Search results for "Drug Interaction"

showing 10 items of 246 documents

Modulation by nitric oxide of spontaneous mechanical activity in rat proximal colon.

1999

Summary 1 In order to examine the role of nitric oxide (NO) in the tonic neural inhibition in rat proximal colon, the effects of Nω-nitro- l-arginine methyl ester (L-NAME) were studied on the spontaneous contractions of circular muscle (monitored as intraluminal pressure changes) and of longitudinal muscle (detected as isometric tension changes). 2 L-NAME (3 × 10−−6–3 × 10−−4 m) caused a concentration-dependent increase in the amplitude of circular contractions, without affecting those of longitudinal muscle. This effect was prevented by l-arginine (1–5 × 10−−3 m), but not d-arginine. 3 In the presence of tetrodotoxin (10−−6 m), which per se induced increase of the pressure waves, L-NAME (1…

Malemedicine.medical_specialtyColonIsometric exerciseNeurotransmissionIn Vitro TechniquesInhibitory postsynaptic potentialNitric OxideNitric oxideTonic (physiology)chemistry.chemical_compoundInternal medicineIsometric ContractionmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats WistarGuanethidinePharmacologyDose-Response Relationship DrugGeneral NeuroscienceMuscle SmoothRatsEndocrinologyNG-Nitroarginine Methyl EsterchemistryTetrodotoxinHexamethoniummedicine.drugMuscle ContractionJournal of autonomic pharmacology
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Modulation of adrenergic contraction of dog pulmonary arteries by nitric oxide and prostacyclin.

1999

Abstract The aim of this work was to investigate the influence of endothelium-derived nitric oxide and prostaglandins on the contractile responses of isolated dog pulmonary arteries to electrical field stimulation and noradrenaline. Electrical field stimulation (1–8 Hz, 20 v, 0.25 ms duration, for 30 s) produced frequency-dependent contractions that were abolished by tetrodotoxin, guanethidine and, prazosin (all at 10−6 M). Noradrenaline induced concentration-dependent contractions with an EC50 of 1.85 × 10−6 M. The increases in tension induced by electrical stimulation and noradrenaline were of greater magnitude in arteries denuded of endothelium. In segments with endothelium, NG-nitro- l …

Malemedicine.medical_specialtyEndotheliumArginineIndomethacinAdrenergicProstacyclinStimulationIn Vitro TechniquesPulmonary ArteryNitric OxideNitric oxidechemistry.chemical_compoundNorepinephrineDogsInternal medicinePrazosinmedicineAnimalsCyclooxygenase InhibitorsDrug InteractionsEnzyme InhibitorsGuanethidineAntihypertensive AgentsPharmacologyEpoprostenolElectric Stimulationmedicine.anatomical_structureEndocrinologyNG-Nitroarginine Methyl EsterchemistryVasoconstrictionProstaglandinsFemalemedicine.drugGeneral pharmacology
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Contribution of both ?- and ?-adrenoceptors to the inotropic effects of catecholamines in the rabbit heart

1992

The functional role of alpha-adrenoceptors was investigated in different parts of the rabbit heart. Phenylephrine (PE) caused a marked increase in force of contraction (Fc) and a prolongation of the action potential (AP) in preparations from the left atrium and the right ventricle. The response was less pronounced in the right atrium and in the left ventricle, whereas APs of spontaneously beating sinoatrial preparations remained completely unchanged. Phentolamine as well as the diesters phorbol 12,13 dibutyrate (PDBu) or 12-O-tetradecanoyl-phorbol-13-acetate (TPA) eliminated the effects of PE. The contribution of alpha-adrenoceptors to the effects of adrenaline (Adr) and noradrenaline (NA) …

Malemedicine.medical_specialtyEpinephrineAction PotentialsStimulationPropranololNorepinephrinechemistry.chemical_compoundPhentolamineInternal medicineIsoprenalinePhorbol EstersReceptors Adrenergic betamedicineAnimalsDrug InteractionsPhentolaminePhenylephrinePharmacologyChemistryMyocardiumHeartGeneral MedicineReceptors Adrenergic alphaMyocardial ContractionPropranololEndocrinologymedicine.anatomical_structureVentricleCirculatory systemPhorbolFemaleRabbitsmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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The Oxidative Stress Concept of Nitrate Tolerance and the Antioxidant Properties of Hydralazine

2005

The hemodynamic and anti-ischemic effects of nitroglycerin (NTG) are rapidly blunted as a result of the development of nitrate tolerance. With initiation of NTG therapy, it is possible to detect neurohormonal activation and intravascular volume expansion. These so-called pseudotolerance mechanisms may compromise the vasodilatory effects of NTG. Long-term nitrate treatment also is associated with decreased vascular responsiveness caused by changes in intrinsic mechanisms of the tolerant vasculature itself. According to the oxidative stress concept, increased vascular superoxide (O 2 − ) production and an increased sensitivity to vasoconstrictors secondary to activation of protein kinase C co…

