Search results for "Dependovirus"

showing 9 items of 19 documents

Sero-epidemiological analysis of the risk of virus infections for childhood leukaemia.

1996

Virus infections have been thought to be involved in the development of childhood leukaemia. In order to address this issue we determined, in a case-control study, the prevalence of antibodies to viruses infecting blood or bone-marrow cells [Epstein-Barr virsus (EBV), human herpes virus type 6 (HHV-6), parvovirus B19] as well as to the human virus known for its tumour-suppressive properties, the adeno-associated virus type 2 (AAV-2), in the sera of 121 children with leukaemia in Germany, and in 197 control individuals, hospitalized for other reasons, and matched for age and gender to the cases. In addition, we developed a questionnaire to be answered by the children's parents, in order to g…

MaleRiskCancer Researchmedicine.medical_specialtyHerpesvirus 4 HumanAdolescentvirusesHerpesvirus 6 Humanmedicine.disease_causeAntibodies ViralVirusHerpesviridaePaternal AgeSerologyImmunophenotypingParvoviridae Infectionshemic and lymphatic diseasesGermanyEpidemiologymedicineParvovirus B19 HumanHumansChildLeukemiabiologyParvovirusInfantHerpesviridae InfectionsDependovirusbiology.organism_classificationmedicine.diseaseEpstein–Barr virusLeukemiaOncologyVirus DiseasesCase-Control StudiesChild PreschoolImmunologyFemaleHuman VirusMaternal AgeInternational journal of cancer
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Glial Promoter Selectivity following AAV-Delivery to the Immature Brain

2013

Recombinant adeno-associated virus (AAV) vectors are versatile tools for gene transfer to the central nervous system (CNS) and proof-of-concept studies in adult rodents have shown that the use of cell type-specific promoters is sufficient to target AAV-mediated transgene expression to glia. However, neurological disorders caused by glial pathology usually have an early onset. Therefore, modelling and treatment of these conditions require expanding the concept of targeted glial transgene expression by promoter selectivity for gene delivery to the immature CNS. Here, we have investigated the AAV-mediated green fluorescent protein (GFP) expression driven by the myelin basic protein (MBP) or gl…

MouseCanavan DiseaseGene ExpressionDevelopmental and Pediatric NeurologyPediatricsGreen fluorescent protein0302 clinical medicineGene expressionNeurobiology of Disease and RegenerationTransgenesPromoter Regions GeneticCells Cultured0303 health sciencesMultidisciplinaryGlial fibrillary acidic proteinQStatisticsRAge FactorsBrainGenomicsGene TherapyAnimal ModelsDependovirusOligodendrogliamedicine.anatomical_structureNeurologyOrgan SpecificityMedicineGenetic EngineeringResearch ArticleBiotechnologyScienceTransgeneCentral nervous systemGenetic VectorsGreen Fluorescent ProteinsGene deliveryBiologyBiostatistics03 medical and health sciencesModel OrganismsGenomic MedicineDevelopmental NeuroscienceNeuroglial DevelopmentGlial Fibrillary Acidic ProteinmedicineGeneticsAnimalsBiology030304 developmental biologyClinical GeneticsMyelin Basic ProteinGenetic TherapyMolecular biologyOligodendrocyteMyelin basic proteinMice Inbred C57BLAnimals NewbornAstrocytesbiology.protein030217 neurology & neurosurgeryMathematicsTransgenicsNeurosciencePLoS ONE
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Intravitreal delivery of AAV-NDI1 provides functional benefit in a murine model of Leber hereditary optic neuropathy.

2012

Leber hereditary optic neuropathy (LHON) is a mitochondrially inherited form of visual dysfunction caused by mutations in several genes encoding subunits of the mitochondrial respiratory NADH-ubiquinone oxidoreductase complex (complex I). Development of gene therapies for LHON has been impeded by genetic heterogeneity and the need to deliver therapies to the mitochondria of retinal ganglion cells (RGCs), the cells primarily affected in LHON. The therapy under development entails intraocular injection of a nuclear yeast gene NADH-quinone oxidoreductase (NDI1) that encodes a single subunit complex I equivalent and as such is mutation independent. NDI1 is imported into mitochondria due to an e…

