Search results for "Drug interactions"

showing 10 items of 229 documents

Sympathetic nervous activity and the pressor effect of noradrenaline under chronic?-?-adrenoceptor blockade with labetalol in hypertension

1983

In 14 patients with essential hypertension, the influence of the alpha- and beta-adrenoceptor blocking drug labetalol on blood pressure, heart rate, plasma renin, plasma noradrenaline and pressor effect of exogenous noradrenaline was investigated during long-term treatment. During the initial four weeks of treatment, labetalol at a dose of 400 mg/day showed a slight effect only on supine blood pressure, whereas upright blood pressure was already lowered effectively after the second week of treatment (p less than 0.01). An increase in the mean dose to 850 mg/day had an additional blood pressure-lowering effect (p less than 0.001), whereby a preferential decrease of the orthostatic blood pres…

Adultmedicine.medical_specialtySympathetic Nervous SystemSupine positionPostureAlpha (ethology)Blood PressureEssential hypertensionPlasma renin activityPlacebosNorepinephrineInternal medicineReceptors Adrenergic betaReninDrug DiscoveryHeart ratemedicineHumansDrug InteractionsLabetalolLabetalolGenetics (clinical)Plasma noradrenalineDose-Response Relationship Drugbusiness.industryGeneral MedicineMiddle AgedReceptors Adrenergic alphamedicine.diseaseReceptors AdrenergicEndocrinologyBlood pressureEthanolaminesChronic DiseaseHypertensionMolecular Medicinebusinesscirculatory and respiratory physiologymedicine.drugKlinische Wochenschrift
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Drug-drug interactions in a cohort of hospitalized elderly patients.

2013

PurposeThe aim of this study is to assess the prevalence of patients exposed to potentially severe drug-drug interactions (DDIs) at hospital admission and discharge and the related risk of in-hospital mortality and adverse clinical events, readmission, and all-cause mortality at 3months. MethodsThis cross-sectional, prospective study was held in 70 Italian internal medicine and geriatric wards. Potentially severe DDIs at hospital admission and discharge; risk of in-hospital mortality and of adverse clinical events, readmission, and all-cause mortality at 3-month follow-up. ResultsAmong 2712 patients aged 65years or older recruited at hospital admission, 1642 (60.5\%) were exposed to at leas…

Aged 80 and overMaledrug interactionpharmacoepidemiologyDrug-Related Side Effects and Adverse ReactionsmortalityCohort StudiesHospitalizationCross-Sectional StudiesItalyMultivariate AnalysisPolypharmacyHumansDrug InteractionsFemaleAged
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Effect of adolescent exposure to WIN 55212-2 on the acquisition and reinstatement of MDMA-induced conditioned place preference.

2009

The present study employs a conditioned place preference procedure (CPP) to examine the effects of exposure to the cannabinoid agonist WIN 55212-2 (WIN) (0.1 and 0.5mg/kg) during adolescence on the reinforcing properties of +/-3,4-methylenedioxymetamphetamine hydrochloride (MDMA) (1.25 and 2.5mg/kg) in mice. On postnatal day (PD) 27, animals received a daily injection of the assigned treatment on 5 consecutive days, and three days later the place conditioning procedure was initiated (PD 35). The results suggest that pre-exposure to cannabinoids strengthens the properties of MDMA and favors reinstatement of the craving for the drug, which endorses the gateway hypothesis.

AgonistMaleReinforcement ScheduleTime Factorsmedicine.drug_classmedicine.medical_treatmentMorpholinesN-Methyl-34-methylenedioxyamphetamineSpatial BehaviorCravingPharmacologyNaphthalenesDevelopmental psychologyExtinction PsychologicalMiceRimonabantPiperidinesmedicineAnimalsDrug InteractionsCannabinoid Receptor AntagonistsBiological PsychiatryPharmacologyAnalysis of VarianceDose-Response Relationship DrugMDMAExtinction (psychology)Calcium Channel BlockersConditioned place preferenceBenzoxazinesAnimals NewbornHallucinogensCannabinoid receptor antagonistConditioning OperantPyrazolesCannabinoidmedicine.symptomRimonabantPsychologypsychological phenomena and processesmedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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Influence of hormonal treatment on the response of the rat isolated uterus to histamine and histamine receptor agonists.