Malemedicine.medical_specialtyMaximum Tolerated Dosegenetic structuresDrug ResistanceMyocardial IschemiaPharmacologyCoronary Angiographymedicine.disease_causeSeverity of Illness IndexDrug Administration ScheduleNitric oxideNitroglycerinchemistry.chemical_compoundInternal medicinemedicineAnimalsHumansDrug Interactionschemistry.chemical_classificationClinical Trials as TopicReactive oxygen speciesDose-Response Relationship Drugbusiness.industryHydralazineHydralazineLong-Term Careeye diseasesDisease Models AnimalOxidative StresschemistryHeart Function TestsExercise TestCardiologyFemaleVascular ResistanceEndothelium Vascularsense organsSodium nitroprussideCardiology and Cardiovascular MedicineSoluble guanylyl cyclasebusinessNicotinamide adenine dinucleotide phosphatePeroxynitriteOxidative stressmedicine.drugThe American Journal of Cardiology
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Disposition of acamprosate in the rat: Influence of probenecid

2002

The purpose of the present study was to investigate the disposition of acamprosate (calcium bis acetyl-homotaurine) in the rat. Initially, we studied the linearity of acamprosate disposition and the fraction of acamprosate excreted unchanged in the urine of the animals. Rats received 9.3, 36.6 or 73.3 mg/kg of the drug as an intravenous bolus. The statistical analysis of the pharmacokinetic parameters did not reveal any significant difference, indicating that acamprosate disposition was linear within the range of the doses assayed. On average, 95% of the administered dose was excreted unchanged in the urine of the animals in the 0-6 h post-administration period indicating that renal excreti…

Malemedicine.medical_specialtyMetabolic Clearance RateTaurineAcamprosatePharmaceutical ScienceRenal functionUrinePharmacologyPharmacokineticsInternal medicinemedicineAnimalsDrug InteractionsPharmacology (medical)Rats WistarPharmacologyKidneyProbenecidChemistryGeneral MedicineDrug interactionRatsProbenecidEndocrinologymedicine.anatomical_structureAcamprosateRenal physiologyInjections Intravenousmedicine.drugBiopharmaceutics & Drug Disposition
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The effects of phorbol 12,13-diacetate on responses of guinea-pig isolated trachea to methylxanthines, isoprenaline and ryanodine

1994

1. Using guinea-pig isolated trachea, we have studied how phorbol 12,13-diacetate (PDA) modulates mechanical responses of the tissue to methylxanthines, isoprenaline and ryanodine. 2. Caffeine (10 microM-5 mM), theophylline (10 microM-5 mM) and isoprenaline (1 nM-1 microM), each inhibited the spontaneous tone of the trachea. Pretreatment with PDA (0.1-10 microM) converted relaxant responses to high concentrations of the methylxanthines into contractions. PDA produced no equivalent effect against isoprenaline. Pretreatment with verapamil (1 or 10 microM), nifedipine (0.1 microM) or incubation with Ca(2+)-free, EGTA (0.1 mM)-containing physiological salt solution (PSS) suppressed the contract…

Malemedicine.medical_specialtyMuscle RelaxationGuinea PigsMepyramineIn Vitro TechniquesCalcium Chloridechemistry.chemical_compoundTheophyllineCaffeineIsoprenalineInternal medicinePhorbol EstersmedicineAnimalsDrug InteractionsTheophyllinePharmacologyRyanodineRyanodine receptorIsoproterenolMuscle SmoothCold TemperatureTracheaEndocrinologyMuscle relaxationVerapamilchemistryMuscle SpasticityXanthinesPotassiumTrachealis muscleVerapamilFemaleCaffeineResearch ArticleHistamineMuscle Contractionmedicine.drugBritish Journal of Pharmacology
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Cardiac effects of isoliquiritigenin

1997

The effects of isoliquiritigenin on force of contraction (Fc), L-type Ca2+ current (I(Ca)) and intracellular Ca2+ concentration ([Ca2+]i) were investigated in rat ventricular heart muscle. Isoliquiritigenin increased Fc and I(Ca) and, after longer exposure times, resting tension and [Ca2+]i. The effect of isoliquiritigenin (100 microM) on I(Ca) was diminished by Rp-cAMPS (30 microM). 1H-[1,2,4]oxa- diazolo[4,3-a]quinoxalin-1-one (50 microM) did not influence the effects of isoliquiritigenin on Fc and I(Ca). The positive inotropic effects of isoprenaline and forskolin, but not of 3-isobutyl-1-methylxanthine, were potentiated by isoliquiritigenin (100 microM). In the presence of milrinone (10…

