Search results for "Decapentaplegic"

showing 7 items of 7 documents

Abstract B5: A BMP7 variant inhibits angiogenesis in vitro and in vivo in part by downregulating VEGFR2 and FGFR1 expression in endothelial cells.

2013

Abstract Glioblastoma multiforme (GBM), the most aggressive glioma, requires active angiogenesis for growth and survival. Bone morphogenetic proteins (BMPs), members of the TGF-β superfamily, have numerous biological activities including control of growth, differentiation, and vascular development. Previously, we demonstrated the use of a BMP7 variant (BMP7v) to differentiate glioblastoma stem-like cells (GSLCs) and significantly reduce their tumorigenic potential (Tate and Pallini et al. 2012). Using an in vitro co-culture endothelial cord formation assay, a surrogate of angiogenesis, and its cognate in vivo model, we investigated the role of BMP7v in VEGF, basic FGF (bFGF), tumor-driven a…

Cancer ResearchMatrigelbiologyAngiogenesisSMADFibroblast growth factorReceptor tyrosine kinaseEndothelial stem cellOncologyIn vivoMothers against decapentaplegic homolog 4Immunologybiology.proteinCancer researchMolecular Cancer Therapeutics
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optomotor-blind suppresses instability at the A/P compartment boundary of the Drosophila wing.

2008

Formation and function of the A/P compartment boundary of the Drosophila wing have been studied intensely. The boundary prevents mingling of A and P cells, is characterized by an expression discontinuity of several genes like engrailed, Cubitus interruptus, hedgehog and decapentaplegic and is essential for patterning the wing. Compared with segmental or compartmental boundaries in several other systems which generally manifest as folds or clefts, the wing A/P boundary is morphologically inconspicuous in both the larval and adult stage. We show here that the Drosophila wing A/P boundary, too, is susceptible to fold and cleft formation and that these processes are suppressed by the T-box tran…

GeneticsEmbryologyanimal structuresWingDecapentaplegicMorphogenesisGene Expression Regulation DevelopmentalNerve Tissue ProteinsBiologyMicrotubulesengrailedCell biologyAdherens junctionCompartment (development)AnimalsDrosophila ProteinsWings AnimalDrosophilaEnhancerT-Box Domain ProteinsHedgehogDevelopmental BiologyBody PatterningSequence DeletionMechanisms of development
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The use of whole-mountin situhybridization to illustrate gene expression regulation

2014

In situ hybridization is a widely used technique for studying gene expression. Here, we describe two experiments addressed to postgraduate genetics students in which the effect of transcription factors on gene expression is analyzed in Drosophila embryos of different genotypes by whole-mount in situ hybridization. In one of the experiments, students analyzed the repressive effect of Snail over rhomboid expression using reporter lines containing different constructs of the rhomboid neuroectodermal enhancer fused to the lacZ gene. In the second experiment, the epistatic relationship between the cabut and decapentaplegic genes was analyzed. These simple experiments allowed students to (1) unde…

GeneticsRegulation of gene expressionDecapentaplegicved/biologyved/biology.organism_classification_rank.speciesIn situ hybridizationBiologyBiochemistryGene expressionEnhancerModel organismMolecular BiologyGeneTranscription factorBiochemistry and Molecular Biology Education
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Spatial discontinuity of Optomotor-blind expression in the Drosophila wing imaginal disc disrupts epithelial architecture and promotes cell sorting

2010

Abstract Background Decapentaplegic (Dpp) is one of the best characterized morphogens, required for dorso-ventral patterning of the Drosophila embryo and for anterior-posterior (A/P) patterning of the wing imaginal disc. In the larval wing pouch, the Dpp target gene optomotor-blind (omb) is generally assumed to be expressed in a step function above a certain threshold of Dpp signaling activity. Results We show that the transcription factor Omb forms, in fact, a symmetrical gradient on both sides of the A/P compartment boundary. Disruptions of the Omb gradient lead to a re-organization of the epithelial cytoskeleton and to a retraction of cells toward the basal membrane suggesting that the O…

GeneticsWinganimal structuresbiologyDecapentaplegicMorphogenesisNerve Tissue ProteinsCell sortingbiology.organism_classificationCell biologyImaginal discDrosophila melanogasterlcsh:Biology (General)Research articleAnimalsDrosophila ProteinsWings AnimalCompartment (development)Drosophila melanogasterT-Box Domain Proteinslcsh:QH301-705.5Drosophila ProteinSignal TransductionDevelopmental BiologyBMC Developmental Biology
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TGF-β Signaling Pathways in Different Compartments of the Lower Airways of Patients With Stable COPD

2017

Background: The expression and localization of transforming growth factor-β (TGF-β) pathway proteins in different compartments of the lower airways of patients with stable COPD is unclear. We aimed to determine TGF-β pathway protein expression in patients with stable COPD. Methods: The expression and localization of TGF-β pathway components was measured in the bronchial mucosa and peripheral lungs of patients with stable COPD (n = 44), control smokers with normal lung function (n = 24), and control nonsmoking subjects (n = 11) using immunohistochemical analysis. Results: TGF-β1, TGF-β3, and connective tissue growth factor expression were significantly decreased in the bronchiolar epithelium…

