Search results for "Dative"

showing 10 items of 2381 documents

Oxidative Stress and Microvascular Alterations in Diabetic Retinopathy: Future Therapies

2019

Diabetes is a disease that can be treated with oral antidiabetic agents and/or insulin. However, patients’ metabolic control is inadequate in a high percentage of them and a major cause of chronic diseases like diabetic retinopathy. Approximately 15% of patients have some degree of diabetic retinopathy when diabetes is first diagnosed, and most will have developed this microvascular complication after 20 years. Early diagnosis of the disease is the best tool to prevent or delay vision loss and reduce the involved costs. However, diabetic retinopathy is an asymptomatic disease and its development to advanced stages reduces the effectiveness of treatments. Today, the recommended treatment for…

Adult0301 basic medicineAgingmedicine.medical_specialtymedicine.medical_treatmentReview ArticleDiseasemedicine.disease_causeBiochemistryAsymptomaticDiabetic Eye DiseaseYoung Adult03 medical and health sciences0302 clinical medicineRisk FactorsInternal medicineDiabetes mellitusmedicineHumanslcsh:QH573-671AgedDiabetic Retinopathybusiness.industrylcsh:CytologyInsulinCell BiologyGeneral MedicineDiabetic retinopathyMiddle Agedmedicine.diseaseOxidative Stress030104 developmental biologyMetabolic control analysis030221 ophthalmology & optometrymedicine.symptombusinessOxidative stressOxidative Medicine and Cellular Longevity
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Redox and autonomic responses to acute exercise-post recovery following Opuntia ficus-indica juice intake in physically active women

2021

Background: The aim of this study was to investigate if the supplementation with Opuntia ficus-indica (OFI) juice may affect plasma redox balance and heart rate variability (HRV) parameters following a maximal effort test, in young physically active women. Methods: A randomized, double blind, placebo controlled and crossover study comprising eight women (23.25 ± 2.95 years, 54.13 ± 9.05 kg, 157.75 ± 0.66 cm and BMI of 21.69 ± 0.66 kg/m2) was carried out. A juice containing OFI diluted in water and a Placebo solution were supplied (170 ml; OFI = 50 ml of OFI juice + 120 ml of water; Placebo = 170 ml beverage without Vitamin C and indicaxanthin). Participants consumed the OFI juice or Placebo…

Adult0301 basic medicineCactus pear juice supplementationOpuntia ficusTotal antioxidant capacityClinical nutritionmedicine.disease_causePlaceboYoung Adult03 medical and health scienceschemistry.chemical_compoundRedox balanceOxygen Consumption0302 clinical medicineAnimal scienceDouble-Blind MethodHigh frequencyHeart RatemedicineHumansHeart rate variabilityAutonomic nervous systemExerciseCross-Over StudiesNutrition and DieteticsSettore M-EDF/02 - Metodi E Didattiche Delle Attivita' SportiveVitamin Cbusiness.industryOpuntiaHydrogen Peroxide030229 sport sciencesCrossover studyFruit and Vegetable JuicesHydroperoxideHydroperoxides030104 developmental biologychemistryOxidative stressOxidative streFemalebusinessLow frequencyOxidation-ReductionIndicaxanthinSettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieOxidative stressResearch ArticleFood Science
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Redox lipidomics to better understand brain aging and function

2019

Human prefrontal cortex (PFC) is a recently evolutionary emerged brain region involved in cognitive functions. Human cognitive abilities decline during aging. Yet the molecular mechanisms that sustain the preservation or deterioration of neurons and PFC functions are unknown. In this review, we focus on the role of lipids in human PFC aging. As the evolution of brain lipid concentrations is particularly accelerated in the human PFC, conferring a specific lipid profile, a brief approach to the lipidome of PFC was consider along with the relationship between lipids and lipoxidative damage, and the role of lipids in human PFC aging. In addition, the specific targets of lipoxidative damage in h…

AdultAgingPrefrontal CortexGlycerophospholipidsBiologymedicine.disease_causeBiochemistryCognitionPhysiology (medical)LipidomicsmedicineHumansCognitive DysfunctionCognitive declineMitochondrionPrefrontal cortexExerciseCaloric RestrictionNeuronsSphingolipidsCognitionEnergy metabolismMiddle AgedLipidomeAdvanced lipoxidation end productsLipid MetabolismCytoskeletal ProteinsOxidative StressProteostasisnervous systemLipidomicsFatty Acids UnsaturatedEnergy MetabolismNeuroscienceFunction (biology)Oxidative stress
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Increased thioredoxin levels are related to insulin resistance in familial combined hyperlipidaemia

2015

BACKGROUND Thioredoxins (TRX) are major cellular protein disulphide reductases that are critical for redox regulation. Oxidative stress and inflammation play promoting roles in the genesis and progression of atherosclerosis, but until now scarce data are available considering the influence of TRX activity in familial combined hyperlipidaemia (FCH). Since FCH is associated with high risk of cardiovascular disease, the objective of the present study was to assess oxidative stress status in FCH patients, and evaluate the influence of insulin resistance (IR). MATERIALS AND METHODS A cohort of 35 control subjects and 35 non-related FCH patients were included, all of them nondiabetic, normotensiv…

