Search results for "Dative"

showing 10 items of 2381 documents

Circulating inflammation-related factors are correlated with systemic redox status in IgA nephropathy; a case-control study.

2020

Abstract Background IgA nephropathy (IGAN) is characterized by oxidative stress and inflammation. In the present study, we explored the relationship of redox status vs. that of circulating inflammation-related factors with other biomarkers in patients with IGAN. Methods This is a case-control study comparing patients with IGAN (Stage 1–4) to healthy controls. Forty patients and 40 controls were matched for age and sex. Two circulating dynamic redox parameters were analysed: oxidized free cysteine (Cys) and nitrate. Thirty-seven inflammation-related factors were measured in serum. Results The patients had elevated levels of oxidized free Cys and nitrate, indicating the presence of oxidative …

0301 basic medicinemedicine.medical_specialtyParathyroid hormoneRenal functionInflammationmedicine.disease_causeBiochemistryNephropathy03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)Internal medicinemedicineHumansOsteopontinInflammationCreatininebiologybusiness.industryCase-control studyGlomerulonephritis IGAmedicine.disease030104 developmental biologyEndocrinologychemistryCase-Control Studiesbiology.proteinmedicine.symptombusinessOxidation-Reduction030217 neurology & neurosurgeryOxidative stressBiomarkersGlomerular Filtration RateFree radical biologymedicine
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Gliptins Suppress Inflammatory Macrophage Activation to Mitigate Inflammation, Fibrosis, Oxidative Stress, and Vascular Dysfunction in Models of Nona…

2017

Abstract Aims: Nonalcoholic steatohepatitis (NASH) is characterized by steatosis, panlobular inflammation, liver fibrosis, and increased cardiovascular mortality. Dipeptidyl peptidase-4 inhibitors (gliptins) are indirect glucagon-like peptide 1 agonists with antidiabetic and anti-inflammatory activity, used for the treatment of type 2 diabetes. Their potential and underlying mechanisms to treat metabolic liver inflammation and fibrosis as well as the associated vascular dysfunction remain to be explored. Results: In the methionine/choline-deficient (MCD) diet and Mdr2−/− models of NASH and liver fibrosis, treatment with sitagliptin and linagliptin significantly decreased parameters of steat…

0301 basic medicinemedicine.medical_specialtyPhysiologyClinical BiochemistryAnti-Inflammatory AgentsGene ExpressionInflammationType 2 diabetes030204 cardiovascular system & hematologymedicine.disease_causeBiochemistryAntioxidantsProinflammatory cytokineMice03 medical and health sciences0302 clinical medicineNon-alcoholic Fatty Liver DiseaseFibrosisInternal medicinemedicineAnimalsMyeloid CellsMolecular BiologyDipeptidyl peptidase-4General Environmental ScienceInflammationMice KnockoutDipeptidyl-Peptidase IV Inhibitorsbusiness.industryMacrophagesCell BiologyMacrophage Activationmedicine.diseaseFibrosisDietDisease Models AnimalOxidative Stress030104 developmental biologyEndocrinologyLiverNADPH Oxidase 2General Earth and Planetary SciencesTumor necrosis factor alphaSteatosismedicine.symptomReactive Oxygen SpeciesbusinessBiomarkersOxidative stressAntioxidants & Redox Signaling
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The Adverse Effects of Environmental Noise Exposure on Oxidative Stress and Cardiovascular Risk

2018

Abstract Epidemiological studies have provided evidence that traffic noise exposure is linked to cardiovascular diseases such as arterial hypertension, myocardial infarction, and stroke. Noise is a nonspecific stressor that activates the autonomous nervous system and endocrine signaling. According to the noise reaction model introduced by Babisch and colleagues, chronic low levels of noise can cause so-called nonauditory effects, such as disturbances of activity, sleep, and communication, which can trigger a number of emotional responses, including annoyance and subsequent stress. Chronic stress in turn is associated with cardiovascular risk factors, comprising increased blood pressure and …

0301 basic medicinemedicine.medical_specialtyPhysiologyClinical Biochemistrytraffic noise exposure030204 cardiovascular system & hematologyenvironmental risk factorsmedicine.disease_causeBiochemistryendothelial dysfunctionCoronary artery disease03 medical and health sciences0302 clinical medicinestress hormonesComprehensive Invited ReviewRisk FactorsInternal medicineDiabetes mellitusmedicineHumansChronic stressMyocardial infarctionEndothelial dysfunctionMolecular BiologyStrokeGeneral Environmental Sciencebusiness.industryaircraft noise exposureEnvironmental ExposureCell Biologymedicine.diseaseStrokeOxidative Stress030104 developmental biologyBlood pressureCardiovascular DiseasesHypertensionCardiologyGeneral Earth and Planetary SciencesEnvironmental PollutantsNoiseReactive Oxygen SpeciesbusinessOxidative stressAntioxidants & Redox Signaling
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The Mitochondria-Targeted Antioxidant MitoQ Modulates Mitochondrial Function and Endoplasmic Reticulum Stress in Pancreatic β Cells Exposed to Hyperg…