Oxidoreductase complexRetinal Ganglion CellsretinaSaccharomyces cerevisiae Proteinsgenetic structuresGenetic enhancementProtein subunitGenetic VectorsOptic Atrophy Hereditary LeberBiologyMitochondrionmedicine.disease_causeRetinal ganglionArticleMiceLHONGeneticsmedicineAnimalsHumansGenetics (clinical)GeneticsRetinaMutationElectron Transport Complex IGenetic heterogeneityAAVDependovirusgene therapyeye diseasesmitochondriaDisease Models AnimalOptic AtrophyNDI1medicine.anatomical_structureIntravitreal InjectionsCancer researchsense organsMicrotubule-Associated ProteinsEuropean journal of human genetics : EJHG
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AAV-Mediated Clarin-1 Expression in the Mouse Retina: Implications for USH3A Gene Therapy

2015

Usher syndrome type III (USH3A) is an autosomal recessive disorder caused by mutations in clarin-1 (CLRN1) gene, leading to progressive retinal degeneration and sensorineural deafness. Efforts to develop therapies for preventing photoreceptor cell loss are hampered by the lack of a retinal phenotype in the existing USH3 mouse models and by conflicting reports regarding the endogenous retinal localization of clarin-1, a transmembrane protein of unknown function. In this study, we used an AAV-based approach to express CLRN1 in the mouse retina in order to determine the pattern of its subcellular localization in different cell types. We found that all major classes of retinal cells express AAV…

Photoreceptors0301 basic medicineRetinal degenerationSensory ReceptorsPhysiologyUsher syndromeCell Membraneslcsh:MedicineSocial SciencesNervous SystemPhotoreceptor cellMicechemistry.chemical_compound0302 clinical medicineAnimal CellsMedicine and Health SciencesPsychologylcsh:ScienceNeuronsRegulation of gene expressionGeneticsMultidisciplinaryRetinal DegenerationAnimal ModelsDependovirusCell biologyElectrophysiologymedicine.anatomical_structureSensory PerceptionCellular TypesAnatomyCellular Structures and OrganellesUsher SyndromesResearch ArticleSignal TransductionCell typeImaging TechniquesOcular AnatomyNeurophysiologyOuter plexiform layerMouse ModelsBiologyResearch and Analysis MethodsRetina03 medical and health sciencesModel OrganismsOcular SystemFluorescence ImagingmedicineAnimalsHumansRetinalcsh:RMembrane ProteinsBiology and Life SciencesAfferent NeuronsRetinalGenetic TherapyCell Biologymedicine.diseaseDisease Models Animal030104 developmental biologyGene Expression RegulationchemistrySynapsesEyeslcsh:QHead030217 neurology & neurosurgeryNeurosciencePLOS ONE
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Rapid, reproducible transduction of select forebrain regions by targeted recombinant virus injection into the neonatal mouse brain

2009

Viral vectors can mediate long-term gene expression in different regions of the brain. Recombinant adeno-associated virus (rAAV) and Lenti virus (LV) have both gained prominence due to their ability to achieve specific transduction of various neuronal populations. Whilst widespread gene delivery has been obtained by targeted injection of rAAV in various brain structures, LV has also been utilized for infection of stem cell populations for cell lineage tracing. Both viral vector systems are most commonly used for gene delivery in mature brains, but the great potential of somatic gene delivery into the neonate brain has not been systematically exploited. Here we provide a systematic guideline…

Rostral migratory streamvirusesGenetic VectorsSubventricular zoneMice TransgenicGene deliveryBiologyRecombinant virusInjectionsViral vectorMiceTransduction (genetics)ProsencephalonNeuroblastTransduction GeneticmedicineAnimalsTissue DistributionNeuronsGeneral NeuroscienceDependovirusMolecular biologyRecombinant ProteinsMice Inbred C57BLmedicine.anatomical_structureAnimals NewbornGene TargetingForebrainJournal of Neuroscience Methods
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Functional roles of the membrane-associated AAV protein MAAP

2021

AbstractWith a limited coding capacity of 4.7 kb, adeno-associated virus (AAV) genome has evolved over-lapping genes to maximise the usage of its genome. An example is the recently found ORF in the cap gene, encoding membrane-associated accessory protein (MAAP), located in the same genomic region as the VP1/2 unique domain, but in a different reading frame. This 13 KDa protein, unique to the dependovirus genus, is not homologous to any known protein. Our studies confirm that MAAP translation initiates from the first CTG codon found in the VP1 ORF2. We have further observed MAAP localised in the plasma membrane, in the membranous structures in close proximity to the nucleus and to the nuclea…

