Search results for "Disposition"

showing 10 items of 832 documents

Extraversion and performance approach goal orientation : An integrative approach to personality

2019

Abstract Research shows that extraversion is unrelated to performance approach goal orientation, both at the trait- and the state-level. However, since previous studies have either focused on the trait- or the state-level, such a conclusion may be premature. Building upon the idea that acting against one’s trait consumes self-control resources, we reason that within-person deviations from one’s level of trait extraversion might negatively relate to performance approach goal orientation. Using experience sampling data from 47 employees across 10 days (N = 307), we found that deviations from one’s trait extraversion levels are associated with lower levels of performance approach goal orientat…

Experience sampling methodSocial Psychologytavoiteorientaatiomedia_common.quotation_subject050109 social psychologyintegrative approach to personality050105 experimental psychologyBehavioral concordance modelbehavioral concordance modelkäyttäytymismallitperformance approach goal orientationPersonality0501 psychology and cognitive sciencesgoalsekstroversioPsychology(all)curvilinear relationshipsGeneral Psychologymedia_commonExtraversion and introversionperformance approachGoal orientation05 social sciencesgoal orientationpersoonallisuustavoitteetsuoriutuminenextraversionTraitcounterdispositional behaviorPsychologyCognitive psychology
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Counterdispositional Conscientiousness and Wellbeing: How Does Acting Out of Character Relate to Positive and Negative Affect at Work?

2020

Conscientiousness is typically seen as a positive or desired personality trait in the workplace, with the overall assumption being “the more, the better”. Drawing on the behavioral concordance model, we challenge this assumption, expecting that the highest level of positive affect and the lowest level of negative affect will correspond at the point where state and trait conscientiousness converge. Using an experience sampling study and an event reconstruction study, we show that deviations from one’s level of trait conscientiousness relate to variations in positive and negative affect, but not in a straightforward way. While wellbeing was lower when people behaved less conscientiously than …

Experience sampling methodorganizational psychologyConcordancemedia_common.quotation_subjecthyvinvointi050109 social psychology050105 experimental psychologywellbeingPersonality0501 psychology and cognitive sciencesWithin- and between- person personalityBig Five personality traitsconscientiousnesswithin- and between- person personalitymedia_commonpositive and negative affect05 social sciencesConscientiousnessTraitcounterdispositional behaviorConscientiousnessPositive psychologyIndustrial and organizational psychologyPsychologySocial psychologytunnollisuusSocial Sciences (miscellaneous)
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Interactions between dietary n-3 fatty acids and genetic variants and risk of disease

2012

Nutritional genomics has undergone rapid development and the concept is now very popular with the general public. Therefore, there is increasing demand for knowledge on adapting dietary composition to the genome. Our aim has been to undertake a systematic review so as to find out the level of evidence existing on whether the effects of n-3 fatty acids on health can be modulated by genetic variation. A systematic literature search was conducted on studies that jointly analyse the effect of one or more genetic variants in candidate genes and n-3 fatty acids. Both observational and experimental studies were included. Results are classified in accordance with whether the study was undertaken on…

Fatty Acid DesaturasesCandidate geneNutritional genomicsGenotypeFADS1FADS2Medicine (miscellaneous)BiologyNutrigeneticsArticleDelta-5 Fatty Acid DesaturaseNutrigenomicsRisk FactorsGenetic variationFatty Acids Omega-3HumansGenetic Predisposition to Diseasechemistry.chemical_classificationGeneticsNutrition and DieteticsFatty acidGenetic VariationDietNutrigenomicsPhenotypechemistry
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RNA sequencing-based transcriptome profiling of cardiac tissue Implicados novela putative disease mechanisms in FLNC-associated arrhythmogenic cardio…

2020

Arrhythmogenic cardiomyopathy (ACM) encompasses a group of inherited cardiomyopathies including arrhythmogenic right ventricular cardiomyopathy (ARVC) whose molecular disease mechanism is associated with dysregulation of the canonical WNT signalling pathway. Recent evidence indicates that ARVC and ACM caused by pathogenic variants in the FLNC gene encoding filamin C, a major cardiac structural protein, may have different molecular mechanisms of pathogenesis. We sought to identify dysregulated biological pathways in FLNC-associated ACM. RNA was extracted from seven paraffin-embedded left ventricular tissue samples from deceased ACM patients carrying FLNC variants and sequenced. Transcript le…

