Search results for "Dendritic cells."

showing 10 items of 360 documents

Human dendritic cells transfected with allergen-DNA stimulate specific immunoglobulin G4 but not specific immunoglobulin E production of autologous B…

2007

Atopic/allergic diseases are characterized by T helper 2 (Th2)-dominated immune responses resulting in immunoglobulin E (IgE) production. DNA-based immunotherapies have been shown to shift the immune response towards Th1 in animal models. In further studies we showed that human dendritic cells (DC) transfected with allergen-DNA are able to stimulate autologous CD4(+) T cells from atopic individuals to produce Th1 instead of Th2 cytokines and to activate interferon-gamma (IFN-gamma)-producing CD8(+) T cells. The aim of this study was to analyse whether DC transfected with allergen-DNA are also able to influence immunoglobulin production of B cells from atopic donors. For this purpose, human …

CD4-Positive T-LymphocytesHypersensitivity ImmediateAllergyImmunologyCD8-Positive T-Lymphocytesmedicine.disease_causeImmunoglobulin ELymphocyte ActivationTransfectionAllergenImmune systemmedicineImmunology and AllergyHumansCells CulturedCell ProliferationPlant ProteinsRhinitisB-LymphocytesCD40biologyTransfectionOriginal ArticlesDendritic CellsAllergensImmunoglobulin ETh1 Cellsmedicine.diseaseCoculture TechniquesImmunoglobulin GImmunologybiology.proteinCytokinesAntibodyCD8T-Lymphocytes Cytotoxic
researchProduct

Comparison of allergen-stimulated dendritic cells from atopic and nonatopic donors dissecting their effect on autologous naive and memory T helper ce…

2000

Abstract Background: Because of their production of IL-12, mature dendritic cells (DC) are potent inducers of T H 1 responses. However, recent reports have demonstrated that DCs can also induce T H 2 differentiation. Objective: In the current study we investigated which immune response is induced by DCs in naive CD45RA + or memory CD45R0 + CD4 + T cells from atopic individuals (patients with grass pollen, birch pollen, or house dust mite allergy) compared with nonatopic control subjects. Methods: Immature DCs, generated from peripheral blood monocytes from atopic and nonatopic donors, were pulsed with the respective allergen and fully matured. Then the mature DCs were cocultured in vitro wi…

CD4-Positive T-LymphocytesHypersensitivity ImmediateAllergymedicine.medical_treatmentImmunologyAntigen presentationImmunoglobulin ETh2 CellsImmune systemmedicineHumansImmunology and AllergyB-LymphocytesbiologyAntibodies MonoclonalDendritic CellsT-Lymphocytes Helper-InducerT lymphocyteDendritic cellAllergensImmunoglobulin Emedicine.diseaseInterleukin-12PhenotypeCytokineImmunologybiology.proteinInterleukin 12CytokinesLeukocyte Common AntigensImmunologic MemoryCell DivisionJournal of Allergy and Clinical Immunology
researchProduct

Inhibition of human allergic T-cell responses by IL-10–treated dendritic cells: Differences from hydrocortisone-treated dendritic cells

2001

Abstract Background: Dendritic cells (DCs) are able to induce human allergic T H 1 responses as well as T H 2 responses. Objective: In this study, we examined the effect of antiinflammatory agents such as IL-10 and hydrocortisone (HC) on the accessory function of DCs and the resulting T-cell response, especially that of T H 2 cells. Methods: Naive and memory CD4 + T cells from atopic donors were stimulated with autologous allergen-pulsed DCs generated from CD14 + monocytes by culture with GM-CSF/IL-4 and fully matured with IL-1β, TNF-α, and PGE 2 in the presence or absence of IL-10 or HC. Results: IL-10–treated DCs and, to a lesser extent, HC-treated DCs showed a decreased expression of MHC…

CD4-Positive T-LymphocytesHypersensitivity ImmediateHydrocortisoneT-LymphocytesCD14T cellImmunologyAntigen presentationAnti-Inflammatory Agentschemical and pharmacologic phenomenaBiologyInterferon-gammaTh2 CellsmedicineHumansImmunology and AllergyAntigen-presenting cellCD86Antigen PresentationModels Immunologicalhemic and immune systemsDendritic CellsDendritic cellT lymphocyteAllergensInterleukin-10Interleukin 10medicine.anatomical_structureImmunologyCytokinesInterleukin-4Interleukin-5Immunologic MemoryJournal of Allergy and Clinical Immunology
researchProduct

Modification of the human allergic immune response by allergen-DNA-transfected dendritic cells in vitro.

