Search results for "DYSTROPHY"

showing 10 items of 268 documents

The Neuropsychological Profile of Infantile Duchenne Muscular Dystrophy

2011

It has been shown that children with Duchenne muscular dystrophy (DMD) exhibit specific cognitive deficits. However, the neuropsychological profile has not yet been fully characterized. In order to control for the contribution of motor impairments as a confounding variable that is usually present when assessing children with muscular pathologies, we compared children with DMD to a group of children with an autoimmune pathology that does not entail either brain or cognitive dysfunction but does imply motor impairment: juvenile rheumatoid arthritis (JRA). An extensive neuropsychological evaluation was administered, including intelligence, perception, language, memory and learning, attention, …

MaleDevelopmental DisabilitiesDuchenne muscular dystrophyIntelligenceNeuropsychological TestsPraxiJuvenile rheumatoid arthritiDevelopmental psychologySettore M-PSI/04 - Psicologia Dello Sviluppo E Psicologia Dell'EducazioneExecutive FunctionDiscrimination PsychologicalDevelopmental and Educational PsychologyNeuropsychological assessmentMuscular dystrophyChildmedia_commonmedicine.diagnostic_testMedicine (all)NeuropsychologyPhonological analysiCognitionVerbal LearningPsychiatry and Mental healthClinical PsychologyMemory Short-TermNeuropsychology and Physiological PsychologyChild PreschoolNeuropsychological TestFemalePsychologyHumanClinical psychologyDuchenne muscular dystrophymusculoskeletal diseasesDevelopmental Disabilitiemedia_common.quotation_subjectVerbal learningNeuropsychological assessmentStatistics NonparametricCognition DisorderArts and Humanities (miscellaneous)PerceptionmedicineHumansDiscrimination (Psychology)Settore M-PSI/02 - Psicobiologia E Psicologia Fisiologicamedicine.diseaseArthritis JuvenileMuscular Dystrophy DuchennePerceptionCognition DisordersJuvenile rheumatoid arthritis
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Mild Aerobic Exercise Training Hardly Affects the Diaphragm of mdx Mice

2016

In the mdx mice model of Duchenne Muscular Dystrophy (DMD), mild endurance exercise training positively affected limb skeletal muscles, whereas few and controversial data exist on the effects of training on the diaphragm. The diaphragm was examined in mdx (C57BL/10ScSn-Dmdmdx) and wild-type (WT, C57BL/10ScSc) mice under sedentary conditions (mdx-SD, WT-SD) and during mild exercise training (mdx-EX, WT-EX). At baseline, and after 30 and 45 days (training: 5 d/wk for 6 weeks), diaphragm muscle morphology and Cx39 protein were assessed. In addition, tissue levels of the chaperonins Hsp60 and Hsp70 and the p65 subunit of nuclear factor-kB (NF-kB) were measured in diaphragm, gastrocnemius, and q…

MaleDuchenne muscular dystrophychaperoninTime FactorsDiaphragmSettore MED/10 - Malattie Dell'Apparato RespiratorioSettore BIO/09 - FisiologiaConnexinsMitochondrial ProteinsNecrosisendurance trainingAnimalsGenetic Predisposition to DiseaseHSP70 Heat-Shock Proteinsstress markersMuscle StrengthNF-kBSettore BIO/16 - Anatomia UmanaTranscription Factor RelAChaperonin 60Settore CHIM/06 - Chimica OrganicaExercise TherapyMuscular Dystrophy DuchenneDisease Models AnimalPhenotypeMice Inbred mdxPhysical Endurance
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Altered electrical activity in colonic smooth muscle cells from dystrophic (mdx) mice

2001

Because the colon from dystrophic (mdx) mice shows an altered motor pattern, probably due to neural disorders, our aim was to examine the electrophysiological properties of muscle cells and the functionality of nitrergic transmission in circular muscle from normal and mdx colon. Normal colonic cells (resting membrane potential [RMP] about -50 mV) showed spontaneous hyperpolarizations (inhibitory junction potentials; IJPs) and cyclic slow depolarizations were sometimes recorded. Mdx colon had a depolarized RMP (about -36 mV) and spontaneous IJPs, but the cyclic activity was never observed. In the normal colon, Nomega-nitro-L-arginine methyl ester (L-NAME) induced depolarization and abolished…