SciencevirusesGenetic VectorsBiologyVirus ReplicationGenomeinfektiotArticleVirusViral Proteins03 medical and health scienceschemistry.chemical_compoundCapsidGene therapyPlasmidProtein sequencingHumansGeneparvovirukset030304 developmental biology0303 health sciencesMultidisciplinaryMolecular engineeringVirus Assembly030302 biochemistry & molecular biologyQVirionRMembrane ProteinsTranslation (biology)DependovirusCell biologyCapsidchemistryMedicineCapsid ProteinsproteiinitDNAPlasmidskapsidi
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Direct and long-term detection of gene doping in conventional blood samples

2010

The misuse of somatic gene therapy for the purpose of enhancing athletic performance is perceived as a coming threat to the world of sports and categorized as 'gene doping'. This article describes a direct detection approach for gene doping that gives a clear yes-or-no answer based on the presence or absence of transgenic DNA in peripheral blood samples. By exploiting a priming strategy to specifically amplify intronless DNA sequences, we developed PCR protocols allowing the detection of very small amounts of transgenic DNA in genomic DNA samples to screen for six prime candidate genes. Our detection strategy was verified in a mouse model, giving positive signals from minute amounts (20 μl)…

Vascular Endothelial Growth Factor ACandidate geneAthletic PerformanceBiologyPolymerase Chain ReactionDNA sequencinglaw.inventionMicelawGene dopingGeneticsAnimalsHumansTransgenesMolecular BiologyGenePolymerase chain reactionDoping in SportsGeneticsGenetic transferGenetic TherapyNucleic acid amplification techniqueDependovirusgenomic DNAGene ComponentsMolecular MedicineNucleic Acid Amplification TechniquesGene Therapy
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Combined effect of AAV-U7-induced dystrophin exon skipping and soluble activin Type IIB receptor in mdx mice.

2012

Adeno-associated virus (AAV)-U7-mediated skipping of dystrophin-exon-23 restores dystrophin expression and muscle function in the mdx mouse model of Duchenne muscular dystrophy. Soluble activin receptor IIB (sActRIIB-Fc) inhibits signaling of myostatin and homologous molecules and increases muscle mass and function of wild-type and mdx mice. We hypothesized that combined treatment with AAV-U7 and sActRIIB-Fc may synergistically improve mdx muscle function. Bioactivity of sActRIIB-Fc on skeletal muscle was first demonstrated in wild-type mice. In mdx mice we show that AAV-U7-mediated dystrophin restoration improved specific muscle force and resistance to eccentric contractions when applied a…

musculoskeletal diseasesmdx mousemedicine.medical_specialtycongenital hereditary and neonatal diseases and abnormalitiesDuchenne muscular dystrophyActivin Receptors Type IIGenetic VectorsMyostatinBiologyDystrophin03 medical and health sciencesMice0302 clinical medicineInternal medicineGeneticsmedicineMyocyteAnimalsMuscular dystrophyMuscle SkeletalMolecular Biology030304 developmental biology0303 health sciencesBody WeightSkeletal muscleExonsGenetic TherapyDependovirusMuscular Dystrophy Animalmedicine.diseasemusculoskeletal system3. Good healthMice Inbred C57BLEndocrinologymedicine.anatomical_structureImmunologybiology.proteinMice Inbred mdxMolecular MedicineITGA7Dystrophin030217 neurology & neurosurgeryMuscle ContractionHuman gene therapy
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Impact of VP1-Specific Protein Sequence Motifs on Adeno-Associated Virus Type 2 Intracellular Trafficking and Nuclear Entry

2012

ABSTRACT Adeno-associated virus type 2 (AAV2) has gained much interest as a gene delivery vector. A hallmark of AAV2-mediated gene transfer is an intracellular conformational change of the virus capsid, leading to the exposure of infection-relevant protein domains. These protein domains, which are located on the N-terminal portion of the structural proteins VP1 and VP2, include a catalytic phospholipase A 2 domain and three clusters of basic amino acids. We have identified additional protein sequence motifs located on the VP1/2 N terminus that also proved to be obligatory for virus infectivity. These motifs include signals that are known to be involved in protein interaction, endosomal sort…

virusesImmunologyProtein domainAmino Acid MotifsMolecular Sequence DataSequence alignmentBiologyMicrobiologyVirusCell LineParvoviridae InfectionsVirologyHumansAmino Acid SequenceAdeno-Associated Virus Type 2Peptide sequenceCell NucleusDependovirusMolecular biologyTransport proteinCell biologyVirus-Cell InteractionsProtein TransportCapsidInsect ScienceCapsid ProteinsSequence motifSequence Alignment
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