2004

Abstract Background Atopic-allergic diseases are characterized by T H 2-dominated immune responses, resulting in IgE production. DNA-based immunotherapies have been shown to shift the immune response toward a T H 1-type response in animal models. Objective The aim of the study was to analyze whether dendritic cells (DCs) transfected with allergen-DNA conjugates are able to stimulate human autologous CD4 + T cells, CD8 + T cells, or both from atopic individuals to produce T H 1 cytokines instead of T H 2 cytokines. Methods For this purpose, human mature DCs from atopic donors were transfected with an adenovirus encoding the allergen Phl p 1. Autologous CD4 + and CD8 + T cells were stimulated…

CD4-Positive T-LymphocytesHypersensitivity Immediatemedicine.medical_treatmentImmunologyGenetic Vectorschemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationTransfectionInterleukin 21Interferon-gammaImmune systemmedicineImmunology and AllergyCytotoxic T cellHumansIL-2 receptorAntigen-presenting cellCells CulturedPlant ProteinsAdenoviruses HumanDendritic cellDendritic CellsAllergensTh1 CellsMolecular biologyCytokineImmunologyCytokinesCD8The Journal of allergy and clinical immunology
researchProduct

Identification of NY-ESO-1 epitopes presented by human histocompatibility antigen (HLA)-DRB4*0101-0103 and recognized by CD4(+) T lymphocytes of pati…

2000

NY-ESO-1 is a member of the cancer-testis family of tumor antigens that elicits strong humoral and cellular immune responses in patients with NY-ESO-1–expressing cancers. Since CD4+ T lymphocytes play a critical role in generating antigen-specific cytotoxic T lymphocyte and antibody responses, we searched for NY-ESO-1 epitopes presented by histocompatibility leukocyte antigen (HLA) class II molecules. Autologous monocyte-derived dendritic cells of cancer patients were incubated with recombinant NY-ESO-1 protein and used in enzyme-linked immunospot (ELISPOT) assays to detect NY-ESO-1–specific CD4+ T lymphocyte responses. To identify possible epitopes presented by distinct HLA class II allele…

CD4-Positive T-LymphocytesImmunologyMolecular Sequence DataAntigen-Presenting Cells10050 Institute of Pharmacology and Toxicology610 Medicine & healthHuman leukocyte antigenBiologyEpitopeCell LineAntigenAntigens NeoplasmImmunology and AllergyCytotoxic T cellHumansAmino Acid SequenceAntigen-presenting cellMelanomaHLA-DRB4Alleles2403 ImmunologyHLA class II–restricted NY-ESO-1 epitopesMembrane ProteinsProteinsT lymphocyteDendritic CellsHLA-DR AntigensVirologyRecombinant ProteinsHistocompatibilityImmunologyCD4+ T cell recognition2723 Immunology and Allergy570 Life sciences; biologyOriginal ArticleHLA-DRB4 Chains
researchProduct

CD4(+) and CD8(+) anergic T cells induced by interleukin-10-treated human dendritic cells display antigen-specific suppressor activity.

2002

Interleukin-10 (IL-10)–treated dendritic cells (DCs) induce an alloantigen- or peptide-specific anergy in various CD4+ and CD8+ T-cell populations. In the present study, we analyzed whether these anergic T cells are able to regulate antigen-specific immunity. Coculture experiments revealed that alloantigen-specific anergic CD4+ and CD8+ T cells suppressed proliferation of syngeneic T cells in a dose-dependent manner. The same effect was observed when the hemagglutinin-specific CD4+T-cell clone HA1.7 or tyrosinase-specific CD8+ T cells were cocultured with anergic T cells of the same specificity. Anergic T cells did not induce an antigen-independent bystander inhibition. Suppression was depe…

CD4-Positive T-LymphocytesIsoantigensImmunoconjugatesImmunologyAntigen-Presenting Cellschemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationBiochemistryT-Lymphocytes RegulatoryAbataceptInterleukin 21Antigens CDAntigens NeoplasmCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorLeukapheresisAntigen-presenting cellMelanomaCells CulturedClonal AnergyImmunosuppression TherapyMonophenol MonooxygenaseCD28Cell BiologyHematologyDendritic cellT lymphocyteDendritic CellsNatural killer T cellAntigens DifferentiationCoculture TechniquesCell biologyInterleukin-10ImmunologyCD4 AntigensLeukocytes MononuclearCell DivisionBlood
researchProduct

Interleukin 1α Promotes Th1 Differentiation and Inhibits Disease Progression in Leishmania major–susceptible BALB/c Mice

2003

Protective immunity against pathogens such as Leishmania major is mediated by interleukin (IL)-12–dependent Th1-immunity. We have shown previously that skin-dendritic cells (DCs) from both resistant C57BL/6 and susceptible BALB/c mice release IL-12 when infected with L. major, and infected BALB/c DCs effectively vaccinate against leishmaniasis. To determine if cytokines other than IL-12 might influence disease outcome, we surveyed DCs from both strains for production of a variety of cytokines. Skin-DCs produced significantly less IL-1α in response to lipopolysaccharide/interferon γ or L. major when expanded from BALB/c as compared with C57BL/6 mice. In addition, IL-1α mRNA accumulation in l…

