Search results for "EBE"

showing 10 items of 929 documents

Capturing the musical brain with Lasso: Dynamic decoding of musical features from fMRI data.

2013

We investigated neural correlates of musical feature processing with a decoding approach. To this end, we used a method that combines computational extraction of musical features with regularized multiple regression (LASSO). Optimal model parameters were determined by maximizing the decoding accuracy using a leave-one-out cross-validation scheme. The method was applied to functional magnetic resonance imaging (fMRI) data that were collected using a naturalistic paradigm, in which participants' brain responses were recorded while they were continuously listening to pieces of real music. The dependent variables comprised musical feature time series that were computationally extracted from the…

AdultMaleCognitive NeuroscienceSpeech recognitionAuditory cortexbehavioral disciplines and activitiesBrain mappingHippocampusSuperior temporal gyrusYoung AdultGyrusCerebellummedicineImage Processing Computer-AssistedMusic information retrievalHumansAuditory CortexNeural correlates of consciousnessBrain Mappingmedicine.diagnostic_testSignal Processing Computer-AssistedMagnetic Resonance Imaginghumanitiesmedicine.anatomical_structureNeurologyta6131Auditory PerceptionFemalePsychologyFunctional magnetic resonance imagingDecoding methodsMusicNeuroImage
researchProduct

The what and how of observational learning

2007

Abstract Neuroimaging evidence increasingly supports the hypothesis that the same neural structures subserve the execution, imagination, and observation of actions. We used repetitive transcranial magnetic stimulation (rTMS) to investigate the specific roles of cerebellum and dorsolateral prefrontal cortex (DLPFC) in observational learning of a visuomotor task. Subjects observed an actor detecting a hidden sequence in a matrix and then performed the task detecting either the previously observed sequence or a new one. rTMS applied over the cerebellum before the observational training interfered with performance of the new sequence, whereas rTMS applied over the DLPFC interfered with performa…

AdultMaleCognitive Neurosciencemedicine.medical_treatmentrTMS cerebellum DLPFCPrefrontal CortexExperimental and Cognitive PsychologyCognitive neurosciencecerebellum; frontal cortex; observational learning; tmsbehavioral disciplines and activitiesTask (project management)NOBehavioral NeuroscienceMental ProcessesNeuroimagingtmsReference ValuesCerebellummental disordersmedicineBiological neural networkHumansObservational learningReference Values; Analysis of Variance; Humans; Cerebellum; Neural Inhibition; Prefrontal Cortex; Motor Skills; Imitative Behavior; Problem Solving; Social Perception; Imagination; Mental Processes; Adult; Transcranial Magnetic Stimulation; Female; MaleProblem SolvingAnalysis of VarianceSettore M-PSI/02 - Psicobiologia E Psicologia Fisiologicafrontal cortexNeural InhibitionCognitionImitative BehaviorTranscranial Magnetic StimulationDorsolateral prefrontal cortexTranscranial magnetic stimulationobservational learningmedicine.anatomical_structureSocial Perceptionnervous systemMotor SkillsImaginationSettore MED/26 - NeurologiaFemalePsychologyNeurosciencepsychological phenomena and processesCognitive psychology
researchProduct

Surgical Treatment in Symptomatic Chiari Malformation Type I: A Series of 25 Adult Patients Treated with Cerebellar Tonsil Shrinkage

2019

Background: The variety of symptoms and radiological findings in patients with Chiari malformation type I makes both the indication for surgery and the technical modality controversial. We report our 5-year experience, describing our technique and critically evaluating the clinical results. Methods: Between 2012 and 2016, 25 patients (15 female and 10 male; mean age 39.2 years) underwent posterior fossa decompression for Chiari malformation type I. Their clinical complaints included headache, nuchalgia, upper limb weakness or numbness, instability, dizziness and diplopia. Syringomyelia was present in 12 patients (48%). Suboccipital craniectomy was completed in all cases with C1 laminectomy …

AdultMaleDecompressive Craniectomymedicine.medical_specialtyCerebellar Vermi030218 nuclear medicine & medical imaging03 medical and health sciencesChiari malformation type I0302 clinical medicineCHIARI MALFORMATION TYPE IElectrocoagulationmedicineDuraplastyIn patientSurgical treatmentCerebellar tonsil shrinkageAdult patientsSettore MED/27 - Neurochirurgiabusiness.industryLaminectomyDecompression Surgicalmedicine.diseaseMagnetic Resonance ImagingSyringomyeliaArnold-Chiari MalformationSurgeryPosterior fossa decompressionTreatment Outcomemedicine.anatomical_structureRadiological weaponCerebellar tonsilFemalebusiness030217 neurology & neurosurgerySyringomyeliaHuman
researchProduct

Cognitive and social cognitive functioning in spinocerebellar ataxia : a preliminary characterization

