Search results for "EDEMA"

showing 10 items of 625 documents

Classification of diabetes-related retinal diseases using a deep learning approach in optical coherence tomography

2019

Background and objectives: Spectral Domain Optical Coherence Tomography (SD-OCT) is a volumetric imaging technique that allows measuring patterns between layers such as small amounts of fluid. Since 2012, automatic medical image analysis performance has steadily increased through the use of deep learning models that automatically learn relevant features for specific tasks, instead of designing visual features manually. Nevertheless, providing insights and interpretation of the predictions made by the model is still a challenge. This paper describes a deep learning model able to detect medically interpretable information in relevant images from a volume to classify diabetes-related retinal d…

Volumetric imagingComputer scienceProfundo InterpretabilidadConvolutional neural network030218 nuclear medicine & medical imagingPattern Recognition Automatedchemistry.chemical_compoundMacular Degeneration[SPI]Engineering Sciences [physics]0302 clinical medicineDeep learning modelsInterpretabilityModelos de aprendizajeAged 80 and overArtificial neural networkmedicine.diagnostic_testMedical findings KeyWords Plus:MACULAR DEGENERATIONAngiographyMiddle AgedRetinal diseases3. Good healthComputer Science ApplicationsArea Under CurveTomographyMedical findingsAlgorithmsTomography Optical CoherenceAprendizaje - ModelosDiabetic macular edemaHealth InformaticsHallazgos médicosMacular Edema03 medical and health sciencesDeep LearningOptical coherence tomographymedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingDeep InterpretabilityHumans[INFO]Computer Science [cs]Enfermedades de la retinaRetinopathyAgedDiabetic RetinopathyOptical coherence tomographybusiness.industryDeep learningReproducibility of ResultsRetinalPattern recognitionMacular degenerationmedicine.diseasechemistryArtificial intelligenceNeural Networks ComputerLa tomografía de coherencia ópticabusinessClassifier (UML)030217 neurology & neurosurgerySoftware
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Clinical Utility of a Unique Genome-Wide DNA Methylation Signature for KMT2A-Related Syndrome

2022

Wiedemann–Steiner syndrome (WDSTS) is a Mendelian syndromic intellectual disability (ID) condition associated with hypertrichosis cubiti, short stature, and characteristic facies caused by pathogenic variants in the KMT2A gene. Clinical features can be inconclusive in mild and unusual WDSTS presentations with variable ID (mild to severe), facies (typical or not) and other associated malformations (bone, cerebral, renal, cardiac and ophthalmological anomalies). Interpretation and classification of rare KMT2A variants can be challenging. A genome-wide DNA methylation episignature for KMT2A-related syndrome could allow functional classification of variants and provide insights into the pathoph…

Wiedemann–Steiner syndromeQH301-705.5Intellectual disability[SDV.BC]Life Sciences [q-bio]/Cellular BiologyCatalysisInorganic ChemistryKMT2A geneNeurodevelopmental disorderGrowth DisorderAbnormalities Multiple[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Biology (General)Physical and Theoretical ChemistryEpisignatureQD1-999[SDV.BC] Life Sciences [q-bio]/Cellular BiologyMolecular BiologySpectroscopyDNA methylationOrganic ChemistryNeurodevelopmental disordersCraniofacial AbnormalitieEpigeneticHypertrichosiGeneral MedicineFacieComputer Science Applications<i>KMT2A</i> geneChemistryepigenetics; DNA methylation; episignature; Wiedemann–Steiner syndrome; <i>KMT2A</i> gene; intellectual disability; neurodevelopmental disordersPhenotype[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]EpigeneticsHuman
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Missense mutations of coagulation factor XII in hereditary angioedema with normal complement C1 inhibitor

2007

biologybusiness.industryImmunologyCoagulation Factor XIImedicine.diseaseComplement (complexity)C1-inhibitorHereditary angioedemaImmunologymedicinebiology.proteinMissense mutationbusinessMolecular BiologyMolecular Immunology
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VALUTAZIONE DELL’INCIDENZA DEL LINFEDEMA SECONDARIO ALLA PROCEDURA DELLA BIOPSIA DEL LINFONODO SENTINELLA NEL TRATTAMENTO DEL MELANOMA DEL TRONCO E D…

biopsia del linfonodo sentinella melanoma linfoscintigrafia incidenza linfedema
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Sepsis-associated encephalopathy

2012

Summary Sepsis-associated encephalopathy (SAE) is defined as a diffuse or multifocal cerebral dysfunction induced by the systemic response to the infection without clinical or laboratory evidence of direct brain infection. Its pathogenesis is multifactorial. SAE generally occurs early during severe sepsis and precedes multiple-organ failure. The most common clinical feature of SAE is the consciousness alteration which ranges from mildly reduced awareness to unresponsiveness and coma. Diagnosis of SAE is primarily clinical and depends on the exclusion of other possible causes of brain deterioration. Electroencephalography (EEG) is almost sensitive, but it is not specific for SAE. Computed To…

brain edemaBloodparasitic diseasesBrain edemaSepsis-associated encephalopathyblood brain barrierReviewS100B
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C1-Esterase Inhibitor Concentrate For Acute Attacks Of Laryngeal Edema In Hereditary Angioedema (HAE): Fixed Dosing Vs Body Weight-Adjusted Dosing

2014

business.industryAnesthesiaImmunologyHereditary angioedemaImmunology and AllergyMedicineDosingbusinessBody weightLaryngeal Edemamedicine.diseaseC1 esteraseJournal of Allergy and Clinical Immunology
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Spreading Depression Induces Permanent Cell Swelling Under Penumbra Conditions

2000

Background. Spreading depression (SD) is known to go along with temporary breakdown of ion gradients and cell swelling which spontaneously normalizes. Here, the effects of SD at reduced flow conditions as encountered in the ischemic penumbra are examined.

business.industryBrain edemaCell swellingPenumbraCortical spreading depressionBiophysicsMedicinebusinessTissue impedance
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Treatment of Hereditary Angioedema with the Mutation c.988A&gt;G (p.K330E) in the Plasminogen Gene

2020

business.industryImmunologyMutation (genetic algorithm)Plasminogen GeneHereditary angioedemamedicineImmunology and Allergymedicine.diseasebusinessMolecular biologyJournal of Allergy and Clinical Immunology
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Neuron-Glial Interaction During Injury and Edema of the CNS

1994

During injury and ischemia of the CNS mediator compounds are released or activated which cause secondary swelling and damage of nerve cells. Such mediators are glutamate, acidosis, free fatty acids, or high extracellular potassium. Glial homeostatic mechanisms are activated to prevent the secondary injury from these mediators. The glial clearance mechanisms have been studied in detail using in vitro systems allowing for a close control of the glial environment. Current evidence suggests glial swelling to occur together with glutamate uptake or in response to extracellular acidosis. Glial swelling, therefore, is rather the result of homeostatic mechanisms than an indication of glial demise.

business.industryIschemiaGlutamate receptormedicine.diseaseCell biologymedicine.anatomical_structureMediatornervous systemEdemamedicineExtracellularNeuronmedicine.symptombusinessHomeostasisAcidosis
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Hereditary Angioedema with Normal C1-INH with Versus without a Specific Mutation in the F12 Gene

2015

business.industrySpecific mutationImmunologyHereditary angioedemaImmunologymedicineImmunology and Allergymedicine.diseasebusinessGeneJournal of Allergy and Clinical Immunology
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