Malemedicine.medical_specialtyPatch-Clamp TechniquesFura-2In Vitro TechniquesMembrane PotentialsRats Sprague-Dawleychemistry.chemical_compoundChalconeChalconesAldehyde ReductaseInternal medicineIsoprenalinemedicineAnimalsDrug InteractionsEnzyme InhibitorsCyclic GMPPharmacologyPlants MedicinalForskolinMyocardiumPhosphodiesteraseHeartCyclic AMP-Dependent Protein KinasesMyocardial ContractionRatsElectrophysiologyEndocrinologychemistryGuanylate CyclaseMilrinoneCalciumFemalemedicine.symptomSoluble guanylyl cyclaseIsoliquiritigeninMuscle contractionmedicine.drugEuropean Journal of Pharmacology
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INTERACTIONS OF ISOPRENALINE AND PROSTAGLANDIN E2 WITH RESPECT TO MYOCARDIAL CONTRACTILE FORCE, CORONARY VASCULAR RESISTANCE AND MYOCARDIAL OXYGEN CO…

1976

Left ventricular pressure (LVP), left ventricular pressure derivative (LV dp/dtmax), coronary vascular resistance (CVR) and myocardial oxygen consumption (Qo2) were measured simultaneously in isolated, electrically driven hearts of guinea-pigs at constant perfusion rate. 2 LVP, LV dp/dtmax, CVR and Qo2 were greatly decreased by either the addition of prostaglandin E2 (50 ng/ml) to the perfusion fluid or pretreatment of the animals with reserpine. 3 Isoprenaline (0.5 nM to 100 nM) induced increases in LVP, LV dp/dtmax and Qo2. In the presence of prostaglandin E2, there was a parallel shift of the isoprenaline concentration-response curve for LVP and LV dp/dtmax. This effect was not seen, aft…

Malemedicine.medical_specialtyReserpinemedicine.medical_treatmentGuinea PigsIndomethacinProstaglandinBlood PressureNorepinephrinechemistry.chemical_compoundOxygen ConsumptionInternal medicineIsoprenalinemedicineAnimalsDrug InteractionsProstaglandin E2PharmacologyDose-Response Relationship Drugbusiness.industryMyocardiumProstaglandins EIsoproterenolReserpineCoronary VesselsMyocardial ContractionBlood pressuremedicine.anatomical_structurechemistryCardiologyVentricular pressureVascular resistanceFemaleVascular ResistancebusinessResearch Articlemedicine.drugProstaglandin EBritish Journal of Pharmacology
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Insulin and raclopride combine to decrease short-term intake of sucrose solutions.

2000

We have previously reported that the hormone insulin can modulate synaptic function of dopamine neurons. To evaluate whether insulin can alter performance of a task which is dependent on intact dopaminergic signaling, we tested rats in a five minute lick rate task, with a range of concentrations of sucrose or oil solutions. Rats received either ip (t -15 min) saline or the D2 receptor antagonist raclopride (50 microg/kg), and intraventricular (t -4 h) saline or insulin (5 mU). Although ineffective on its own, insulin combined with raclopride treatment resulted in significant suppression of sucrose lick rates compared to the saline/saline group. The overall results are consistent with our hy…

Malemedicine.medical_specialtySucrosePhysiologymedicine.medical_treatmentDopamineBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundEndocrinologyDopamineInternal medicinemedicineAnimalsInsulinDrug InteractionsSalineRacloprideBehavior AnimalChemistryInsulinDopaminergicRatsSynaptic functionDopamine D2 Receptor AntagonistsEndocrinologyRacloprideDopamine AntagonistsHormonemedicine.drugPeptides
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Effects of CGS 10746B on hyperactivity and place preference induced by morphine

2001

The effects of CGS 10746B, a dopamine release inhibitor, on spontaneous locomotor activity, morphine-induced hyperactivity, acquisition of conditioned place paradigm and morphine-induced conditioned place preference (CPP) was evaluated in male mice. In experiment 1, animals treated with CGS 10746B (0.5, 1, 2, 4, 8, 16, 24 and 32 mg/kg), morphine (40 mg/kg) or morphine (40 mg/kg) plus CGS 10746B (0.5, 1, 2, 4, 8, 16, 24 and 32 mg/kg) were placed in an actimeter during a period of 90 min. In experiment 2, animals treated with CGS 10746B (0.5, 1, 3 and 10 mg/kg), morphine (40 mg/kg) or morphine (40 mg/kg) plus CGS 10746B (0.5, 1, 3 and 10 mg/kg) were conditioned following a procedure unbiased …

Malemedicine.medical_specialtyThiazepinesDopamineConditioning ClassicalMale miceMice Inbred StrainsSocial EnvironmentChoice BehaviorMiceBehavioral Neurosciencechemistry.chemical_compoundDopamineInternal medicinemedicineAnimalsDrug InteractionsNeurotransmitterDose-Response Relationship DrugMorphineChemistryAssociation LearningBrainConditioned place preferenceDose–response relationshipEndocrinologyMorphineCatecholamineConditioningNeuroscienceAntipsychotic Agentsmedicine.drugBehavioural Brain Research
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