Search results for "Dihydropyridine"

showing 10 items of 70 documents

U-46619-induced potentiation of noradrenergic constriction in the human saphenous vein: antagonism by thromboxane receptor blockade.

2001

Objective: We investigated the potentiating effect of U-46619, a synthetic analogue of thromboxane A2 (TXA2), on the adrenergic responses in human saphenous vein. Methods: Saphenous vein rings were obtained from 35 patients undergoing coronary artery bypass surgery. The rings were suspended in organ bath chambers for isometric recording of tension. Results: U-46619 (10−10–3×10−7 mol/l) produced concentration-dependent and endothelium-independent contractile responses. U-46619 (10−10 mol/l) potentiated the contractions elicited by electrical stimulation and potassium chloride, and produced leftward shifts of the concentration–response curve for noradrenaline. The TXA2 receptor antagonist SQ-…

Malemedicine.medical_specialtyDihydropyridinesNifedipinePhysiologymedicine.drug_classThromboxaneReceptors ThromboxaneAdrenergicIn Vitro TechniquesPotassium ChlorideThromboxane receptorThromboxane A2chemistry.chemical_compoundNorepinephrineThromboxane A2Physiology (medical)Internal medicinemedicineHumansVasoconstrictor AgentsSaphenous VeinAgedVoltage-dependent calcium channelDose-Response Relationship DrugChemistryCalcium channelDihydropyridineDrug SynergismMiddle AgedReceptor antagonistCalcium Channel BlockersElectric StimulationStimulation ChemicalEndocrinology15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidFemaleEndothelium VascularCardiology and Cardiovascular Medicinemedicine.drugCardiovascular research
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Anti-inflammatory effects of cerebrocrast in a model of rat paw edema and on mononuclear THP-1 cells.

2002

Cerebrocrast (IOS 1.1212; 4-[2-(difluoromethoxy)phenyl]-2,6-dimethyl-1,4-dihydropyridine-3,5-dicarboxylic acid di(2-propoxyethyl) diester) is a novel derivative of 1,4-dihydropyridine, which does not antagonize Ca(2+) influx in neuronal tissues. Since several classical dihydropyridines possess anti-inflammatory properties, we first studied the effects of cerebrocrast in a model of rat paw edema induced by carrageenan. Cerebrocrast had a preventative effect in this model of inflammation, with maximal activity (32-45% inhibition) in the 0.1-0.25 mg kg(-1) range. It was ineffective when added after the injection of carrageenan. Subsequent in vitro experiments showed that cerebrocrast in the mi…

Malemedicine.medical_specialtyDihydropyridinesmedicine.drug_classmedicine.medical_treatmentAnti-Inflammatory AgentsInflammationNeuroprotectionAnti-inflammatoryMonocyteschemistry.chemical_compoundInternal medicineForelimbmedicineTumor Cells CulturedAnimalsEdemaHumansTHP1 cell lineRats WistarPharmacologyDose-Response Relationship Drugbusiness.industryBiological activityCarrageenanRatsDisease Models AnimalEndocrinologyCytokinechemistryTumor necrosis factor alphamedicine.symptombusinessEuropean journal of pharmacology
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Voltage-Dependent Effects of Barnidipine in Rat Vascular Smooth Muscle

2003

The effects of the dihydropyridine nifedipine and its more lipophilic congener, barnidipine, were investigated in smooth muscle preparations from the rat in resting and depolarizing conditions. Both drugs relaxed precontracted aortic rings more potently in depolarizing conditions, barnidipine being more potent than nifedipine. Currents through Ca 2+ channels in rat vascular smooth muscle cells (A7r5) and in isolated rat cardiomyocytes were reduced more potently by both drugs at a holding potential of-40 mV than at -80 mV. However, barnidipine and nifedipine were more effective in reducing the current in A7r5 cells than in cardiomyocytes. The IC 50 obtained in aortic rings and in A7r5 cells …

Malemedicine.medical_specialtyPatch-Clamp TechniquesVascular smooth muscleBarnidipineNifedipinechemistry.chemical_elementPharmacologyCalciumMuscle Smooth VascularRats Sprague-DawleyNifedipineInternal medicinemedicineAnimalsMyocyteCells CulturedPharmacologyChemistryDihydropyridineDepolarizationCalcium Channel BlockersRatsEndocrinologyMechanism of actioncardiovascular systemFemalemedicine.symptomCardiology and Cardiovascular Medicinemedicine.drugJournal of Cardiovascular Pharmacology
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Comparative study of taurine and tauropyrone: GABA receptor binding, mitochondrial processes and behaviour.

