Search results for "Different"

showing 10 items of 8549 documents

Epithelium‐specific MyD88 signaling, but not DCs or macrophages, control acute intestinal infection with Clostridium difficile

2019

Infection with Clostridium difficile is one of the major causes of health care acquired diarrhea and colitis. Signaling though MyD88 downstream of TLRs is critical for initiating the early protective host response in mouse models of C. difficile infection (CDI). In the intestine, MyD88 is expressed in various tissues and cell types, such as the intestinal epithelium and mononuclear phagocytes (MNP), including DC or macrophages. Using a genetic gain-of-function system, we demonstrate here that restricting functional MyD88 signaling to the intestinal epithelium, but also to MNPs is sufficient to protect mice during acute CDI by upregulation of the intestinal barrier function and recruitment o…

0301 basic medicineCell typeImmunologyBiologyMice03 medical and health sciences0302 clinical medicineDownregulation and upregulationmedicineAnimalsImmunology and AllergyIntestinal MucosaColitisEnterocolitis PseudomembranousBarrier functionClostridioides difficileMacrophagesDendritic CellsClostridium difficilemedicine.diseaseIntestinal epitheliumPhenotypeEpitheliumDisease Models Animal030104 developmental biologymedicine.anatomical_structureHost-Pathogen InteractionsMyeloid Differentiation Factor 88ImmunologySignal Transduction030215 immunologyEuropean Journal of Immunology
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Taking Advantage of Nature’s Gift: Can Endogenous Neural Stem Cells Improve Myelin Regeneration?

2016

Irreversible functional deficits in multiple sclerosis (MS) are directly correlated to axonal damage and loss. Neurodegeneration results from immune-mediated destruction of myelin sheaths and subsequent axonal demyelination. Importantly, oligodendrocytes, the myelinating glial cells of the central nervous system, can be replaced to some extent to generate new myelin sheaths. This endogenous regeneration capacity has so far mainly been attributed to the activation and recruitment of resident oligodendroglial precursor cells. As this self-repair process is limited and increasingly fails while MS progresses, much interest has evolved regarding the development of remyelination-promoting strateg…

0301 basic medicineCell typeMultiple Sclerosisgliaadult neural stem cellsoligodendrocytesReviewBiologyRegenerative MedicineCatalysisInorganic ChemistryWhite matterlcsh:Chemistry03 medical and health sciencesMyelin0302 clinical medicineNeural Stem CellsmedicineAnimalsHumansPhysical and Theoretical ChemistryRemyelinationMolecular Biologylcsh:QH301-705.5SpectroscopyMyelin SheathMultiple sclerosisRegeneration (biology)Organic ChemistryEndogenous regenerationGeneral Medicinedifferentiationmedicine.diseaseNeural stem cellComputer Science ApplicationsNerve Regeneration030104 developmental biologymedicine.anatomical_structureremyelinationlcsh:Biology (General)lcsh:QD1-999nervous systemprecursor cellsImmunologyNeurosciencecell fate determinationwhite matter030217 neurology & neurosurgeryInternational Journal of Molecular Sciences
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Trans-presentation of IL-6 by dendritic cells is required for the priming of pathogenic TH17 cells

2016

The cellular sources of interleukin 6 (IL-6) that are relevant for differentiation of the TH17 subset of helper T cells remain unclear. Here we used a novel strategy for the conditional deletion of distinct IL-6-producing cell types to show that dendritic cells (DCs) positive for the signaling regulator Sirpα were essential for the generation of pathogenic TH17 cells. Using their IL-6 receptor α-chain (IL-6Rα), Sirpα+ DCs trans-presented IL-6 to T cells during the process of cognate interaction. While ambient IL-6 was sufficient to suppress the induction of expression of the transcription factor Foxp3 in T cells, trans-presentation of IL-6 by DC-bound IL-6Rα (called 'IL-6 cluster signaling'…

0301 basic medicineCell typebiologyCellular differentiationImmunologyLymphocyte differentiationFOXP3Priming (immunology)medicine.disease_cause3. Good healthCell biologyAutoimmunity03 medical and health sciences030104 developmental biology0302 clinical medicineImmunologybiology.proteinmedicineImmunology and AllergyInterleukin 6Transcription factor030215 immunologyNature Immunology
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A High Throughput Phenotypic Screening reveals compounds that counteract premature osteogenic differentiation of HGPS iPS-derived mesenchymal stem ce…

2016

AbstractHutchinson-Gilford progeria syndrome (HGPS) is a rare fatal genetic disorder that causes systemic accelerated aging in children. Thanks to the pluripotency and self-renewal properties of induced pluripotent stem cells (iPSC), HGPS iPSC-based modeling opens up the possibility of access to different relevant cell types for pharmacological approaches. In this study, 2800 small molecules were explored using high-throughput screening, looking for compounds that could potentially reduce the alkaline phosphatase activity of HGPS mesenchymal stem cells (MSCs) committed into osteogenic differentiation. Results revealed seven compounds that normalized the osteogenic differentiation process an…