MaleCCN2 connective tissue growth factorSmad Proteinsairway inflammationCritical Care and Intensive Care MedicineTRAP-1 transforming growth factor-β receptor-associated binding proteinPulmonary Disease Chronic ObstructiveLAP latency-associated peptideSMAD small mother against decapentaplegicBAMBI CTGF SMAD TGF-B airway inflammation autoimmunityLungTGF transforming growth factorLLC large latent complexBAMBI CTGF SMAD TGF-β Airway Inflammation AutoimmunityautoimmunityMiddle Agedrespiratory systemLTBP latent transforming growth factor-β binding proteinImmunohistochemistryTGIF 5′-TG-3′-interacting factorECM extracellular matrixTGFBI transforming growth factor-β-induced proteinFemaleCardiology and Cardiovascular MedicinePI3K phosphoinositide 3-kinaseSignal TransductionTGF-βPulmonary and Respiratory MedicineTGF-βR TGF-β receptorSocio-culturaleBronchiRespiratory MucosaArticleTGF-BTransforming Growth Factor beta1Transforming Growth Factor beta3Macrophages AlveolarHumansAgedBAMBI; CTGF; SMAD; TGF-β; airway inflammation; autoimmunityBAMBIMembrane ProteinsCTGFBMP bone morphogenetic proteinBAMBI; CTG; SMAD; TGF-β; airway inflammation; autoimmunityCTGBAMBI bone morphogenetic proteins and activin membrane-bound inhibitorrespiratory tract diseasesairway inflammation; autoimmunity; BAMBI; CTGF; SMAD; TGF-β; Pulmonary and Respiratory Medicine; Critical Care and Intensive Care Medicine; Cardiology and Cardiovascular MedicineCase-Control StudiesBiomarkersMAPK mitogen-activated protein kinaseSMAD
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Bone morphogenetic protein-4 is required for mesoderm formation and patterning in the mouse.

1995

Bone morphogenetic protein-4 (BMP-4) is a member of the TGF-beta superfamily of polypeptide signaling molecules, closely related to BMP-2 and to Drosophila decapentaplegic (DPP). To elucidate the role of BMP-4 in mouse development the gene has been inactivated by homologous recombination in ES cells. Homozygous mutant Bmp-4tm1blh embryos die between 6.5 and 9.5 days p.c., with a variable phenotype. Most Bmp-4tm1blh embryos do not proceed beyond the egg cylinder stage, do not express the mesodermal marker T(Brachyury), and show little or no mesodermal differentiation. Some homozygous mutants develop to the head fold or beating heart/early somite stage or beyond. However, they are development…

MaleMesodermBrachyuryHeterozygoteanimal structuresMolecular Sequence DataBiologyCell LineMesodermEmbryonic and Fetal DevelopmentMiceGeneticsmedicineParaxial mesodermAnimalsCrosses GeneticDecapentaplegicBase SequenceChimeraStem CellsHomozygoteProteinsGastrulaCell biologyMice Inbred C57BLmedicine.anatomical_structureBone morphogenetic protein 5PhenotypeBone morphogenetic protein 4GDF6embryonic structuresMesoderm formationBone Morphogenetic ProteinsGene TargetingFemaleDevelopmental BiologyGenesdevelopment
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Modulation of Hedgehog target gene expression by the Fused serine-threonine kinase in wing imaginal discs

1998

0925-4773 doi: DOI: 10.1016/S0925-4773(98)00130-0; The Fused (Fu) serine–threonine kinase and the Suppressor of fused (Su(fu)) product are part of the Hedgehog (Hh) signalling pathway both in embryos and in imaginal discs. In wing imaginal discs, the Hh signal induces Cubitus interruptus (Ci) accumulation and activates patched (ptc) and decapentaplegic (dpp) expression along the anterior/posterior (A/P) boundary. In this paper, we have examined the role of the Fu and Su(fu) proteins in the regulation of Hh target gene expression in wing imaginal discs, by using different classes of fu alleles and an amorphic Su(fu) mutation. We show that, at the A/P boundary, Fu kinase activity is involved …

PatchedEmbryologyanimal structuresReceptors Cell SurfaceBiologyProtein Serine-Threonine KinasesSignal transductionCubitus interruptusImaginal disc developmentMorphogenesisAnimalsDrosophila ProteinsWings AnimalHedgehog ProteinsKinase activitySuppressor of fusedGeneticsSerine/threonine-specific protein kinaseHomeodomain ProteinsDecapentaplegicFusedGene Expression Regulation DevelopmentalMembrane ProteinsCi proteinHedgehog signaling pathwayCell biologyDNA-Binding ProteinsRepressor ProteinsImaginal discDrosophila melanogasterInsect ProteinsDrosophilaHedgehogMorphogenTranscription FactorsDevelopmental Biology
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