCell ProliferationTissue EngineeringEndothelial CellsBiomaterialHydroxylapatiteSilicon DioxideIn vitroEndothelial stem cellchemistryBone SubstitutesBiophysicsGelsBiomedical engineeringBiomedical Materials
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In vitro evaluation of biomimetic chitosan-calcium phosphate scaffolds with potential application in bone tissue engineering.

2013

This work reports on the physicochemical properties and in vitro cytotoxicity assessment of chitosan–calcium phosphate (Cs–CP) scaffolds for bone tissue engineering, which were synthesized by a novel biomimetic co-precipitation method. X-ray diffraction (XRD) along with scanning electron microscopy (SEM) analysis confirmed the porous morphology of the scaffolds and the amorphous nature of the inorganic phase with different crystallite sizes and the formation of various forms of calcium phosphate. Compressive mechanical testing revealed that the Young’s modulus of the biomaterials is in the range of human trabecular bone. In vitro tests were performed on the biomaterials for up to 14 days to…

Calcium PhosphatesMaterials scienceCompressive StrengthCell SurvivalBiomedical EngineeringBioengineeringBone remodelingCell LineBiomaterialschemistry.chemical_compoundIn vivoBiomimetic MaterialsHardnessElastic ModulusMaterials TestingmedicineHumansViability assayCytotoxicityChitosanOsteoblastsOsteoblastIn vitroVascular endothelial growth factormedicine.anatomical_structurechemistryCell cultureBone SubstitutesBiophysicsBiomedical engineeringBiomedical materials (Bristol, England)
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Microstructure, mechanical characteristics and cell compatibility of β-tricalcium phosphate reinforced with biodegradable Fe–Mg metal phase

2015

The use of beta-tricalcium phosphate (β-TCP) ceramic as a bioresorbable bone substitute is limited to non-load-bearing sites by the material׳s brittleness and low bending strength. In the present work, new biocompatible β-TCP-based composites with improved mechanical properties were developed via reinforcing the ceramic matrix with 30 vol% of a biodegradable iron-magnesium metallic phase. β-TCP-15Fe15Mg and β-TCP-24Fe6Mg (vol%) composites were fabricated using a combination of high energy attrition milling, cold sintering/high pressure consolidation of powders at room temperature and annealing at 400 °C. The materials synthesized had a hierarchical nanocomposite structure with a nanocrystal…

Calcium PhosphatesMaterials scienceIronComposite numberBiomedical EngineeringSinteringBiocompatible Materials02 engineering and technology010402 general chemistryCeramic matrix composite01 natural sciencesCell LineBiomaterialsFlexural strengthMaterials TestingHumansMagnesiumCeramicComposite materialMechanical PhenomenaOsteoblastsNanocompositeEndothelial Cells021001 nanoscience & nanotechnologyMicrostructureNanocrystalline material0104 chemical sciencesMechanics of Materialsvisual_artvisual_art.visual_art_medium0210 nano-technologyJournal of the Mechanical Behavior of Biomedical Materials
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Influence of β-tricalcium phosphate granule size and morphology on tissue reaction in vivo.

2010

In this study the tissue reaction to five different β-tricalcium phosphate (β-TCP)-based bone substitute materials differing only in size, shape and porosity was analyzed over 60 days, at 3, 10, 15, 30 and 60 days after implantation. Using the subcutaneous implantation model in Wistar rats both the inflammatory response within the implantation bed and the resulting vascularization of the biomaterials were qualitatively and quantitatively assessed by means of standard and special histological staining methods. The data from this study showed that all investigated β-TCP bone substitutes induced the formation of multinucleated giant cells. Changes in size, shape and porosity influenced the int…

Calcium PhosphatesVascular Endothelial Growth Factor AChemokineMaterials scienceCellBiomedical EngineeringNeovascularization PhysiologicBiocompatible MaterialsBiochemistryGiant CellsBiomaterialschemistry.chemical_compoundImplants ExperimentalX-Ray DiffractionIn vivomedicineAnimalsParticle SizeRats WistarMolecular BiologybiologyGranule (cell biology)Acid phosphataseBiomaterialGeneral MedicineAnatomyImmunohistochemistryRatsVascular endothelial growth factormedicine.anatomical_structurechemistryGiant cellOrgan SpecificityBone Substitutesbiology.proteinBiophysicsMicroscopy Electron ScanningBiotechnologyActa biomaterialia
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CD90+ liver cancer cells modulate endothelial cell phenotype through the release of exosomes containing H19 lncRNA

2015

Background CD90+ liver cancer cells have been described as cancer stem-cell-like (CSC), displaying aggressive and metastatic phenotype. Using two different in vitro models, already described as CD90+ liver cancer stem cells, our aim was to study their interaction with endothelial cells mediated by the release of exosomes. Methods Exosomes were isolated and characterized from both liver CD90+ cells and hepatoma cell lines. Endothelial cells were treated with exosomes, as well as transfected with a plasmid containing the full length sequence of the long non-coding RNA (lncRNA) H19. Molecular and functional analyses were done to characterize the endothelial phenotype after treatments. Results …