0301 basic medicineCell typecongenital hereditary and neonatal diseases and abnormalitiesPhenotypic screeningInduced Pluripotent Stem CellsRetinoic acidTretinoinBiologyArticle03 medical and health scienceschemistry.chemical_compoundProgeriaOsteogenesis[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]medicineHumansInduced pluripotent stem cellChildIsotretinoinGeneticsProgeriaMultidisciplinaryintegumentary systemGuided Tissue RegenerationMesenchymal stem cellnutritional and metabolic diseasesAging PrematureCell DifferentiationMesenchymal Stem Cellsmedicine.diseaseProgerinAlkaline PhosphataseLamin Type A3. Good healthCell biologyHigh-Throughput Screening Assays030104 developmental biologychemistryGene Expression Regulation[ SDV.BBM.GTP ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]Alkaline phosphataseScientific Reports
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Long Term Culture of the A549 Cancer Cell Line Promotes Multilamellar Body Formation and Differentiation towards an Alveolar Type II Pneumocyte Pheno…

2016

Pulmonary research requires models that represent the physiology of alveolar epithelium but concerns with reproducibility, consistency and the technical and ethical challenges of using primary or stem cells has resulted in widespread use of continuous cancer or other immortalized cell lines. The A549 'alveolar' cell line has been available for over four decades but there is an inconsistent view as to its suitability as an appropriate model for primary alveolar type II (ATII) cells. Since most work with A549 cells involves short term culture of proliferating cells, we postulated that culture conditions that reduced proliferation of the cancer cells would promote a more differentiated ATII ce…

0301 basic medicineCellular differentiationCell Culture Techniqueslcsh:MedicineGene ExpressionPolymerase Chain ReactionBiochemistry0302 clinical medicineAnimal ProductsMedicine and Health SciencesCell Cycle and Cell Divisionlcsh:ScienceOligonucleotide Array Sequence Analysiseducation.field_of_studyMultidisciplinaryCell CycleCell DifferentiationAgricultureCell cyclerespiratory systemLipidsCell biologyPhenotypeCell Processes030220 oncology & carcinogenesisStem cellResearch ArticleMeatPopulationBiology03 medical and health sciencesExtraction techniquesMicroscopy Electron TransmissionGeneticsHumansGene RegulationeducationNutritionA549 celllcsh:RBiology and Life SciencesCell BiologyLipid MetabolismRNA extractionHamDietResearch and analysis methods030104 developmental biologyMetabolismGene Expression RegulationCell cultureA549 CellsFoodAlveolar Epithelial CellsCancer celllcsh:QImmortalised cell lineDevelopmental BiologyPloS one
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MOBP levels are regulated by Fyn kinase and affect the morphological differentiation of oligodendrocytes.

2015

Oligodendrocytes are the myelinating glial cells of the central nervous system (CNS). Myelin is formed by extensive wrapping of oligodendroglial processes around axonal segments which ultimately allows a rapid saltatory conduction of action potentials within the CNS and sustains neuronal health. The non-receptor tyrosine kinase Fyn is an important signaling molecule in oligodendrocytes. It controls the morphological differentiation of oligodendrocytes and is an integrator of axon-glial signaling cascades leading to localized synthesis of Myelin Basic Protein (MBP) which is essential for myelin formation. The abundant Myelin-Associated Oligodendrocytic Basic Protein (MOBP) resembles MBP in s…

0301 basic medicineCellular differentiationCentral nervous systemGene ExpressionBiologyProto-Oncogene Proteins c-fyn03 medical and health sciencesMyelinFYNmedicineAnimalsCell ShapeCells CulturedSaltatory conductionCell DifferentiationCell BiologyOligodendrocyteMyelin basic proteinCell biologyMice Inbred C57BLOligodendroglia030104 developmental biologymedicine.anatomical_structurenervous systemBiochemistryProtein Biosynthesisbiology.proteinTyrosine kinaseMyelin ProteinsJournal of cell science
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Epigenetic Regulation of Cardiac Differentiation of Embryonic Stem Cells and Tissues.

2016

International audience; Specific gene transcription is a key biological process that underlies cell fate decision during embryonic development. The biological process is mediated by transcription factors which bind genomic regulatory regions including enhancers and promoters of cardiac constitutive genes. DNA is wrapped around histones that are subjected to chemical modifications. Modifications of histones further lead to repressed, activated or poised gene transcription, thus bringing another level of fine tuning regulation of gene transcription. Embryonic Stem cells (ES cells) recapitulate within embryoid bodies (i.e., cell aggregates) or in 2D culture the early steps of cardiac developme…

