Search results for "Dopamine receptor"

showing 10 items of 125 documents

Shell/core differences in mu- and delta-opioid receptor modulation of dopamine efflux in nucleus accumbens

2008

The mu- and delta-opioid receptors located at the terminal level in nucleus accumbens are involved in the opiate modulation of dopamine release in this brain area. However, recent studies suggest that the effects of opioid drugs on the core subregion of nucleus accumbens may completely differ from those observed in the shell. We used in vivo microdialysis to simultaneously apply selective mu- and delta-opioid receptor agonists and to measure extracellular levels of dopamine in three subregions of the accumbens, namely shell, core, and the transition zone between them. The regional analysis of these subregions of the accumbens demonstrated that basal levels of dopamine and its metabolites we…

MaleAgonistTime FactorsEnkephalinmedicine.drug_classDopamineMicrodialysisReceptors Opioid muPharmacologyNucleus accumbensNucleus Accumbensδ-opioid receptorCellular and Molecular Neurosciencechemistry.chemical_compoundDopamine receptor D1DopamineReceptors Opioid deltamedicineAnimalsRats WistarPharmacologyDopaminergicHomovanillic AcidEnkephalin Ala(2)-MePhe(4)-Gly(5)-RatsAnalgesics OpioidDAMGOchemistry34-Dihydroxyphenylacetic AcidEnkephalin D-Penicillamine (25)-medicine.drugNeuropharmacology
researchProduct

Synthesis and evaluation in rats of the dopamine D2/3 receptor agonist 18F-AMC20 as a potential radioligand for PET

2015

Dopamine D2/3 receptor (D2/3R) agonist PET tracers are better suited for the imaging of synaptic dopaminergic neurotransmission than D2/3R antagonists and may also offer the opportunity to study in vivo the high-affinity state of D2/3R (D2/3RHigh). With the aim to develop 18F-labeled D2/3R agonists suitable for widespread clinical application, we report here on the synthesis and in vitro and in vivo evaluation of a D2/3R agonist ligand from the aminomethyl chromane (AMC) class-(R)-2-[(4- 18Fluorobenzylamino)methyl]chroman-7-ol (18F-AMC20). Methods: In vitro affinities of AMC20 toward dopaminergic receptor subtypes were measured in membrane homogenates prepared from HEK293 cells expressing h…

MaleBenzylaminesChemistry Techniques SyntheticPharmacologyLigandsRats Sprague-DawleyCricetinaeBINDINGRadioligandIN-VIVORacloprideRadiochemistryChemistryDopaminergicBrainStereoisomerismLigand (biochemistry)SERIESADDICTIONDopamine receptorDopamine AgonistsHIGH-AFFINITY STATEmedicine.drugAgonistmedicine.drug_classDRUG-ABUSECHO CellsPOSITRON-EMISSION-TOMOGRAPHYCricetulusFIn vivoDopamine receptor D2medicineAnimalsHumansRadiology Nuclear Medicine and imagingBenzopyransChromansReceptors Dopamine D2F-18Receptors Dopamine D3Biological TransportAgonist tracerRatsKineticsHEK293 CellsPETDopamine receptorRaclopridePositron-Emission TomographyRADIOTRACERSYSTEMJournal of Nuclear Medicine
researchProduct

Role of dopamine and glutamate receptors in cocaine-induced social effects in isolated and grouped male OF1 mice.

2005

Cocaine administration in paired male mice decreases social contacts as well as increases avoidance and flee elements. As dopamine (DA) and glutamate seem to be involved in some of cocaine's effects, an attempt was made to assess whether a range of associated receptors influenced the social impacts of this drug of abuse. The NMDA antagonist memantine (10 and 40 mg/kg); the AMPA antagonist CNQX (1 and 20 mg/kg); the DA release inhibitor CGS 10746b (2 and 8 mg/kg): the DA D1 antagonist SCH 23390 (0.05 and 0.5 mg/kg); and the DA D2/D3 antagonist raclopride (0.03 and 0.3 mg/kg) were administered prior to 25 mg/kg of cocaine and behaviour was evaluated during an encounter between an experimental…