1992

The response of the isolated uterus to histamine and histamine agonists was investigated in progesterone- and oestrogen-treated rats. The uterine inhibitory responses to histamine and 4-methylhistamine (a histamine H2 receptor agonist) were similar in KCl-contracted uteri from progesterone- and oestrogen-treated rats. The histamine H1 receptor agonist, 2-pyridyl-ethylamine, produced a relaxant response only in progesterone dominant uterus. This was inhibited by the histamine H1 receptor antagonist. In the rat isolated uterus which was not preconstricted by KCl, neither histamine, 4-methylhistamine, nor 2-pyridyl-ethylamine produced any effect in the presence or absence of ranitidine. Raniti…

Agonistmedicine.medical_specialtymedicine.drug_classPyridinesHistamine H1 receptorBiologyRanitidineHistamine agonistPotassium ChlorideRanitidinechemistry.chemical_compoundHistamine receptorUterine ContractionHistamine H2 receptorInternal medicinemedicineAnimalsDrug InteractionsHistamine H4 receptorProgesteronePharmacologyMethylhistaminesUterusEstrogensRats Inbred StrainsRatsEndocrinologychemistryReceptors HistamineFemaleHistaminemedicine.drugHistamineEuropean journal of pharmacology
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Effects of environmental and occupational lead toxicity and its association with iron metabolism

2021

Abstract Background Discrepancies are present in the findings from clinical trials evaluating a physiological role of iron status in the lead-exposed population. Objective The purpose of this article was to summarize the current understanding of cellular mechanisms of lead toxicity and present a comprehensive review of existing clinical trials related to associations of lead poisoning and iron status. Although an association of iron metabolism pathways that are affected by lead intoxication has been studied, there are still aspects that remain to be elucidated. The existence of additional Pb uptake pathways besides DMT1 transporter-mediated is postulated to non-specifically regulate lead ab…

AnemiaIronPopulationToxicologyLead poisoningOccupational ExposureEnvironmental healthStatistical significancemedicineHumansDrug InteractionsLead (electronics)educationPharmacologyeducation.field_of_studymedicine.diagnostic_testbusiness.industrylead poisoningEnvironmental Exposurelead toxicityIron metabolismmedicine.diseaseClinical trialLeadToxicityEnvironmental PollutantsBlood lead levelbusinessToxicology and Applied Pharmacology
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Bevacizumab, a humanized anti-angiogenic monoclonal antibody for the treatment of colorectal cancer

2007

Angiogenesis is the process by which new blood vessels are created from pre-existing vessels. It is essential for the growth and development of normal cells and tissues during embryonic and neonatal development and of tumour cells. Solid tumours rely on having an extensive network of blood vessels for growth and survival. The key mediator of angiogenesis, vascular endothelial growth factor-A (VEGF-A), is critical for the growth of tumours and their subsequent metastasis and is known to initiate angiogenesis. Bevacizumab is a humanized immunoglobulin G monoclonal antibody that binds to VEGF with high specificity, thereby blocking VEGF-mediated signalling pathways and thus angiogenesis. Clini…

Antimetabolites AntineoplasticBevacizumabColorectal cancerAngiogenesisNeovascularization PhysiologicAngiogenesis InhibitorsAntibodies Monoclonal HumanizedMetastasisNeovascularizationchemistry.chemical_compoundHumansMedicineDrug InteractionsPharmacology (medical)Neoplasm MetastasisSurvival ratePharmacologybusiness.industryAntibodies Monoclonalmedicine.diseaseBevacizumabSurvival RateVascular endothelial growth factorchemistryImmunologyMonoclonalCancer researchDrug Therapy CombinationFluorouracilmedicine.symptomColorectal Neoplasmsbusinessmedicine.drugJournal of Clinical Pharmacy and Therapeutics
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Effect of polyphenols on enniatins-induced cytotoxic effects in mammalian cells

2012

Enniatins (ENs) are fungal secondary metabolites produced by genus Fusarium. The ENs exert antimicrobial and insecticidal effect, and has also been demonstrated cytotoxic effects on several mammalian cell lines. On the other hands, it has been proved that natural polyphenols have antioxidant effect. In this study, cell effects at low levels of exposure of four ENs (A, A(1), B and B(1)) and five polyphenols (quercetin, quercetin-3-β-D-glucoside, rutin, myricetin and t-pterostilbene) present in wine; and the cytoprotective effect of these polyphenols exposed simultaneously with ENs in Chinese Hamster Ovary (CHO-K1) cells, were studied. Cell effects were determined by the MTT test after 24 h o…

AntioxidantCell SurvivalHealth Toxicology and Mutagenesismedicine.medical_treatmentCell Culture TechniquesCHO CellsBiologyToxicologyAntioxidantschemistry.chemical_compoundRutinCricetulusCricetinaeDepsipeptidesmedicineAnimalsDrug InteractionsCytotoxicityIC50Molecular StructureChinese hamster ovary cellPolyphenolsfood and beverageschemistryBiochemistryCytoprotectionPolyphenolMyricetinQuercetinToxicology Mechanisms and Methods
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Endotoxin inhibits gastric emptying in rats via a capsaicin-sensitive afferent pathway.