MaleDuchenne muscular dystrophymedicine.medical_specialtyInhibitory junction potentialColonPhysiologyDuchenne muscular dystrophyInhibitory postsynaptic potentialSynaptic TransmissionSettore BIO/09 - FisiologiaProximal colonMembrane PotentialsMiceSmooth muscleInternal medicinemedicineAnimalsMyocyteEnzyme InhibitorsMembrane potentialNeuroscience (all)Endocrine and Autonomic SystemsChemistryGastroenterologyMuscle SmoothNitric oxideDepolarizationMuscular Dystrophy AnimalHyperpolarization (biology)medicine.diseaseElectric StimulationElectrophysiologyMice Inbred C57BLMuscular Dystrophy DuchenneMdx miceElectrophysiologyNG-Nitroarginine Methyl EsterEndocrinologyMice Inbred mdxSodium nitroprussideNeurosciencemedicine.drugNeurogastroenterology and Motility
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Absence of dysferlin alters myogenin expression and delays human muscle differentiation 'in vitro'

2006

Mutations in dysferlin cause a type of muscular dystrophy known as dysferlinopathy. Dysferlin may be involved in muscle repair and differentiation. We compared normal human skeletal muscle cultures expressing dysferlin with muscle cultures from dysferlinopathy patients. We quantified the fusion index of myoblasts as a measure of muscle development and conducted optic and electronic microscopy, immunofluorescence, Western blot, flow cytometry, and real-time PCR at different developmental stages. Short interference RNA was used to corroborate the results obtained in dysferlin-deficient cultures. A luciferase reporter assay was performed to study myogenin activity in dysferlin-deficient cultur…

MaleDysferlinopathyMuscle ProteinsIn Vitro TechniquesBiochemistryMuscular DystrophiesDysferlinmedicineMyocyteHumansMuscular dystrophyMuscle SkeletalMolecular BiologyDysferlinMyogeninCells CulturedbiologyMyogenesisMusclesSkeletal muscleMembrane ProteinsCell DifferentiationCell Biologymedicine.diseaseMolecular biologyCD56 Antigenmedicine.anatomical_structureGene Expression RegulationCase-Control Studiesbiology.proteinFemaleMyogeninMyofibrilSignal Transduction
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Muscle NAD+ depletion and Serpina3n as molecular determinants of murine cancer cachexia—the effects of blocking myostatin and activins

2020

Objective Cancer cachexia and muscle loss are associated with increased morbidity and mortality. In preclinical animal models, blocking activin receptor (ACVR) ligands has improved survival and prevented muscle wasting in cancer cachexia without an effect on tumour growth. However, the underlying mechanisms are poorly understood. This study aimed to identify cancer cachexia and soluble ACVR (sACVR) administration-evoked changes in muscle proteome. Methods Healthy and C26 tumour-bearing (TB) mice were treated with recombinant sACVR. The sACVR or PBS control were administered either prior to the tumour formation or by continued administration before and after tumour formation. Muscles were an…

MaleEXPRESSIONActivin receptor; APR; C26; Cancer cachexia; Nrk2; OXPHOSlcsh:Internal medicineCachexiaREVERSALActivin ReceptorsMETABOLISMactivin receptorOxidative PhosphorylationCell Line TumorAnimalsMuscle Skeletallcsh:RC31-1245aineenvaihduntaSerpinslihassolut318 Medical biotechnologyNrk2Cancer cachexiaMyostatinNADOXPHOSMUSCULAR-DYSTROPHYActivinsMitochondriaActivin receptorDisease Models AnimalMuscular AtrophyMICESIRTUINSOriginal ArticlesyöpätauditproteiinitC26lihassurkastumasairaudetAPRAcute-Phase Proteinscancer cachexia
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In-frame deletion in the seventh immunoglobulin-like repeat of filamin C in a family with myofibrillar myopathy.