2019

Background/aims Mitochondria-targeted antioxidants such as mitoquinone (MitoQ) have demonstrated protective effects against oxidative damage in several diseases. The increase in reactive oxygen species (ROS) production during glucose metabolism in β cells can be exacerbated under hyperglycaemic conditions such as type 2 diabetes (T2D), thus contributing to β cell function impairment. In the present work, we aimed to evaluate the effect of MitoQ on insulin secretion, oxidative stress, endoplasmic reticulum (ER) stress and nuclear factor kappa B (NFκB) signalling in a pancreatic β cell line under normoglycaemic (NG, 11.1 mM glucose), hyperglycaemic (HG, 25 mM glucose) and lipidic (palmitic ac…

0301 basic medicinemedicine.medical_specialtyPhysiologyUbiquinoneCarbohydrate metabolismmedicine.disease_causeMitoQlcsh:PhysiologyPancreatic β cellsAntioxidantsProinflammatory cytokinelcsh:Biochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineOrganophosphorus CompoundsInternal medicineCell Line TumorInsulin-Secreting CellsmedicineAnimalslcsh:QD415-436chemistry.chemical_classificationReactive oxygen speciesMitoQlcsh:QP1-981Endoplasmic reticulumGlutathioneEndoplasmic Reticulum StressType 2 DiabetesMitochondriaRatsOxidative Stress030104 developmental biologyEndocrinologyGlucosechemistry030220 oncology & carcinogenesisHyperglycemiaUnfolded protein responseER stressMitochondrial dysfunctionReactive Oxygen SpeciesOxidative stressSignal Transduction
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Evaluation of an Antioxidant and Anti-inflammatory Cocktail Against Human Hypoactivity-Induced Skeletal Muscle Deconditioning

2020

International audience; Understanding the molecular pathways involved in the loss of skeletal muscle mass and function induced by muscle disuse is a crucial issue in the context of spaceflight as well as in the clinical field, and development of efficient countermeasures is needed. Recent studies have reported the importance of redox balance dysregulation as a major mechanism leading to muscle wasting. Our study aimed to evaluate the effects of an antioxidant/anti-inflammatory cocktail (741 mg of polyphenols, 138 mg of vitamin E, 80 mu g of selenium, and 2.1 g of omega-3) in the prevention of muscle deconditioning induced by long-term inactivity. The study consisted of 60 days of hypoactivi…

0301 basic medicinemedicine.medical_specialtyPhysiologymedicine.medical_treatment[SDV]Life Sciences [q-bio]Context (language use)Sciences du Vivant [q-bio]/Médecine humaine et pathologieBed restmedicine.disease_causelcsh:Physiology03 medical and health sciencesSciences du Vivant [q-bio]/Autre [q-bio.OT]0302 clinical medicineAtrophyDeconditioningInternal medicinePhysiology (medical)medicineoxidative stresscell signalingWastingOriginal Research[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologylcsh:QP1-981business.industrySkeletal musclemuscle wastingmedicine.disease3. Good health[SDV] Life Sciences [q-bio]030104 developmental biologyEndocrinologymedicine.anatomical_structureantioxidantsinactivityFisiologia humanamedicine.symptombusinessHypoactivity030217 neurology & neurosurgeryOxidative stress[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Obesogenic Diets Cause Alterations on Proteins and Theirs Post-Translational Modifications in Mouse Brains

2021

Obesity constitutes a major global health threat and is associated with a variety of diseases ranging from metabolic and cardiovascular disease, cancer to neurodegeneration. The hallmarks of neurodegeneration include oxidative stress, proteasome impairment, mitochondrial dysfunction and accumulation of abnormal protein aggregates as well as metabolic alterations. As an example, in post-mortem brain of patients with Alzheimer’s disease (AD), several studies have reported reduction of insulin, insulin-like growth factor 1 and insulin receptor and an increase in tau protein and glycogen-synthase kinase-3β compared to healthy controls suggesting an impairment of metabolism in the AD patient’s …

0301 basic medicinemedicine.medical_specialtyRC620-627Endocrinology Diabetes and Metabolismmedicine.medical_treatmentTau proteinObesity nutrition brain impairment proteomics post-translational modificationsBrain damageMitochondrionProteomicsmedicine.disease_causeSettore BIO/09 - Fisiologia03 medical and health sciencesproteomics0302 clinical medicineInternal medicinepost-translational modificationsmedicineTX341-641ObesityNutritional diseases. Deficiency diseasesOriginal ResearchSettore MED/04 - Patologia GeneraleNutrition and DieteticsbiologyNutrition. Foods and food supplyInsulinNeurodegenerationmedicine.diseasebrain impairmentInsulin receptornutrition030104 developmental biologyEndocrinologybiology.proteinmedicine.symptom030217 neurology & neurosurgeryOxidative stressFood ScienceNutrition and Metabolic Insights
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Hypoxie et régénération cardiaque : une nouvelle approche paradoxale de la cardioprotection

