Search results for "Dizocilpine"

showing 4 items of 14 documents

Acute ammonia toxicity is mediated by the NMDA type of glutamate receptors

1992

AbstractPrevious experiments in our laboratory suggested that ammonium toxicity could be mediated by the NMDA type of glutamate receptors. To assess this hypothesis we tested if MK-801, a specific antagonist of the NMDA receptor, is able to prevent ammonium toxicity. Mice and rats were injected i.p. with 12 and 7 mmol/kg of ammonium acetate, respectively, 73% of the mice and 70% of the rats died. However, when the animals were injected i.p. with 2 mg/kg of MK-801, 15 min before ammonium injection, only 5% of the mice and 15% of the rats died. The remarkable protection afforded by MK-801 indicates that ammonia toxicity is mediated by the NMDA receptor.

Malemedicine.medical_specialtyBiophysicsGlutamic AcidReceptors N-Methyl-D-AspartateBiochemistryAmmonia toxicityMicechemistry.chemical_compoundGlutamatesAmmoniaStructural BiologyInternal medicineGeneticsmedicineAnimalsHyperammonemiaNeurotransmitter metabolismAmmoniumReceptorMolecular BiologyMK-801Glutamate receptorRats Inbred StrainsValineCell BiologyGlutamic acidNMDA receptorRatsReceptors NeurotransmitterEndocrinologyReceptors GlutamatechemistryToxicityNMDA receptorDizocilpine MaleateAmmonium acetateFEBS Letters
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The NMDA receptor complex: a promising target for novel antiepileptic strategies.

2001

Antiepileptic drugs (AEDs) cover a broad spectrum of pathological conditions ranging from seizures following congenital or acquired brain disorders to behavioural and psychiatric disorders and recently neuropathic pain. The need for novel antiepileptics raises from the expanding field of indications as well as from the fact, that special seizure types are refractory to common AEDs. In addition, many of the conventional antiepileptic drugs exhibit an unfavourable side-effect profile. Since there is growing evidence, that NMDA receptor activation might play a crucial role in epilepsy, NMDA receptor antagonists have become compounds of interest in preventing and treating seizures. This review …

N-Methylaspartatemedicine.medical_treatmentPharmacologyBiochemistryReceptors N-Methyl-D-AspartateFelbamatechemistry.chemical_compoundEpilepsyDrug DiscoveryIfenprodilmedicineAnimalsHumansRemacemidePharmacologyEpilepsybusiness.industryOrganic ChemistryMemantinemedicine.diseaseDizocilpineAnticonvulsantchemistryMolecular MedicineAnticonvulsantsbusinessEliprodilmedicine.drugCurrent medicinal chemistry
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Oxygen and glucose deprivation induces major dysfunction in the somatosensory cortex of the newborn rat

2005

The mechanisms and functional consequences of ischemia-induced injury during perinatal development are poorly understood. Subplate neurons (SPn) play a central role in early cortical development and a pathophysiological impairment of these neurons may have long-term detrimental effects on cortical function. The acute and long-term consequences of combined oxygen and glucose deprivation (OGD) were investigated in SPn and compared with OGD-induced dysfunction of immature layer V pyramidal cortical neurons (PCn) in somatosensory cortical slices from postnatal day (P)0-4 rats. OGD for 50 min followed by a 10-24-h period of normal oxygenation and glucose supply in vitro or in culture led to pron…

medicine.medical_specialtyPatch-Clamp TechniquesTolbutamideIn Vitro TechniquesBiologySomatosensory systemMembrane PotentialsInternal medicineSubplatemedicineExtracellularAnimalsHypoglycemic AgentsMagnesiumEnzyme InhibitorsHypoxiaOuabainNeuronsMembrane potentialCaspase 3General NeuroscienceDose-Response Relationship RadiationDepolarizationSomatosensory CortexHyperpolarization (biology)ImmunohistochemistryElectric StimulationRatsGlucoseNeuroprotective AgentsEndocrinologymedicine.anatomical_structureAnimals Newbornnervous systemApoptosisCaspasesNMDA receptorDizocilpine MaleateEuropean Journal of Neuroscience
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Addition of NMDA-receptor antagonist MK801 during oxygen/glucose deprivation moderately attenuates the upregulation of glucose uptake after subsequen…

2011

During stroke the blood–brain barrier (BBB) is damaged which can result in vasogenic brain edema and inflammation. The reduced blood supply leads to decreased delivery of oxygen and glucose to affected areas of the brain. Oxygen and glucose deprivation (OGD) can cause upregulation of glucose uptake of brain endothelial cells. In this letter, we investigated the influence of MK801, a non-competitive inhibitor of the NMDA-receptor, on the regulation of the glucose uptake and of the main glucose transporters glut1 and sglt1 in murine BBB cell line cerebEND during OGD. mRNA expression of glut1 was upregulated 68.7- fold after 6 h OGD, which was significantly reduced by 10 μM MK801 to 28.9-fold.…

medicine.medical_specialtyTime FactorsSchlaganfallNMDA-RezeptorGlucose uptakeNMDA-AntagonistInflammationCarbohydrate metabolismTritiumBlood–brain barrierReceptors N-Methyl-D-AspartateMice03 medical and health sciences0302 clinical medicineDownregulation and upregulationInternal medicinemedicineBlut-Hirn-SchrankeAnimalsddc:610RNA MessengerHypoxia030304 developmental biology0303 health sciencesDose-Response Relationship DrugbiologyGeneral NeuroscienceGlucosetransportproteineGlucose transporterBrainEndothelial CellsUp-RegulationOxygencarbohydrates (lipids)Glucosemedicine.anatomical_structureEndocrinologynervous systembiology.proteinNMDA receptorGLUT1Dizocilpine Maleatemedicine.symptomExcitatory Amino Acid Antagonists030217 neurology & neurosurgeryNeuroscience Letters
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