MaleClinical BiochemistryPharmacologyToxicologyBiochemistryReceptors DopamineBehavioral Neurosciencechemistry.chemical_compoundMiceCocainemedicineAnimalsBiological PsychiatryPharmacologyRacloprideSCH-23390Behavior AnimalMemantineDopamine antagonistAntagonistchemistryReceptors GlutamateSocial IsolationDopamine receptorCNQXNMDA receptorPsychologymedicine.drugPharmacology, biochemistry, and behavior
researchProduct

Self-Regulation Therapy to Reproduce Drug Effects:A Suggestion Technique to Change Personality and theDRD3Gene Expression

2013

This study proposes a strategy, based on self-regulation therapy, to change personality and its biological substrate, the DRD3 gene expression. It has been demonstrated that acute doses of stimulating drugs, like methylphenidate, are able to change personality and the expression of certain genes in the short term. On the other hand, self-regulation therapy has been proven to reproduce the effects of drugs. Thus, it is feasible to hope that self-regulation therapy is equally effective as methylphenidate in changing personality and the gene expression. This is a preliminary study with a single-case experimental design with replication in which 2 subjects participated. The results and potentia…

MaleComplementary and Manual TherapyDrugHypnosisPsychotherapistPersonality InventoryC-Fosmedia_common.quotation_subjectGene ExpressionMessenger RNA expressionD-3 receptorGene expressionmedicineHumansPersonalityPeripheral blood lymphocytesSuggestionmedia_commonMethylphenidateReceptors Dopamine D3Middle AgedClinical PsychologyExpression (architecture)MethylphenidateCentral Nervous System StimulantsFemalesense organsDopamine receptor geneMATEMATICA APLICADAPsychologyPersonalitymedicine.drugInternational Journal of Clinical and Experimental Hypnosis
researchProduct

Involvement of Dopamine D2 Receptors in Addictive-Like Behaviour for Acetaldehyde

2014

Acetaldehyde, the first metabolite of ethanol, is active in the central nervous system, where it exerts motivational properties. Acetaldehyde is able to induce drinking behaviour in operant-conflict paradigms that resemble the core features of the addictive phenotype: drug-intake acquisition and maintenance, drug-seeking, relapse and drug use despite negative consequences. Since acetaldehyde directly stimulates dopamine neuronal firing in the mesolimbic system, the aim of this study was the investigation of dopamine D2-receptors' role in the onset of the operant drinking behaviour for acetaldehyde in different functional stages, by the administration of two different D2-receptor agonists, q…

MaleIndoleslcsh:MedicinePharmacologyBehavioral Neurosciencechemistry.chemical_compoundquinpiroleMedicine and Health Scienceslcsh:ScienceNeuropharmacologyDrug DependenceMultidisciplinaryDopaminergicD2 dopamine receptorsAcetaldehyde; Operant self-administration; D2 dopamine receptors; quinpiroleNeurologyBehavioral PharmacologyDopamine AgonistsSignal TransductionResearch Articlemedicine.drugAlcohol DrinkingDrug-Seeking BehaviorAcetaldehydeAddictive-Like BehaviourNeuropharmacologyQuinpiroleDopamineDopamine receptor D2medicineAnimalsRats WistarAcetaldehyde; Addictive-Like Behaviour; Dopamine D2 ReceptorsPharmacologyOperant self-administrationEthanolReceptors Dopamine D2Neurotransmissionlcsh:RAcetaldehydeBiology and Life SciencesDopamine D2 ReceptorsRatsRopinirolePharmacodynamicschemistrySettore BIO/14 - FarmacologiaConditioning Operantlcsh:QNeurosciencePLoS ONE
researchProduct

Pharmacodynamic effects of aripiprazole and ziprasidone with respect to p-glycoprotein substrate properties.

