Search results for "Doubling time"

showing 4 items of 14 documents

Hsp70 is required for optimal cell proliferation in mouse A6 mesoangioblast stem cells.

2009

Mouse Hsp70 (70 kDa heat shock protein) is preferentially induced by heat or stress stimuli. We previously found that Hsp70 is constitutively expressed in A6 mouse mesoangioblast stem cells, but its possible role in these cells and the control of its basal transcription remained unexplored. Here we report that in the absence of stress, Ku factor is able to bind the HSE (heat shock element) consensus sequence in vitro, and in vivo it is bound to the proximal hsp70 promoter. In addition, we show that constitutive hsp70 transcription depends on the co-operative interaction of different factors such as Sp1 (specificity protein 1) and GAGA-binding protein with Ku factor, which binds the HSE cons…

Gene knockdownMesoangioblastBinding SitesGeneral transcription factorCell growthStem CellsCell BiologyBiologyFlow CytometryBiochemistryMolecular biologyHsp70MiceTranscription (biology)Heat shock proteinAnimalsBlood VesselsHSP70 Heat-Shock ProteinsRNA InterferenceStem cellmesoangioblast RNAi doubling timePromoter Regions GeneticMolecular BiologyCell ProliferationTranscription FactorsThe Biochemical journal
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Hepatocellular Carcinoma Presenting at Contrast-Enhanced Multi–Detector-Row Computed Tomography or Gadolinium-Enhanced Magnetic Resonance Imaging as …

2012

OBJECTIVE: The objective of the study was to measure growth rate and to determine the optimal interval time for imaging follow-up of hepatocellular carcinomas (HCCs) presenting at multi-detector-row computed tomography (MDCT) or magnetic resonance imaging (MRI) as small, indeterminate lesions. METHODS: We included patients with cirrhosis with HCC initially presenting as indeterminate lesion of 2 cm or less at MDCT or MRI August 2005 to August 2009 and with available imaging follow-up. Measures of tumor growth included tumor volume doubling time (TVDT), tumor percentual diameter increase, and tumor percentual volume increase. RESULTS: We examined 48 patients (mean age, 64 years) with 69 HCCs…

Liver CirrhosisMalemedicine.medical_specialtyCarcinoma HepatocellularTime FactorsCirrhosisGadoliniumVolume Doubling TimeContrast Mediahepatocellular carcinomaschemistry.chemical_elementStatistics NonparametricLesionImage Interpretation Computer-AssistedmedicineHumansRadiology Nuclear Medicine and imagingAgedRetrospective StudiesAged 80 and overmedicine.diagnostic_testbusiness.industryLiver NeoplasmsNodule (medicine)Magnetic resonance imagingMiddle Agedmedicine.diseaseMagnetic Resonance ImagingchemistryHepatocellular carcinomaDisease ProgressionFemaleRadiologymedicine.symptomTomography X-Ray ComputedIndeterminatebusinessNuclear medicinefollow-up; small hepatocellular carcinoma; mri; ctJournal of Computer Assisted Tomography
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Characterization of lymphokine-mediated activation of macrophages for antigen presentation: studies with long-term cultured bone marrow-derived macro…

1984

In cultures of bone marrow (BM) supplemented with L cell-derived colony-stimulating factor a pure population of macrophages (M phi) differentiates, which can be further propagated with a doubling time of 3.8 days. "Young" BMM phi obtained on day 8 of culture were shown to act as antigen-presenting cells inducing the antigen-specific proliferation of the cloned T cell line ST2/K.9, whereas "old" M phi had lost this ability. However, at any time tested (up to 132 days) the presentation function of old BMM phi could be completely restored by pulsing the cells with lymphokines (LK). A duration of 11 hr for the LK-pulse was sufficient to trigger the M phi to exert an optimal presentation functio…

Time FactorsT cellT-LymphocytesImmunologyPopulationAntigen presentationAntigen-Presenting CellsBone Marrow CellsBiologyLymphocyte ActivationInterferon-gammaMiceImmune systemAntigenmedicineImmunology and AllergyDoubling timeAnimalseducationCells Culturededucation.field_of_studyLymphokinesLymphokineHematologyMacrophage ActivationMolecular biologymedicine.anatomical_structureImmunologyBone marrowImmunobiology
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Treatment of B-CLL Cells with Bortezomib and Rituximab Reduces Cell Viability In Vitro..

2004

Introduction : In B-CLL, somatic mutation of IgVH genes defines a group of patients with favorable prognosis, whereas the absence of IgVH mutations is correlated with a worse outcome. Our previous data suggested that BCL-6 mutations identify a subgroup of patients with high risk of progression despite the presence of mutated IgVH gene, but the clinical significance of this molecular alteration remains uncertain. New approaches are now being tested for the treatment of B-CLL. Proteasome inhibitor, Bortezomib (Btz), and monoclonal antibodies specific for surface antigens, Rituximab (Rtx), represent potential therapeutic strategy. Objetives : To study the effects of Btz and Rtx on viability of…

medicine.diagnostic_testBortezomibLymphocyteImmunologyCell BiologyHematologyBiochemistryMolecular biologyFlow cytometrychemistry.chemical_compoundmedicine.anatomical_structurechemistryAntigenImmunologymedicineProteasome inhibitorDoubling timePropidium iodideViability assaymedicine.drugBlood
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