2001

The effects of endotoxin on gastric emptying of a solid nutrient meal and the neural mechanisms involved in such a response were investigated in conscious rats. The intraperitoneal (i.p.) administration of E. coli endotoxin (40 mug/kg) significantly reduced the 4-h rate of gastric emptying of a standard solid nutrient meal. Ablation of primary afferent neurons by systemic administration of high doses of capsaicin (20+30+50 mg/kg s.c.) to adult rats did not modify the rate of gastric emptying in control animals but prevented the delay in gastric transit induced by endotoxin. Local application of capsaicin to the vagus nerve rather than application of capsaicin to the celiac ganglion signific…

AtropineLipopolysaccharidesMaleendotoxinmedicine.medical_specialtyCalcitonin Gene-Related PeptidePharmacology toxicologyMuscarinic AntagonistscapsaicinRats Sprague-Dawleychemistry.chemical_compoundgastric emptyingtransitNeurons EfferentCalcitonin Gene-Related Peptide Receptor AntagonistsInternal medicinemedicineAnimalsDrug InteractionsCGRPNeurons AfferenttachykininsPhentolamineAfferent PathwayAdrenergic alpha-AntagonistsPharmacologyMealAfferent PathwaysGastric emptyingdigestive oral and skin physiologyGeneral MedicinePeptide FragmentsRatsEndocrinologychemistryGastric EmptyingCapsaicinCapsaicinNaunyn-Schmiedeberg's archives of pharmacology
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Cholinesterase activity and exposure time to acetylcholine as factors influencing the muscarinic inhibition of [3H]-noradrenaline overflow from guine…

1985

Guinea-pig isolated atria were incubated and loaded with [3H]-noradrenaline. The release of 3H and of [3H]-noradrenaline was induced by field stimulation (6-9 trains of 150 pulses at 5 Hz). The stimulation-evoked overflows of 3H and of [3H]-noradrenaline were determined. In the absence of an inhibitor of acetylcholinesterase, acetylcholine (12 min preincubation before nerve stimulation, up to 10 microM) failed to inhibit the evoked [3H]-noradrenaline overflow. In the presence of atropine, an increase by acetylcholine of evoked release was observed in the same atria. In contrast, the selective muscarinic agonist methacholine significantly decreased the evoked overflow. The inhibition was ant…

AtropinePhysostigminemedicine.medical_specialtyTime FactorsPhysostigmineGuinea PigsHexamethonium CompoundsIn Vitro TechniquesHexamethoniumMuscarinic agonistNorepinephrinechemistry.chemical_compoundCocaineInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsDrug InteractionsHeart AtriaPhentolamineMethacholine ChlorideCholinesterasePharmacologybiologyHeartPropranololReceptors MuscarinicAcetylcholineAtropineEndocrinologychemistryAcetylcholinesterasebiology.proteinMethacholineHexamethoniumCorticosteroneAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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Acetylcholine via Muscarinic Receptors Inhibits Histamine Release from Human Isolated Bronchi

1997

Human bronchi were incubated in organ baths to measure histamine release. The calcium ionophore A23187 (10 mumol/L; 1 min) stimulated histamine release by 148 +/- 28% (n = 11) above the prestimulation level but was ineffective in epithelium-denuded bronchi. Neither bradykinin (0.1 mumol/L) nor compound 48/80 (10 micrograms/ml) triggered the release of histamine from epithelium-intact bronchi. Acetylcholine did not affect spontaneous histamine release (about 2 nmol/g x 5 min) but inhibited A23187-evoked histamine release in an atropine-sensitive manner. Already a concentration as low as 0.1 nmol/L acetylcholine was effective, the maximal inhibition (by 89%) occurred at 100 nmol/L, whereas a …

AtropinePulmonary and Respiratory MedicineAgonistPhysostigminemedicine.medical_specialtyTime Factorsmedicine.drug_classPhysostigmineBradykininBronchiMuscarinic AntagonistsMuscarinic AgonistsCritical Care and Intensive Care MedicineHistamine Releasechemistry.chemical_compoundCulture TechniquesInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineHumansDrug InteractionsCalcimycinDose-Response Relationship DrugIonophoresbusiness.industryOxotremorineImmunoglobulin EReceptors MuscarinicAcetylcholineEndocrinologychemistryAcetylcholinesterase inhibitorDepression ChemicalCholinesterase InhibitorsbusinessAcetylcholineHistaminemedicine.drugAmerican Journal of Respiratory and Critical Care Medicine
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