2011

Abstract Objectives Taurine, a sulfur-containing amino acid, has high hydrophilicity and is poorly absorbed. Tauropyrone, a taurine-containing 1,4-dihydropyridine derivative, is suggested to have greater activity than taurine owing to improved physicochemical properties that facilitate delivery of the compound to target cells. The aim of this study was to determine whether the 1,4-dihydropyridine moiety in tauropyrone improves the pharmacological efficacy of taurine in vitro and in vivo. Methods The effects of taurine and tauropyrone, as well as of the 1,4-dihydropyridine moiety were compared in in-vitro experiments to determine the binding to GABA receptors and influence on mitochondrial p…

Malemedicine.medical_specialtyTaurineDihydropyridinesGABA receptor bindingTaurinePharmaceutical SciencePharmacologyMotor ActivityBicucullinechemistry.chemical_compoundMiceStructure-Activity RelationshipIn vivoSeizuresInternal medicinemedicineStructure–activity relationshipAnimalsRats WistarReceptorPharmacologychemistry.chemical_classificationMice Inbred ICRDiazepamBehavior AnimalEthanolChemistryGABAA receptorBicucullineReceptors GABA-AAmino acidMitochondriaRatsEndocrinologyMuscle TonusRotarod Performance TestEnergy MetabolismHydrophobic and Hydrophilic Interactionsmedicine.drugProtein BindingThe Journal of pharmacy and pharmacology
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Distinct effects of atypical 1,4-dihydropyridines on 1-methyl-4-phenylpyridinium-induced toxicity.

2006

Our previous data obtained from in vivo experiments demonstrated high neuroprotective effects of three novel atypical neuronal non-calcium antagonistic 1,4-dihydropyridine (DHP) derivatives cerebrocrast, glutapyrone and tauropyrone. The present studies were carried out in vitro to clarify, at least in part, their mechanism of action in primary culture of cerebellar granule cells by use of 1-methyl-4-phenylpyridinium (MPP+) as a neurotoxic agent which causes dramatic oxidative stress. Cerebrocrast (highly lipophilic, with a classical two-ring structure) dose-dependently (0.01-10.0 microM, EC50 = 13 nM) reduced MPP+-induced cell death. At the same time, the calcium antagonist nimodipine (refe…

Membrane potentialNeurons1-Methyl-4-phenylpyridiniumDihydropyridinesCell DeathMolecular Structure1-Methyl-4-phenylpyridiniumClinical BiochemistryCell BiologyGeneral MedicineMitochondrionPharmacologyBiologymedicine.disease_causeBiochemistryNeuroprotectionMechanism of actionmedicinemedicine.symptomMode of actionReactive Oxygen SpeciesOxidative stressIntracellularCells CulturedCell biochemistry and function
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CCDC 915981: Experimental Crystal Structure Determination

2014

Related Article: Inguna Goba, Baiba Turovska, Sergey Belyakov, Edvards Liepinsh|2014|J.Mol.Struct.|1074|549|doi:10.1016/j.molstruc.2014.06.044

Methyl 35-diacetyl-126-trimethyl-14-dihydropyridine-4-carboxylateSpace GroupCrystallographyCrystal SystemCrystal StructureCell ParametersExperimental 3D Coordinates
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CCDC 907992: Experimental Crystal Structure Determination

2014

Related Article: Inguna Goba, Baiba Turovska, Sergey Belyakov, Edvards Liepinsh|2014|J.Mol.Struct.|1074|549|doi:10.1016/j.molstruc.2014.06.044

Methyl 35-dicyano-1246-tetramethyl-14-dihydropyridine-4-carboxylateSpace GroupCrystallographyCrystal SystemCrystal StructureCell ParametersExperimental 3D Coordinates
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CCDC 885313: Experimental Crystal Structure Determination

2013

Related Article: I.Goba,B.Turovska,S.Belyakov,E.Liepinsh|2013|Khim.Get.Soedin.,SSSR||778|

Methyl 3-acetyl-5-cyano-126-trimethyl-14-dihydropyridine-4-carboxylateSpace GroupCrystallographyCrystal SystemCrystal StructureCell ParametersExperimental 3D Coordinates
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Barnidipine block of L-type Ca2+ channel currents in rat ventricular cardiomyocytes

2000

The effects of barnidipine and nifedipine on L-type Ca2+ current (ICa(L)) were investigated in ventricular cardiomyocytes from rats. Both barnidipine and nifedipine reduced ICa(L) in a concentration and voltage dependent manner; the EC50 were 80 and 130 nM at a holding potential of −80 mV, respectively, and 18 and 6 nM at −40 mV, respectively. Both drugs induced a leftward shift of the steady-state inactivation curve of ICa(L). Using a twin pulse protocol, the relationships between the amount of block of ICa(L) by either drug, seen during the second pulse, and the length of the first pulse were described by monoexponential functions reflecting onset of block, dependent on drug concentration…

PharmacologyMembrane potentialBarnidipinePulse (signal processing)ChemistryDihydropyridineCardiac musclePharmacologyElectrophysiologymedicine.anatomical_structureNifedipinemedicinePatch clampmedicine.drugBritish Journal of Pharmacology
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CCDC 991747: Experimental Crystal Structure Determination

2014

Related Article: Inguna Goba, Baiba Turovska, Sergey Belyakov, Edvards Liepinsh|2014|J.Mol.Struct.|1074|549|doi:10.1016/j.molstruc.2014.06.044

Space GroupCrystallography5-acetyl-26-dimethyl-4-phenyl-14-dihydropyridine-3-carbonitrileCrystal SystemCrystal StructureCell ParametersExperimental 3D Coordinates
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