0301 basic medicineCellular differentiationGeneral Chemical Engineering[SDV]Life Sciences [q-bio]Human Embryonic Stem Cellscardiac developmentcardiac differentiationEmbryoid bodychromatin immunoprecipitationBiologyGeneral Biochemistry Genetics and Molecular BiologyEpigenesis GeneticHistones03 medical and health sciencesMiceIssue 112AnimalsHumansEpigeneticsEnhancerTranscription factorGeneticsGeneral Immunology and MicrobiologyGeneral NeurosciencePromoterCell DifferentiationHeartgene transcription regulationEmbryonic stem cellES cellsCell biology[SDV] Life Sciences [q-bio]030104 developmental biologyEpigeneticsChromatin immunoprecipitationDevelopmental Biology
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Bifunctional Hydrogels Containing the Laminin Motif IKVAV Promote Neurogenesis

2017

Summary Engineering of biomaterials with specific biological properties has gained momentum as a means to control stem cell behavior. Here, we address the effect of bifunctionalized hydrogels comprising polylysine (PL) and a 19-mer peptide containing the laminin motif IKVAV (IKVAV) on embryonic and adult neuronal progenitor cells under different stiffness regimes. Neuronal differentiation of embryonic and adult neural progenitors was accelerated by adjusting the gel stiffness to 2 kPa and 20 kPa, respectively. While gels containing IKVAV or PL alone failed to support long-term cell adhesion, in bifunctional gels, IKVAV synergized with PL to promote differentiation and formation of focal adh…

0301 basic medicineCellular differentiationHYDROGELSCELL DIFFERENTIATION02 engineering and technologyBiochemistry//purl.org/becyt/ford/1 [https]MiceNeural Stem CellsIKVAVlcsh:QH301-705.5Cells Culturedlcsh:R5-920β(1)-integrinNeurogenesisHydrogelsMouse Embryonic Stem Cells021001 nanoscience & nanotechnologyNeural stem cellCell biologyStem celllcsh:Medicine (General)0210 nano-technologyCIENCIAS NATURALES Y EXACTASbiomaterialsPOLYLYSINENeurogenesisBiologyNEUROGENESISCiencias BiológicasFocal adhesion03 medical and health sciencesBiología Celular MicrobiologíalamininReportGeneticsΒ1-INTEGRINAnimalsProgenitor cell//purl.org/becyt/ford/1.6 [https]BIOMATERIALSCell adhesionFocal AdhesionsbioengineeringTissue Engineeringβ1-integrinCell BiologypolylysineNEURAL STEM CELLSMolecular biologyEmbryonic stem cellElasticityPeptide FragmentsBIOENGINEERINGLAMININMice Inbred C57BLcell differentiation030104 developmental biologylcsh:Biology (General)Developmental BiologyStem Cell Reports
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The activation of Wnt signaling by a STAT6-dependent macrophage phenotype promotes mucosal repair in murine IBD

2016

The complete repair of the mucosa constitutes a key goal in inflammatory bowel disease (IBD) treatment. The Wnt signaling pathway mediates mucosal repair and M2 macrophages that coordinate efficient healing have been related to Wnt ligand expression. Signal transducer and activator of transcription 6 (STAT6) mediates M2 polarization in vitro and we hypothesize that a STAT6-dependent macrophage phenotype mediates mucosal repair in acute murine colitis by activating the Wnt signaling pathway. Our results reveal an impaired mucosal expression of M2 macrophage-associated genes and delayed wound healing in STAT6(-/-) mice treated with 2,4,6-trinitrobenzenesulfonic acid (TNBS). These mice also ex…

0301 basic medicineCellular differentiationImmunologyBiology03 medical and health sciencesMice0302 clinical medicineImmunology and AllergyAnimalsHumansIntestinal MucosaCells CulturedSTAT6Mice KnockoutMice Inbred BALB CWound HealingWnt signaling pathwayLGR5LRP5Cell DifferentiationColitisInflammatory Bowel DiseasesCell biologyWnt Proteins030104 developmental biologyPhenotypeTrinitrobenzenesulfonic AcidImmunologySTAT proteinMacrophages PeritonealSignal transductionWound healingSTAT6 Transcription Factor030215 immunologySignal Transduction
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Candida albicansis able to use M cells as a portal of entry across the intestinal barrierin vitro

2015

Candida albicans is the most frequent yeast responsible for systemic infections in humans. These infections mainly originate from the gastrointestinal tract where C. albicans can invade the gut epithelial barrier to gain access to the bloodstream. Along the gut, pathogens can use Microfold (M) cells as a portal of entry to cross the epithelial barrier. M cells are specialized cells mainly located in the follicule-associated epithelium of Peyer patches. In this study, we used scanning electron and fluorescence microscopy, adhesion and invasion assays and fungal mutants to investigate the interactions of C. albicans with M cells obtained in an established in vitro model whereby enterocyte-lik…

0301 basic medicineCellular differentiationImmunologyBiologyEndocytosisbiology.organism_classificationMicrobiologyIn vitroEpitheliumCorpus albicansMicrobiology03 medical and health sciences030104 developmental biologymedicine.anatomical_structureTranscytosisVirologyImmunologymedicineCandida albicansMicrofold cellCellular Microbiology
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