CD4-Positive T-LymphocytesLipopolysaccharidedendritic cellT helper cell type 1/T helper cell type 2 immune responsemedicine.medical_treatmentImmunologyLeishmaniasis CutaneousMice Inbred StrainsLymphocyte ActivationArticleBALB/cMicechemistry.chemical_compoundCutaneous leishmaniasismedicineAnimalsImmunology and AllergyLeishmania majorLeishmania majorMice Inbred BALB CCD11b AntigenbiologyIL-1InterleukinDendritic Cellsbiology.organism_classificationmedicine.diseaseLeishmaniainfectionDisease Models AnimalCytokinechemistryImmunologyLymphInterleukin-1Journal of Experimental Medicine
researchProduct

Inhibitors of β-catenin affect the immuno-phenotype and functions of dendritic cells in an inhibitor-specific manner

2015

Many tumors are characterized by mutation-induced constitutive activation of β-catenin which promotes tumor growth and survival. Consequently, the development of specific β-catenin inhibitors for tumor therapy has come into the focus of drug development. β-Catenin was also shown to contribute to the tolerance-promoting function of unstimulated dendritic cells (DCs). In response to activation, DCs acquire potent T cell stimulatory capacity and induce profound tumor antigen-specific immune responses. Here we asked for effects of pre-clinically established β-catenin inhibitors (CCT-031374, iCRT-5, PNU-75654) on mouse bone marrow-derived (BM)DCs. All three inhibitors moderately increased surfac…

CD4-Positive T-LymphocytesLipopolysaccharides0301 basic medicineCell SurvivalOvalbuminT cellImmunologyPopulationAntineoplastic AgentsBone Marrow CellsMice Transgenicchemical and pharmacologic phenomena03 medical and health sciencesImmune systemmedicineAnimalsImmunology and AllergyeducationCells Culturedbeta CateninCell ProliferationPharmacologyCD86education.field_of_studyCD40biologyFollicular dendritic cellsCell growthhemic and immune systemsDendritic CellsCell biologyMice Inbred C57BLPhenotype030104 developmental biologymedicine.anatomical_structurebiology.proteinCytokinesCD80International Immunopharmacology
researchProduct

Glycoprotein 96-activated dendritic cells induce a CD8-biased T cell response.

2005

Heat shock proteins (Hsps) are able to induce protective immune responses against pathogens and tumors after injection into immunocompetent hosts. The activation of components of the adaptive immune system, including cytotoxic T lymphocytes specific for pathogen- or tumor-derived peptides, is crucial for the establishment of immuno- protection. Hsps acquire these peptides during intracellular protein degradation and when released during necrotic cell death, facilitate their uptake and Minor Histocompatibility Complex (MHC)-restricted representation by professional antigen-presenting cells (APCs). In addition, the interaction of Hsps with APCs, including the Endoplasmatic Reticulum (ER)-resi…

CD4-Positive T-LymphocytesLipopolysaccharidesAntigen-Presenting CellsBone Marrow CellsMice TransgenicReceptors Cell SurfaceBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexLymphocyte ActivationBiochemistryMiceImmune systemHeat shock proteinCytotoxic T cellAnimalsHumansAntigen-presenting cellCells CulturedMembrane GlycoproteinsToll-Like ReceptorsCell DifferentiationCell BiologyDendritic cellDendritic CellsOriginal ArticlesAcquired immune systemLymphocyte SubsetsCell biologyMice Inbred C57BLToll-Like Receptor 4biology.proteinInflammation MediatorsCD8Signal TransductionCell stresschaperones
researchProduct

Blue light irradiation suppresses dendritic cells activationin vitro

2013

Blue light is a UV-free irradiation suitable for treating chronic skin inflammation, for example, atopic dermatitis, psoriasis, and hand- and foot eczema. However, a better understanding of the mode of action is still missing. For this reason, we investigated whether dendritic cells (DC) are directly affected by blue light irradiation in vitro. Here, we report that irradiation neither induced apoptosis nor maturation of monocyte-derived and myeloid DC. However, subsequent DC maturation upon LPS/IFNγ stimulation was impaired in a dose-dependent manner as assessed by maturation markers and cytokine release. Moreover, the potential of this DC to induce cytokine secretion from allogeneic CD4 T …

CD4-Positive T-LymphocytesLipopolysaccharidesLightUltraviolet Raysmedicine.medical_treatmentStimulationInflammationCell SeparationDermatologyLymphocyte ActivationBiochemistryInterferon-gammaPsoriasismedicineHumansIrradiationMolecular BiologyImmunosuppression TherapyInflammationChemistryDendritic Cellsmedicine.diseaseCoculture TechniquesIn vitroCell biologyCytokineApoptosisImmunologyCytokinesCytokine secretionmedicine.symptomExperimental Dermatology
researchProduct