Search results for "DPH"

showing 10 items of 361 documents

Energetic coupling between plastids and mitochondria drives CO2 assimilation in diatoms.

2015

International audience; Diatoms are one of the most ecologically successful classes of photosynthetic marine eukaryotes in the contemporary oceans. Over the past 30 million years, they have helped to moderate Earth's climate by absorbing carbon dioxide from the atmosphere, sequestering it via the biological carbon pump and ultimately burying organic carbon in the lithosphere. The proportion of planetary primary production by diatoms in the modern oceans is roughly equivalent to that of terrestrial rainforests. In photosynthesis, the efficient conversion of carbon dioxide into organic matter requires a tight control of the ATP/NADPH ratio which, in other photosynthetic organisms, relies prin…

Aquatic Organismschemistry.chemical_compoundAdenosine TriphosphateSettore BIO/04 - Fisiologia VegetaleCYCLIC ELECTRON FLOWPlastidsPhotosynthesisPHAEODACTYLUM-TRICORNUTUMPlant Proteinschemistry.chemical_classificationMultidisciplinarymicroalgaeRespirationCarbon fixationEnergetic interactionsProton-Motive ForceMitochondriametabolic mutantPhenotypeATP/NADPH ratioOXYGEN PHOTOREDUCTIONCarbon dioxideOxidoreductasesOxidation-ReductionOceanOceans and SeasElectron flowMarine eukaryotesBiologyPhotosynthesisCHLAMYDOMONAS-REINHARDTIICarbon cycleCarbon CycleMitochondrial ProteinsEnergetic exchangesBotanyOrganic matterEcosystem[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology14. Life underwaterPlastidEcosystemDiatomsChemiosmosisfungiECSCarbon Dioxidechemistry13. Climate actionNADP
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On the feasibility of personal audio systems over a network of distributed loudspeakers

2018

Los sistemas de reproducción de audio personal se ocupan de la creación de zonas sonoras personales dentro de una habitación sin necesidad de utilizar auriculares. Estos sistemas utilizan un conjunto de altavoces y diseñan los filtros necesarios en cada altavoz con el fin de que la señal de audio deseada llegue a cada persona en la sala lo más libre de interferencias posible. Existen propuestas muy interesantes en la literatura que hacen uso de arrays circulares o lineales, pero en este trabajo estudiamos el problema considerando una red de altavoces distribuidos controlados por un conjunto de nodos acústicos, que pueden intercambiar información a través de una red. Enunciamos el modelo de …

Audio signalbusiness.product_category:CIENCIAS TECNOLÓGICAS [UNESCO]MicrophoneComputer scienceAcoustics020206 networking & telecommunications02 engineering and technologypersonal audio systemsUNESCO::CIENCIAS TECNOLÓGICASGeneralLiterature_MISCELLANEOUSSignal-to-noise ratioSound reinforcement system0202 electrical engineering electronic engineering information engineeringElectronic engineering020201 artificial intelligence & image processingLoudspeakerDirectional soundwireless acoustic sensor networksbusinessHeadphones
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MTHFR 677C → T genotype modulates the effect of a 5-year supplementation with B-vitamins on homocysteine concentration: The SU.FOL.OM3 randomized con…

2018

Aims To study how MTHFR 677C→T genotype modulates the effect of supplementation with B-vitamins on total homocysteine (tHcy) and B-vitamin concentrations. Methods 2381 patients with a personal history of cardiovascular disease were randomly assigned to one of four groups: 1) B-vitamins alone (560 μg of 5-methyl-THF, 3 mg of vitamin B6 and 20 μg of vitamin B12), 2) n-3 fatty acids alone (600 mg of EPA and DHA in a 2:1 ratio), 3) B-vitamins and n-3 fatty acids, and 4) placebo. Participants were followed up for 4.7 years. At baseline and annually thereafter, biological parameters were assessed. Multivariate and linear mixed models were fit to study the interaction between B-vitamins and MTHFR …