2013

Introduction Aripiprazole, an atypical antipsychotic drug with mixed antagonism and agonism on dopamine D2 and serotonin receptors, is a substrate of the efflux transporter P-glycoprotein (P-gp). Here we tested the pharmacodynamic consequences of these properties in a P-gp deficient mouse model by studying the effects of aripiprazole and of ziprasidone on motor coordination. Methods The motor behaviour of wild-type (WT) and P-gp deficient [abcb1ab(-/-)] mice was investigated on a RotaRod. Mice received acute injections of either aripirazole or ziprasidone. For comparison, the dopamine receptor antagonist haloperidol and serotonin receptor ligands buspirone and ketanserin were also applied. …

MaleKetanserinmedicine.drug_classAripiprazoleAtypical antipsychoticPharmacologyMotor ActivityQuinolonesRotarod performance testPiperazinesBuspironeMiceDopamine receptor D2medicineAnimalsPharmacology (medical)ZiprasidoneATP Binding Cassette Transporter Subfamily B Member 1Mice KnockoutChemistryGeneral MedicineBuspironeSerotonin Receptor AgonistsPsychiatry and Mental healthThiazolesDopamine receptorRotarod Performance TestHaloperidolAripiprazoleKetanserinSerotonin Antagonistsmedicine.drugAntipsychotic AgentsPharmacopsychiatry
researchProduct

Understanding Cannabinoid Psychoactivity with Mouse Genetic Models

2007

Marijuana and its main psychotropic ingredient Δ9-tetrahydrocannabinol (THC) exert a plethora of psychoactive effects through the activation of the neuronal cannabinoid receptor type 1 (CB1), which is expressed by different neuronal subpopulations in the central nervous system. The exact neuroanatomical substrates underlying each effect of THC are, however, not known. We tested locomotor, hypothermic, analgesic, and cataleptic effects of THC in conditional knockout mouse lines, which lack the expression of CB1 in different neuronal subpopulations, including principal brain neurons, GABAergic neurons (those that release γ aminobutyric acid), cortical glutamatergic neurons, and neurons expres…

MaleMESH: Body TemperatureCannabinoid receptormedicine.medical_treatmentGene ExpressionMESH: Receptor Cannabinoid CB1NeocortexMESH: gamma-Aminobutyric AcidMESH: CatalepsyPharmacologyHippocampusMESH: Mice KnockoutMESH: Corpus StriatumBody TemperatureMESH: Autonomic Nervous SystemMESH: NeocortexMice0302 clinical medicineReceptor Cannabinoid CB1MESH: Behavior AnimalCannabinoid receptor type 1MESH: AnimalsMESH: Gene SilencingDronabinolMESH: NociceptorsBiology (General)gamma-Aminobutyric AcidMice Knockout0303 health sciencesBehavior Animalmusculoskeletal neural and ocular physiologyGeneral NeuroscienceMESH: Pain ThresholdNociceptorsMESH: Glutamic AcidMESH: InterneuronsMESH: Motor Activity3. Good healthGABAergicMESH: TetrahydrocannabinolGeneral Agricultural and Biological SciencesResearch Articlemedicine.drugPain ThresholdMESH: Gene ExpressionMESH: Psychotropic DrugsQH301-705.5Glutamic AcidMotor ActivityBiologyAutonomic Nervous SystemGeneral Biochemistry Genetics and Molecular Biologygamma-Aminobutyric acid03 medical and health sciencesGlutamatergicDopamine receptor D1InterneuronsCannabinoid Receptor Modulatorsmental disorders[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineAnimalsGenetic Predisposition to Disease[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyGene SilencingTetrahydrocannabinolMESH: MiceAnesthesiology and Pain Management030304 developmental biologyPharmacologyCatalepsyPsychotropic DrugsModels GeneticGeneral Immunology and MicrobiologyCannabinoidsIllicit Drugsorganic chemicalsMESH: MaleCorpus StriatumPrimerDisease Models Animalnervous systemCannabinoidNervous System Diseases030217 neurology & neurosurgeryNeurosciencePLoS Biology
researchProduct