Cancer ResearchAngiogenesisAngiogenesis; CD90+ liver cancer cells; Exosomes; Long-non-coding RNA H19; Antigens Thy-1; Cell Adhesion; Cell Line Tumor; Endothelial Cells; Exosomes; Human Umbilical Vein Endothelial Cells; Humans; Liver Neoplasms; RNA Long Noncoding; Phenotype; Molecular Medicine; Oncology; Cancer ResearchBiologyCD90+ liver cancer cellsExosomesCell LineSettore BIO/13 - Biologia ApplicataCancer stem cellCell Line TumormedicineCell AdhesionHuman Umbilical Vein Endothelial CellsHumansCD90AntigensThy-1TumorExosomes Long-non-coding RNA H19 CD90+ liver cancer cells AngiogenesisResearchLiver NeoplasmsCancerEndothelial Cellsmedicine.diseaseMicrovesiclesCell biologyEndothelial stem cellPhenotypeOncologyembryonic structuresThy-1 AntigensRNAMolecular MedicineRNA Long NoncodingLong NoncodingAngiogenesisStem cellLiver cancerLong-non-coding RNA H19Molecular Cancer
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Role of exosomes released by chronic myelogenous leukemia cells in angiogenesis

2012

The present study is designed to assess if exosomes released from Chronic Myelogenous Leukemia (CML) cells may modulate angiogenesis. We have isolated and characterized the exosomes generated from LAMA84 CML cells and demonstrated that addition of exosomes to human vascular endothelial cells (HUVEC) induces an increase of both ICAM-1 and VCAM-1 cell adhesion molecules and interleukin-8 expression. The stimulation of cell-cell adhesion molecules was paralleled by a dose-dependent increase of adhesion of CML cells to a HUVEC monolayer. We further showed that the treatment with exosomes from CML cells caused an increase in endothelial cell motility accompanied by a loss of VE-cadherin and β-ca…

Cancer ResearchAngiogenesisVascular Cell Adhesion Molecule-1BiologyExosomesArticleExosomes Chronic Myelogenous Leukemia Cells Endothelial cells Tumor MicroenvironmentMiceAntigens CDCell Movementhemic and lymphatic diseasesCell Line TumorLeukemia Myelogenous Chronic BCR-ABL PositivemedicineCell AdhesionHuman Umbilical Vein Endothelial CellsTumor MicroenvironmentAnimalsHumansCell adhesionbeta CateninMatrigelTumor microenvironmentNeovascularization PathologicCell adhesion moleculeInterleukin-8medicine.diseaseCadherinsIntercellular Adhesion Molecule-1MicrovesiclesCell biologyEndothelial stem cellDrug CombinationsOncologyGene Expression RegulationCancer researchProteoglycansCollagenLamininChronic myelogenous leukemia
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Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge ahead

2015

Goodson, William H. et al.

Cancer ResearchCarcinogenesis[SDV]Life Sciences [q-bio]METHOXYCHLOR-INDUCED ALTERATIONSReviewPharmacologyMESH: Carcinogens EnvironmentalCarcinogenic synergiesChemical mixturesNeoplasmsMESH: AnimalsMESH: NeoplasmsCarcinogenesiRisk assessmentCancerACTIVATED PROTEIN-KINASESMedicine (all)Low dose1. No povertyCumulative effectsBREAST-CANCER CELLSGeneral MedicineEnvironmental exposureMESH: CarcinogenesisBIO/10 - BIOCHIMICAEPITHELIAL-MESENCHYMAL TRANSITION3. Good health[SDV] Life Sciences [q-bio]Environmental CarcinogenesisESTROGEN-RECEPTOR-ALPHARisk assessmentHumanMESH: Environmental ExposureENDOCRINE-DISRUPTING CHEMICALSTARGETING TISSUE FACTOR[SDV.CAN]Life Sciences [q-bio]/CancerBiologyPrototypical chemical disruptorsExposure[SDV.CAN] Life Sciences [q-bio]/CancerEnvironmental healthmedicine[SDV.EE.SANT] Life Sciences [q-bio]/Ecology environment/HealthCarcinogenEnvironmental carcinogenesis[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/HealthMESH: HumansAnimalPOLYBROMINATED DIPHENYL ETHERSCancerEnvironmental Exposuremedicine.diseaseMESH: Hazardous SubstancesCarcinogens EnvironmentalMIGRATION INHIBITORY FACTORVASCULAR ENDOTHELIAL-CELLSHazardous SubstanceNeoplasmCarcinogenesis
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Osteosarcoma cell-derived exosomes affect tumor microenvironment by specific packaging of microRNAs

2018

Abstract Bone microenvironment provides growth and survival signals essential for osteosarcoma (OS) initiation and progression. OS cells regulate communications inside tumor microenvironment through different ways and, among all, tumor-derived exosomes support cancer progression and metastasis. To define the contribution of OS-derived exosomes inside the microenvironment, we investigated the effects induced in bone remodeling mechanism and tumor angiogenesis. We demonstrated that exosomes promoted osteoclasts differentiation and bone resorption activity. Furthermore, exosomes potentiated tube formation of endothelial cells and increased angiogenic markers expression. We therefore investigat…

Cancer ResearchCellBone NeoplasmsBiologyExosomesmedicine.disease_causeCell MovementSettore BIO/13 - Biologia ApplicataosteosarcomamicroRNABiomarkers TumormedicineHumansexosometumor microenvironmentTelomerase reverse transcriptaseCells CulturedCell ProliferationTube formationTumor microenvironmentNeovascularization PathologicGene Expression ProfilingGeneral Medicinemedicine.diseaseMicrovesiclesGene Expression Regulation NeoplasticMicroRNAsmedicine.anatomical_structureCancer researchmicroRNAs profilingOsteosarcomaEndothelium VascularCarcinogenesis
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