B VitaminsMaleHomocysteinePhysiologylcsh:Medicine[SDV.GEN] Life Sciences [q-bio]/Genetics030204 cardiovascular system & hematologyBiochemistryGastroenterologychemistry.chemical_compound0302 clinical medicineBlood plasmaGenotypeMedicine and Health Sciences030212 general & internal medicinelcsh:ScienceHomocysteine[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyMultidisciplinarybiologyOrganic CompoundsFatty AcidsPyridoxineVitaminsMiddle AgedLipidsBody Fluids3. Good healthChemistryBloodCardiovascular DiseasesCreatininePhysical SciencesVitamin B ComplexFemaleAnatomyResearch Articlemedicine.medical_specialtyGenotypePlaceboBlood PlasmaCobalamins03 medical and health sciencesFolic AcidDouble-Blind MethodInternal medicine[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyVitamin B12Methylenetetrahydrofolate Reductase (NADPH2)[SDV.GEN]Life Sciences [q-bio]/GeneticsCreatininePolymorphism Geneticbusiness.industryOrganic Chemistrylcsh:RChemical CompoundsBiology and Life Sciences[SDV.AEN] Life Sciences [q-bio]/Food and NutritionB vitaminschemistry[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieMethylenetetrahydrofolate reductaseDietary Supplementsbiology.protein[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologielcsh:Qbusiness[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBiomarkers[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyPLoS ONE
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Short-term e-cigarette vapour exposure causes vascular oxidative stress and dysfunction: evidence for a close connection to brain damage and a key ro…

2019

Abstract Aims Electronic (e)-cigarettes have been marketed as a ‘healthy’ alternative to traditional combustible cigarettes and as an effective method of smoking cessation. There are, however, a paucity of data to support these claims. In fact, e-cigarettes are implicated in endothelial dysfunction and oxidative stress in the vasculature and the lungs. The mechanisms underlying these side effects remain unclear. Here, we investigated the effects of e-cigarette vapour on vascular function in smokers and experimental animals to determine the underlying mechanisms. Methods and results Acute e-cigarette smoking produced a marked impairment of endothelial function in chronic smokers determined b…

Behavioural risk factorInflammationElectronic Nicotine Delivery Systems030204 cardiovascular system & hematologyPharmacologymedicine.disease_causeVascular MedicineLifestyle drugNicotineLipid peroxidationMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBasic ScienceAnimalsHumansMedicineEndothelial dysfunction030212 general & internal medicineEndothelial dysfunctionMacitentanNADPH oxidasebiologybusiness.industryBrainNADPH Oxidasesmedicine.diseaseE-cigarette vapourEditor's ChoiceLeukemia Myeloid AcuteOxidative Stressmedicine.anatomical_structurechemistryE-Cigarette VaporNADPH Oxidase 2Neoplastic Stem Cellsbiology.proteinmedicine.symptomCardiology and Cardiovascular MedicinebusinessOxidative stressmedicine.drugBlood vesselEuropean Heart Journal
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New evidence for the multiplicity of ubiquinone- and inhibitor-binding sites in the mitochondrial complex I.

2000

Determination of the number of ubiquinone- and inhibitor-binding sites in the mitochondrial complex I (NADH:ubiquinone oxidoreductase) is a controversial question with a direct implication for elaborating a suitable model to explain the bioenergetic mechanism of this complicated enzyme. We have used combinations of both selective inhibitors and common ubiquinone-like substrates to demonstrate the multiplicity of the reaction centers in the complex I in contrast with competition studies that have suggested the existence of a unique binding site for ubiquinone. Our results provide new evidence for the existence of at least two freely exchangeable ubiquinone-binding sites with different specif…

BioenergeticsStereochemistryUbiquinoneSubmitochondrial ParticlesBiophysicsBiologyIn Vitro TechniquesBiochemistryModels BiologicalMitochondria HeartSubstrate SpecificityOxidoreductaseAnimalsNADH NADPH OxidoreductasesBinding siteMultiplicity (chemistry)Molecular Biologychemistry.chemical_classificationNADH-Ubiquinone OxidoreductaseBinding SitesElectron Transport Complex IKineticsEnzymechemistryBiochemistryCattleEnergy MetabolismMitochondrial Complex IArchives of biochemistry and biophysics
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Hypothalamic reactive oxygen species are required for insulin-induced food intake inhibition: an NADPH oxidase-dependent mechanism