FilaminsDNA Mutational Analysis030204 cardiovascular system & hematologyGene mutationFilaminArticleTranscriptome03 medical and health sciences0302 clinical medicineHumansMedicineGenetic Predisposition to Disease030212 general & internal medicineJAM2FLNCGeneArrhythmogenic Right Ventricular Dysplasiabusiness.industryGene Expression ProfilingDNAArrhythmogenic cardiomyopathy Filamin C Focal adhesion pathway Integrin linked kinase pathway RNA sequencingActin cytoskeletonPatologiaCell biologyPhenotypeMutationCardiology and Cardiovascular MedicinebusinessMYL7
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G-protein-coupled receptor kinase 5 polymorphism and Takotsubo cardiomyopathy.

2015

BACKGROUND: Takotsubo cardiomyopathy (TTC) is an increasingly reported clinical syndrome that mimics acute myocardial infarction without obstructive coronary artery disease and is characterized by transient systolic dysfunction of the apical and/or mid-segments of the left ventricle. The syndrome mainly occurs in postmenopausal women with high adrenergic state conditions. Nowadays, the pathophysiology of TTC is not yet known and the possibility of a genetic predisposition is controversial. AIMS: The purpose of this study was to assess the genetic susceptibility to TTC through analysis of the L41Q polymorphism of the G-protein-coupled receptor kinase 5 (GRK5). METHODS AND RESULTS: In a cohor…

G-Protein-Coupled Receptor Kinase 5Malemedicine.medical_specialtyCardiomyopathyInfarctionCohort StudiesGene FrequencyG-Protein-Coupled Receptor Kinase 5Takotsubo CardiomyopathyInternal medicineGenotypemedicineGenetic predispositionHumansSettore MED/05 - Patologia ClinicaGenetic Predisposition to Diseasecardiovascular diseasesgenotype G-protein-coupled receptor kinase 5 gene polymorphism Takotsubo cardiomyopathyAgedPolymorphism Geneticbusiness.industryCase-control studyGeneral MedicineMiddle Agedmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato Cardiovascolaremedicine.anatomical_structureVentricleCase-Control StudiesCardiologyFemaleCardiology and Cardiovascular MedicinebusinessCohort study
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La limitation des compléments de service imposés aux enseignants du secondaire

2007

International audience

GESTION ADMINISTRATIVE DES AGENTS PUBLICS[SHS.DROIT]Humanities and Social Sciences/Law[SHS.DROIT] Humanities and Social Sciences/LawDisposition statutaireChef de serviceCirculaireComputingMilieux_MISCELLANEOUS
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Glutathione peroxidase-1 in health and disease: from molecular mechanisms to therapeutic opportunities.

2011

Reactive oxygen species, such as superoxide and hydrogen peroxide, are generated in all cells by mitochondrial and enzymatic sources. Left unchecked, these reactive species can cause oxidative damage to DNA, proteins, and membrane lipids. Glutathione peroxidase-1 (GPx-1) is an intracellular antioxidant enzyme that enzymatically reduces hydrogen peroxide to water to limit its harmful effects. Certain reactive oxygen species, such as hydrogen peroxide, are also essential for growth factor-mediated signal transduction, mitochondrial function, and maintenance of normal thiol redox-balance. Thus, by limiting hydrogen peroxide accumulation, GPx-1 also modulates these processes. This review explor…

GPX1AntioxidantPhysiologyProtein Conformationmedicine.medical_treatmentClinical BiochemistryMolecular Sequence DataGene ExpressionBiologymedicine.disease_causeBiochemistryDiabetes mellitus geneticschemistry.chemical_compoundGlutathione Peroxidase GPX1Risk FactorsComprehensive Invited ReviewNeoplasmsmedicineDiabetes MellitusAnimalsHumansGenetic Predisposition to DiseaseAmino Acid SequenceEnzyme InhibitorsHydrogen peroxideMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationReactive oxygen speciesGlutathione PeroxidasePolymorphism GeneticCell DeathSuperoxideCell BiologyGlutathioneSelenocysteineOxidative StresschemistryBiochemistryGene Expression RegulationCardiovascular DiseasesGeneral Earth and Planetary SciencesReactive Oxygen SpeciesOxidation-ReductionOxidative stressAntioxidantsredox signaling
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H2-Mβ1 and H2-Mβ2 Heterodimers Equally Promote CLIP Removal in I-Aq Molecules from Autoimmune-prone DBA/1 Mice