AdultBlood GlucoseMale0301 basic medicinemedicine.medical_specialtyanimal structuresmedicine.medical_treatmentClinical BiochemistryHyperlipidemia Familial Combined030204 cardiovascular system & hematologymedicine.disease_causeBiochemistry03 medical and health scienceschemistry.chemical_compoundThioredoxins0302 clinical medicineInsulin resistanceInternal medicinemedicineHumansTriglyceridesGlutathione Disulfidemedicine.diagnostic_testbusiness.industryInsulinCholesterol HDLCase-control studyCholesterol LDLGeneral MedicineGlutathioneMiddle Agedmedicine.diseaseGlutathioneOxidative Stress030104 developmental biologyEndocrinologychemistryCardiovascular DiseasesCase-Control StudiesGlutathione disulfideFemaleInsulin ResistanceThioredoxinLipid profilebusinessOxidative stressEuropean Journal of Clinical Investigation
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Behavior of the total antioxidant status in a group of subjects with metabolic syndrome.

2014

AIM: Our purpose was to examine the total antioxidant status (TAS) in subjects with metabolic syndrome (MS) subdivided according to the presence or not of diabetes mellitus. METHODS: We enrolled 106 subjects (45 women, 61 men) with MS subsequently subdivided in diabetics (14 women, 29 men) and nondiabetics (31 women, 29 men). TAS was obtained using an Assay kit which relies on the ability of plasma antioxidant substances to inhibit the oxidation of 2,2'-azino-bis(3-ethylbenzthiazoline sulfonic acid) to the radical ABTS+. RESULTS: In the group of MS subjects a significant decrease in TAS (p<0.05) in comparison with normal controls was evident. This difference was present between normal subje…

AdultBlood GlucoseMalemedicine.medical_specialtyanimal structuresAntioxidantSettore MED/09 - Medicina InternaBilirubinEndocrinology Diabetes and Metabolismmedicine.medical_treatmentBlood PressureAntioxidantschemistry.chemical_compoundInternal medicineDiabetes mellitusInternal MedicinemedicineHumansAgedMetabolic SyndromeABTSbusiness.industryBilirubinGeneral MedicineMiddle Agedmedicine.diseasePathophysiologyTOTAL ANTIOXIDANT STATUS METABOLIC SYNDROMEUric AcidAntioxidant capacityOxidative StressEndocrinologychemistryDiabetes Mellitus Type 2ItalyLinear ModelsUric acidFemaleMetabolic syndromebusiness
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In renal transplanted patients inflammation and oxidative stress are interrelated.

2006

Introduction: The inflammatory state plays a well-documented role to cause oxidative stress, especially in end-stage renal disease (ESRD) patients, wherein several cardiovascular risk factors are amplified by the coexistence of a microinflammatory state with increased oxidative stress. Methods: We measured serum concentrations of high sensitivity C-reactive protein (CRP), tumor necrosis factor α (TNFα), 8-iso-prostaglandin F2α (8-iso-PGF2α-in vivo oxidative stress marker) in 15 chronic renal failure (CRF) and 15 transplant patients versus 15 healthy controls. Exclusion criteria were: age 65 years as well as a diagnosis of diabetes or cardiovascular diseases. We evaluated systolic (SBP) and …

AdultBlood GlucoseMalemedicine.medical_specialtyrenal transplantation oxidative stress inflammationRenal functionInflammationBlood Pressuremedicine.disease_causeWhite PeopleBody Mass Indexchemistry.chemical_compoundDiabetes mellitusInternal medicinemedicineCadaverHumansInflammationTransplantationCreatinineAnalysis of Variancebusiness.industryMiddle Agedmedicine.diseaseKidney TransplantationTissue DonorsOxidative StressBlood pressureEndocrinologyC-Reactive ProteinchemistryItalyKidney Failure ChronicSurgeryTumor necrosis factor alphamedicine.symptombusinessBody mass indexOxidative stressBiomarkersGlomerular Filtration Rate
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Dimethyl fumarate treatment restrains the antioxidative capacity of T cells to control autoimmunity

2021

Abstract Dimethyl fumarate, an approved treatment for relapsing-remitting multiple sclerosis, exerts pleiotropic effects on immune cells as well as CNS resident cells. Here, we show that dimethyl fumarate exerts a profound alteration of the metabolic profile of human CD4+ as well as CD8+ T cells and restricts their antioxidative capacities by decreasing intracellular levels of the reactive oxygen species scavenger glutathione. This causes an increase in mitochondrial reactive oxygen species levels accompanied by an enhanced mitochondrial stress response, ultimately leading to impaired mitochondrial function. Enhanced mitochondrial reactive oxygen species levels not only result in enhanced T…