2009

1939-327X (Electronic) Journal Article Research Support, Non-U.S. Gov't; OBJECTIVE: Insulin plays an important role in the hypothalamic control of energy balance, especially by reducing food intake. Emerging data point to a pivotal role of reactive oxygen species (ROS) in energy homeostasis regulation, but their involvement in the anorexigenic effect of insulin is unknown. Furthermore, ROS signal derived from NADPH oxidase activation is required for physiological insulin effects in peripheral cells. In this study, we investigated the involvement of hypothalamic ROS and NADPH oxidase in the feeding behavior regulation by insulin. RESEARCH DESIGN AND METHODS: We first measured hypothalamic RO…

Blood GlucoseMaleReactive Oxygen Species/*metabolismHypothalamusHomeostasis/drug effects/physiologyInbred C57BLCerebral VentriclesCerebral Ventricles/drug effects/*physiologyMiceHomeostasisInsulinAnimalsBlood Glucose/metabolismHypothalamus/*physiologyInsulin/administration & dosage/blood/*pharmacologyNADPH OxidasesEnergy Intake/drug effects/*physiologyNADPH Oxidase/*metabolismGlutathioneGlutathione/metabolismMice Inbred C57BLOriginal ArticleEnergy IntakeReactive Oxygen SpeciesEnergy MetabolismSignal Transduction
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Time course of asymmetric dimethylarginine (ADMA) and oxidative stress in fructose-hypertensive rats: A model related to metabolic syndrome

2011

Asymmetric dimethylarginine (ADMA) is an endogenous modulator of endothelial function and oxidative stress, and increased levels of this molecule have been reported in some metabolic disorders and cardiovascular diseases. The aim of this work was to analyze the time course of dimethylarginine compounds and oxidative stress levels and the relationship between these and cardiovascular function in fructose-hypertensive rats.90 male Sprague-Dawley rats were randomized into 2 groups, fed for 3 months with standard (C) chow supplemented or not with fructose (F, 60%). After sacrifice at different weeks (W), the aorta and plasma were harvested to assess the vascular and biochemical parameters. Our …

Blood GlucoseMaleTime FactorsVasodilator AgentsNitric Oxide Synthase Type IIBlood Pressure030204 cardiovascular system & hematologymedicine.disease_causeRats Sprague-Dawleychemistry.chemical_compound0302 clinical medicineHeart RateEnzyme InhibitorsAortaComputingMilieux_MISCELLANEOUSMetabolic Syndrome0303 health sciencesOxidase testVasodilationNAD(P)H oxidaseHypertensionCardiology and Cardiovascular Medicinemedicine.medical_specialtyFructoseArginine03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicinemedicine.arterymedicineAnimals030304 developmental biologyAortaDose-Response Relationship Drugbusiness.industryVascular diseaseBody WeightNADPH OxidasesFructosemedicine.diseaseRatsDisease Models AnimalOxidative StressEndocrinologychemistryTyrosineMetabolic syndromebusinessAsymmetric dimethylarginineOxidative stressAtherosclerosis
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Vascular Dysfunction in Streptozotocin-Induced Experimental Diabetes Strictly Depends on Insulin Deficiency

2010

<i>Objective:</i> In previous studies we and others have shown that streptozotocin (STZ)-induced diabetes in rats is associated with vascular oxidative stress and dysfunction. In the present study, we sought to determine whether vascular dysfunction and oxidative stress strictly depend on insulin deficiency. <i>Methods:</i> The effects of insulin (2.5 U/day s.c., 2 weeks) therapy on vascular disorders in STZ-induced (60 mg/kg i.v., 8 weeks) diabetes mellitus (type I) were studied in Wistar rats. The contribution of NADPH oxidase to overall oxidative stress was investigated by in vivo (30 mg/kg/day s.c., 4 days) and in vitro treatment with apocynin. <i>Results:&…