2001

Antigen-presenting cells degrade endocytosed antigens, e.g. collagen type II, into peptides that are bound and presented to arthritogenic CD4(+) helper T cells by major histocompatibility complex (MHC) class II molecules. Efficient loading of many MHC class II alleles with peptides requires the assistance of H2-M (HLA-DM in humans), a heterodimeric MHC class II-like molecule that facilitates CLIP removal from MHC class II molecules and aids to shape the peptide repertoire presented by MHC class II to CD4(+) T cells. In contrast to the HLA-DM region in humans, the beta-chain locus is duplicated in mice, with the H2-Mb1 beta-chain distal to H2-Mb2 and the H2-Ma alpha-chain gene. H2-M alleles …

Gene isoformAntigen PresentationMHC class IICD74ArthritisHistocompatibility Antigens Class IICD1AutoimmunityCell BiologyMHC restrictionBiologyMajor histocompatibility complexBiochemistryMolecular biologyCell LineAntigens Differentiation B-LymphocyteMiceAntigenMice Inbred DBAMHC class Ibiology.proteinAnimalsHumansGenetic Predisposition to DiseaseMolecular BiologyJournal of Biological Chemistry
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Genetic background in nonalcoholic fatty liver disease: A comprehensive review

2015

In the Western world, nonalcoholic fatty liver disease (NAFLD) is considered as one of the most significant liver diseases of the twenty-first century. Its development is certainly driven by environmental factors, but it is also regulated by genetic background. The role of heritability has been widely demonstrated by several epidemiological, familial, and twin studies and case series, and likely reflects the wide inter-individual and inter-ethnic genetic variability in systemic metabolism and wound healing response processes. Consistent with this idea, genome-wide association studies have clearly identified Patatin-like phosholipase domain-containing 3 gene variant I148M as a major player i…

Genetic MarkersCandidate geneGenome-wide association studieHeredityPatatin-like phospholipase domain-containing 3Genome-wide association studyDiseaseReviewBiologymedicine.disease_causeGeneticNon-alcoholic Fatty Liver DiseaseRisk FactorsHeredityNonalcoholic fatty liver diseasemedicineHumansNonalcoholic fatty liver diseaseGenetic Predisposition to DiseaseGenetic variabilityGenetic associationGeneticsFatty liverGastroenterologyGenetic VariationGeneral Medicinemedicine.diseaseCandidate gene studiePedigreePhenotypeNonalcoholic steatohepatitiTransmembrane 6 superfamily member 2Candidate gene studies; Genetics; Genome-wide association studies; Nonalcoholic fatty liver disease; Nonalcoholic steatohepatitis; Patatin-like phospholipase domain-containing 3; Transmembrane 6 superfamily member 2Genome-Wide Association Study
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Interest of exome sequencing trio-like strategy based on pooled parental DNA for diagnosis and translational research in rare diseases.

2021

Abstract Background Exome sequencing (ES) has become the most powerful and cost‐effective molecular tool for deciphering rare diseases with a diagnostic yield approaching 30%–40% in solo‐ES and 50% in trio‐ES. We applied an innovative parental DNA pooling method to reduce the parental sequencing cost while maintaining the diagnostic yield of trio‐ES. Methods We pooled six (Agilent‐CRE‐v2–100X) or five parental DNA (TWIST‐HCE–70X) aiming to detect allelic balance around 8–10% for heterozygous status. The strategies were applied as second‐tier (74 individuals after negative solo‐ES) and first‐tier approaches (324 individuals without previous ES). Results The allelic balance of parental‐pool v…

Genetic MarkersCost effectivenessTranslational researchBiologyQH426-470Sensitivity and SpecificityWorkflowTranslational Research Biomedicalchemistry.chemical_compoundsymbols.namesakeExome SequencingFalse positive paradoxGeneticsHumansDna poolingGenetic Predisposition to DiseaseGenetic TestingAlleleMolecular BiologyGenetics (clinical)Exome sequencingtrio‐like strategy; parental‐pool strategyGeneticsSanger sequencingcost effectivenessReproducibility of Resultsrare diseasesSequence Analysis DNAOriginal ArticleschemistryResearch DesignsymbolsOriginal ArticleDNAGenome-Wide Association StudyMolecular geneticsgenomic medicine
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