2009

Myofibrillar myopathies (MFMs) are an expanding and increasingly recognized group of neuromuscular disorders caused by mutations in DES, CRYAB, MYOT, and ZASP. The latest gene to be associated with MFM was FLNC; a p.W2710X mutation in the 24th immunoglobulin-like repeat of filamin C was shown to be the cause of a distinct type of MFM in several German families. We studied an International cohort of 46 patients from 39 families with clinically and myopathologically confirmed MFM, in which DES, CRYAB, MYOT, and ZASP mutations have been excluded. In patients from an unrelated family a 12-nucleotide deletion (c.2997_3008del) in FLNC resulting in a predicted in-frame four-residue deletion (p.Val…

MaleFilaminsDNA Mutational AnalysisImmunoblottingMolecular Sequence DataImmunoglobulinsmacromolecular substancesBiologymedicine.disease_causeFilaminArticle03 medical and health sciences0302 clinical medicineContractile ProteinsMuscular DiseasesMyofibrilsGeneticsmedicineHumansFLNCAmino Acid SequenceMyopathyRepeated sequenceMuscle SkeletalGenePeptide sequenceGenetics (clinical)030304 developmental biologyRepetitive Sequences Nucleic AcidSequence DeletionGeneticsFamily Health0303 health sciencesMutationSequence Homology Amino AcidMicrofilament Proteinsmedicine.diseaseMolecular biologyImmunohistochemistry3. Good healthMicroscopy ElectronMutationFemalemedicine.symptom030217 neurology & neurosurgeryLimb-girdle muscular dystrophyEuropean journal of human genetics : EJHG
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Giant axonal neuropathy and leukodystrophy

1991

Abstract An 11-year-old Persian boy, born to consanguineous parents, manifested a progressive gait abnormality beginning at 5 years of age. A severe cerebellar disorder developed with associated dysfunction of the peripheral nervous system, but no sign of mental impairment. The sensory and motor nerve conduction velocities were greatly reduced, especially in the lower extremities. Cerebrospinal fluid protein was normal. Computed tomography and magnetic resonance imaging revealed leukoencephalopathy, especially in the cerebellum, but also in periventricular areas. The diagnosis of giant axonal neuropathy was established by biopsy of the sural nerve. The few previous histologic examinations h…

MaleIntermediate FilamentsMotor nerveGenes RecessiveSural nerveCerebral VentriclesLeukoencephalopathyConsanguinityDevelopmental NeuroscienceCerebellummedicineHumansCerebellar disorderGliosisPeripheral NervesChildMyelin SheathSpinocerebellar DegenerationsGiant axonal neuropathybusiness.industryLeukodystrophyAnatomymedicine.diseaseMagnetic Resonance ImagingAxonsMicroscopy Electronmedicine.anatomical_structurenervous systemNeurologyPeripheral nervous systemPediatrics Perinatology and Child HealthGait abnormalityNeurology (clinical)medicine.symptomHereditary Sensory and Motor NeuropathybusinessPediatric Neurology
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Impact of 7-Ketocholesterol and Very Long Chain Fatty Acids on Oligodendrocyte Lipid Membrane Organization: Evaluation Via LAURDAN and FAMIS Spectral…

2011

International audience; In the context of multiple sclerosis and X-linked adrenoleukodystrophy, 7-ketocholesterol (7KC) and very long chain fatty acids (C24:0, C26:0) are supposed to induce side effects respectively on oligodendrocytes which are myelin (which is a lipoproteic complex) synthesizing cells. The effects of 7KC (25, 50 mu M), C24:0 and C26:0 (10, 20 mu M) on cell viability and lipid membrane organization were investigated on 158N murine oligodendrocytes. Concerning 7KC and fatty acids (at 20 mu M only):1) cell growth was strongly inhibited; 2) marked induction of cell death was revealed with propidium iodide (PI); 3) no apoptotic cells were found with C24:0 and C26:0 (absence of…

MaleMYELINlaw.inventionchemistry.chemical_compoundMice0302 clinical medicinelawFAMIS2-Naphthylamine[SDV.IDA]Life Sciences [q-bio]/Food engineeringEnzyme InhibitorsLipid bilayerKetocholesterols0303 health sciencesMicroscopy ConfocalOXYSTEROLSFatty AcidsMULTIPLE-SCLEROSISvery long chain fatty acidsCell biologyPEROXISOMAL DISORDERSAPOPTOSISOligodendrogliaX-LINKED ADRENOLEUKODYSTROPHYmedicine.anatomical_structureMembraneCHOLESTEROL OXIDESlipids (amino acids peptides and proteins)Laurdanalpha-CyclodextrinsHistologyContext (language use)BiologyMETABOLISMPathology and Forensic Medicine158N oligodendrocytes03 medical and health sciencesMembrane LipidsConfocal microscopymedicineAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringViability assayPropidium iodideLAURDAN7-ketocholesterol030304 developmental biologyFluorescent DyesCell MembraneCENTRAL-NERVOUS-SYSTEMCell BiologyOligodendrocytechemistryCELLSmono-photon confocal microscopy030217 neurology & neurosurgeryLaurates
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Irregularity of the posterior corneal surface after curved interface femtosecond laser-assisted versus microkeratome-assisted descemet stripping auto…