AdultCD4-Positive T-LymphocytesMaleDimethyl FumarateT cellAutoimmunityCD8-Positive T-Lymphocytesmedicine.disease_causeAntioxidantsCohort StudiesMiceYoung Adultchemistry.chemical_compoundMultiple Sclerosis Relapsing-RemittingImmune systemmedicineAnimalsHumanschemistry.chemical_classificationReactive oxygen speciesDimethyl fumarateExperimental autoimmune encephalomyelitisGlutathioneMiddle Agedmedicine.diseaseCell biologyMice Inbred C57BLmedicine.anatomical_structurechemistryFemaleNeurology (clinical)Immunosuppressive AgentsOxidative stressCD8Brain
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Distribution of betalain pigments in red blood cells after consumption of cactus pear fruits and increased resistance of the cells to ex vivo induced…

2005

Betalain pigments are bioavailable phytochemicals recently acknowledged as natural radical scavengers. This work, which extends previous research on the postabsorbitive fate of dietary betalains, investigated the distribution of betanin and indicaxanthin in red blood cells (RBCs) isolated from healthy volunteers (n = 8), before and during the 1-8 h interval after a cactus pear fruit meal, and the potential antioxidative activity of the pigments in these cells. A peak concentration of indicaxanthin (1.03 +/- 0.2 microM) was observed in RBCs isolated at 3 h after fruit feeding, whereas the concentration at 5 h was about half, and even smaller amounts were measured at 8 h. Indicaxanthin was no…

AdultCactaceaeMaleAntioxidantErythrocytesIndolesPyridinesmedicine.medical_treatmentBetalainsindicaxanthinred blood cellBiologyHemolysischemistry.chemical_compoundBetalainBotanymedicineHumansFood sciencecactus pear; betalains; betanin; indicaxanthin; red blood cell; oxidative hemolysis; bioavailable phytochemicalsBetaninbioavailable phytochemicalsbetaninbetalainGeneral Chemistrymedicine.diseaseHemolysisBetaxanthinsDietQuaternary Ammonium CompoundsRed blood celloxidative hemolysiKineticsmedicine.anatomical_structurechemistryCumene hydroperoxideFruitcactus pearFemaleBetacyaninsGeneral Agricultural and Biological SciencesIndicaxanthinEx vivoJournal of agricultural and food chemistry
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Impairment of antioxidant enzymes, lipid peroxidation and 8-oxo-2'-deoxyguanosine in advanced epithelial ovarian carcinoma of a Spanish community.

2006

In the present study, we describe the changes of antioxidant enzyme activities and other oxidative stress-related parameters in a mediterranean cohort of women affected with epithelial ovarian carcinoma (EOC). For that purpose, the most representative enzymatic activities, such as superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GPx) and the oxidized/reduced glutathione (GSSG/GSH) ratio have been analyzed in tumor tissue biopsies and compared with the normal tissue of the same patient. As oxidation products, the levels of malondialdehyde (MDA) as an indication of lipid peroxidation, and the DNA damaged base 8-oxo-2'-deoxyguanosine (8-oxo-dG) have been also measured. Ad…

AdultCancer Research8-Oxo-2'-deoxyguanosinemedicine.disease_causeAndrologyLipid peroxidationSuperoxide dismutasechemistry.chemical_compoundmedicineHumansNeoplasms Glandular and EpithelialAgedNeoplasm Stagingchemistry.chemical_classificationAged 80 and overOvarian NeoplasmsGlutathione PeroxidasebiologySuperoxide DismutaseGlutathione peroxidase8-Hydroxy-2'-deoxyguanosineDeoxyguanosineGlutathioneMiddle AgedMalondialdehydeCatalaseGlutathioneOncologyBiochemistrychemistry8-Hydroxy-2'-Deoxyguanosinebiology.proteinFemaleLipid PeroxidationOxidative stressDNA DamageCancer letters
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Induction of heme oxygenase-1 and adaptive protection against the induction of DNA damage after hyperbaric oxygen treatment.

2000

Hyperbaric oxygen (HBO) treatment of human subjects (i.e. exposure to 100% oxygen at a pressure of 2.5 ATA for a total period of 3 x 20 min) caused clear and reproducible DNA damage in lymphocytes, as detected with the comet assay (single cell gel electrophoresis). Induction of DNA damage was found only after the first HBO exposure and not after further treatments of the same individuals. Furthermore, blood taken 24 h after HBO treatment was significantly protected against the induction of DNA damage by hydrogen peroxide (H(2)O(2)) in vitro, indicating that adaptation occurred due to induction of antioxidant defenses. The cells were not significantly protected against the genotoxic effects …

AdultCancer ResearchDNA RepairDNA repairDNA damageCarbon-Oxygen LyasesBiologymedicine.disease_causeSuperoxide dismutasemedicineDNA-(Apurinic or Apyrimidinic Site) LyaseHumansLymphocytesDNA Polymerase betachemistry.chemical_classificationReactive oxygen speciesHyperbaric OxygenationSuperoxide DismutaseMembrane ProteinsGeneral MedicineHydrogen PeroxideCatalaseMolecular biologyDNA-(apurinic or apyrimidinic site) lyaseAdaptation PhysiologicalDeoxyribonuclease IV (Phage T4-Induced)Comet assayOxidative StresschemistryBiochemistryCatalaseEnzyme InductionHeme Oxygenase (Decyclizing)biology.proteinOxidative stressHeme Oxygenase-1DNA DamageCarcinogenesis
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