2006

INTRODUCTION : The spinocerebellar ataxias (SCAs), are rare neurodegenerative disorders caused by distinct genetic mutations. Clinically, the SCAs are characterised by progressive ataxia and a variety of other features, including cognitive dysfunction. The latter is consistent with a growing body of evidence supporting a cognitive as well as motor role for the cerebellum. Recent suggestions of cerebellar involvement in social cognition have not been extensively explored in these conditions. The availability of definitive molecular diagnosis allows genetically defined subgroups of SCA patients, with distinct patterns of cerebellar and extracerebellar involvement, to be tested comparatively u…

AdultMaleEmotionsNeuropsychological TestsSocial Environmentcognitive functioningDisability EvaluationCognitionSocial cognitionCerebellumTheory of mindmedicineHumansSpinocerebellar AtaxiasCognitive skillSocial BehaviorAgedIntelligence TestsVerbal BehaviorCognitive disorderNeuropsychologyRecognition PsychologyCognitionMachado-Joseph DiseaseMiddle Agedmedicine.diseaseNeurologyMental RecallSpinocerebellar ataxiaAutismFemaleAtaxiaNeurology (clinical)PsychologyNeurosciencePsychomotor Performance
researchProduct

A preliminary characterisation of cognition and social cognition in spinocerebellar ataxia types 2, 1, and 7.

2010

Over the last decade, studies have implicated the cerebellum not only in motor functioning, but also in cognition and social cognition. Although some aspects of cognition have been explored in the five most common forms of Spinocerebellar Ataxia (SCA), social cognition in these patients has rarely been examined. The present study provides a preliminary characterisation of the severity of cognitive and social cognitive impairments in patients with SCA2, SCA1 and SCA7 using an identical battery to the one previously used in SCA3 and SCA6 patients for comparison. The cognitive profiles of SCA1 and SCA7 patients were comparable to that of SCA6 patients; SCA1 patients had relatively intact profi…

AdultMaleEmotionsTheory of MindemotionNeurosciences. Biological psychiatry. NeuropsychiatryGeneral MedicineMiddle AgedNeuropsychological TestsNeuropsychology and Physiological PsychologyCognitionNeurologySocial PerceptionHumansSpinocerebellar AtaxiasSpinocerebellar ataxia (SCA)FemaleOtherNeurology (clinical)EmpathyErratumSocial BehaviorPsychomotor PerformanceRC321-571Behavioural neurology
researchProduct

Performing allocentric visuospatial judgments with induced distortion of the egocentric reference frame: an fMRI study with clinical implications

2003

The temporary improvement of visuospatial neglect during galvanic vestibular stimulation (Scand. J. Rehabil. Med. 31 (1999)117) may result from correction of the spatial reference frame distorted by the responsible lesion. Prior to an investigation of the neural basis of this effect in neurological patients, exploration of the neural mechanisms underlying such procedures in normals is required to provide insight into the physiological basis thereof. Despite their clinical impact, the neural mechanisms underlying the interaction of galvanic (and other) vestibular manipulations with visuospatial processing (and indeed the neural bases of how spatial reference frames are computed in man) remai…

AdultMaleEye MovementsCognitive Neurosciencemedia_common.quotation_subjectVisuospatial neglectNeglectPerceptual DisordersPremotor cortexCerebellumImage Processing Computer-AssistedmedicineHumansGalvanic vestibular stimulationmedia_commonCerebral CortexVestibular systemMagnetic Resonance ImagingElectric StimulationOxygenNormal volunteersmedicine.anatomical_structureNeurologySpace PerceptionVisual PerceptionRight posteriorFemaleVestibule LabyrinthPsychologyNeurosciencePhotic StimulationPsychomotor PerformanceReference frameCognitive psychologyNeuroImage
researchProduct

Mutations in the gene encoding the basal body protein RPGRIP1L, a nephrocystin-4 interactor, cause Joubert syndrome.

2007

Peters, T.A./0000-0001-8443-5500; van Beersum, Sylvia E.C./0000-0002-4552-2908; Cremers, Frans/0000-0002-4954-5592; Roepman, Ronald/0000-0002-5178-8163 WOS: 000247619800019 PubMed: 17558407 Protein- protein interaction analyses have uncovered a ciliary and basal body protein network that, when disrupted, can result in nephronophthisis ( NPHP), Leber congenital amaurosis, Senior- Loken syndrome ( SLSN) or Joubert syndrome ( JBTS)(1-6). However, details of the molecular mechanisms underlying these disorders remain poorly understood. RPGRIP1- like protein ( RPGRIP1L) is a homolog of RPGRIP1 ( RPGR-interacting protein 1), a ciliary protein defective in Leber congenital amaurosis(7,8). We show t…