2013

During donor tissue preparation for Descemet stripping automated endothelial keratoplasty (DSAEK), either microkeratome or femtosecond laser can be used for intrastromal cutting. We compared morphological and functional outcomes after DSAEK using both cutting techniques.In this retrospective study, 22 uneventful DSAEK surgeries were reviewed. Eight donor corneas were prepared for DSAEK using the VisuMax femtosecond laser (Carl Zeiss Meditec AG, Jena, Germany). Fourteen corneas were processed using an Amadeus II microkeratome (Ziemer Ophthalmic Systems AG, Port, Switzerland). The postoperative best spectacle-corrected visual acuity was measured. Furthermore, corneal optical coherence tomogra…

MaleMaterials sciencegenetic structuresDonor tissueVisual AcuityCell Countlaw.inventionOpticsOrgan Culture TechniqueslawMicrokeratomeHumansCorneal surfaceAgedRetrospective StudiesAged 80 and overbusiness.industryEndothelium CornealFuchs' Endothelial DystrophyCorneal TopographyMiddle AgedDescemet stripping automated endothelial keratoplastyLaser assistedLasereye diseasesTissue DonorsOphthalmologyFemtosecondFemalesense organsLaser TherapybusinessDescemet Stripping Endothelial KeratoplastyTomography Optical CoherenceBiomedical engineeringCornea
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Noninvasive assessment of respiratory muscle strength and activity in Myotonic dystrophy

2017

Objective To evaluate sensitivity/specificity of the maximum relaxation rate (MRR) of inspiratory muscles, amplitude of electromyographic activity of the sternocleidomastoid (SCM), scalene (SCA), parasternal (2ndIS) and rectus abdominis (RA) muscles; lung function and respiratory muscle strength in subjects with Myotonic dystrophy type 1 (DM1) compared with healthy subjects. Design and methods Quasi-experimental observational study with control group. MRR of inspiratory muscles, lung function and amplitude of the electromyographic activity of SCM, SCA, 2ndIS and RA muscles during maximum inspiratory pressure (PImax), maximum expiratory pressure (PEmax) and sniff nasal inspiratory pressure (…

MaleMuscle PhysiologyTime FactorsMuscle FunctionsPhysiologyMuscle RelaxationRespiratory Systemlcsh:MedicineElectromyographyPulmonary function testing0302 clinical medicineThoracic DiaphragmMedicine and Health SciencesMyotonic DystrophyMedicineRespiratory systemlcsh:ScienceMusculoskeletal SystemAbdominal MusclesMultidisciplinaryAnthropometrymedicine.diagnostic_testMusclesMuscle AnalysisRespiratory MusclesRespiratory Function TestsBioassays and Physiological AnalysisMuscle relaxationInhalationGenetic DiseasesExhalationParasternal lineCardiologyFemaleAnatomyMuscle ElectrophysiologyResearch ArticleAdult; Anthropometry; Electromyography; Exhalation; Female; Humans; Inhalation; Male; Muscle Relaxation; Muscle Strength; Myotonic Dystrophy; Pressure; ROC Curve; Respiratory Function Tests; Respiratory Muscles; Sample Size; Time FactorsAdultmedicine.medical_specialtyRespiratory physiologyResearch and Analysis Methods03 medical and health sciencesInternal medicineRespiratory musclesPressureRespiratory muscleHumansRespiratory PhysiologyMuscle StrengthClinical GeneticsElectromyographic activityElectromyographybusiness.industryElectrophysiological Techniqueslcsh:RBiology and Life SciencesExhalationSkeletal MusclesROC Curve030228 respiratory systemSample Sizelcsh:Qbusiness030217 neurology & neurosurgeryPLOS ONE
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