Clozapine: Strong antiaggressive effects with minimal motor impairment

1992

Abstract Clinical studies have shown clozapine to be effective in the treatment of schizophrenia and associated with an extremely low incidence of extrapiramidal side effects. Diverse studies indicate that clozapine is an atypical neuroleptic with a preferential activity on the mesolimbic structures and a lower affinity for striatal D2 receptors than the classical antipsychotics. The purpose of this study was to assess the behavioral properties of clozapine, especially its effects on aggressive and motor behaviors. Individually housed male mice of the OF1 strain were exposed to anosmic “standard opponents” 30 minutes after the last drug administration. One category of animals received a sin…

MaleMale miceExperimental and Cognitive PsychologyAtypical neurolepticMotor ActivityPharmacologyMiceBehavioral NeuroscienceDopamine receptor D2medicineAnimalsClozapineClozapineDose-Response Relationship DrugDrug administrationMotor impairmentmedicine.diseaseAggressionLower affinityMotor SkillsSchizophreniaAnesthesiaArousalPsychologyPsychomotor Performancemedicine.drugPhysiology & Behavior
researchProduct

Tetrahydroisoquinolines as dopaminergic ligands: 1-Butyl-7-chloro-6-hydroxy-tetrahydroisoquinoline, a new compound with antidepressant-like activity …

2009

International audience; Three series of 1-substituted-7-chloro-6-hydroxy-tetrahydroisoquinolines (1-butyl-, 1-phenyl- and 1-benzyl derivatives) were prepared to explore the influence of each of these groups at the 1-position on the affinity for dopamine receptors. All the compounds displayed affinity for D(1)-like and/or D(2)-like dopamine receptors in striatal membranes, and were unable to inhibit [(3)H]-dopamine uptake in striatal synaptosomes. Different structure requirements have been observed for adequate D(1) or D(2) affinities. This paper details the synthesis, structural elucidation, dopaminergic binding assays, structure-activity relationships (SAR) of these three series of isoquin…

MaleModels MolecularStereochemistryDopamineClinical BiochemistryPharmaceutical ScienceMotor Activity010402 general chemistryLigands01 natural sciencesBiochemistryReceptors Dopaminechemistry.chemical_compoundMiceStructure-Activity RelationshipDopamineTetrahydroisoquinolinesDrug DiscoverymedicineStructure–activity relationshipAnimalsReceptorMolecular Biology010405 organic chemistryTetrahydroisoquinolineReceptors Dopamine D2Receptors Dopamine D1[SCCO.NEUR]Cognitive science/NeuroscienceOrganic ChemistryDopaminergicAntagonistAntidepressive AgentsCorpus Striatum3. Good health0104 chemical sciencesRatschemistryDopamine receptorHydroxyquinolinesMolecular MedicineLead compoundmedicine.drugProtein Binding
researchProduct

The effects of dopamine D 2 and D 3 antagonists on spontaneous motor activity and morphine-induced hyperactivity in male mice

1999

Rationale: Dopaminergic neurotransmission, in particular the mesolimbic pathway, is involved in spontaneous locomotor activity and in morphine-induced hyperactivity, since the drugs acting on DA receptors can modify the action of morphine and this effect could be dependent on the type of DA receptor affected. Objective: In this study, the action of U-99194A maleate, haloperidol, sulpiride and morphine (5, 10, 20, 40 mg/kg) on locomotor activity in male mice was evaluated. Likewise, the effects of these dopaminergic antagonists on morphine-induced hyperactivity were studied. Methods: Animals treated with U-99194A maleate (2.5, 5, 10, 20 mg/kg), haloperidol (0.075, 0.1 mg/kg), sulpiride (20, …

MaleNarcoticsmedicine.medical_specialtyMesolimbic pathwayMotor ActivityPharmacologyMiceDopamine receptor D2Internal medicineHaloperidolmedicineAnimalsPharmacologyMorphineChemistryDopaminergicReceptors Dopamine D3AntagonistDopamine D2 Receptor AntagonistsEndocrinologyMechanism of actionIndansMorphineDopamine AntagonistsHaloperidolSulpiridemedicine.symptomSulpiridemedicine.drugPsychopharmacology
researchProduct