AdultMaleHealth aging / healthy living [IGMD 5]Eye DiseasesGenetics and epigenetic pathways of disease [NCMLS 6]TMEM67Molecular Sequence DataMembrane transport and intracellular motility [NCMLS 5]Biologymedicine.disease_causeJoubert syndromeCell LineGenomic disorders and inherited multi-system disorders [IGMD 3]NephronophthisisCerebellar DiseasesGeneticsmedicinePerception and Action [DCN 1]Basal bodyAnimalsHumansNeurosensory disorders [UMCN 3.3]CiliaAdaptor Proteins Signal TransducingRenal disorder [IGMD 9]GeneticsMutationCiliumCiliary transition zoneProteinsSyndromemedicine.diseasePedigreeRatsCytoskeletal ProteinsGenetic defects of metabolism [UMCN 5.1]RPGRIP1LFemaleKidney DiseasesFunctional Neurogenomics [DCN 2]Ciliary Motility Disorders
researchProduct

Evidence of Wallerian degeneration in normal appearing white matter in the early stages of relapsing-remitting multiple sclerosis

2003

Objective: Wallerian degeneration in normal appearing white matter in early relapsing-remitting multiple sclerosis (RRMS), and its correlation with the number of relapses and disease duration. Background Recent pathological studies have demonstrated Wallerian degeneration in normal appearing white matter (NAWM) in multiple sclerosis (MS), in established RRMS, and in chronic MS. However, the presence of Wallerian degeneration early in the disease and its correlation with relapse and with disease duration has not been studied. Methods: We performed proton magnetic resonance spectroscopic imaging in 21 MS patients, and 4 healthy controls, age and gender matched, aged under 45 years, with a max…

AdultMaleIn vivo magnetic resonance spectroscopyWallerian degenerationPathologymedicine.medical_specialtyMagnetic Resonance SpectroscopyTime FactorsNeurologyAdolescentWhite matterCentral nervous system diseaseMultiple Sclerosis Relapsing-RemittingmedicineHumansAspartic AcidMultiple sclerosisBrainmedicine.diseaseMagnetic Resonance ImagingPonsRadiographymedicine.anatomical_structurenervous systemNeurologyCerebellar peduncleCreatinineFemaleNeurology (clinical)Wallerian DegenerationPsychologyJournal of Neurology
researchProduct

KIAA0556 is a novel ciliary basal body component mutated in Joubert syndrome

2015

Background Joubert syndrome (JBTS) and related disorders are defined by cerebellar malformation (molar tooth sign), together with neurological symptoms of variable expressivity. The ciliary basis of Joubert syndrome related disorders frequently extends the phenotype to tissues such as the eye, kidney, skeleton and craniofacial structures. Results Using autozygome and exome analyses, we identified a null mutation in KIAA0556 in a multiplex consanguineous family with hallmark features of mild Joubert syndrome. Patient-derived fibroblasts displayed reduced ciliogenesis potential and abnormally elongated cilia. Investigation of disease pathophysiology revealed that Kiaa0556-/- null mice possess…

AdultMaleK04F10.2KIAA0556MicrotubuleMicrotubulesRetinaMiceJoubert syndromeCerebellumAnimalsHumansAbnormalities MultipleExomeCiliaEye AbnormalitiesSensory disorders Radboud Institute for Molecular Life Sciences [Radboudumc 12]Caenorhabditis elegansChildCells CulturedAdenosine TriphosphatasesADP-Ribosylation FactorsResearchBrainMetabolic Disorders Radboud Institute for Molecular Life Sciences [Radboudumc 6]Kidney Diseases CysticBasal BodiesPedigreeMice Inbred C57BLRenal disorders Radboud Institute for Molecular Life Sciences [Radboudumc 11]Basal bodyChild PreschoolMutationFemaleKataninMicrotubule-Associated ProteinsProtein BindingGenome Biology
researchProduct

A longitudinal investigation into cognition and disease progression in spinocerebellar ataxia types 1, 2, 3, 6, and 7

2016

Background The natural history of clinical symptoms in the spinocerebellar ataxias (SCA)s has been well characterised. However there is little longitudinal data comparing cognitive changes in the most common SCA subtypes over time. The present study provides a preliminary longitudinal characterisation of the clinical and cognitive profiles in patients with SCA1, SCA2, SCA3, SCA6 and SCA7, with the aim of elucidating the role of the cerebellum in cognition. Methods 13 patients with different SCAs all caused by CAG repeat expansion (SCA1, n = 2; SCA2, n = 2; SCA3, n = 2; SCA6, n = 4; and SCA7, n = 3) completed a comprehensive battery of cognitive and mood assessments at two time points, a mea…

AdultMaleMedicine(all)Settore M-PSI/02 - Psicobiologia E Psicologia FisiologicaResearchNeuroimagingMiddle AgedCognitionNeuropsychologyDisease ProgressionHumansSpinocerebellar AtaxiasFemaleAtaxiaGenetics(clinical)Pharmacology (medical)Longitudinal StudiesSpinocerebellar ataxiaAgedAtaxia; Cognition; Spinocerebellar ataxiaOrphanet Journal of Rare Diseases
researchProduct