Blood GlucoseMalemedicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumPhysiologymedicine.medical_treatmentmedicine.disease_causeStreptozocinDiabetes Mellitus Experimentalchemistry.chemical_compoundInternal medicineDiabetes mellitusmedicineAnimalsInsulinRats WistarEndothelial dysfunctionNADPH oxidasebiologybusiness.industryMyocardiumInsulinAcetophenonesNADPH OxidasesStreptozotocinmedicine.diseaseRatsOxidative StressNG-Nitroarginine Methyl EsterEndocrinologymedicine.anatomical_structurechemistryApocyninbiology.proteinEndothelium VascularCardiology and Cardiovascular MedicinebusinessDiabetic AngiopathiesOxidative stressmedicine.drugJournal of Vascular Research
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AT1-receptor blockade by telmisartan upregulates GTP-cyclohydrolase I and protects eNOS in diabetic rats.

2008

Several enzymatic sources of reactive oxygen species (ROS) were described as potential reasons of eNOS uncoupling in diabetes mellitus. In the present study, we investigated the effects of AT1-receptor blockade with chronic telmisartan (25 mg/kg/day, 6.5 weeks) therapy on expression of the BH4-synthesizing enzyme GTP-cyclohydrolase I (GCH-I), eNOS uncoupling, and endothelial dysfunction in streptozotocin (STZ, 60 mg/kg iv, 7 weeks)-induced diabetes mellitus (type I). Telmisartan therapy did not modify blood glucose and body weight. Aortas from diabetic animals had vascular dysfunction as revealed by isometric tension studies (acetylcholine and nitroglycerin potency). Vascular and cardiac RO…

Blood GlucoseMalemedicine.medical_specialtyNitric Oxide Synthase Type IIImedicine.disease_causeBiochemistryBenzoatesReceptor Angiotensin Type 1chemistry.chemical_compoundEnosPhysiology (medical)Internal medicinemedicineDiabetes MellitusAnimalsTelmisartanEndothelial dysfunctionRats WistarXanthine oxidaseGTP CyclohydrolaseNADPH oxidasebiologySuperoxideBody WeightNADPH Oxidasesmedicine.diseaseStreptozotocinbiology.organism_classificationMitochondriaRatsUp-RegulationEnzyme ActivationOxidative StressEndocrinologychemistrybiology.proteinBenzimidazolesTelmisartanAngiotensin II Type 1 Receptor BlockersOxidative stressmedicine.drugFree radical biologymedicine
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Vascular Dysfunction in Experimental Diabetes Is Improved by Pentaerithrityl Tetranitrate but Not Isosorbide-5-Mononitrate Therapy

2011

OBJECTIVE Diabetes is associated with vascular oxidative stress, activation of NADPH oxidase, and uncoupling of nitric oxide (NO) synthase (endothelial NO synthase [eNOS]). Pentaerithrityl tetranitrate (PETN) is an organic nitrate with potent antioxidant properties via induction of heme oxygenase-1 (HO-1). We tested whether treatment with PETN improves vascular dysfunction in the setting of experimental diabetes. RESEARCH DESIGN AND METHODS After induction of hyperglycemia by streptozotocin (STZ) injection (60 mg/kg i.v.), PETN (15 mg/kg/day p.o.) or isosorbide-5-mononitrate (ISMN; 75 mg/kg/day p.o.) was fed to Wistar rats for 7 weeks. Oxidative stress was assessed by optical methods and o…

Blood GlucoseMalemedicine.medical_specialtyXanthine OxidaseEndocrinology Diabetes and MetabolismVasodilator AgentsOxidative phosphorylationIsosorbide Dinitratemedicine.disease_causeWeight GainNitric oxideDiabetes Mellitus Experimentalchemistry.chemical_compoundEnosInternal medicineInternal MedicinemedicineAnimalsPentaerythritol TetranitrateGene SilencingEndothelial dysfunctionRats WistarXanthine oxidaseGTP CyclohydrolaseNADPH oxidasebiologyNADPH Oxidasesmedicine.diseasebiology.organism_classificationStreptozotocinPharmacology and TherapeuticsRatsOxidative StressEndocrinologychemistryVasoconstrictionbiology.proteinEndothelium VascularReactive Oxygen SpeciesOxidative stressHeme Oxygenase-1medicine.drugDiabetes
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