2017

IF 2.331; International audience

0301 basic medicinemedicine.medical_specialtyReactive oxygen species metabolismTranscription GeneticCardiac pathologyEnergy metabolismCardiac metabolismCardioprotectionHypoxie03 medical and health sciencesInternal medicinemedicineRégénérationAnimalsHumansRegenerationMyocytes CardiacTissue survivalHypoxiaCoeurCell ProliferationCardioprotectionHeart FailureTissue Survivalbusiness.industryOxygen metabolismHeartGeneral MedicineRecovery of FunctionHypoxia (medical)[ SDV.MHEP.CSC ] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemCell HypoxiaOxygenOxidative Stress030104 developmental biologyGene Expression RegulationAnesthesiaCardiologymedicine.symptomCardiology and Cardiovascular MedicinebusinessEnergy MetabolismReactive Oxygen SpeciesSignal TransductionArchives of cardiovascular diseases
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Comparison of total antioxidant capacity of saliva in women with gestational diabetes mellitus and non-diabetic pregnant women

2017

Background Pregnancy is considered a stressful event, results in higher levels of oxidative stress and considerable changes in physiological and metabolic functions such as gestational diabetes mellitus (GDM). Due to the cumulative effect of antioxidants and considering the controversies in this area, this study was undertaken to investigate the total antioxidant capacity (TAC) of saliva in pregnant women whit gestational diabetes in comparison to non-diabetic pregnant women. Material and methods In this cross-sectional study (2015-16), a total of 31 women with a diagnosis of GDM and 59 non-diabetic pregnant women were included in the diabetic and control groups, respectively. Salivary samp…

0301 basic medicinemedicine.medical_specialtySalivaPhysiologymedicine.disease_cause03 medical and health sciences0302 clinical medicineInternal medicineDiabetes mellitusMedicineGeneral DentistryCumulative effectPregnancyOral Medicine and Pathologybusiness.industryResearchmedicine.disease:CIENCIAS MÉDICAS [UNESCO]Gestational diabetesAntioxidant capacity030104 developmental biologyEndocrinology030220 oncology & carcinogenesisUNESCO::CIENCIAS MÉDICASbusinessOxidative stressNon diabetic
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Atorvastatin but not pravastatin impairs mitochondrial function in human pancreatic islets and rat β-cells. Direct effect of oxidative stress

2017

AbstractStatins are a class of drugs widely prescribed as frontline therapy for lowering plasma LDL-cholesterol in cardiovascular risk prevention. Several clinical reports have recently suggested an increased risk of type 2 diabetes associated with chronic use of these drugs. The pathophysiology of this effect remains to be fully elucidated but impaired β-cell function constitutes a potential mechanism. The aim of this study was to explore the effect of a chronic treatment with lipophilic and hydrophilic statins on β-cell function, using human pancreatic islets and rat insulin-secreting INS-1 cells; we particularly focused on the role of mitochondria and oxidative stress. The present study …

0301 basic medicinemedicine.medical_specialtyStatinmedicine.drug_classmedicine.medical_treatmentAtorvastatinPancreatic isletslcsh:MedicineType 2 diabetes030204 cardiovascular system & hematologyMitochondrionPharmacologymedicine.disease_causeArticle03 medical and health sciences0302 clinical medicineInternal medicinemedicinelcsh:ScienceMultidisciplinarybusiness.industryPancreatic isletsInsulinlcsh:RStatinmedicine.disease030104 developmental biologyEndocrinologymedicine.anatomical_structurelcsh:Qlipids (amino acids peptides and proteins)Statins; Pancreatic isletsbusinessOxidative stressPravastatinmedicine.drug
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Metabolic syndrome enhances endoplasmic reticulum, oxidative stress and leukocyte–endothelium interactions in PCOS

2017

Polycystic ovary syndrome (PCOS) is associated with insulin resistance, which can lead to metabolic syndrome (MetS). Oxidative stress and leukocyte-endothelium interactions are related to PCOS. Our aim was to evaluate whether the presence of MetS in PCOS patients can influence endoplasmic reticulum (ER) and oxidative stress and leukocyte-endothelium interactions.This was a prospective controlled study conducted in an academic medical center. The study population consisted of 148 PCOS women (116 without/32 with MetS) and 112 control subjects (87 without / 25 with MetS). Metabolic parameters, reactive oxygen species (ROS) production, ER stress markers (GRP78, sXBP1, ATF6), leukocyte-endotheli…

0301 basic medicinemedicine.medical_specialtyXBP1Endocrinology Diabetes and Metabolism030209 endocrinology & metabolismBiologymedicine.disease_cause03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceInternal medicineCell AdhesionLeukocytesmedicineHumansProspective StudiesEndoplasmic Reticulum Chaperone BiPMetabolic SyndromeInterleukin-6ATF6Endoplasmic Reticulum StressIntercellular Adhesion Molecule-1medicine.diseasePolycystic ovaryOxidative Stress030104 developmental biologyEndocrinologyUnfolded protein responseHomeostatic model assessmentCytokinesFemaleEndothelium VascularMetabolic syndromeReactive Oxygen SpeciesCell Adhesion MoleculesOxidative stressPolycystic Ovary SyndromeMetabolism
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